ライフサイエンスコーパス: O157

1 ric culture (0.7%), with 65 cases of E. coli O157.

2 are an important pathogenicity trait of EHEC O157.

3 for epidemiological surveillance of E. coli O157.

4 lated, 28 (90%) were attributable to E. coli O157 and 3 (10%) were attributable to non-O157 STEC.

5 Cattle are a reservoir host of EHEC O157 and a major source of human exposure through contam

6 nce for the geographic divergence of E. coli O157 and for a prominent role of stx2a in total Stx prod

7 EIA and/or culture and distinguishes between O157 and non-O157 STEC in clinical samples and that E. c

8 targeting stx1 and stx2 for the detection of O157 and non-O157 STEC in diarrheal stool samples enrich

9 E. coli O157 and non-O157 STEC were detected in 35 and 18 cases,

10 ce) and cultured in attempts to recover both O157 and non-O157 STEC.

11 erococcus faecalis, Salmonella spp., E. coli O157 and porcine parvovirus in bioreduction vessels cont

12 Interestingly, the IEE sequences from O157 and the top 10 non-O157 STEC serotypes fell into cl

13 associated with highly pathogenic serotypes (O157 and top non-O157 Shiga toxin-producing Escherichia

14 and epidemiological surveillance of E. coli O157, and the data were used to identify discernible ass

15 Cattle are the main reservoir for E. coli O157, and vaccines for cattle now exist.

16 o identify sublineages and clades of E. coli O157, and when they were correlated with the clinical ou

17 prevent substantial numbers of human E. coli O157 cases.

18 ential cultivar-specific response to E. coli O157 colonization, although importantly there was no rel

19 bacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, Listeria monocytogenes, and Salmonella spp

20 modified by enzymes in the E. coli serotype O157 (E. coli O157) O-PS biosynthetic pathway.

21 found espW in the sequenced O157:H7 and non-O157 EHEC strains as well as in Shigella boydii Furtherm

22 Most EPEC and non-O157 EHEC strains express lymphostatin (also known as Li

23 genomic lineages, the O157/O55 EHEC1 and non-O157 EHEC2.

24 -based simulation model for Escherichia coli O157 environmental transmission in cattle to simulate fo

25 LD-PCR differentiated STEC O157 from non-O157 using rfbEO157, and LD-PCR results pr

26 differential association of Escherichia coli O157 genotypes with animal and human hosts has recently

27 Adherence of E. coli O157 : H-expressing flagella of serotype H7, H6 or H48 t

28 s of endotoxins and bacterial cells (E. coli O157:H19).

29 or both glucose (0.08+/-0.02muM) and E. coli O157:H7 ( approximately 4 CFUmL(-1)) were competitive wi

30 ion of the food contaminant Escherichia coli O157:H7 (E. coli O157:H7) in complex food products due t

31 etection of food pathogenic Escherichia coli O157:H7 (E.coli O157:H7), a dangerous strain among 225 E

32 PCR) was used to test for genes from E. coli O157:H7 (eaeO157), shiga-toxin producing E. coli (stx2),

33 ation is a fitness trait of Escherichia coli O157:H7 (EcO157) that provides ample time for the pathog

34 Enterohemorrhagic Escherichia coli O157:H7 (EHEC) is a foodborne pathogen that causes blood

35 licated type I locus within Escherichia coli O157:H7 (EHEC), which we have named the gene pairs zorO-

36 including enterohemorrhagic Escherichia coli O157:H7 (EHEC).

37 ly, a homologue of lymphostatin from E. coli O157:H7 (ToxB; L7095) was also found to possess comparab

38 Extremely low concentration of E. coli O157:H7 (~10 CFU/mL) could be detected within 1h and 3h

39 Positive selection for E. coli O157:H7 across the farms identified only one positive is

40 inase (PI3K)/Akt signaling increased E. coli O157:H7 adherence to HT-29 cells.

41 Similarly, E. coli O157:H7 adhesion to cattle colonic explants was reduced

42 inflammatory response to attenuated E. coli O157:H7 adhesion, mucin 2 (MUC2) expression was analyzed

43 units (cfu)/mL, or approximately 500 E. coli O157:H7 and approximately 500 E. coli K12 cfu/mL.

