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1                                              O2 binding to the Fe(II) heme complex of its N-terminal
2                                              O2 plasma etching increased the sensitivity due to incre
3                                              O2 plasma etching was performed by a microwave plasma sy
4 e cells adapted to a hypoxic environment (1% O2), those cultured in 5% O2 still mobilized sufficient
5  Our data support a new model in which a (1) O2 retrograde signal, generated by chlorophyll precursor
6 ncer cells, whereas provides an elevated (1) O2 environment in the mitochondria.
7 luorescent nanoprobe for singlet oxygen ((1) O2 ) detection in biological systems was designed, synth
8 t enables tumor-specific singlet oxygen ((1) O2 ) generation for cancer therapy, based on a Fenton-li
9 t specifically generates singlet oxygen ((1) O2 ) in mitochondria under white light irradiation that
10 atives exhibiting strong singlet oxygen ((1) O2 ) quenching (HCP2, HCP3) and another capable of dissi
11 vitro and in vivo through tumor-specific (1) O2 generation and subsequent ROS mediated mechanism.
12 rchitectures to optimize the response to (1) O2 .
13                                           (1)O2 generation by BADs in living cells enables visualizat
14 M generated similar levels of (3)DOM* and (1)O2, enhancing degradation of CTC, ROX, and SMX.
15                        Finally, PhiRI for (1)O2 and (3)DOM* correlated negatively with antioxidant ac
16                   Imidazole, a well-known (1)O2 scavenger, was incorporated in the hydrophobic core o
17 in the BODIPY subunit defines the site of (1)O2 addition.
18 enhanced fluorescence and singlet oxygen ((1)O2) production upon laser irradiation.
19 molecular oxygen produces singlet oxygen ((1)O2), which reacts with the anthracene moiety yielding hi
20 mited diffusion radius of singlet oxygen ((1)O2).
21 yer via the production of singlet oxygen ((1)O2).
22 s a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)2].
23 zed and converted into their endoperoxides 1-O2 upon oxidation with singlet oxygen.
24  min after three 5 min hypoxic episodes (11% O2, H1-H3).
25 d that continuously breathing normobaric 11% O2 from an early age prevents neurological disease and d
26 nally, we show that breathing normobaric 11% O2 in mice with late-stage encephalopathy reverses their
27 tion reactions carried out with labeled (18) O2 unambiguously show that the oxygen atoms present in t
28 e of an unusual (S)P species [(MeAN)2Cu(II)2(O2(2-))](2+) ((S)P(MeAN), MeAN: N-methyl-N,N-bis[3-(dime
29  similar -40 degrees C, cis-[Pd(IPr)2(eta(2)-O2)] adds a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)
30 on to reaction with O2, cis-[Pd(IPr)2(eta(2)-O2)] reacts at low temperature with H2O in methanol/ethe
31 e to further stabilize cis-[Pd(IMes)2(eta(2)-O2)].
32 ing of the NHC plane in cis-[Pd(IPr)2(eta(2)-O2)].
33 ia formation of a transient [Cu(I)(NH3)2](+)-O2-[Cu(I)(NH3)2](+) intermediate.
34  promising cathode materials, Na(Li1/3 M2/3 )O2 (M: transition metals featuring stabilized M(4+) ), f
35 tion mechanism in Li2 MnO3 , Na(Li1/3 Mn2/3 )O2 is designed as an example of a new class of promising
36          Human MSCs, grown chronically at 5% O2, were administered intravenously.
37                       Culture of cells in 5% O2 (>5 d) decreased histamine- but not shear stress-stim
38 ic environment (1% O2), those cultured in 5% O2 still mobilized sufficient Ca(2+) to activate AMPK.
39 r, increased dephosphorylation of eNOS in 5% O2 was Ca(2+)-sensitive and reversed by okadaic acid or
40 brane targeting of PP2A-C was observed in 5% O2, resulting in greater interaction with eNOS in respon
41 and biofilm formation under microaerobic (5% O2) conditions.
42 ban was increased in cells cultured under 5% O2.
