ライフサイエンスコーパス: OVX

1 OVX caused a 17% decrease in plasma glucose, which was c

2 OVX caused a reduction in osteocyte density in alveolar

3 OVX female mice had increased lung SPP1 expression in re

4 OVX increased ischemic damage in +/? mice; estrogen redu

5 OVX mice had increased adiposity that was prevented with

6 OVX NTG, CRPtg, and CRPtg lacking Fc gamma RI, Fc gamma

7 OVX was associated with a significant decline in perform

8 OVX+E mice exhibit negative feedback in the A.M. and pos

9 OVX-Diet rats showed enhanced osteoblastogenesis and ost

10 OVX-induced bone loss was associated with increased oste

11 (Intact, 0.26 +/- 0.01; OVX, 0.22 +/- 0.01; OVX+E(2), 0.28 +/- 0.01 mg/min per 100 g) and increase i

12 creatinine clearance (Intact, 0.26 +/- 0.01; OVX, 0.22 +/- 0.01; OVX+E(2), 0.28 +/- 0.01 mg/min per 1

13 ) and increase in BUN (Intact, 20.3 +/- 2.1; OVX, 32.6 +/- 5.1; OVX+E(2), 24.3 +/- 2.4 mg/dl).

14 UN (Intact, 20.3 +/- 2.1; OVX, 32.6 +/- 5.1; OVX+E(2), 24.3 +/- 2.4 mg/dl).

15 modifications in BV/TV (intact, 28.7+/-1.6; OVX, 16.3+/-0.9; OVX+E2, 25.7+/-1.4%).

16 llowed the same pattern (intact, 33.1+/-1.6; OVX, 34.4+/-3.7; OVX+E2, 42.4+/-2.1%).

17 attern (intact, 33.1+/-1.6; OVX, 34.4+/-3.7; OVX+E2, 42.4+/-2.1%).

18 en replacement (intact, 55+/-8; OVX, 59+/-7; OVX+E2, 92+/-4%).

19 d with estrogen replacement (intact, 82+/-7; OVX, 61+/-9; OVX+E2, 90+/-4%), which corresponded with s

20 d with estrogen replacement (intact, 55+/-8; OVX, 59+/-7; OVX+E2, 92+/-4%).

21 BV/TV (intact, 28.7+/-1.6; OVX, 16.3+/-0.9; OVX+E2, 25.7+/-1.4%).

22 en replacement (intact, 82+/-7; OVX, 61+/-9; OVX+E2, 90+/-4%), which corresponded with similar modifi

23 he depolarizing sag and was increased across OVX --> diestrous --> proestrous mice.

24 changes in synaptic functions, we used adult OVX rats to evaluate the consequences of short-term (7-1

25 the liver estrogen receptor alpha, E2 after OVX limited adiposity but failed to improve insulin sens

26 omotor activity and energy expenditure after OVX can explain these metabolic changes.

27 after sham surgery compared to OVX or after OVX plus estrogen replacement compared to OVX plus place

28 to 14 months (M14) after laparotomy or after OVX-Diet, with intermediate time points at M3 and M12.

29 d in mice 1-3 weeks (but not 10 weeks) after OVX by the selective ERbeta agonist, LY3201, given as co

30 ltiple beneficial cognitive effects in aged, OVX rhesus macaque females.

31 lation in obese animals prior to OVX plus an OVX-induced positive energy imbalance might cooperate to

32 ns between ovariectomized (OVX) controls and OVX animals treated with a silastic capsule containing E

33 were recorded in GnRH neurons from OVX+E and OVX mice in coronal and sagittal slices.

34 owever, both VCD-induced ovarian failure and OVX led to a dramatic reduction in the extent of excitot

35 ve protective effect of estrogen, female and OVX plus estrogen mice were relatively resistant to MPTP

36 y high systemic estrogen (intact females and OVX females and males administered estrogen subcutaneous

37 male rats, ovariectomized (OVX) females and OVX females treated with 17beta-estradiol (E2).