44 hip with a case testing positive for E. coli O157:H7 and coincident diarrheal illness.

45 antly increased survival of E. coli, E. coli O157:H7 and Ent. faecalis in the water column.

46 actively transposes and proliferates in EHEC O157:H7 and enterotoxigenic E. coli (ETEC) O139 and O149

47 d to a reduction in the transport of E. coli O157:H7 and K12 from 98 to 10% and from 95 to 70%, respe

48 anise oil reduced the population of E. coli O157:H7 and L. monocytogenes by 1.48 and 0.47 log cfu/ml

49 nise oil nanoemulsion (AO75) reduced E. coli O157:H7 and L. monocytogenes count by 2.51 and 1.64 log

50 We found espW in the sequenced O157:H7 and non-O157 EHEC strains as well as in Shigella

51 the transport of Escherichia coli pathogenic O157:H7 and nonpathogenic K12 strains in water-saturated

52 ulated (Mat) fimbriae) for E. coli serotypes O157:H7 and O18:K1:H7.

53 7BL/6 mice were also inoculated with E. coli O157:H7 and only 1 of 14 developed disease, whereas 10 o

54 s testing of the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic aci

55 ow as 20 nm was capable of detecting E. coli O157:H7 and Salmonella sp. in complex hamburger and cucu

56 In Escherichia coli O157:H7 and Salmonella, QseC has been proposed as the ad

57 covalently grafting an anti-Escherichia coli O157:H7 antibody onto SAM-modified gold electrodes.

58 e feasibility was demonstrated using E. coli O157:H7 as a model analyte, this approach could be easil

59 d decimal reduction times of Escherichiacoli O157:H7 at different heating temperatures were used in e

60 nsor was able to specifically detect E. coli O157:H7 at the low concentration within 10 min in pure c

61 the passenger domain of the Escherichia coli O157:H7 autotransporter EspP have been shown to cause st

62 he complete assembly of the Escherichia coli O157:H7 autotransporter EspP in vitro.

63 ed to the C terminus of the Escherichia coli O157:H7 autotransporter EspP to test this hypothesis.

64 nalysis of mutations in the Escherichia coli O157:H7 autotransporter EspP.

65 nhanced the sensitivity for Escherichia coli O157:H7 bacteria detection.

66 ghly sensitive detection of Escherichia coli O157:H7 bacteria.

67 The E. coli O157:H7 bacteriophage PhiV10 was modified to express Nan

68 ic substrates were anti-adhesive for E. coli O157:H7 binding to human HT29 cells.

69 tation and genome diversification in E. coli O157:H7 but also contributing to the development of path

70 QS columns enhanced the transport of E. coli O157:H7 by 3.1 fold compared to the unoxidized counterpa

71 , we reported the rapid detection of E. coli O157:H7 by using calcium signaling of the B cell upon ce

72 E. coli O157:H7 can cause bloody diarrhea, hemolytic uremic synd

73 rical properties once the pathogenic E. coli O157:H7 captured on the sensor surface.

74 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 causes hemorrhagic diarrhea and potentially fata

75 s of antibodies with single Escherichia coli O157:H7 cells and demonstrated a capability of determini

76 ulation onto plants, the majority of E. coli O157:H7 cells either die or are no longer culturable.

77 e (with PMA) and total (without PMA) E. coli O157:H7 cells on growth chamber and field-grown lettuce.

78 Fewer E. coli O157:H7 cells survived when applied onto plants in dropl

79 en enterohemorrhagic Escherichia coli (EHEC) O157:H7 codes for two interacting DNA binding proteins,

80 rotect host epithelial cells against E. coli O157:H7 colonization, at least in part, by promoting muc

81 lular immune responses of cattle during EHEC O157:H7 colonization.

82 en enterohemorrhagic Escherichia coli (EHEC) O157:H7 colonizes the rectoanal junction (RAJ) in cattle

83 157:H7 from cultures containing 1000 E. coli O157:H7 colony-forming units (cfu)/mL, or approximately

84 ion of color was correlated with the E. coli O157:H7 concentration.

