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1 cialized intercellular structures (fungi and oomycetes).
2 t the fungal kingdom, and in the fungus-like oomycetes.
3 e are shared with plant-associated fungi and oomycetes.
4  from the same or closely related species of oomycetes.
5 , SWEETs had not been identified in fungi or oomycetes.
6 or future P450 annotations in newly explored oomycetes.
7  and 31 P450 subfamilies were newly found in oomycetes.
8 ee hundred and fifty-six P450s were found in oomycetes.
9 homology data revealed P450 family blooms in oomycetes.
10 ution of phytopathogenic traits in fungi and oomycetes.
11 gen-associated molecular patterns (PAMPs) in oomycetes.
12 oactivity against plant pathogenic fungi and oomycetes.
13  independently in plant pathogenic fungi and oomycetes.
14 , protects plants against diseases caused by oomycetes.
15 infection structures of pathogenic fungi and oomycetes.
16 production and bioactivity against fungi and oomycetes.
17 ted for RxLR-effectors from plant pathogenic oomycetes.
18 ttern of cross-kingdom HGT between fungi and oomycetes.
19 to pathogen Phytophthora infestans and other oomycetes.
20 s is not the case for several subfamilies in oomycetes.
21 ced by plant pathogenic bacteria, fungi, and oomycetes.
22 ved in the perception of bacteria, fungi and oomycetes.
23 e encoded by the genomes of plant pathogenic oomycetes.
24 e population structure within these obligate oomycetes.
25 or delivery are uncharacterized in fungi and oomycetes.
26 ies and catalogues the effector secretome of oomycetes.
27  in avirulence proteins from three different oomycetes.
28 t appear to be widespread and diverse in the oomycetes.
29 hogen of potato and a model organism for the oomycetes, a distinct lineage of fungus-like eukaryotes
30 diseases of plants and animals are caused by oomycetes, a group of eukaryotic pathogens important to
31                                              Oomycetes accomplish parasitic colonization of plants by
32 o mutualism) with their hosts include fungi, oomycetes and actinomycete bacteria.
33 her than the so-called Crinkler effectors of oomycetes and fungi, these effectors are encoded by othe
34 onal secreted proteins from plant pathogenic oomycetes and its similarity to a host-targeting signal
35  last 150 yr between plant pathogens (fungi, oomycetes and plasmodiophorids) and vascular plants.
36 d nematodes), common host-targeting signals (oomycetes and protozoans) and specialized intercellular
37 d economical impact of the animal pathogenic oomycetes and review the recent advances in this emergin
38 he understanding of the relationship between oomycetes and their host plants.
39  The resource is focused on fungi, protists (oomycetes) and bacterial plant pathogens that have genom
40 olite not previously shown to be produced by oomycetes, and two proteins with homology to vertebrate
41                                              Oomycetes are a phylogenetically distinct group of organ
42 sequences similar to those seen in fungi and oomycetes are also found in the animal kingdom, but rath
43                                              Oomycetes are fungal-like eukaryotic microbes in the kin
44 in bacteria, equivalent systems in fungi and oomycetes are poorly understood.
45                                              Oomycetes are responsible for multi-billion dollar damag
46  able to identify known sequences present in oomycetes as well as identify novel sequences.
47 e, we report the identification of SWEETs in oomycetes as well as SWEETs and a potential SemiSWEET in
48 toplasm, consistent with the hypothesis that oomycetes, as is the case with bacteria and fungi, activ
49 keleton confers resistance against fungi and oomycetes, AtADF4 is not involved in resistance against
50 ggests a molecular "arms race" as plants and oomycetes attempt to achieve and evade detection, respec
51 are consistent with the hypothesis that some oomycetes became successful plant parasites by multiple
52                       Research revealed that oomycetes belonging to different orders contain distinct
53 icals to control plant and animal pathogenic oomycetes cannot be used anymore; due to resistance in t
54          The three examined plant pathogenic oomycetes carry complex and diverse sets of RXLR effecto
55                                              Oomycetes cause devastating plant diseases of global imp
56                              Many species of oomycetes cause economic and environmental damage owing
57                              Phytopathogenic oomycetes cause some of the most devastating diseases af
58 T has played a role in shaping how fungi and oomycetes colonize plant hosts.
59                                          The oomycetes form a phylogenetically distinct group of euka
60                                          The oomycetes form one of several lineages within the eukary
61 ubset of these genes is conserved in related oomycetes from the Phytophthora genus.