38 administration, intact males and females and OVX+E but not OVX+Veh females were less sensitive to the

39 Untreated ovarian-intact and OVX animals were compared with OVX animals receiving est

40 es using expression profiles from intact and OVX mice from a panel of inbred strains.

41 fects of OFQ were dose dependent in male and OVX animals and were reversibly antagonized by UFP-101 (

42 Male and OVX female rats were significantly more sensitive than i

43 istant to MPTP toxicity compared to male and OVX plus placebo mice, respectively.

44 aspect of energy storage was manipulated and OVX itself had no overall effect on post-breeding surviv

45 d GnRH neuron response in cells from OVX and OVX+E mice in the morning but not afternoon.

46 ute in vitro treatment of cells from OVX and OVX+E mice with estradiol rapidly increased HVA currents

47 its metabolites between OVX plus placebo and OVX plus estrogen mice.

48 tes a statistical difference in the sham and OVX D-spacing distributions (P<0.01).

49 In sham and OVX mice, HFD feeding induced fatty liver, and insulin r

50 r neuron was equivalent in young OVX+Veh and OVX+E groups.

51 acological inhibition of ERRalpha attenuated OVX-induced bone loss in mice.

52 vels of caffeine and its metabolites between OVX plus placebo and OVX plus estrogen mice.

53 mediate between the high levels of bilateral OVX (no reproductive investment) and the low levels of S

54 he expression of four modules was altered by OVX, including module 23 whose expression was decreased

55 module 23 whose expression was decreased by OVX across all strains.

56 n young rats, vasodilation was diminished by OVX and restored with estrogen replacement (intact, 82+/

57 old animals, vasodilation was unaffected by OVX but enhanced with estrogen replacement (intact, 55+/

58 tion of caffeine/ovariectomy; or 4) caffeine/OVX = ingestion of caffeine/ovariectomy.

59 The OVX and caffeine/OVX groups presented a greater number of TRAP(+) cells a

60 hate; OMSC, orofacial mesenchymal stem cell; OVX, ovariectomized.

61 In contrast, OVX animals that were chronically replaced with either e

62 In contrast, OVX+E cells recorded in the p.m. showed an increased mea

63 In contrast, OVX+E mice showed a large LH surge (8- to 124-fold relat

64 experienced either short-term (10 d, control OVX) or long-term (5 months, OVX(LT)) ovarian hormone de

65 In the control OVX neurons, E(2) acutely increased IE and reduced the s

66 protein than adipocytes of pairfed control (OVX) mice, and this difference was associated with enhan

67 r duration of severe seizures among control, OVX and VCD-treated mice, OVX+E mice exhibited seizures

68 variectomized and received a deficient diet (OVX-Diet).

69 e expression is significantly reduced in DIO OVX rats.

70 were ovariectomized, randomized to estrogen (OVX-E2) or control pellet implants (OVX-C), and pairfed

71 In addition, following OVX, there was an increase in plasma ghrelin that was te

72 lary, and mandibular alveolar bone following OVX, yet was increased in lingual mandibular alveolar bo

73 ge in the efficacy for E2 5 months following OVX.

74 E2) therapy when initiated shortly following OVX but not after substantial delay.

75 lacement therapy initiated shortly following OVX but not after substantial delay.

76 increased GnRH neuron response in cells from OVX and OVX+E mice in the morning but not afternoon.

77 Acute in vitro treatment of cells from OVX and OVX+E mice with estradiol rapidly increased HVA

78 urnal changes in PSC frequency in cells from OVX mice in either slice orientation.

79 X+E mice were lower than those in cells from OVX mice in the A.M. but were higher in the P.M.

80 was no time-of-day difference in cells from OVX mice.

81 ice; this response was blunted in cells from OVX mice.

82 with time of day; HVA currents in cells from OVX+E mice were lower than those in cells from OVX mice

83 ts (PSCs) were recorded in GnRH neurons from OVX+E and OVX mice in coronal and sagittal slices.