85 All samples testing positive for E. coli O157:H7 contained deer feces, and 5 tested farm fields h

86 munosensor for specific detection of E. coli O157:H7 contamination with the use of sandwich assay eva

87 tphone based fluorescence device for E. coli O157:H7 detection.

88 al-time monitoring method for viable E. coli O157:H7 developed in this study can be used to enrich an

89 ction experiments showed that 73% of E. coli O157:H7 died within 2 h with a disinfection rate constan

90 lity that E. coli Nissle 1917 can starve the O157:H7 E. coli strain EDL933 of gluconeogenic nutrients

91 157:H7 when tested against a panel of 15 non-O157:H7 E. coli.

92 Tir from enterohemorrhagic Escherichia coli O157:H7 expressed in epithelial cells induced a loss of

93 Concentrations of E. coli O157:H7 from 3x10(1) to 3x10(7)cfu/mL could be detected.

94 ction and identification of Escherichia coli O157:H7 from colony isolates in a colorimetric multiplex

95 tely 95%) in isolating live Escherichia coli O157:H7 from cultures containing 1000 E. coli O157:H7 co

96 sensitive tools for the detection of E. coli O157:H7 from food as they are host-specific and able to

97 assay shows promise for detection of E. coli O157:H7 from food in a simple, fast and sensitive manner

98 Enrichment assays using E. coli O157:H7 grown in LB broth with a reporter phage concentr

99 t is critical to eliminate or reduce E. coli O157:H7 gut colonization.

100 Escherichia coli O157:H7 has been shown to express heme uptake and transp

101 ted with different concentrations of E. coli O157:H7 have been tested using anti-E. coli O157-magneti

102 A specific DNA sequence from E. coli O157:H7 having 22 mers as an amine-terminated probe ssDN

103 upon cellular membrane anchors anti-E. coli O157:H7 IgM.

104 The food-borne pathogen Escherichia coli O157:H7 impacts the transcription of a subset of NF-kapp

105 Presence of higher than 1cfu E.coli O157:H7 in 25g of food has been considered as a dangerou

106 oli strains prevents colonization of E. coli O157:H7 in a mouse model.

107 id method which can detect low level E. coli O157:H7 in foods at real-time.

108 The detection of E. coli O157:H7 in foods has held the attention of many research

109 g contaminated food, most frequently E. coli O157:H7 in ground beef or fresh produce.

110 sensor was used for the detection of E. coli O157:H7 in ground beef samples.

111 ction and quantification of Escherichia coli O157:H7 in meat and water samples based on the electroca

112 complete inhibition of the growth of E. coli O157:H7 in the later 24 h irradiation.

113 rating the assay's ability to detect E. coli O157:H7 in the presence of high levels of background DNA

114 Limits of detection of Escherichia coli O157:H7 in undiluted milk were determined to be 6x10(4),

115 nts, survival of FIB but not that of E. coli O157:H7 increased in disinfected treatments, indicating

116 ding and uptake within the gut after E. coli O157:H7 infection should result in greater disease sever

117 ppaB inflammatory signaling, whereas E. coli O157:H7 infection suppressed this pathway by inhibiting

118 strawberries as a novel vehicle for E. coli O157:H7 infection, implicated deer feces as the source o

119 tect consumers from deadly foodborne E. coli O157:H7 infection, it is vital to develop a simple, reli

120 reased by TNF-alpha treatment and by E. coli O157:H7 infection.

121 measured Ang-1/2 in 77 children with E. coli O157:H7 infection.

122 was investigated in a mouse model of E. coli O157:H7 infection.

123 nt complex, was protective in murine E. coli O157:H7 infection.

124 In a recent cluster of Escherichia coli O157:H7 infections attributed to salad bar exposures and

125 This multistate outbreak of STEC O157:H7 infections was associated with consumption of ro

126 dates potentially useful for preventing EHEC O157:H7 infections.

127 nvestigated a multistate outbreak of E. coli O157:H7 infections.

128 nflammatory response acts to perturb E. coli O157:H7 intestinal colonization.