62 yotic proteins, which include effectors from oomycetes, fungi and other parasites.
63 ve major pathogen groups (viruses, bacteria, oomycetes, fungi, and nematodes), has contributed to our
64 ety of eukaryotic plant pathogens, including oomycetes, fungi, and nematodes.
65        Although research on plant pathogenic oomycetes has flourished in recent years, the animal pat
66 ished in recent years, the animal pathogenic oomycetes have received less attention.
67 he presence or absence of the mycelia of the oomycetes in both shaking and static conditions.
68  only form of extracellular SOD in fungi and oomycetes, in stark contrast to the extracellular Cu/Zn-
69               The eukaryotic microbes called oomycetes include many important saprophytes and pathoge
70       The genus Aphanomyces (Saprolegniales, Oomycetes) includes species with a variety of ecologies
71                        When plant-pathogenic oomycetes infect their hosts, they employ a large arsena
72     Current research is helping us learn how oomycetes interact with host and environment, understand
73 tion of RxLR effectors from plant pathogenic oomycetes into the cytoplasm of their host is currently
74 nt that the effector secretome of pathogenic oomycetes is more complex than expected, with perhaps se
75  largest group of translocated proteins from oomycetes is the RxLR effectors, defined by their conser
76 TIR-NBS-LRR R genes specifying resistance to oomycetes, is dependent on a functional EDS1 allele for
77 te having limited secondary metabolism, many oomycetes make chemicals for communicating within their
78  unparalleled opportunities to determine how oomycetes manipulate hosts to establish infection.
79 ngdom as well as in phylogenetically distant oomycetes or "pseudofungi" species.
80                               The eukaryotic oomycetes, or water molds, contain several species that
81 on between Phytophthora pathogens, which are oomycetes, phylogenetically distinct from fungi, has bee
82                 One type of plant pathogens, oomycetes, produces effector proteins with N-terminal RX
83 rasites, including viruses, bacteria, fungi, oomycetes, protozoa, insects and nematodes.
84                                          Two oomycetes, Pythium oligandrum and Pythium aphanidermatum
85                                              Oomycetes secrete both extracellular and intracellular e
86                                    Fungi and oomycetes secrete many specialized effector proteins for
87 rium catenoides, a free-living sister of the oomycetes, shows that these transfers largely converge w
88                                              Oomycetes such as these Phytophthora species share the k
89                             Plant pathogenic oomycetes, such as the Irish potato famine pathogen Phyt
90                             Plant pathogenic oomycetes, such as the potato (Solanum tuberosum) and to
91 guish isolates within several species of the oomycetes that cause downy mildew diseases.
92                                    Fungi and oomycetes that colonize living plant tissue form extensi
93 ers largely converge within the radiation of oomycetes that colonize plant tissues.
94 ng function has been recruited by pathogenic oomycetes to facilitate their own invasion.
95 ed by eukaryotic microbes, such as fungi and oomycetes, to host plants and contribute to the establis
96                            During infection, oomycetes translocate effector proteins into host cells,
97 ning and biochemical studies have shown that oomycetes, which belong to the kingdom Stramenopila, sec
98                                       Unlike oomycetes, which employ RXLR effectors to suppress host
99 quences and other sequenced plant pathogenic oomycetes with 91% of the hybrid assembly derived sequen
100 ering effectors, have emerged from comparing oomycetes with different genome characteristics, parasit
101  plant defenses against pathogenic fungi and oomycetes with limited, indirect evidence.
102 lacking chromalveolates such as ciliates and oomycetes would be explained by plastid loss in these li

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