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1                                              P. syringae 508 was also surveyed for the presence of ef
2                                              P. syringae carrying hopW1-1 have restricted host range
3                                              P. syringae effector proteins encounter a pH gradient as
4                                              P. syringae pv. tomato DC3000 HrpP has a C-terminal, put
5                                              P. syringae was able to attach and extensively colonize
6                                              P. syringae was defective in porous-paper colonization w
7                                            A P. syringae hopD1 mutant and ETI-inducing P. syringae st
8                                            A P. syringae pv tomato DC3000 mutant lacking about one-th
9                                   In 1991, a P. syringae pathovar tomato (Pst) strain, DC3000, was re
10                               In addition, a P. syringae strain defective in coronatine synthesis sho
11 abidopsis eukaryotic activator of AvrRpt2, a P. syringae effector that is a cysteine protease.
12 d P. fluorescens heterologously expressing a P. syringae TTSS and AvrPto1(PtoJL1065).
13 ome of Por(1_6) is the first sequenced for a P. syringae isolate that is a pathogen of monocots, and,
14 ctive oxygen burst, and enhances growth of a P. syringae hrpA mutant that fails to secrete effectors.
15 oB-mediated anti-PCD activity, and abrogates P. syringae pathogenesis of susceptible tomato plants.
16  amount of NAD(P) leaking into the ECC after P. syringae pv. tobacco DC3000/avrRpt2 infection is suff
17 howed significantly reduced expression after P. syringae interference.
18 nce gene-mediated inducible defenses against P. syringae (and possibly other pathogens) by playing a
19 ent that orchestrates plant defenses against P. syringae.
20 t not Pto possessed greater immunity against P. syringae than tomatoes lacking Prf.
21 stemic resistance response in plants against P. syringae pv tomato DC3000.
22 oteins and contributed to resistance against P. syringae pv tomato.
23 ce of PRR2 in plant immune responses against P. syringae and suggest a novel function for this partic
24 NLR induces strong defense responses against P. syringae and X. campestris The P. syringae T3SE HopZ1
25     Plants in turn defend themselves against P. syringae by activating the salicylic acid (SA)-mediat
26 a constitutive GFP marker to account for all P. syringae cells on a leaf.
27 that RhpRS is a global regulator that allows P. syringae to sense and respond to environmental change
28 s, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A restored HR elicitati
29 a gene in P. syringae pv. syringae B728a and P. syringae pv. tomato DC3000, termed phcA, that has hom
30 inding protein (MBP) in Escherichia coli and P. syringae pv. syringae.
31 esions, challenge infections with DC3000 and P. syringae pv. tabaci 11528, respectively.
32 2 is transcriptionally upregulated by SA and P. syringae, enhances SA biosynthesis and SA signalling
33 -host pathogens P. syringae pv. syringae and P. syringae pv. tabaci.
34      The interaction between A. thaliana and P. syringae DC3000 highly induced the secretion of sever
35 athogens Pseudomonas syringae pv. tomato and P. syringae pv. maculicola.
36 athogens Pseudomonas syringae pv. tomato and P. syringae pv. maculicola.
37 onella enterica, it was effective in another P. syringae strain and Ralstonia solanacearum.
38 ore Pseudomonas genome and 365 ORFs that are P. syringae specific.
39 s a selective advantage for microbes such as P. syringae that are adapted to maximally exploit cholin
40        The HopS2 effector possessed atypical P. syringae TTSS N-terminal characteristics and was tran
41  of cytosolic calcium triggered by avirulent P. syringae was compromised in crt1-2 crh1-1 plants, but
42 were compromised for resistance to avirulent P. syringae and more susceptible to virulent strains tha
43 s have increased susceptibility to avirulent P. syringae strains, are unable to activate systemic acq
44 t DC3000) and avirulent (Pst DC3000 AvrRPM1) P. syringae strains, conserving typical hypersensitive r
45 ulture and injected into plant cells by both P. syringae and Q8r1-96 T3SSs.
46 response (HR) to effector proteins from both P. syringae and the fungal pathogen, Cladosporium fulvum
47  WRKY60 made plants more susceptible to both P. syringae and B. cinerea.
48  AvrPtoB did not prevent the HR activated by P. syringae pv. tomato DC3000 + avrB, avrRpm1, avrRps4 o
49 f exogenous auxin enhances disease caused by P. syringae strain DC3000.
50 ssion at specific promoter configurations by P. syringae.