129 Escherichia coli O157:H7 is a notorious foodborne pathogen due to its low

130 E. coli O157:H7 is an enterohemorrhagic bacteria responsible for

131 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 is an important food-borne pathogen responsible

132 The pathogenicity of E. coli O157:H7 is mainly caused by the expression of Shiga-like

133 n of the foodborne pathogen Escherichia coli O157:H7 is of vital importance for public health worldwi

134 n of food contaminated with Escherichia coli O157:H7 is one of the major concerns in ensuring food sa

135 Escherichia coli O157:H7 is one of the most notorious foodborne pathogens

136 Enterohemorrhagic Escherichia coli O157:H7 is responsible for many outbreaks of gastrointes

137 higa toxin-producing Escherichia coli (STEC) O157:H7 is the causal agent for more than 96,000 cases o

138 red the virulence of K3995 to those of other O157:H7 isolates and an isogenic Stx2 mutant in rabbits

139 ubsequent disease after ingestion of E. coli O157:H7 may depend, at least in part, on individual diet

140 site platform was modified with anti-E. coli O157:H7 monoclonal antibody.

141 f these options is most relevant for E. coli O157:H7 on leafy green produce, we developed and applied

142 Otherwise, E. coli O157:H7 only growth 50% when PBE was added to the cultur

143 In 2006, a deadly Escherichia coli O157:H7 outbreak in bagged spinach was traced to Califor

144 Shiga toxin-producing Escherichia coli O157:H7 primarily resides in cattle asymptomatically, an

145 fected by the PhiV10 reporter phage, E. coli O157:H7 produces a strong bioluminescent signal upon add

146 Given that E. coli O157:H7 produces effectors that attenuate inflammatory s

147 as developed based on the ability of E. coli O157:H7 proteases to change the optical response of a su

148 While the amounts of culturable E. coli O157:H7 rapidly decline after introduction onto lettuce

149 lting from contamination of Escherichia coli O157:H7 remain a serious concern in food safety.

150 57 STEC in clinical samples and that E. coli O157:H7 remains the predominant cause of HUS in our inst

151 A 2006 spinach-associated outbreak of STEC O157:H7 resulted in higher hospitalization and HUS rates

152 ntext of the overall E. coli species and the O157:H7 sequence type 11 (ST11) subgroup.

153 n strategy for pathogenic strains of E. coli O157:H7 serotype based on a conserved signature insertio

154 ory effect of C70-TiO2 thin films on E. coli O157:H7 showed a decrease of the bacterial concentration

155 ors that were biofunctionalized with E. coli O157:H7 specific antibodies for sensitive pathogenic bac

156 r detecting and quantifying Escherichia coli O157:H7 specific eaeA gene.

157 a lineage distinct from previously reported O157:H7 ST11 EHEC and was not a member of the hypervirul

158 E. coli microdiversity analysis yielded one (O157:H7 str.

159 infection, while none of those infected with O157:H7 strain 2812 (Stx1a(+) Stx2a(+)) died or showed p

160 w (2%) fiber diets and infected with E. coli O157:H7 strain 86-24 (Stx2+).

161 with K3995 died than did those infected with O157:H7 strain 86-24 (Stx2a(+)).

162 lturable amounts of the nontoxigenic E. coli O157:H7 strain ATCC 700728 and the virulent strain EC404

163 We previously showed that enterohemorrhagic O157:H7 strain E. coli EDL933 colonizes a niche in the s

164 The E. coli O157:H7 strain EDL933 carries two copies of non-locus of

165 f the LEE, encoded by ecs1581 in the E. coli O157:H7 strain Sakai genome and present but not annotate

166 ne based capacitors were specific to E. coli O157:H7 strain with a sensitivity as low as 10-100 cells

167 roge et al. reported that E. coli 86-24, an O157:H7 strain, activates the expression of virulence ge

168 This study characterized an E. coli O157:H7 strain, designated PA2, that belongs to the hype

169 hen it is confronted with E. coli EDL933, an O157:H7 strain.

170 rtant for colonization of E. coli EDL933, an O157:H7 strain.

171 quid cultures using a bioluminescent E. coli-O157:H7 strain.