43 dothelial cells cultured in physiologic (5%) O2 and stimulated with histamine or shear stress.
44 re chopped, modified atmosphere packaged (70%O2/30%CO2) and maintained under retail conditions (4+/-0
45 device based on the model system Gd0.1 Ce0.9 O2-delta /Er2 O3 to set and tune the property of "memris
46 o CO molecules, while Ag binds and activates O2 , and Ag/Pt surface proximity disfavors poisoning by
47                                Additionally, O2(*-) appears to be formed by the reduction of O2 at ei
48 ped SWCNTs with ionic strength, pH, adsorbed O2, and ascorbic acid.
49 of O2 with Al2O3 also activates the adsorbed O2 for receiving electrons from the photoexcited dyes.
50  of the Ru was still in molecular form after O2 evolution.
51 and the relatively hydrophilic surface after O2 plasma etching provided better resistance to fouling
52 in-between NIV sessions, was superior to air/O2 in reducing NIV failure (25-15%) in severe hypercapni
53 he decrease in matrix [dicarboxylate] allows O2 access to reduced site IIf, thereby making electron d
54 noxygenic phototrophic bacteria use BchE, an O2-sensitive [4Fe-4S] cluster protein, whereas plants, c
55 from Toxoplasma gondii is hydroxylated by an O2-dependent prolyl-4-hydroxylase (PhyA), and the result
56 s are identical to those created when (i) an O2 molecule accepts an electron from a near-surface dopa
57 e of NO consumption by metabolizing NO in an O2-dependent manner with decreased NO consumption in phy
58 a, and some phototrophic bacteria possess an O2-dependent enzyme, the major catalytic component of wh
59 ligated (deoxy), CO-inhibited (carboxy), and O2-bound (oxy) hemes in myoglobin (MB) and hemoglobin (H
60 treatment as it biologically couples CO2 and O2 fluxes.
61 l example, Schonecker showed that copper and O2 promoted the hydroxylation of steroid-containing liga
62 ngs through accompanying electrolytic H2 and O2 production were accounted for.
63  flue gas components (SO2, NO, NO2, H2O, and O2) on vanadium at 500-600 degrees C were investigated b
64 nability, relying on green oxidants H2O2 and O2 as the ultimate oxygen source.
65 ve activation of peroxide intermediates, and O2 evolution.
66 s on electronic charge of both heme iron and O2 , resulting in increased O2 dissociation and reduced
67 ing CO when triggered with visible light and O2.
68 ng gradual depletion of TAN (NH3 + NH4+) and O2 .
69  monooxygenase, all of which can bind NO and O2.
70  catalyze OH radical formation from H2 O and O2 at high temperatures.
71 uring simultaneously high CO2 resistance and O2 permeability and the exploitation of phase reactions
72  lipid peroxidation and of superoxide anion (O2(* horizontal line )) were higher in Prdx6 (-/-) than
73 etries of monochloramine (NH2Cl) and aqueous O2 consumption, derived (18)O-kinetic isotope effects ((
74 cts ((18)O-KIE) for the reactions of aqueous O2, and studied the impact of radical scavengers on NDMA
75 actions conducted with air rather than 1 atm O2.
76 thology and the concentration of atmospheric O2 Future work on glaciation-weathering-carbon cycle fee
77 ither assembling into spoked wheels, 1-5 bar O2, or closely packed in parallel lines, above 2.2 bar.
78 to use our previously reported gelatin-based O2-controllable hydrogels that can provide hypoxic micro
79 hniques, reflecting a tight coupling between O2 production and consumption by aerobic processes under
80 sed for detection and discrimination between O2 and H2O at low temperature.
81                                        Blood O2 saturation (r(2) = 0.80, P < 0.0001) and plasma gluco
82 otion of direct oxidation of palladium(0) by O2, bypassing the typical requirement for Cu salts or re
83 ddition to -C( identical withN), followed by O2 addition to -C(OH) horizontal lineN., internal H-shif
84                     However, inactivation by O2 remains an obstacle that prevents them being used in
85 )E and D2:(246)M are oxidatively modified by O2(*-) formed by the reduction of O2 either by PheoD1(*-
86 roduct resulting from the oxidation of NO by O2 under aerobic conditions.