84 e membrane potential of arcuate neurons from OVX+E mice; this response was blunted in cells from OVX

85 In both kisspeptin neuron populations from OVX mice, the frequency of GABAergic spontaneous postsyn

86 identified GnRH neurons in brain slices from OVX+E or OVX female mice were recorded during the mornin

87 Furthermore, OVX increased food intake and body weight in wild-type m

88 mplant (OVX), 17-beta estradiol (E) implant (OVX+E) or E implant plus cyclic oral progesterone (OVX+E

89 ent with either a subcutaneous sham implant (OVX), 17-beta estradiol (E) implant (OVX+E) or E implant

90 subset was treated with estradiol implants (OVX+E).

91 or OVX and treated with estradiol implants (OVX+E).

92 strogen (OVX-E2) or control pellet implants (OVX-C), and pairfed for 40 days.

93 In OVX rats, 5-min intracerebroventricular infusion of a PA

94 In OVX+E cells in both orientations, PSC frequency was low

95 age chemoattractants (CINC-2alpha, MCP-1) in OVX+V arteries and E2-induced inhibition of CINC-2alpha

96 behavior in female rats at proestrous and in OVX rats after E2 treatment.

97 tation induces activated T-cell apoptosis in OVX mice via Fas ligand (FasL)-mediated Fas pathway acti

98 anges in any aspect of HVA-mediated I(Ca) in OVX mice.

99 In contrast, in OVX+E mice, HVA-mediated currents varied with time of da

100 sponse to vascular injury provoked by CRP in OVX CRPtg depends on Fc gamma RI and probably requires i

101 tic and pharmacodynamic properties of DVS in OVX rats.

102 o exacerbate thermoregulatory dysfunction in OVX rats.

103 iol, and produced a dose-dependent effect in OVX females treated with 1 ng to 100 microg of estradiol

104 Experiments in OVX mice given estradiol replacement identified an estra

105 ssel number and stimulates bone formation in OVX mice.

106 atment showed TTX decreased PSC frequency in OVX+E cells in sagittal slices, but not coronal slices.

107 tenuated the orexigenic action of ghrelin in OVX female and male rats.

108 ut not the CA3, region of the hippocampus in OVX compared to intact rats.

109 sponsive, and following s.c. implantation in OVX versus OVX + E(2) mice, E(2) action on the host comp

110 ve stress, a process known to be involved in OVX-induced bone loss.

111 on-genomic actions of E2 promote leanness in OVX mice independently of reduced energy intake.

112 LH in OVX mice showed no time-of-day difference.

113 A1), piriform cortex, and BNST were lower in OVX+E2 females compared to females without E2.

114 e mass marker) were down-regulated at M12 in OVX-Diet rats.

115 ggest that cognitive impairments observed in OVX rats may be associated with morphological changes in

116 ion of F4/80+CRP+ cells was revealed only in OVX CRPtg.

117 ast development in vitro and osteoporosis in OVX rat models.

118 and ameliorates the osteopenia phenotype in OVX mice.

119 ation would be sufficient to restore pLTF in OVX rats.

120 for restoring thermoregulatory processes in OVX rats.

121 ient in Snca and observed a 40% reduction in OVX-induced bone loss.

122 of mirtazapine on temperature regulation in OVX rat models and explore further the role of 5-HT(2A)

123 oss associated with oestrogen replacement in OVX rats, we food restricted a separate group of OVX rat

124 ng enhanced bone formation and resorption in OVX-Diet rats.

125 was also higher than ShA control subjects in OVX+E but not OVX+Veh females after ExA self-administrat

126 ynapse/microm3) only slightly higher than in OVX control rats.

127 ood restricted rats were similar to those in OVX rats treated with the oil vehicle.

128 Thus, in OVX rats, DVS has good pharmacokinetic properties, rapid

129 ficantly suppressed by estrogen treatment in OVX ERalpha-/AA mice.

130 y relevant regimen of estradiol treatment in OVX rats increases Pet-1 and 5-HTT mRNA levels in the mi

131 and protects mice from ovariectomy-induced (OVX-induced) osteoporosis.