172 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 strains are major human food-borne pathogens, re

173 a probiotic E. coli strain that outcompetes O157:H7 strains for gluconeogenic nutrients could render

174 greater virulence of K3995 compared to other O157:H7 strains in rabbits and mice.

175 was less effective against Escherichia coli O157:H7 than free LAE, which was correlated with the ava

176 cision enhancer (IEE) was discovered in EHEC O157:H7 that promoted the excision of IS3 family members

177 nce by consuming nutrients needed by E. coli O157:H7 to colonize, thus preventing this first step in

178 The binding of E. coli O157:H7 to the IgM on B cell surface activates the B cel

179 p and tested for immunogenicity against EHEC O157:H7 using a murine model of gastrointestinal infecti

180 id technique to detect low levels of E. coli O157:H7 using membrane filtration and silver intensifica

181 the detection of pathogenic Escherichia coli O157:H7 using TCEP-reduced antibody with native antibody

182 (2) The retention of E. coli O157:H7 was 3.3 fold higher than that of E. coli K12 in

183 tocatalytic disinfection of Escherichia coli O157:H7 was achieved by using a C70 modified TiO2 (C70-T

184 sed Stx2 accumulation has been reported when O157:H7 was cocultured with phage-susceptible nonpathoge

185 detection system for viable Escherichia coli O157:H7 was developed using a piezoelectric biosensor-qu

186 or the common food pathogen Escherichia coli O157:H7 was developed.

187 An outbreak of Escherichia coli O157:H7 was identified in Oregon through an increase in

188 on limit, 100 CFU g(-1) dry soil) of E. coli O157:H7 was observed in the soils from Salinas Valley, C

189 s with 10(3), 10(4) and 10(5) CFU/mL E. coli O157:H7 were 106.98, 96.52 and 102.65 (in yogurt) and 10

190 hia coli, Enterococcus faecalis, and E. coli O157:H7 were compared in fresh (river) water and sedimen

191 sence of UV-A light against Escherichia coli O157:H7 were investigated.

192 O157:H7, the sensitive detection of E. coli O157:H7 were realized both in standard samples and real

193 .0 x 10(7) genome copies and was specific to O157:H7 when tested against a panel of 15 non-O157:H7 E.

194 ducing, food-borne pathogen Escherichia coli O157:H7 will develop a life-threatening sequela called t

195 confirmed cases as persons from whom E. coli O157:H7 with the outbreak PFGE pattern was cultured duri

196 We enrolled children infected with E. coli O157:H7 within 1 week of the onset of diarrhea in this p

197 and thus, enable visual detection of E. coli O157:H7 without instrumentation.

198 e assay truly discriminated Escherichia coli O157:H7's 16s rRNA from closely related bacteria with a

199 ontaminant Escherichia coli O157:H7 (E. coli O157:H7) in complex food products due to the recent outb

200 pathogenic Escherichia coli O157:H7 (E.coli O157:H7), a dangerous strain among 225 E. coli unique se

201 (Listeria monocytogenesis 19115 and E. coli O157:H7), clinical isolates (methicillin-resistant S. au

202 the gram-negative organism (Escherichia coli O157:H7), preventing unbiased detection and quantitation

203 (Salmonella enteritidis and Escherichia coli O157:H7).

204 ion of pathogenic bacteria (Escherichia coli O157:H7).

205 Escherichia coli O157:H7, a major Shiga toxin-producing pathogen, has a l

206 CFT073 and UTI89, enterohemorrhagic E. coli O157:H7, and enterotoxigenic E. coli O78:H11, compared t

207 der: Enterococcus faecalis, Escherichia coli O157:H7, and Escherichia coli D21f2.

208 uding Campylobacter jejuni, Escherichia coli O157:H7, and multidrug resistant Klebsiella pneumoniae.

209 f cattle is the primary reservoir of E. coli O157:H7, and thus, it is critical to eliminate or reduce

210 teraction between AuNPs and Escherichia coli O157:H7, AuNPs attached to the surface of the bacteria a

211 experiments using meats spiked with E. coli O157:H7, colicins efficiently reduced the population of

212 gens (Campylobacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, Listeria monocytogenes, and S

213 desirable for specific detection of E. coli O157:H7, one of the leading bacterial pathogens causing

214 biota could enhance the virulence of E. coli O157:H7, particularly a subset of clade 8 strains.