87                  Abiotic Fe(II) oxidation by O2 commonly occurs in the presence of mineral sorbents a
88 alkyl halides, as well as rapid oxidation by O2, to generate detectable Ni(III) and/or Ni(IV) interme
89                           Therefore, cardiac O2 consumption is controlled by endothelial NO in a para
90 ystins in sensing and responding to cellular O2 levels.
91  with molecular oxygen: for instance, CH2I + O2 --> CH2OO + I.
92 bon nanotube yarn microelectrodes (CNTYMEs): O2 plasma etching and antistatic gun treatment.
93      The futile redox cycle rapidly consumes O2, rendering standard assays of Krebs cycle turnover un
94 diments to artificial groundwater containing O2 or NO3(-) under diffusion-limited conditions.
95 ding antibacterial activity of cycloviolacin O2 against A. baumannii.
96 uced arterial hypoxaemia (EIAH) can decrease O2 delivery and exacerbate exercise-induced quadriceps f
97 y inhibits mitochondrial complex I-dependent O2 consumption and reverse electron transfer-mediated re
98 of transition-metal complexes with dioxygen (O2 ) is important for understanding oxidation in biologi
99                                       Direct O2 utilization suffers from intrinsic challenges imposed
100 eal reaction mechanism, which permits direct O2 formation in single collisions of energetic water ion
101 indow of pH and in the presence of dissolved O2, but occurs independently of this optical quenching.
102 antified the functional significance of each O2 pathway defect by calculating the improvement in exer
103 en envisioned because they provide efficient O2 reduction with almost no overpotential.
104 parate electron inventories of four-electron O2 reduction and two-electron substrate oxidation.
105                            Finally, elevated O2 pressures are shown to contribute to background oxida
106 al cofactor in oxygen metabolism, especially O2 production via photosynthesis and the disposal of sup
107 ell as in situ collection of locally evolved O2 by photosystem 2 using a positioned scanning electroc
108 ut) and functional capacity by peak exercise O2 consumption.
109                             An extracellular O2 concentration of approximately 0.007 mm could supply
110 ffusion distances and permit relatively fast O2 uptake kinetics [4].
111 ential EPTM, 2-chloro-H2Q, allows for faster O2 reduction rates at higher applied potential.
112                              The emerging Fe-O2 bonding situation includes in essence a ferrous iron
113 oproteins and are also used as models for Fe-O2 systems.
114 the development of efficient biocathodes for O2 reduction relying both on direct and mediated electro
115 ip surpass the 2008 Robeson upper bounds for O2/N2, H2/N2, CO2/N2, H2/CH4 and CO2/CH4, with the poten
116 st that the adsorption mechanisms differ for O2 and H2O adsorption on ZnO, and are governed by the su
117                                         Free O2 levels in this layer were, however, undetectable by c
118  present in the Mn(IV) dimers originate from O2 .
119 ts origin was ascribed to primordial gaseous O2 incorporated into the nucleus during the comet's form
120 iO2 surface, we show that the tip-generated (O2)(-) radicals are identical to those created when (i)
121                           The rates of gross O2 production and carbon fixation in the SCM were found
122           The molecular basis for the O1 --&gt; O2 transition and how ChR2 modulates selectivity between
123         Furthermore, the generated acidic H2 O2 can oxidize l-Arginine (l-Arg) into NO for enhanced g
124 zonides from 1,5-dicarbonyl compounds and H2 O2 .
125 124 promoted anthocyanin accumulation and H2 O2 detoxification in response to cold.
126 e using nearly stoichiometric 3 % aqueous H2 O2 with a turnover frequency (TOF) of 16 000 h(-1) .
127 e ring system are developed as endogenous H2 O2 sensors.
128 ddition of ADMA reduced NOx and increased H2 O2 levels (p<0.001).