132 In the a.m., OVX+E cells showed changes in GnRH neuron firing reflect

133 ompared with OVX cells, whereas in the p.m., OVX+E cells exhibited changes suggesting positive feedba

134 res among control, OVX and VCD-treated mice, OVX+E mice exhibited seizures of a significantly longer

135 (10 d, control OVX) or long-term (5 months, OVX(LT)) ovarian hormone deficiency.

136 Nevertheless, OVX females treated with vehicle exhibited a substantial

137 r than ShA control subjects in OVX+E but not OVX+Veh females after ExA self-administration, confirmin

138 , intact males and females and OVX+E but not OVX+Veh females were less sensitive to the effects of D1

139 Adipocytes of OVX-E2 mice contained >3-fold more perilipin protein tha

140 sed (2 to 5000 times) in injured arteries of OVX+V rats at 2 hours and was reduced by 24 hours.

141 insulin sensitivity and body composition of OVX rats bred for low-running capacity.

142 In contrast, the average T(CORE) of OVX, KNDy-ablated rats was lower than OVX control rats a

143 eated female rats, as well as the effects of OVX on plasma ghrelin and hypothalamic orexigneic neurop

144 and biomechanics confirmed bone fragility of OVX-Diet rats, and quantitative RT-PCR revealed a higher

145 rats, we food restricted a separate group of OVX rats and evaluated Isop-induced changes in MAP, HR a

146 ds to increased tumor burden in the lungs of OVX + E(2) mice.

147 ys and with performance levels below that of OVX+E animals.

148 re also found to be reduced in the tibias of OVX mice.

149 ty that was prevented with E2 at the time of OVX.

150 ntact, 3) young ovariectomized (OVX), 4) old OVX, 5) young OVX plus estrogen replacement (OVX+E2), an

151 us estrogen replacement (OVX+E2), and 6) old OVX+E2.

152 ow endogenous estrogen (i.e., intact and old OVX), vasodilation was correlated with BV/TV (R(2) = 0.6

153 ed on neurons from approximately 7-month-old OVX rats that experienced either short-term (10 d, contr

154 stent and protective effects of SPI diets on OVX-induced bone loss were associated with down-regulati

155 male C57BL/6J mice underwent sham operation, OVX, or OVX with estradiol (E2) treatment and were fed a

156 Increased energy intake in both HFD and/or OVX groups, and decreased locomotor activity and energy

157 n mesenteric adipose tissue after HFD and/or OVX, independent of previous postnatal programming, yet

158 d GnRH neurons in brain slices from OVX+E or OVX female mice were recorded during the morning or afte

159 adult mice that were ovariectomized (OVX) or OVX and treated with estradiol implants (OVX+E).

160 -aged rats that were ovariectomized (OVX) or OVX and treated with estradiol.

161 BL/6J mice underwent sham operation, OVX, or OVX with estradiol (E2) treatment and were fed an HFD.

162 ologic treatment of intact aged male rats or OVX female rats with Scl-Ab had no effect on morphologic

163 had higher bone density as compared to other OVX mice groups.

164 Ovariectomized (OVX) female rats were given estradiol or cholesterol imp

165 Ovariectomized (OVX) female rats with and without estradiol replacement

166 Ovariectomized (OVX) mice treated or not with estradiol (E) were studied

167 Ovariectomized (OVX) rats (n=5-6/group) were treated with 0, 2, or 10 mi

168 Ovariectomized (OVX) rats 24 h (but not 6 or 72 h) after estradiol benzo

169 f caffeine/sham surgery); 3) ovariectomized (OVX) = non-ingestion of caffeine/ovariectomy; or 4) caff

170 cl-Ab) in aged male rats and ovariectomized (OVX) female rats were used to study the effects of scler

171 , intact male and intact and ovariectomized (OVX) female rats with and without estradiol replacement

172 female (freely cycling), and ovariectomized (OVX) females treated with either estrogen benzoate (EB;

173 Intact males, females, and ovariectomized (OVX) females with and without estradiol (vulnerable, OVX

174 ChR-positive neurons between ovariectomized (OVX) controls and OVX animals treated with a silastic ca

175 drawal in morphine-dependent ovariectomized (OVX) rats (MD model), while the second model relies on t

176 ertaken in brain slices from ovariectomized (OVX), diestrous, and proestrous kisspeptin-GFP mice.