215 us aureus, Bacillus cereus, Escherichia coli O157:H7, Pseudomonas aeruginosa and Salmonella typhimuri

216 ilk), the LOD was under 5 CFU/mL for E. coli O157:H7, S. typhimurium and L. monocytogenes.

217 ccus faecalis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, St

218 mL of the tested pathogens (Escherichia coli O157:H7, Salmonella typhimurium, and Listeria monocytoge

219 E. coli O157:H7, suspended in 0.1% peptone, was inoculated onto

220 specific recognition of antibody to E. coli O157:H7, the sensitive detection of E. coli O157:H7 were

221 mune responses during colonization with EHEC O157:H7, the temporality of which is strain dependent, w

222 Irrespective of the presence of E. coli O157:H7, TNF-alpha enhanced activation of p65, the key m

223 higa toxin-producing E. coli [STEC], E. coli O157:H7, Vibrio cholerae, Yersinia enterocolitica, and t

224 Three strains of E. coli, BL 21, ATCC8739, O157:H7, when spiked into UHT milk and fermented palm ju

225 pathogenic species such as Escherichia coli O157:H7, which is a highly infectious and lethal member

226 fter gastrointestinal infection with E. coli O157:H7, which produces Shiga toxins (Stx) that cause he

227 Escherichia coli O157:H7-associated hemolytic-uremic syndrome (HUS) is ch

228 1,25(OH)2D3 altered E. coli O157:H7-induced reductions in transepithelial electrical

229 ly associated with enterohemorrhagic E. coli O157:H7-mediated hemorrhagic colitis that sometimes prog

230 is gram negative pathogenic species E. coli O157:H7.

231 nged with enterohemorrhagic Escherichia coli O157:H7.

232 nd resulted in decreased adhesion of E. coli O157:H7.

233 llness caused by strains of Escherichia coli O157:H7.

234 cause injury to the cell membrane of E. coli O157:H7.

235 amples from farm A were positive for E. coli O157:H7.

236 he gut from colonization by incoming E. coli O157:H7.

237 nsor for the detection of pathogenic E. coli O157:H7.

238 sensitive and selective detection of E. coli O157:H7.

239 Listeria monocytogenes and Escherichia coli O157:H7.

240 n even in low concentration range of E. coli O157:H7.

241 visual and quantitative detection of E. coli O157:H7.

242 ing the imbalance of redox status of E. coli O157:H7.

243 LT-1 from the complex cell lysate of E. coli O157:H7.

244 ains, including the enterohemorragic E. coli O157:H7.

245 stolytica, Cryptosporidium spp., and E. coli O157:H7; 95% for Giardia lamblia; 94% for ETEC and STEC;

246 te stand-alone test for detection of E. coli O157 in clinical samples.

247 ng enterohemorrhagic Escherichia coli (EHEC) O157 in seeded stool samples.

248 tool to inform national surveillance of STEC O157 in terms of identifying linked cases and clusters a

249 lora can influence the outcome of an E. coli O157 infection in mice.

250 we have determined that a subset of E. coli O157 infections will not be detected if an agar-based me

251 oratory characterization of Escherichia coli O157 is essential to inform epidemiological investigatio

252 toxin-producing Escherichia coli O157 (STEC O157), is key to rapidly identifying linked cases in the

253 rt vector machine analysis of bovine E. coli O157 isolate sequences can be applied to predict their z

254 nor subset (less than 10%) of bovine E. coli O157 isolates analyzed in our datasets were predicted to

255 E. coli O157 isolates clearly segregated into SNP lineages that

256 orphism (SNP) assay to segregate 148 E. coli O157 isolates from Australia, Argentina, and the United

257 enome sequencing was used to compare E. coli O157 isolates from host reservoirs (cattle and sheep) fr

258 olution of the relationships between E. coli O157 isolates than that provided by MLVA.

259 differences between human and bovine E. coli O157 isolates were due to the relative abundances of hun

260 Among E. coli O157 isolates, 57 (88%) were identified by both SMAC aga

261 comparison of human PT8 and animal PT21 VTEC O157 isolates.