129 bsequently maintaining hydrogen peroxide (H2 O2 ) homeostasis in Arabidopsis.
130 -CoAs into trans-2-enoyl-CoA and produced H2 O2 .
131 nal excitation, which yields a remarkable H2 O2 -NO cooperative anticancer effect with minimal advers
132 s are active, although moderately, toward H2 O2 reduction.
133 n of glucose into gluconic acid and toxic H2 O2 , a novel treatment paradigm of starving-like therapy
134 of carbonyl and dicarbonyl compounds with H2 O2 were not detected because the structural distortions
135 plateaued to a rate similar to the U + H2O + O2 reaction.
136  (MAP), in our case, aerobic, vacuum or high O2, to extend the shelf life of beef.
137 that the best packaging conditions were high-O2 atmosphere in combination with REO.
138  baseline, O1A titres were 4.6 times higher, O2 titres were 9.4 times higher, O6A titres were 4.9 tim
139                  The Mo layer likely hinders O2 gas permeation, impeding contact with active Pt.
140  stress responses and thermogenesis, and how O2 deficiency leads to metabolic reprograming in cancer.
141 HIm)4](+), and LS-3DCHIm, [(DCHIm)F8Fe(III)-(O2(2-))-Cu(II)(DCHIm)3](+) (F8 = tetrakis(2,6-difluoroph
142 -Cu complexes, LS-4DCHIm, [(DCHIm)F8Fe(III)-(O2(2-))-Cu(II)(DCHIm)4](+), and LS-3DCHIm, [(DCHIm)F8Fe(
143 ction, as indicated by the level of impaired O2 extraction from arterial blood during peak exercise.
144 stive and capacitive responses to changes in O2 and H2O.
145 extent to which this reflects differences in O2 storage fluctuations and/or contributions from oxidat
146 -dependent structural dynamics of FeO NSs in O2.
147  after 10000 potential cycles (0.6-1.0 V) in O2 saturated acid.
148 the hypothesis that age-associated increased O2(*-) and resulting DNA damage mediate the increased su
149 th heme iron and O2 , resulting in increased O2 dissociation and reduced O2 affinity at high E degree
150          A sputter beam, consisting of large O2 clusters, was used to record depth profiles of alkali
151                          The lithium-air (Li-O2 ) battery has been deemed one of the most promising n
152 ncompetitive transport, the textile-based Li-O2 cathode exhibits a high discharge capacity of 8.6 mAh
153 ns of cathodic reactions in a liquid-cell Li-O2 microbattery in the presence of the redox mediator te
154 ies or a replacement for lithium metal in Li-O2 and Li-S batteries.
155 y of the discharging/charging products in Li-O2 cells.
156 st of all battery chemistries (lower than Li/O2 and Mg/O2 but comparable to Li/S), and Mg metal allow
157  charge potentials mainly takes place at Li2 O2 /electrolyte interfaces and has obvious correspondenc
158 otonation of the base and protonation of Li2 O2 .
159 y chemistries based on LiOH, rather than Li2 O2 , have been recently reported in systems with added w
160                     Discharge in the lithium-O2 battery is known to occur either by a solution mechan
161 radical, however, these peroxy radicals lose O2 in competition with bimolecular reactions.
162 +), whereas nitrifier denitrification at low O2 levels was stimulated by NO2(-) at levels as low as 0
163 h both production pathways stimulated by low O2, independent of NO2(-) concentrations.
164  inherent CO2 resistance, typically have low O2 permeability but this can be improved via different a
165                                 Hypoxia (low O2) is a fundamental microenvironmental determinant of b
166                How cancer cells adapt to low O2 has been illuminated by numerous studies, with "repro
167 es bacteria possibly contributed to lowering O2 levels in leaf pockets but did not release detectable
168 easonable faradaic efficiencies for measured O2 production.