51       Expression of HDA19 is also induced by P. syringae, and the stability of its induced transcript
52  resistance signaling following infection by P. syringae expressing the Cys protease Type III effecto
53 he main characterized surfactant produced by P. syringae.
54              The protein HrpJ is secreted by P. syringae and is required for a fully functional T3SS.
55  A tyrosine phosphatase, HopAO1, secreted by P. syringae, reduces EFR phosphorylation and prevents su
56 s being the only siderophores synthesized by P. syringae pv. syringae B728a.
57 arched for homologs to 44 known or candidate P. syringae type III effectors and two effector chaperon
58                This system includes a cloned P. syringae hrp gene cluster and the model plant Nicotia
59               Here, we show that a conserved P. syringae virulence protein, HopM1, targets an immunit
60                         COR and the critical P. syringae type III effector HopM1 target distinct sign
61 gher in leaves challenged with COR-deficient P. syringae or in the more resistant JA receptor mutant
62 eins restores virulence of a HopM1-deficient P. syringae mutant, providing a link between HopM1 and t
63 y divergent protein in the T3SS of different P. syringae pathovars, hrpP from P. syringae pv. syringa
64 r this tail; this function was confirmed for P. syringae BetT using deletion derivatives.
65 t high temperature, HopI1 is dispensable for P. syringae pathogenicity, unless excess Hsp70 is provid
66 3000 effectors and a central requirement for P. syringae pathogenesis.
67                         The primary role for P. syringae type-III effectors is the suppression of pla
68 gesting an epidemic population structure for P. syringae.
69 of effectors and related T3SS substrates for P. syringae pv. tomato DC3000 and three other sequenced
70 ins from P. syringae pv. tomato, and 19 from P. syringae pv. phaseolicola race 6.
71 contributes to virulence after delivery from P. syringae in leaves of susceptible soybean plants, and
72          We previously identified hopA1 from P. syringae pv syringae strain 61 as an avirulence gene
73 f different P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseoli
74 0 PFPs along with pDC3000A and pDC3000B from P. syringae pv. tomato encoded a type IVB T4SS (tra syst
75 from P. syringae pv. syringae, pPh1448B from P. syringae pv. phaseolicola, and pPMA4326A from P. syri
76 yringae pv. phaseolicola, and pPMA4326A from P. syringae pv. maculicola encoded a type IVA T4SS (VirB
77            Twelve PFPs along with pPSR1 from P. syringae pv. syringae, pPh1448B from P. syringae pv.
78 l criteria defined 29 type III proteins from P. syringae pv. tomato, and 19 from P. syringae pv. phas
79 omparative genomic analyses to elucidate how P. syringae subverts the attack and defense responses of
80  a detailed mechanistic understanding of how P. syringae transitions from reliance on exogenously der
81 nome, high-throughput screen for identifying P. syringae type III effector genes.
82 rnitine/choline family transporter (BCCT) in P. syringae pv. tomato strain DC3000 that mediates the t
83 n of P. syringae pv. glycinea PG4180 CmaA in P. syringae pv. syringae FF5 as a FLAG-tagged protein an
84  homologues occur at very low frequencies in P. syringae populations on A. thaliana.
85               Here we characterize a gene in P. syringae pv. syringae B728a and P. syringae pv. tomat
86 zed the hopO1-1, hopS1, and hopS2 operons in P. syringae pv. tomato DC3000; these operons encode thre
87               * hopAS1 is broadly present in P. syringae strains, contributes to virulence in tomato,
88     Three alleles are known to be present in P. syringae, with HopZ1a most resembling the ancestral a
89 d stringent response plays a central role in P. syringae virulence and survival and indicated that pp
90 e data indicate that HrpJ has a dual role in P. syringae: inside bacterial cells HrpJ controls the se
91 enes coregulated with the Hrp T3SS system in P. syringae pv. tomato DC3000 have predicted lytic trans
92 loring multiple aspects of the Hrp system in P. syringae.
93       To identify additional RhpR targets in P. syringae, we performed chromatin immunoprecipitation
94 e global role of rppH in thermoregulation in P. syringae, RNA sequencing was used to compare the tran
95 1 supported increased growth of ETI-inducing P. syringae strains compared with wild-type Arabidopsis.