177 ects of exogenous ghrelin in ovariectomized (OVX) and estradiol (E2)-treated female rats, as well as

178 reased cell proliferation in ovariectomized (OVX) animals, an effect that was reversed by the adminis

179 activity was not enhanced in ovariectomized (OVX) female mice as a result of cardiac stress, but admi

180 n catecholaminergic areas in ovariectomized (OVX) female rats.

181 prefrontal cortex (dlPFC) in ovariectomized (OVX) female rhesus monkeys, and that E induces a corresp

182 lar injury is exaggerated in ovariectomized (OVX) human C-reactive protein transgenic mice (CRPtg) co

183 ive tumor cells implanted in ovariectomized (OVX) mice that contain s.c. implants of placebo (OVX) or

184 nisms mediating bone loss in ovariectomized (OVX) mice, a model of human menopause, using co-expressi

185 (2) pLTF would be absent in ovariectomized (OVX) rats and in physiological conditions in which serum

186 and the cecal microbiota in ovariectomized (OVX) rats bred for low-running capacity (LCR), a model t

187 inding, in male rats, and in ovariectomized (OVX) rats given estradiol benzoate (EB) or oil vehicle (

188 A previous study in ovariectomized (OVX) rats has suggested that mirtazapine can alleviate t

189 culating levels of AngII, in ovariectomized (OVX) rats treated with oestradiol benzoate (EB).

190 , have not been performed in ovariectomized (OVX) rats, a model that mimics the loss of ovarian hormo

191 However, in ovariectomized (OVX) WT and in ERbeta(-/-) mice, there was a marked redu

192 solitary tract (NTS) of lean ovariectomized (OVX) rodents.

193 croinjection of OFQ in male, ovariectomized (OVX), and diestrous rats produced a significant antinoci

194 rol Spp1+/+ (C57BL/6J) mice, ovariectomized (OVX) female mice, and estrogen-treated male mice were tr

195 ore temperature (T(CORE)) of ovariectomized (OVX) control rats was significantly elevated, and this v

196 of Hdac4 in the amygdala of ovariectomized (OVX) female mice.

197 Treatment of ovariectomized (OVX) rats with estradiol benzoate (EB) caused rapid and

198 ucleus and MOR in the MPN of ovariectomized (OVX) rats.

199 istered to intact male rats, ovariectomized (OVX) females and OVX females treated with 17beta-estradi

200 In the ovariectomized (OVX) rodent model of menopause, increased adiposity is p

201 nondiabetic (+/?) mice were ovariectomized (OVX) and treated with estrogen or vehicle prior to H/I;

202 Two-month-old rats were ovariectomized (OVX) or had maxillary molars removed from one side to in

203 sted in adult mice that were ovariectomized (OVX) or OVX and treated with estradiol implants (OVX+E).

204 d middle-aged rats that were ovariectomized (OVX) or OVX and treated with estradiol.

205 Mice were ovariectomized (OVX), and a subset was treated with estradiol implants (

206 sk acquisition, animals were ovariectomized (OVX).

207 act, 2) old intact, 3) young ovariectomized (OVX), 4) old OVX, 5) young OVX plus estrogen replacement

208 signaling in rats 1 week after ovariectomy (OVX).

209 ice exhibited elevated LH after ovariectomy (OVX).

210 D via the tail vein ameliorates ovariectomy (OVX)-induced osteopenia by reducing T-helper 1 (Th1) and

211 using unilateral and bilateral ovariectomy (OVX) along with intact (SHAM) control.

212 fects of estrogen deficiency by ovariectomy (OVX) and 17beta-estradiol (E(2)) replacement (OVX+E(2))

213 d hormone deprivation caused by ovariectomy (OVX) in young adult rats prevents the ability of estroge

214 fter weaning, followed later by ovariectomy (OVX; as a model of menopause).