169      Enhanced current densities for mediated O2 reduction are observed with the redox nanoparticle sy
170   Best-fit rates for Citrate-Fe(II) mediated O2 to O2(-) and H2O2 to OH were 3.0 +/- 0.7 and (4.2 +/-
171 battery chemistries (lower than Li/O2 and Mg/O2 but comparable to Li/S), and Mg metal allows reversib
172 ucts stimulated significantly more microbial O2 consumption (113 +/- 4 muM) than either the dark (78
173 total electron flow), even at sub-micromolar O2 concentrations.
174             CPC inhibited both mitochondrial O2 consumption [half maximal inhibitory concentration (I
175 sed SOD2 acetylation, elevated mitochondrial O2(. -), and diminished endothelial nitric oxide.
176 ored, demonstrating a role for mitochondrial O2(*-) in these effects.
177                        METHOD: Mitochondrial O2 consumption and adenosine triphosphate (ATP) synthesi
178 ELs and S3QELs, suppressors of mitochondrial O2()/H2O2 generation that do not inhibit oxidative phosp
179 anism such as the incorporation of molecular O2 are poorly understood.
180 of phoQ Salmonella to RNS requires molecular O2 and coincides with the nitrotyrosine formation, the o
181 ases with a precision better than 1% for N2, O2, CO2, He, Ar, 2% for Kr, 8% for Xe, and 3% for CH4, N
182 tible or easy-to-generate chemicals like N2, O2, CO2, CO, H2, or methane gas to value-added products
183 ding modes including N3-HgII-N3, N4-HgII-N3, O2-HgII-N3 and N3-HgII-O4.
184  (23)Na MAS NMR spectra of sodium-oxygen (Na-O2) cathodes reveals a combination of degradation specie
185 both the presence of H2O2 and the absence of O2 Experiments show that Ccp lacks enough activity to sh
186                            The activation of O2 on metal surfaces is a critical process for heterogen
187 l overlayer involves the mixed adsorption of O2 and H2O on a partially defected surface.
188 nic effect makes Pt favour the adsorption of O2, alleviating CO poisoning and promoting the catalysis
189 aximum (SCM) releases significant amounts of O2 to the otherwise anoxic environment.
190                  The surface complexation of O2 with Al2O3 also activates the adsorbed O2 for receivi
191 of conditions with varying concentrations of O2, NH4(+), and NO2(-).
192  and chemical absorption and dissociation of O2 , especially at tellurium vacancy sites.
193                The mechanism and dynamics of O2 dissociation are also reviewed, including the importa
194 experimental and computational enthalpies of O2 binding.
195 m characterized by physiological extremes of O2 tension and blood flow.
196 asis for the various biological functions of O2-utilizing metalloproteins.
197 n together, we propose that the influence of O2 availability on the levels of active Fur adds a previ
198 ur results suggest that seasonal influxes of O2 and NO3(-) may cause only localized mobilization of U
199 es, high pH, the presence of H2O2 instead of O2 as the initial Fe(II) oxidant, or a combination of al
200 en shown that LPMOs can use H2O2, instead of O2, as a cosubstrate.
201                            Resting levels of O2 in the rodent brain varied between 6.6 +/- 0.7 muM in
202 er conditions of variable cellular levels of O2.
203        Molecular dynamic simulations (MD) of O2 and H2O adsorption energy on ZnO surfaces were perfor
204 ations to uncover the molecular mechanism of O2 diffusion within the enzyme and its reactions at the
205                    The dominant mechanism of O2 transport into silage remains unresolved.
206 vide support for the postulated mechanism of O2(.-) activation at class I b Mn2 RNRs.
207 in the junctions, induced by the presence of O2 and H2 molecules, respectively.
208 the conversion are formed in the presence of O2 and that high temperature together with prolonged act
209  well as terminal oxidant in the presence of O2 as an external oxidant.
210 ngly, the decrease of GSH, the production of O2 , and the formation of nanoDVD are shown to be synerg
211       In this review, we survey the range of O2 activation processes mediated by heme proteins and mo
212 ess of the O-O sigma bond makes reactions of O2, which eventually lead to cleavage of this bond, very
213 ions involving 1- or 2-electron reduction of O2 Although often viewed as dangerous, ROS are now recog
214 *-) appears to be formed by the reduction of O2 at either PheoD1 or QA Early oxidation of D1:(332)H,
215 ither the two- or four-electron reduction of O2 can be explained by the constraint provided by the st
216 odified by O2(*-) formed by the reduction of O2 either by PheoD1(*-) or QA(*-) The identification of
217 s, which catalyze four-electron reduction of O2 to H2O.
