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1 cosylase, Nth protein (endonuclease III) and O6-methylguanine-DNA methyltransferase.
2 ne S-transferase P1, and the DNA repair gene O6-methylguanine-DNA-methyltransferase.
3 uclease protein resulted in the retention of O6-methylguanine DNA methyltransferase activity but loss
4 These results demonstrate that the fusion of O6-methylguanine DNA methyltransferase and apurinic endo
5 udied: 27% (26/95) at p16, 33% (31 of 95) at O6-methylguanine-DNA-methyltransferase; and 18% (17 of 9
6 of the apurinic endonuclease portion of the O6-methylguanine DNA methyltransferase-apurinic endonucl
7 tructed a human fusion protein consisting of O6-methylguanine DNA methyltransferase coupled with an a
8 ges in molecules involved in DNA repair (eg, O6-methylguanine-DNA methyltransferase, DNA topoisomeras
9 ve DNA lesion, O6-methylguanine (O6-MeG), by O6-methylguanine-DNA-methyltransferase (E.C. 2.1.1.63, M
10 ructure-function information about the human O6-methylguanine-DNA methyltransferase (EC 2.1.1.63), as
11 ctional 39 kDa Escherichia coli Ada protein (O6-methylguanine-DNA methyltransferase) (EC 2.1.1.63), p
12 or radiotherapy alone, with consideration of O6-methylguanine-DNA-methyltransferase gene (MGMT) promo
13 ion-specific PCR and included: p16 (CDKN2A), O6-methylguanine-DNA-methyltransferase, glutathione S-tr
14 DNA repair enzymes, DNA polymerase beta and O6-methylguanine-DNA methyltransferase, have been shown
15 t increased levels of the DNA repair protein O6 methylguanine-DNA methyltransferase (MGMT), also refe
18 etinoic acid receptor-beta2 (RAR-beta2), and O6-methylguanine DNA methyltransferase (MGMT) genes were
20 of the gene encoding the DNA repair protein O6-methylguanine DNA methyltransferase (MGMT) might be r
21 ow cells express extremely low levels of the O6-methylguanine DNA methyltransferase (MGMT) protein th
25 tal carcinogens and the protective effect of O6-methylguanine DNA methyltransferase (MGMT), heterozyg
26 etection of four different proteins, avidin, O6-methylguanine DNA methyltransferase (MGMT), SNAP-tag,
31 h-level expression of the DNA repair protein O6-methylguanine-DNA methyltransferase (MGMT) correlates
36 The mechanism whereby the DNA repair protein O6-methylguanine-DNA methyltransferase (MGMT) is silence
37 ed methylation data from a CpG island in the O6-methylguanine-DNA methyltransferase (MGMT) promoter.
38 nation in the disposition of the inactivated O6-methylguanine-DNA methyltransferase (MGMT) protein in
45 rivatives is countered by the repair protein O6-methylguanine-DNA methyltransferase (MGMT), which rem
49 essor gene p16 (CDKN2A), the DNA repair gene O6-methylguanine-DNA-methyltransferase (MGMT) and the pu
51 r of metalloproteinase 3 (TIMP-3), p16INK4a, O6-methylguanine-DNA-methyltransferase (MGMT), death-ass
53 uced cells in vivo, selection based on P140K O6-methylguanine-DNA-methyltransferase (MGMT[P140K]) gen
54 ariate analysis, the factors age, WHO grade, O6-methylguanine-DNA methyltransferase promoter methylat
55 wed prognostic significance, with WHO grade, O6-methylguanine-DNA methyltransferase status, age, and
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