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1 cialized intercellular structures (fungi and oomycetes).
2 t the fungal kingdom, and in the fungus-like oomycetes.
3 e are shared with plant-associated fungi and oomycetes.
4 from the same or closely related species of oomycetes.
5 , SWEETs had not been identified in fungi or oomycetes.
6 or future P450 annotations in newly explored oomycetes.
7 and 31 P450 subfamilies were newly found in oomycetes.
8 ee hundred and fifty-six P450s were found in oomycetes.
9 homology data revealed P450 family blooms in oomycetes.
10 ution of phytopathogenic traits in fungi and oomycetes.
11 gen-associated molecular patterns (PAMPs) in oomycetes.
12 oactivity against plant pathogenic fungi and oomycetes.
13 independently in plant pathogenic fungi and oomycetes.
14 , protects plants against diseases caused by oomycetes.
15 infection structures of pathogenic fungi and oomycetes.
16 production and bioactivity against fungi and oomycetes.
17 ted for RxLR-effectors from plant pathogenic oomycetes.
18 ttern of cross-kingdom HGT between fungi and oomycetes.
19 to pathogen Phytophthora infestans and other oomycetes.
20 s is not the case for several subfamilies in oomycetes.
21 ced by plant pathogenic bacteria, fungi, and oomycetes.
22 ved in the perception of bacteria, fungi and oomycetes.
23 e encoded by the genomes of plant pathogenic oomycetes.
24 e population structure within these obligate oomycetes.
25 or delivery are uncharacterized in fungi and oomycetes.
26 ies and catalogues the effector secretome of oomycetes.
27 in avirulence proteins from three different oomycetes.
28 t appear to be widespread and diverse in the oomycetes.
29 hogen of potato and a model organism for the oomycetes, a distinct lineage of fungus-like eukaryotes
30 diseases of plants and animals are caused by oomycetes, a group of eukaryotic pathogens important to
33 her than the so-called Crinkler effectors of oomycetes and fungi, these effectors are encoded by othe
34 onal secreted proteins from plant pathogenic oomycetes and its similarity to a host-targeting signal
36 d nematodes), common host-targeting signals (oomycetes and protozoans) and specialized intercellular
37 d economical impact of the animal pathogenic oomycetes and review the recent advances in this emergin
39 The resource is focused on fungi, protists (oomycetes) and bacterial plant pathogens that have genom
40 olite not previously shown to be produced by oomycetes, and two proteins with homology to vertebrate
42 sequences similar to those seen in fungi and oomycetes are also found in the animal kingdom, but rath
47 e, we report the identification of SWEETs in oomycetes as well as SWEETs and a potential SemiSWEET in
48 toplasm, consistent with the hypothesis that oomycetes, as is the case with bacteria and fungi, activ
49 keleton confers resistance against fungi and oomycetes, AtADF4 is not involved in resistance against
50 ggests a molecular "arms race" as plants and oomycetes attempt to achieve and evade detection, respec
51 are consistent with the hypothesis that some oomycetes became successful plant parasites by multiple
53 icals to control plant and animal pathogenic oomycetes cannot be used anymore; due to resistance in t
63 ve major pathogen groups (viruses, bacteria, oomycetes, fungi, and nematodes), has contributed to our
68 only form of extracellular SOD in fungi and oomycetes, in stark contrast to the extracellular Cu/Zn-
72 Current research is helping us learn how oomycetes interact with host and environment, understand
73 tion of RxLR effectors from plant pathogenic oomycetes into the cytoplasm of their host is currently
74 nt that the effector secretome of pathogenic oomycetes is more complex than expected, with perhaps se
75 largest group of translocated proteins from oomycetes is the RxLR effectors, defined by their conser
76 TIR-NBS-LRR R genes specifying resistance to oomycetes, is dependent on a functional EDS1 allele for
77 te having limited secondary metabolism, many oomycetes make chemicals for communicating within their
81 on between Phytophthora pathogens, which are oomycetes, phylogenetically distinct from fungi, has bee
87 rium catenoides, a free-living sister of the oomycetes, shows that these transfers largely converge w
95 ed by eukaryotic microbes, such as fungi and oomycetes, to host plants and contribute to the establis
97 ning and biochemical studies have shown that oomycetes, which belong to the kingdom Stramenopila, sec
99 quences and other sequenced plant pathogenic oomycetes with 91% of the hybrid assembly derived sequen
100 ering effectors, have emerged from comparing oomycetes with different genome characteristics, parasit
102 lacking chromalveolates such as ciliates and oomycetes would be explained by plastid loss in these li
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