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1 ted transcriptomic and proteomic analysis of Onchocerca flexuosa to explore the molecular mechanisms
2 series describes the 6 human infections with Onchocerca lupi, a parasite known to infect cats and dog
3 Although of limited veterinary significance, Onchocerca ochengi has become famous as a natural model
4 linical infection models of Brugia malayi or Onchocerca ochengi that elevated exposures of orally-adm
5  cattle infected with the parasitic nematode Onchocerca ochengi, further supporting the presence of t
6        This review summarizes research using Onchocerca spp. in chimpanzees, cattle and mice to gain
7  as to our basic biological understanding of Onchocerca spp., over the past quarter-century.
8 ly based immunophenotyping approach to study Onchocerca volvulus (Ov) population diversity.
9 neuronal structures and a protein present in Onchocerca volvulus (OV).
10 es has been isolated from the human parasite Onchocerca volvulus (OvCYP-16).
11  we analyze patterns of human infection with Onchocerca volvulus (the cause of river blindness) in di
12 romally with Ags from the parasitic helminth Onchocerca volvulus (which causes river blindness).
13 ously reported that rOv-ASP-1, a recombinant Onchocerca volvulus activation associated protein-1, was
14 unized s.c. and injected intrastromally with Onchocerca volvulus Ags.
15  with paramyosin from the tropical parasites Onchocerca volvulus and Brugia malayi.
16                                              Onchocerca volvulus and lymphatic filariae, causing rive
17                                              Onchocerca volvulus antigen (OvAg)-stimulated peripheral
18 detects immunoglobulin G4 to the recombinant Onchocerca volvulus antigen Ov-16 was modified to detect
19  that detects IgG4 antibodies to recombinant Onchocerca volvulus antigen Ov16 with serum samples from
20                                          The Onchocerca volvulus antigen Ov39 is cross-reactive with
21 antigen for cross-reactive antibodies to the Onchocerca volvulus antigen Ov39.
22 rciasis in which Ags from the parasitic worm Onchocerca volvulus are injected into the corneal stroma
23 cle, I review the control and elimination of Onchocerca volvulus as a public health problem.
24 eatment should be guided by what is done for Onchocerca volvulus as there are no data for O. lupi.
25 fection with the parasitic filarial nematode Onchocerca volvulus can lead to severe visual impairment
26      The similar localization of the related Onchocerca volvulus cathepsin Z protein suggests that th
27                                 We evaluated Onchocerca volvulus community microfilarial intensity an
28 of a cysteine protease in the development of Onchocerca volvulus fourth stage larvae (L4) by testing
29 duce successfully the complete life cycle of Onchocerca volvulus has hindered progress towards unrave
30 tective immune response to the filarial worm Onchocerca volvulus in humans.
31           Protective immunity against larval Onchocerca volvulus in mice depends on the development o
32 e of the Wolbachia bacterial endosymbiont of Onchocerca volvulus in these reactions, serum samples co
33                                              Onchocerca volvulus infection has been associated with i
34 lthough the general perception has been that Onchocerca volvulus infection is well on its way towards
35 ant immunity and its potential mechanisms in Onchocerca volvulus infection were examined by analyzing
36 ter tetanus vaccination in 193 subjects with Onchocerca volvulus infection with 85 comparable noninfe
37  of adverse reactions after the treatment of Onchocerca volvulus infection, 40 O. volvulus-infected G
38 ensitive than current methods for diagnosing Onchocerca volvulus infection, and it overcomes many dif
39 tages that are critical for establishment of Onchocerca volvulus infection, the third-stage larvae (L
40 is) is a major cause of visual impairment in Onchocerca volvulus infection.
41                               Infection with Onchocerca volvulus is associated with the prevalence of
42 ro-Ecuadorian individuals from an area where Onchocerca volvulus is hyperendemic have been monitored
43                        The filarial nematode Onchocerca volvulus is the causative organism of river b
44 ies demonstrated that in the murine model of Onchocerca volvulus keratitis, neutrophils and eosinophi
45 microscopic examination for the detection of Onchocerca volvulus microfiladermia (2 of 218 samples po
46 ical disease caused by the filarial nematode Onchocerca volvulus that affects more than 37 million pe
47 ical disease caused by the filarial nematode Onchocerca volvulus that can lead to blindness and chron
48 The responses against antigens prepared from Onchocerca volvulus third-stage larvae (L3), molting L3
49               Mice immunized with irradiated Onchocerca volvulus third-stage larvae developed protect
50 se after early exposure to or infection with Onchocerca volvulus was investigated in an autochthonous
51 a bacteria that infect the filarial nematode Onchocerca volvulus were previously found to have an ess
52 non, we assessed the effect of targeting the Onchocerca volvulus Wolbachia endosymbionts with doxycyc
53 asitic helminth that causes river blindness (Onchocerca volvulus) induces eosinophil recruitment to t
54 yi, Wuchereria bancrofti) or onchocerciasis (Onchocerca volvulus) is doxycycline.
55 essed blisterase from the parasitic nematode Onchocerca volvulus, a major cause of blindness in Afric
56 filarial nematodes Acanthocheilonema viteae, Onchocerca volvulus, and Brugia malayi, strongly support
57 e (L3) larvae of the human filarial parasite Onchocerca volvulus, belongs to the family 18 glycosyl h
58 related cathepsin L in the filarial nematode Onchocerca volvulus, eggshell and cuticle, suggests that
59 itor (SPI) from the human parasitic nematode Onchocerca volvulus, Ov-SPI-1, was identified through th
60  infection with L. loa, Mansonella perstans, Onchocerca volvulus, Strongyloides stercoralis, or Wuche
61 orts have altered the global distribution of Onchocerca volvulus, the agent of river blindness, and f
62 T1 has been implicated in the development of Onchocerca volvulus, the causative agent of onchocercias
63 is a major antigen of the parasitic nematode Onchocerca volvulus, the causative agent of river blindn
64     This includes filarial pathogens such as Onchocerca volvulus, the cause of human onchocerciasis,
65                           Efforts to control Onchocerca volvulus, the etiologic agent of river blindn
66                Extracts of Brugia malayi and Onchocerca volvulus, which contain Wolbachia, directly s
67 nflammation induced by the filarial nematode Onchocerca volvulus, which harbors endosymbiotic Wolbach
68 itiates from the filarial parasitic nematode Onchocerca volvulus, which is transmitted by the blackfl
69                  With the genomes of L. loa, Onchocerca volvulus, Wuchereria bancrofti, and Brugia ma
70 ic filariasis being conducted in areas where Onchocerca volvulus, Wuchereria bancrofti, and L. loa ar
71 ast cell counts were examined in 2 groups of Onchocerca volvulus-infected subjects after ivermectin t
72                       Using a mouse model of Onchocerca volvulus-mediated keratitis (river blindness)
73  the effects of multiple ivermectin doses on Onchocerca volvulus.
74 lic responses to the human helminth pathogen Onchocerca volvulus.
75  stroma with Ags from the parasitic helminth Onchocerca volvulus.
76 ciasis (river blindness), which is caused by Onchocerca volvulus.
77 tigens extracted from the parasitic helminth Onchocerca volvulus.
78 ed filarial species Wuchereria bancrofti and Onchocerca volvulus.

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