ライフサイエンスコーパス: P. intermedia

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1 P. intermedia alone could induce a similar neutrophil ph

2 P. intermedia-induced proliferation was T-cell specific

3 Antibodies raised against P. intermedia type C fimbriae and against whole cells in

4 P. gingivalis, A. actinomycetemcomitans, and P. intermedia, was associated with pronounced volume los

5 importance, P. gingivalis, T. denticola, and P. intermedia were the microorganisms significantly asso

6 , and with coinfections of P. gingivalis and P. intermedia (OR 4.03); P. gingivalis and B. forsythus

7 indings indicate that both P. gingivalis and P. intermedia express heme-repressible proteinaceous hem

8 A. actinomycetemcomitans, P. gingivalis, and P. intermedia in subgingival plaque, at levels above the

9 A. actinomycetemcomitans, P. gingivalis, and P. intermedia.

10 intermedia 27, another clinical isolate, and P. intermedia 25611, the type strain, were not found to

11 domonas maltophilia, Strept, pneumoniae, and P. intermedia were the predominant organisms detected an

12 d as much hemin in the absence of RSA as did P. intermedia.

13 y and greater heme storage capacity than did P. intermedia, suggesting that the former would be ecolo

14 nomycetemcomitans, and 14% were positive for P. intermedia.

15 ections with P. gingivalis, T. forsythensis, P. intermedia, and C. rectus were associated with an inc

16 ycetemcomitans, P. gingivalis, T. forsythia, P. intermedia, and total bacteria significantly decrease

17 elaninogenicus, F. nucleatum, P. gingivalis, P. intermedia, S. mutans, S. sanguis, Selenomonas sputig

18 P. gingivalis, P. intermedia, T. forsythia, and T. denticola were more

19 n teeth for Parvimonas micra, P. gingivalis, P. intermedia, T. forsythia, and Treponema denticola.

20 tion of eukaryotic cells with AdpF increased P. intermedia 17 internalization by 5- and 10-fold for H

21 la, A. actinomycetemcomitans, P. nigrescens, P. intermedia, B. forsythus, and P. gingivalis in 38% to

22 any orange-complex pathogens (F. nucleatum, P. intermedia, and C. rectus), total cancer risk (HR = 1

23 The ability of P. intermedia to stimulate CD4(+)-T-cell proliferation w

24 rAdpF plays a role in the internalization of P. intermedia 17 by a variety of host cells.

25 4 degreesC) inhibited the internalization of P. intermedia 17.

26 orse periodontal status and higher levels of P. intermedia, E. corrodens, F. nucleatum, and IL-1beta

27 ens MH1 and 2) the cell-free cell lysates of P. intermedia MH2.

28 ed from the index teeth, and the presence of P. intermedia, P. gingivalis, A. actinomycetemcomitans,

29 that AdpC is an important invasin protein of P. intermedia 17.

30 rovides evidence that at least one strain of P. intermedia can invade an oral epithelial cell line an

31 collectively suggest that certain strains of P. intermedia can activate Vbeta-specific T cells in a m

32 Streptococcus oralis, P. intermedia, and Selenomonas noxia were elevated in sa

33 that the endodontic pathogens, particularly P. intermedia, can efficiently disable and kill infiltra

34 tion of A. actinomycetemcomitans, C. rectus, P. intermedia/nigrescens, E. corrodens, P. micros, Capno

35 control E. coli BL21 cells expressing a sham P. intermedia 17 protein.

36 Under these conditions, heme-starved P. intermedia cells (two strains) expressed a single bin

37 Among the bacterial strains tested, P. intermedia strain 17, a clinical isolate, induced the

38 Finally, cells exposed to P. intermedia 17 internalized the bacteria more readily

39 Vbeta12, and Vbeta17 expanded in response to P. intermedia strain 17.

40 uman oral epithelial cell line (KB), whereas P. intermedia 27, another clinical isolate, and P. inter

41 te a new virulence mechanism associated with P. intermedia.

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