96  A P. syringae hopD1 mutant and ETI-inducing P. syringae strains exhibited enhanced growth on Arabido
97                    In contrast, ETI-inducing P. syringae strains were unable to overcome PTI-induced
98                  Much existing research into P. syringae-plant interactions has focused on the molecu
99 -LRR genes RPS2, RPM1, and RPS5 and isogenic P. syringae strains expressing single corresponding avir
100                 In the nonpathogenic isolate P. syringae 508 the genomic region that in pathogenic P.
101 a variety of additional genes encoding known P. syringae virulence factors.
102 r TNL class is represented by a single known P. syringae resistance gene, RPS4.
103                    phcA is conserved in many P. syringae strains, but is absent in one of the major c
104                         We propose that many P. syringae type III effectors have more than one target
105                                    Moreover, P. syringae mutants that were deficient in the uptake of
106 cks early MAMP signaling and enables nonhost P. syringae growth.
107                                  Analyses of P. syringae pv. tomato DC3000 mutants indicated that bot
108 at is supported by the common association of P. syringae with plants and the widespread production of
109                               In the case of P. syringae, growth on a nata1 mutant is reduced compare
110     The hopPtoM and avrE genes in the CEL of P. syringae were found to encode suppressors of this SA-
111  and is carried in the functional cluster of P. syringae pv. syringae 61 hrp genes cloned in cosmid p
112 open reading frames (ORFs) within the EEL of P. syringae pv. tomato DC3000.
113 :acyl carrier protein transacylase (FabD) of P. syringae was overproduced and shown to catalyze malon
114 vrRpt2 may be among the virulence factors of P. syringae that modulate host auxin physiology to promo
115 tion was visualized via dual fluorescence of P. syringae cells harboring a transcriptional fusion of
116 erexpressing plants supported more growth of P. syringae and developed more severe disease symptoms t
117 us, flagellin perception restricts growth of P. syringae strains on N. benthamiana.
118                        Conversely, growth of P. syringae strains was reduced in plants expressing a c
119 g serine(s) in two other effectors, HopZ3 of P. syringae and PopP2 of Ralstonia solanacerum, also abo
120 fector as reporter revealed the inability of P. syringae 508 to translocate effectors into plant cell
121              By analyzing the interaction of P. syringae mutants with Arabidopsis thaliana mutants, w
122 tor-triggered immunity in the interaction of P. syringae pv tomato DC3000 and N. benthamiana.
123 lenge inoculation with a virulent isolate of P. syringae.
124                         A shotgun library of P. syringae was screened in the mutant E. coli by growin
125                          The localization of P. syringae bioreporter cells to the surface and interce
126          The enzymatic activities of most of P. syringae effectors and their targets remain obscure.
127 resulted in the loss of swarming motility of P. syringae pv. tomato DC3000 on medium containing a low
128 onas fluorescens, a TTSS-deficient mutant of P. syringae pv. tabaci, or flg22 (a flagellin-derived pe
129      These efforts allowed overproduction of P. syringae pv. glycinea PG4180 CmaA in P. syringae pv.
130  a FLAG-tagged protein and overproduction of P. syringae pv. tomato CmaA in Escherichia coli as a His
131                             The pangenome of P. syringae encodes 57 families of effectors injected by
132 ed and eight known PFPs from 12 pathovars of P. syringae, which belong to four genomospecies.
133  III effector proteins from two pathovars of P. syringae.
134                     However, the presence of P. syringae carrying avrPphB is probably insufficient to
135 ce factor, coronatine, during progression of P. syringae infection of Arabidopsis thaliana.
136  role in Arabidopsis NHR to a broad-range of P. syringae strains.
137 esistance signaling following recognition of P. syringae DC3000-AvrRpt2 by Arabidopsis.
138 of the effectors comprising the secretome of P. syringae pv tomato DC3000 led to the identification o
139 s study, we report on the genome sequence of P. syringae pv. phaseolicola isolate 1448A, which encode
140 as coinoculated with the avirulent strain of P. syringae pv phaseolicola into tobacco leaves.
141 roteins were delivered by the RW60 strain of P. syringae pv. phaseolicola.
142  were more resistant to a virulent strain of P. syringae pv. tabaci and showed an accelerated hyperse
143 by PstAvr, infection by a virulent strain of P. syringae, and low temperature.
144 ced disease severity to a virulent strain of P. syringae, suggesting a role of ATT1 in disease resist
145   In basal resistance to virulent strains of P. syringae and H. arabidopsidis, PAD4 functions togethe
146 nces basal resistance to virulent strains of P. syringae and the oomycete Phytophthora sojae.
147 al phenotypic differences between strains of P. syringae is the range of plant hosts they infect.