218 X4) enzyme, which catalyzes the reduction of O2 to hydrogen peroxide (H2O2), has been implicated in t
219                 The 4H(+)/4e(-) reduction of O2 to water, a key fuel-cell reaction also carried out i
220 that serves as the main in vivo regulator of O2-dependent NO degradation in smooth muscle remains elu
221                               The release of O2 creates a hollow nanostructure with Li2O outer-shell
222 enges imposed by the triplet ground state of O2 and the disparate electron inventories of four-electr
223 ce on [Co] and [AcOH], but no dependence on [O2] or [Fc*].
224 o identify residues that contribute to O1 or O2 selectivity and gating to minimize undesirable effect
225                                     Overall, O2 plasma etching and antistatic gun treatment improve t
226 ndrial energy metabolism (glucose oxidation, O2 consumption, and ATP production), insulin secretion w
227                                      Oxygen (O2) acts as a potent upstream regulator of cell function
228 ene by using a controlled low energy oxygen (O2(+)/O(+))-ion for chemical adsorption and a low energy
229     Then, using isotopically labeled oxygen (O2) as an oxidant in the presence of hydrogen peroxide (
230 -type exoplanets including molecular oxygen (O2), ozone (O3), water vapor (H2O), carbon dioxide (CO2)
231 e eighth key metabolite is molecular oxygen (O2), thermodynamically activated for reduction by one el
232     However, LCHF also increased the oxygen (O2 ) cost of race walking at velocities relevant to real
233  are attributed to the elimination of the P2-O2 phase transition upon cycling to 4.5 V.
234 k cardiac power output and 69% achieved peak O2 consumption within the ranges of healthy controls.
235 ties relevant to real-life race performance: O2 uptake (expressed as a percentage of new VO2 peak ) a
236 ed a substantial fraction (70%) of gas-phase O2 More oxygenated products were formed than the amount
237 nce, photosynthetic CO2 and photorespiratory O2 fixation, and starch synthesis in response to changes
238 timuli that are apparent only in physiologic O2 levels.
239 nd mixtures, with and without physiological [O2].
240 ndidates for the pairing glue in pressurized O2.
241 recursor state, which dissociates to produce O2(-).
242 ost of these structures also develop in pure O2 and are identified as (surface) oxides.
243 ave a direct link to seagrass-derived radial O2 loss and secretion of dissolved organic carbon from t
244 ing in increased O2 dissociation and reduced O2 affinity at high E degrees ' values.
245                             First, reductive O2 activation induces selective oxidative cleavage, reve
246  show that, in general, the way the released O2 is accommodated is linked to lithium-ion diffusion an
247                                  By removing O2 in electrolyte, a dramatic decrease in Tafel slope of
248           Assembly of this cofactor requires O2, Fe(II), and a reducing equivalent.
249 s C, cis-[Pd(IPr)2(eta(2)-O2)] adds a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)2].
250 is was put forward after discounting several O2 production mechanisms in comets, including photolysis
251                                 Steady-state O2-evolution activity assays revealed that substitution
252 he metal center for reduction and subsequent O2 binding.
253 n of exogenous NO and endogenous superoxide (O2(*-)) produced in the electron transport chain.
254 erobic metabolism also generates superoxide (O2()) and hydrogen peroxide (H2O2) as bona fide products
255 nd maximal oxygen uptake ([Formula: see text]O2 max) were determined with the use of indirect calorim
256  MFO was explained by the [Formula: see text]O2 max, sex, and SRPAL with dietary carbohydrate (carboh
257 iability explained by the [Formula: see text]O2 max, sex, and SRPAL; dietary carbohydrate and fat int
258                                We found that O2 levels regulate the subcellular localization and chan
259 ical scavengers (ABTS and trolox) imply that O2 reacted with radical species.
