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1                                              ONJ appears to be time-dependent with higher risk after
2                                              ONJ associated with IV bisphosphonate therapy is extreme
3                                              ONJ occurred posterior to the cuspids (n = 20) mostly in
4                                              ONJ resolved in 60 patients (62%), resolved and recurred
5                                  We found 16 ONJ cases among 572,606 cohort members; seven additional
6                              We enrolled 191 ONJ cases and 573 controls in 119 dental practices.
7  prospectively for a minimum 3.2 years after ONJ.
8       The variables predictive of developing ONJ were dental extraction (P = .009), treatment with pa
9 ered profile in wound-healing markers during ONJ development.
10 ith recurrent/nonhealing than single-episode ONJ (84% v 62%; P = .02).
11 ctice-Based Research Network study estimated ONJ incidence and odds ratios from bisphosphonate exposu
12 xtractions were independent risk factors for ONJ.
13 ate with physicians to minimize the risk for ONJ.
14 ons and suggests that the absolute risks for ONJ from oral bisphosphonates is low.
15 were 15.5 (CI, 6.0-38.7) more likely to have ONJ than non-exposed patients; however, the sparse numbe
16 ce of dental disease and tooth extraction in ONJ pathogenesis and help delineate an altered profile i
17  were 62 men; the median age was 61 years in ONJ patients and 58 years among the rest.
18   The incidence of osteonecrosis of the jaw (ONJ) in the population is low, but specifics are unknown
19                    Osteonecrosis of the jaw (ONJ), a side-effect of bisphosphonate therapy, is charac
20 lation, bone pain, osteonecrosis of the jaw (ONJ), atypical fractures, and osteosarcoma.
21 ral fractures, and osteonecrosis of the jaw (ONJ), the pathophysiological mechanisms involved are not
22 nal development of osteonecrosis of the jaw (ONJ).
23 de a discussion of osteonecrosis of the jaw (ONJ).
24 weigh the risk for osteonecrosis of the jaw (ONJ).
25 n in patients with osteonecrosis of the jaw (ONJ).
26                   Osteonecrosis of the jaws (ONJ) is a rare but severe complication of antiresorptive
27 onate therapy and osteonecrosis of the jaws (ONJ), but neither causality nor specific risks for lesio
28 ients with single, recurrent, and nonhealing ONJ (P = .2).
29 , 60.7 to 84.2 months), 33 possible cases of ONJ were reported, all in the zoledronate-treated patien
30 rmed as being consistent with a diagnosis of ONJ, representing a cumulative incidence of 2.1% (95% CI
31                We report on the frequency of ONJ and investigate oral health-related quality of life
32 r studies of the pathogenesis and healing of ONJ are needed.
33 einitiating bisphosphonates after healing of ONJ is a reasonable option in patients experiencing rela
34  trial is associated with a low incidence of ONJ but does not seem to adversely affect Oral-QoL.
35 osed patients; however, the sparse number of ONJ cases limits firm conclusions and suggests that the
36                 All potential occurrences of ONJ were reported as serious adverse events and centrall
37                               Higher risk of ONJ began within 2 years of bisphosphonate initiation an
38                         More than a third of ONJ patients also suffered from long bone fractures (n =
39                          Dental treatment of ONJ should be conservative and provide relief to the pat
40                   Dental procedures preceded ONJ in 46 patients (47%) and were more common in those w
41                                    Recurrent ONJ followed reinitiation of bisphosphonates in six of 1
42 r infections and four patients had recurrent ONJ.
43  Committee also discusses measures regarding ONJ.
44          Incidence of bisphosphonate-related ONJ in osteoporosis patients is unclear, but several stu
45                    Patients with spontaneous ONJ have a higher risk of nonhealing and recurrence.
46 tal extraction, including 12 patients in the ONJ group.
47             Median time from MM diagnosis to ONJ was 8.4 years for the whole group.
48  Oral and intravenous bisphosphonate-treated ONJ sites had reduced numbers of basal epithelial progen
49 phosphonate use was strongly associated with ONJ (odds ratios [OR] 299.5 {95%CI 70.0-1282.7} for intr
50 l disease have been strongly associated with ONJ development.
51 R = 6.6 {1.6-26.6}) remained associated with ONJ.
52 hosphonate use were strongly associated with ONJ.
53 osis therapies and many adverse events, with ONJ an exception.
54 sphosphonate use and other risk factors with ONJ.
55  examined human tissue from individuals with ONJ and non-bisphosphonate-treated control individuals t
56 portantly, human biopsies from patients with ONJ showed similar findings.

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