148 -depleted plants to nonpathogenic strains of P. syringae supports a defense-promoting role for Hsp70.
149 ible, non-host and non-pathogenic strains of P. syringae.
150 ere we report the existence of a subgroup of P. syringae isolates that do not cause disease on any pl
151 been determined and is compared with that of P. syringae pv. tomato DC3000 (Pst DC3000).
152 n the temperature-dependent transcriptome of P. syringae, affecting the expression of 569 genes at ei
153 ter system for Hrp-mediated translocation of P. syringae TTSS effectors into plant cells.
154 in of the Xcv AvrBs2 protein via the TTSS of P. syringae.
155 side plant cells to promote the virulence of P. syringae pv. tomato strain DC3000 (PstDC3000) on Arab
156 t (p)ppGpp is required for full virulence of P. syringae.
157            GABA may have multiple effects on P. syringae-plant interactions, with elevated levels inc
158 culation with P. syringae DC3000(avrRpm1) or P. syringae DC3000(avrRpt2) induces differential decreas
159  by RLs following challenge by B. cinerea or P. syringae pv tomato.
160 iotic and biotic stresses such as drought or P. syringae infection induced similar increase.
161  defenses against Manduca sexta herbivory or P. syringae pv tomato DC3000 infection rates.
162 influence resistance against virulent Pst or P. syringae pv. maculicola (Psm) pathogens.
163  produced by immunization with Shewanella or P. syringae cells bound to B. anthracis spores but not t
164  common N-terminal characteristic from other P. syringae type III secreted substrates increased HopS2
165 between BAK1 and HopF2 and between two other P. syringae effectors, AvrPto and AvrPtoB, was confirmed
166 ospora parasitica and the bacterial pathogen P. syringae pv. tomato.
167 or repertoire of the sequenced bean pathogen P. syringae pv. syringae (Psy) B728a using bioinformatic
168 lso enhanced the growth of the host pathogen P. syringae pv tabaci by increasing nutrient efflux into
169 enes in the repertoire of the model pathogen P. syringae pv. tomato DC3000 were deleted to produce po
170 rains closely related to the tomato pathogen P. syringae pv. tomato (Pto), including strains isolated
171 ic and most likely evolved from a pathogenic P. syringae ancestor through loss of the T3SS.
172 Pseudomonas syringae sustains but pathogenic P. syringae suppresses early MAMP (microbe-associated mo
173 etions of avrPto and avrPtoB from pathogenic P. syringae reduce its virulence.
174 ae 508 the genomic region that in pathogenic P. syringae strains contains the hrp-hrc cluster coding
175 d are sufficient to transform non-pathogenic P. syringae strains into virulent pathogens in immunodef
176 ot affect plant susceptibility to pathogenic P. syringae bacteria; conversely, expression of the cons
177 ere highly susceptible to non-host pathogens P. syringae pv. syringae and P. syringae pv. tabaci.
178  with a phylogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a strong degree
179 toB acts as a virulence protein by promoting P. syringae pv. tomato growth and enhancing symptoms ass
180 es to disease resistance in response to Pto (P. syringae pathovars tomato) DC3000(avrB), but not agai
181                                This purified P. syringae protein was determined to catalyze the epoxi
182  (localized to chloroplasts) greatly reduces P. syringae-induced PCD, suggesting a pro-PCD role for m
183      Repression of auxin signaling restricts P. syringae growth, implicating auxin in disease suscept
184  induce systemic susceptibility to secondary P. syringae infection in the host plant Arabidopsis thal
185         cmaL is found in all other sequenced P. syringae strains with coronatine biosynthesis genes.
186  type III effector suites from two sequenced P. syringae pathovars and show that type III effector pr
187                                      Several P. syringae effectors require accessory proteins called
188 cens was used to test the ability of several P. syringae pv. tomato DC3000 effectors for their abilit
189 ype plants; however, responses to A. solani, P. syringae, or M. sexta were similar to the wild-type p
190                           Intriguingly, some P. syringae strains also secrete the virulence factor sy
191                                   While some P. syringae type III effectors were acquired recently, o
192 s from the closely related pathogenic strain P. syringae pv. syringae B728a, but none were detected.