260                      Our studies reveal that O2 reduces hydroxyl ion density at catalyst interface, r
261                                          The O2 concentration exerted the strongest control on net N2
262 ovided by the stannoxane core that makes the O2-binding to 1 an entropically unfavorable process.
263 nd pathophysiological microenvironments, the O2 concentration is not uniformly distributed but instea
264                   Systematic analysis of the O2 pathway in HFpEF showed that exercise capacity was un
265                           Examination of the O2 requirements of the CO release step revealed that the
266 rophic bacteria reveals three classes of the O2-dependent enzyme.
267 ermore, both oxides are unstable outside the O2 atmosphere, indicating the presence of active O atoms
268 n species, which damages DNA and reduces the O2 level; (2) decreased cross-membrane proton gradient f
269 rovide direct experimental evidence that the O2 generated during the OER on some highly active oxides
270 eps of oxygen transport and utilization (the O2 pathway) in each patient with HFpEF, identifying the
271 tin complex through its interaction with the O2-sensing prolyl hydroxylase domain containing protein
272 s via their role in Fe(II) oxidation through O2 production, the capacity of their cell surfaces to so
273 ite IIf, thereby making electron donation to O2 possible, explaining the rapid increase in ROS produc
274            How to efficiently oxidize H2O to O2 (oxygen evolution reaction, OER) in photoelectrochemi
275 ), reducing CO2 into CO and oxidizing H2O to O2 with a 64% electricity-to-chemical-fuel efficiency.
276 -fit rates for Citrate-Fe(II) mediated O2 to O2(-) and H2O2 to OH were 3.0 +/- 0.7 and (4.2 +/- 1.7)
277 h lower transport efficiency with respect to O2 consumption.
278 PFM), demonstrate differences in response to O2 and H2O, confirming that different adsorption mechani
279 We propose that the distinctive responses to O2 and H2O adsorption on ZnO could be utilized to statis
280       MoTe2 is found to be ultrasensitive to O2 at elevated temperatures (250 degrees C).
281               Here, multiple sensors tracked O2 and CO2, gas pressure (DeltaP) between internal silag
282 ion of the two unpaired electrons in triplet O2, relative to the unpaired electrons in two hydroxyl r
283 ce stabilization of the pi system of triplet O2, the weakness of the O-O sigma bond makes reactions o
284 r propane ODH after thermal activation under O2 to open a cobalt coordination site and to oxidize Co(
285     Here we combine high-resolution underway O2/Ar, which provides an estimate of net community produ
286  single, steady-state process at 723 K using O2 as an abundantly available oxidant.
287 larly in developing systems that can utilize O2, will be required to develop a practical process that
288  reported can directly or indirectly utilize O2 as the only net coreactant based only on thermodynami
289 xcludes the intermediacy of mer-(ONO(Q))Re(V)O2(IMes) in this oxygen atom transfer reaction.
290 nce functional hypoxia, a situation in which O2 supply is inadequate to meet oxygen demand.
291 e-supported cobalt(I) complex L(tBu) Co with O2 gives a rare example of a side-on dioxygen complex of
292              Whereas amine donors react with O2-chemisorbed AC and nucleophiles to give dehydrogenati
293                           1 was reacted with O2(.-) at -40 degrees C resulting in the formation of a
294 the sensory module, undergoing reaction with O2 that leads to conversion to a [2Fe-2S] form with loss
295                 In addition to reaction with O2, cis-[Pd(IPr)2(eta(2)-O2)] reacts at low temperature
296 chemistry at mild potentials and reacts with O2, CO2, and ethylene via formal [4+2] cycloaddition to
297 2 and CO evolved and has a relationship with O2 evolved from the TMO lattice on the first charge.
298 ples calculations for undoped and O-doped Y2 O2 Bi.
299 superconductivity is satisfied in O-doped Y2 O2 Bi.
300                Several Bi 6p x/y bands of Y2 O2 Bi are raised in energy by oxygen doping because the

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