215 Rats then underwent either ovariectomy (OVX) or sham surgery and thereafter either continued to

216 s of ovarian hormones following ovariectomy (OVX) elevates the risks of cognitive impairment and deme

217 ovarian hormone loss following ovariectomy (OVX) is associated with cognitive impairment.

218 ffeine is influenced by gender, ovariectomy (OVX), and then exogenous estrogen in the mouse 1-methyl-

219 r surgically-induced menopause [ovariectomy (OVX)], on tests of memory and attention.

220 (HFD) when given at the time of ovariectomy (OVX) in mice.

221 Effects of ovariectomy (OVX) on performance of the memory tasks, Object Recognit

222 In the ovariectomy (OVX)-induced osteoporotic mouse model, serum and bone ma

223 we manipulated estrogen through ovariectomy (OVX) and estradiol administration.

224 y, the GNPs-ALD were applied to ovariectomy (OVX)-induced osteoporotic mice and the experiments were

225 uced periodontitis in rats with ovariectomy (OVX) that are or are not treated with estrogen replaceme

226 s of ovarian function (surgical ovariectomy; OVX).

227 VX) or E(2)-containing slow-release pellets (OVX + E(2)).

228 atment and from oophorectomized guinea pigs (OVX-GPs) treated with vehicle, estradiol (E2), medroxypr

229 mice that contain s.c. implants of placebo (OVX) or E(2)-containing slow-release pellets (OVX + E(2)

230 were spayed and either treated with placebo (OVX), estrogen alone (E), progesterone alone (P) or E+P.

231 ared to intact rats beginning at 1 week post OVX for OR and 4 weeks post OVX for OP.

232 g at 1 week post OVX for OR and 4 weeks post OVX for OP.

233 Golgi impregnation at 7 weeks post OVX showed significantly lower spine densities (17-53%)

234 e to the a.m.) in p.m. samples on d 2-5 post-OVX+E.

235 same chronological age as the 19-month post-OVX group, estrogen replacement significantly increased

236 However, by 19 months post-OVX, the same estrogen replacement was unable to induce

237 ity at CA3-CA1 synapses up to 15 months post-OVX.

238 Furthermore, the post-OVX rise in serum LH was significantly suppressed by est

239 ly in early life protected against 1 wk post-OVX-associated bone loss.

240 cortical bone mineral density loss 3 wk post-OVX.

241 ramming effects on bone formation to prevent OVX-induced bone loss in adult female rats.

242 ramming effects on bone formation to prevent OVX-induced bone loss in adult female rats.-Chen, J.-R.,

243 or E implant plus cyclic oral progesterone (OVX+EP).

244 geminal ganglia (TG) of ovariectomized rats (OVX).

245 lper 17 (Th17) cell numbers in the recipient OVX mice.

246 VX) and 17beta-estradiol (E(2)) replacement (OVX+E(2)) on glomerulosclerosis and tubulointerstitial f

247 OVX, 5) young OVX plus estrogen replacement (OVX+E2), and 6) old OVX+E2.

248 This SHED-mediated immunomodulation rescues OVX-induced impairment of bone marrow mesenchymal stem c

249 out estradiol (vulnerable, OVX+E; resistant, OVX+Veh) were given either short access (ShA) (three fix

250 SNCA-OVX mice display age-dependent loss of nigrostriatal dop

251 Aged SNCA-OVX mice exhibit reduced firing of SNc dopamine neurons

252 is longitudinal phenotyping strategy in SNCA-OVX mice thus provides insights into the region-specific

253 artificial chromosome transgenic mice (SNCA-OVX) that express wild-type alpha-synuclein from the com

254 RE) of OVX, KNDy-ablated rats was lower than OVX control rats at 33 degrees C, and not altered by E(2

255 These results suggest that OVX-induced bone loss, in part, is a result of increased

256 These results suggest that OVX-induced bone loss, in part, is a result of increased

257 , but not in Ghsr(-/-) mice, suggesting that OVX increases food intake by releasing ghrelin from a to