193              Luminescence of luxCDABE-tagged P. syringae allows rapid and convenient quantification o
194                       Strikingly, all tested P. syringae strains that are pathogenic in Arabidopsis c
195                             We conclude that P. syringae strains may have evolved large effector repe
196                 In this study, we found that P. syringae pv. tomato strain DC3000 was distinct from m
197 ical evidence supporting the hypothesis that P. syringae pv. syringae B728a produces both of these si
198                                 We show that P. syringae-elicited SIS is caused by the production of
199                   These results suggest that P. syringae has evolved to survive in relatively choline
200                     Our results suggest that P. syringae has the potential to utilize phcA to acquire
201                   These results suggest that P. syringae type III effectors and coronatine act by aug
202                           This suggests that P. syringae 508 supplemented with a T3SS could be used t
203                                          The P. syringae effector AvrRpt2, which targets RPM1 INTERAC
204                                          The P. syringae pv. tomato DC3000 effector HopF2 suppresses
205                                          The P. syringae pv. tomato DC3000 HopK1 type-III effector wa
206                                          The P. syringae pv. tomato OpuC transporter had a high affin
207                                          The P. syringae pv. tomato OpuC transporter was more closely
208                                          The P. syringae TTSS is encoded by hrp-hrc genes that reside
209                                          The P. syringae TTSS is encoded by the hrp-hrc gene cluster.
210                                          The P. syringae-specific HopI1 effector has a putative chlor
211        Adding a plasmid-encoded T3SS and the P. syringae pv. syringae 61 effector gene hopA1 increase
212 n somewhat differently than YscP because the P. syringae T3SS pilus likely varies in length due to di
213  ahlR regulon presence within and beyond the P. syringae pan-genome.
214 st that chloroplast Hsp70 is targeted by the P. syringae HopI1 effector to promote bacterial virulenc
215 use it is poorly secreted in cultures by the P. syringae Hrp system, was translocated into plant cell
216 ure and translocated into plant cells by the P. syringae pv. tomato DC3000 TTSS.
217  order to be effectively translocated by the P. syringae T3SS.
218 es against P. syringae and X. campestris The P. syringae T3SE HopZ1a is an acetyltransferase that ace
219  three protein classes cooperate to form the P. syringae T3SS translocon.
220 rotein (PSPTO_2145), which is located in the P. syringae pyoverdine cluster.
221  one of the major clades, which includes the P. syringae pathovar phaseolicola.
222 peron from Photorhabdus luminescens into the P. syringae chromosome under the control of a constituti
223 I effector genes, which are orthologs of the P. syringae effector genes hopAA1-1 and hopM1, as well a
224 e complex is activated by recognition of the P. syringae effectors AvrPto and AvrPtoB.
225                   Informatic analysis of the P. syringae genome suggests only one putative non-heme i
226 for locations associated with binding of the P. syringae IS sigma factor PSPTO_1203.
227 Por(1_6) helps to define an expansion of the P. syringae pan-genome, a corresponding contraction of t
228                      The availability of the P. syringae pv. tomato DC3000 genome sequence has result
229     Here, the functional significance of the P. syringae T3SS substrate compositional patterns was te
230 two distinct levels in the regulation of the P. syringae TTSS: regulation of assembly of the secreton
231 anslationally modified after delivery of the P. syringae type III effectors AvrRpm1, AvrB, or AvrRpt2
232  structural and regulatory components of the P. syringae type III secretion system (T3SS), essential
233                     We further show that the P. syringae is able to use N. crassa as a sole nutrient
234        We also show that the ability of this P. syringae strain to block antimicrobial exudation is d
235 nduced resistance to H. arabidopsidis and to P. syringae pv tomato whereas jasmonic acid is essential
236 ell as to the fungus Botrytis cinerea and to P. syringae.
237 B only rarely confers a virulence benefit to P. syringae on A. thaliana.
238 g the importance of extracellular choline to P. syringae on leaves.
239 for the superior osmoprotection conferred to P. syringae by choline over glycine betaine when these c
240                         HopD1 contributes to P. syringae virulence in Arabidopsis and reduces effecto
241                       * HopD1 contributes to P. syringae virulence in Arabidopsis and reduces effecto
242                         HopD1 contributes to P. syringae virulence in part by targeting NTL9, resulti
243                       * HopD1 contributes to P. syringae virulence in part by targeting NTL9, resulti
244 uced accumulation of Pip in leaves distal to P. syringae inoculation, they display a considerable sys
245 tibility of WRKY33-over-expressing plants to P. syringae is associated with reduced expression of the
246 re more susceptible than wild-type plants to P. syringae.
247 gae pv maculicola 1 (RPM1) and Resistance to P. syringae 2 (RPS2) disease resistance proteins.