262 tool to inform national surveillance of STEC O157; it can be used in real time to provide the highest

263 tudies of the global distribution of E. coli O157 lineages and the impacts of regionally predominant

264 he impacts of regionally predominant E. coli O157 lineages on the prevalence and severity of disease.

265 O157:H7 have been tested using anti-E. coli O157-magnetic beads conjugate (MBs-pECAb) as a capture p

266 ults prompted successful recovery of E. coli O157 (n = 25) and non-O157 STEC (n = 8) isolates, althou

267 nzymes in the E. coli serotype O157 (E. coli O157) O-PS biosynthetic pathway.

268 arily divided into two genomic lineages, the O157/O55 EHEC1 and non-O157 EHEC2.

269 curred in 12 patients (10 infected with STEC O157, one infected with STEC O125ac, and one with PCR ev

270 timates of linked clusters representing STEC O157 outbreaks in England and Wales increased by 2-fold

271 ocytotoxin-producing Escherichia coli (VTEC) O157 phage types (PTs), such as PT8 and PT2, are associa

272 hi8-positive strains were compared with VTEC O157 possessing BP933W.

273 s randomly sampled from 1002 strains of STEC O157 received by the Gastrointestinal Bacteria Reference

274 atic synthesis of Escherichia coli (serotype O157) RU-PP-Und.

275 iarrhea for non-O157 STEC in addition to the O157 serotype by using a sensitive assay.

276 veloped for the detection of the top six non-O157 Shiga toxin-producing Escherichia coli (STEC) O gro

277 Non-O157 Shiga toxin-producing Escherichia coli (STEC) strai

278 ighly pathogenic serotypes (O157 and top non-O157 Shiga toxin-producing Escherichia coli [STEC]) impl

279 x2) (including specific detection of E. coli O157), Shigella spp./enteroinvasive E. coli, Cryptospori

280 ivity and reliably detected Salmonella, EHEC O157, Shigella, and Campylobacter at concentrations 1- t

281 ul recovery of E. coli O157 (n = 25) and non-O157 STEC (n = 8) isolates, although the primary culture

282 th acute community-acquired diarrhea for non-O157 STEC in addition to the O157 serotype by using a se

283 lture and distinguishes between O157 and non-O157 STEC in clinical samples and that E. coli O157:H7 r

284 1 and stx2 for the detection of O157 and non-O157 STEC in diarrheal stool samples enriched in Gram-ne

285 e IEE sequences from O157 and the top 10 non-O157 STEC serotypes fell into clusters I and II, while l

286 ta showed an incidence rate of 51.2% for non-O157 STEC strains, with 5.8% of patients (1/17) with non

287 E. coli O157 and non-O157 STEC were detected in 35 and 18 cases, respectively

288 red in attempts to recover both O157 and non-O157 STEC.

289 li O157 and 3 (10%) were attributable to non-O157 STEC.

290 ch as Shiga toxin-producing Escherichia coli O157 (STEC O157), is key to rapidly identifying linked c

291 with non-O157 strains and 42.9% (6/14) with O157 strains (P = 0.03) developing hemolytic-uremic synd

292 rains, with 5.8% of patients (1/17) with non-O157 strains and 42.9% (6/14) with O157 strains (P = 0.0

293 were attributable to six "atypical" E. coli O157 strains and included recombinant regions.

294 esence of PT8-like phages in a panel of VTEC O157 strains belonging to different PTs and determined t

295 ich we named Phi8, was more frequent in VTEC O157 strains from human disease than in bovine strains.

296 One hundred five E. coli O157 strains isolated over a 5-year period from human fe

297 Citrobacter rodentium and Escherichia coli O157 triggered similar Th17 responses, whereas adhesion-

298 Subtilase cytotoxin (SubAB), produced by non-O157 type Shiga-toxigenic Escherichia coli (STEC), is an

299 LD-PCR differentiated STEC O157 from non-O157 using rfbEO157, and LD-PCR results prompted success

300 One such global pathogen is Escherichia coli O157, which causes a serious and sometimes fatal gastroi