258 The OVX and caffeine/OVX groups presented a greater number o

259 The OVX animals were then assigned to treatment with either

260 The OVX+E animals also performed significantly better in the

261 The OVX+E animals performed significantly better on the dela

262 The OVX+EP animals also showed improved performance in the d

263 eine group presented the greatest BL and the OVX group the highest number of TRAP-positive (TRAP(+))

264 ts in vitro profile, 21 was evaluated in the OVX and ORX rat models and exhibited an osteoanabolic, t

265 Furthermore, in the OVX mouse model, the mice treated GNPs-ALD had higher bo

266 rized AVPV kisspeptin neurons, except in the OVX PM group in which GABA did not alter membrane potent

267 e regulation of IE by E(2) was absent in the OVX(LT) neurons.

268 ART-immunopositive area in comparison to the OVX control group with Student's t-test, but not with AN

269 In contrast, within the OVX groups, responding was enhanced from control levels

270 Estradiol administration to OVX mice also exerted a significant neuroprotective effe

271 uroprotectant after sham surgery compared to OVX or after OVX plus estrogen replacement compared to O

272 er OVX plus estrogen replacement compared to OVX plus placebo treatment.

273 tabolic regulation in obese animals prior to OVX plus an OVX-induced positive energy imbalance might

274 odents differ in their metabolic response to OVX-induced weight gain, and whether this difference aff

275 the area postrema was greater in EB-treated OVX rats compared to those in OIL-treated OVX and male r

276 ured at 22 h after MCAO in estradiol-treated OVX animals in the presence and absence of STAT3 inhibit

277 Similarly, the endometria of MPA treated OVX-GPs displayed decreased alphaSMA staining and fewer

278 ed OVX rats compared to those in OIL-treated OVX and male rats.

279 basis in ovariectomized, estradiol-treated (OVX+E) mice; GnRH neurons are suppressed in the morning

280 tablished ovariectomized, estradiol-treated (OVX+E) mouse model exhibiting daily surges to investigat

281 As predicted, unilateral OVX (intermediate reproductive investment) induced level

282 without affecting infarct size in untreated OVX rats.

283 nd following s.c. implantation in OVX versus OVX + E(2) mice, E(2) action on the host compartment lea

284 ales with and without estradiol (vulnerable, OVX+E; resistant, OVX+Veh) were given either short acces

285 pLTF was abolished with OVX, but was re-established by acute E2 replacement (3 h

286 in females, that weight gain associated with OVX is ghrelin mediated, and that this endocrine interac

287 fibrosis in the 12M group was augmented with OVX (GSI, 3.27 +/- 0.34; CTIFI, 74.4 +/- 9.2; P < 0.01 v

288 an-intact and OVX animals were compared with OVX animals receiving estradiol (E) alone or E with prog

289 g reflecting negative feedback compared with OVX cells, whereas in the p.m., OVX+E cells exhibited ch

290 fter tone-shock presentations, compared with OVX-homecage control females.

291 the treatment of periodontitis in rats with OVX that are or are not given estrogen replacement thera

292 mal rats; 2) rats with OVX; and 3) rats with OVX with estrogen replacement.

293 o three groups: 1) normal rats; 2) rats with OVX; and 3) rats with OVX with estrogen replacement.

294 treatment increased insulin sensitivity with OVX and HFD.

295 female FVB/NJ mice were ovariectomized with (OVX+E, n=6) or without (OVX, n=8) estrogen replacement.

296 ovariectomized with (OVX+E, n=6) or without (OVX, n=8) estrogen replacement.

297 g ovariectomized (OVX), 4) old OVX, 5) young OVX plus estrogen replacement (OVX+E2), and 6) old OVX+E

298 d by cyclic E administration, although young OVX+Veh monkeys still had a higher complement of small s

299 ative to vehicle control values (group young OVX+Veh) but nonetheless led to a robust increase in spi

300 of spines per neuron was equivalent in young OVX+Veh and OVX+E groups.