248 RIN4b abrogates RPG1-B-derived resistance to P. syringae expressing AvrB.
249 nea) mediates species-specific resistance to P. syringae expressing the avirulence protein AvrB, simi
250  Arabidopsis increased disease resistance to P. syringae Expression of CRK28 in Nicotiana benthamiana
251 ced plant defenses, conferring resistance to P. syringae infection.
252 ng male sterility and enhanced resistance to P. syringae infection.
253 ana benthamiana did not confer resistance to P. syringae pv. tabaci (Pta) expressing avrPto or avrPto
254 vely, of the variance of basal resistance to P. syringae pv. tomato DC3000 in the Col-0 x Fl-1 F(2) p
255  expression of WRKY18 enhanced resistance to P. syringae, its coexpression with WRKY40 or WRKY60 made
256 upted also compromised disease resistance to P. syringae.
257 expression results in enhanced resistance to P. syringae.
258 ural variation that conditions resistance to P. syringae/hopW1-1.
259 s overexpressing IOS1 were more resistant to P. syringae and demonstrated a primed PTI response.
260  mutant were substantially more resistant to P. syringae but more susceptible to B. cinerea than wild
261  reduced in the pbs3-1 mutant in response to P. syringae (avrRpt2) infection, free SA was elevated.
262 nerate a full disease resistance response to P. syringae expressing this type III effector.
263 es in Arabidopsis tissues and in response to P. syringae infection.
264 SA signalling responses; e.g. in response to P. syringae, PRR2 induces the production of SA and the a
265 sis of quantitative variation in response to P. syringae.
266 f three aspects of A. thaliana's response to P. syringae: symptom severity, bacterial population size
267 ween defense pathways mediating responses to P. syringae and necrotrophic pathogens.
268 we analyzed Arabidopsis defense responses to P. syringae through differential coexpression analysis.
269 play a negative role in defense responses to P. syringae.
270 at AvrRpt2 virulence activity is specific to P. syringae.
271 ins result in an increased susceptibility to P. syringae, whereas overexpression of these genes alter
272 mmune deficient and were more susceptible to P. syringae.
273 of Bti9 and SlLyk13 were more susceptible to P. syringae.
274  similarity to Escherichia coli BetT than to P. syringae BetT.
275 s of differences in the osmotolerance of two P. syringae strains, B728a and DC3000.
276 effectors and for host susceptibility to two P. syringae pathogens.
277  staining effect was suppressed by wild-type P. syringae pv. tabaci and P. fluorescens heterologously
278                        HopI1 is a ubiquitous P. syringae virulence effector that acts inside plant ce
279  cell death in susceptible leaves undergoing P. syringae infection.
280                                         Upon P. syringae infection, ACD2 levels and localization chan
281 n mutant was less responsive to BTH and upon P. syringae infection had reduced SA levels and increase
282 ica effector protein ATR13 was delivered via P. syringae by fusing the ATR13 gene with the avrRpm1 ty
283                                     Virulent P. syringae also has the potential to induce net systemi
284                                     Virulent P. syringae strains were able to overcome a PAMP pretrea
285 bivory caused by prior infection by virulent P. syringae.
286 and srfr3, that were susceptible to virulent P. syringae pv. tomato strain DC3000, but resistant to D
287 exhibits enhanced susceptibility to virulent P. syringae strains, suggesting it may impact basal dise
288  WIN2 or WIN3 confers resistance to virulent P. syringae, which is consistent with these proteins bei
289 iation in growth of the universally virulent P. syringae pv. maculicola ES4326 among more than 100 Ar
290  the wild type when challenged with virulent P. syringae.
291 d to defense soon after initial contact with P. syringae, but these proteins were not secreted in the
292 fers a benefit when plants are infected with P. syringae carrying avrPphB2 but also incurs a large co
293 e was observed in irAOX plants infected with P. syringae, which correlated with higher levels of sali
294 ase in free IAA levels during infection with P. syringae pv. tomato strain DC3000 (PstDC3000).
295 t induced when M. sativa was inoculated with P. syringae DC3000.
296 reased significantly when co-inoculated with P. syringae pv. tomato but not when co-inoculated with a
297 tive response when challenge inoculated with P. syringae pv. tomato DC3000.
298                             Inoculation with P. syringae DC3000(avrRpm1) or P. syringae DC3000(avrRpt
299 ion in N. caerulescens, but inoculation with P. syringae did not elicit the defensive oxidative burst
300 susceptibility upon surface inoculation with P. syringae, wider stomatal apertures, and enhanced plas

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