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1 P. syringae 508 was also surveyed for the presence of ef
2 P. syringae carrying hopW1-1 have restricted host range
3 P. syringae effector proteins encounter a pH gradient as
4 P. syringae pv. tomato DC3000 HrpP has a C-terminal, put
5 P. syringae was able to attach and extensively colonize
6 P. syringae was defective in porous-paper colonization w
13 ome of Por(1_6) is the first sequenced for a P. syringae isolate that is a pathogen of monocots, and,
14 ctive oxygen burst, and enhances growth of a P. syringae hrpA mutant that fails to secrete effectors.
15 oB-mediated anti-PCD activity, and abrogates P. syringae pathogenesis of susceptible tomato plants.
16 amount of NAD(P) leaking into the ECC after P. syringae pv. tobacco DC3000/avrRpt2 infection is suff
18 nce gene-mediated inducible defenses against P. syringae (and possibly other pathogens) by playing a
23 ce of PRR2 in plant immune responses against P. syringae and suggest a novel function for this partic
24 NLR induces strong defense responses against P. syringae and X. campestris The P. syringae T3SE HopZ1
25 Plants in turn defend themselves against P. syringae by activating the salicylic acid (SA)-mediat
27 that RhpRS is a global regulator that allows P. syringae to sense and respond to environmental change
28 s, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A restored HR elicitati
29 a gene in P. syringae pv. syringae B728a and P. syringae pv. tomato DC3000, termed phcA, that has hom
32 2 is transcriptionally upregulated by SA and P. syringae, enhances SA biosynthesis and SA signalling
39 s a selective advantage for microbes such as P. syringae that are adapted to maximally exploit cholin
41 of cytosolic calcium triggered by avirulent P. syringae was compromised in crt1-2 crh1-1 plants, but
42 were compromised for resistance to avirulent P. syringae and more susceptible to virulent strains tha
43 s have increased susceptibility to avirulent P. syringae strains, are unable to activate systemic acq
44 t DC3000) and avirulent (Pst DC3000 AvrRPM1) P. syringae strains, conserving typical hypersensitive r
46 response (HR) to effector proteins from both P. syringae and the fungal pathogen, Cladosporium fulvum
48 AvrPtoB did not prevent the HR activated by P. syringae pv. tomato DC3000 + avrB, avrRpm1, avrRps4 o
52 resistance signaling following infection by P. syringae expressing the Cys protease Type III effecto
55 A tyrosine phosphatase, HopAO1, secreted by P. syringae, reduces EFR phosphorylation and prevents su
57 arched for homologs to 44 known or candidate P. syringae type III effectors and two effector chaperon
61 gher in leaves challenged with COR-deficient P. syringae or in the more resistant JA receptor mutant
62 eins restores virulence of a HopM1-deficient P. syringae mutant, providing a link between HopM1 and t
63 y divergent protein in the T3SS of different P. syringae pathovars, hrpP from P. syringae pv. syringa
65 t high temperature, HopI1 is dispensable for P. syringae pathogenicity, unless excess Hsp70 is provid
69 of effectors and related T3SS substrates for P. syringae pv. tomato DC3000 and three other sequenced
71 contributes to virulence after delivery from P. syringae in leaves of susceptible soybean plants, and
73 f different P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseoli
74 0 PFPs along with pDC3000A and pDC3000B from P. syringae pv. tomato encoded a type IVB T4SS (tra syst
75 from P. syringae pv. syringae, pPh1448B from P. syringae pv. phaseolicola, and pPMA4326A from P. syri
76 yringae pv. phaseolicola, and pPMA4326A from P. syringae pv. maculicola encoded a type IVA T4SS (VirB
78 l criteria defined 29 type III proteins from P. syringae pv. tomato, and 19 from P. syringae pv. phas
79 omparative genomic analyses to elucidate how P. syringae subverts the attack and defense responses of
80 a detailed mechanistic understanding of how P. syringae transitions from reliance on exogenously der
82 rnitine/choline family transporter (BCCT) in P. syringae pv. tomato strain DC3000 that mediates the t
83 n of P. syringae pv. glycinea PG4180 CmaA in P. syringae pv. syringae FF5 as a FLAG-tagged protein an
86 zed the hopO1-1, hopS1, and hopS2 operons in P. syringae pv. tomato DC3000; these operons encode thre
88 Three alleles are known to be present in P. syringae, with HopZ1a most resembling the ancestral a
89 d stringent response plays a central role in P. syringae virulence and survival and indicated that pp
90 e data indicate that HrpJ has a dual role in P. syringae: inside bacterial cells HrpJ controls the se
91 enes coregulated with the Hrp T3SS system in P. syringae pv. tomato DC3000 have predicted lytic trans
94 e global role of rppH in thermoregulation in P. syringae, RNA sequencing was used to compare the tran
95 1 supported increased growth of ETI-inducing P. syringae strains compared with wild-type Arabidopsis.
96 A P. syringae hopD1 mutant and ETI-inducing P. syringae strains exhibited enhanced growth on Arabido
99 -LRR genes RPS2, RPM1, and RPS5 and isogenic P. syringae strains expressing single corresponding avir
108 at is supported by the common association of P. syringae with plants and the widespread production of
110 The hopPtoM and avrE genes in the CEL of P. syringae were found to encode suppressors of this SA-
111 and is carried in the functional cluster of P. syringae pv. syringae 61 hrp genes cloned in cosmid p
113 :acyl carrier protein transacylase (FabD) of P. syringae was overproduced and shown to catalyze malon
114 vrRpt2 may be among the virulence factors of P. syringae that modulate host auxin physiology to promo
115 tion was visualized via dual fluorescence of P. syringae cells harboring a transcriptional fusion of
116 erexpressing plants supported more growth of P. syringae and developed more severe disease symptoms t
119 g serine(s) in two other effectors, HopZ3 of P. syringae and PopP2 of Ralstonia solanacerum, also abo
120 fector as reporter revealed the inability of P. syringae 508 to translocate effectors into plant cell
127 resulted in the loss of swarming motility of P. syringae pv. tomato DC3000 on medium containing a low
128 onas fluorescens, a TTSS-deficient mutant of P. syringae pv. tabaci, or flg22 (a flagellin-derived pe
130 a FLAG-tagged protein and overproduction of P. syringae pv. tomato CmaA in Escherichia coli as a His
138 of the effectors comprising the secretome of P. syringae pv tomato DC3000 led to the identification o
139 s study, we report on the genome sequence of P. syringae pv. phaseolicola isolate 1448A, which encode
142 were more resistant to a virulent strain of P. syringae pv. tabaci and showed an accelerated hyperse
144 ced disease severity to a virulent strain of P. syringae, suggesting a role of ATT1 in disease resist
145 In basal resistance to virulent strains of P. syringae and H. arabidopsidis, PAD4 functions togethe
147 al phenotypic differences between strains of P. syringae is the range of plant hosts they infect.
148 -depleted plants to nonpathogenic strains of P. syringae supports a defense-promoting role for Hsp70.
150 ere we report the existence of a subgroup of P. syringae isolates that do not cause disease on any pl
152 n the temperature-dependent transcriptome of P. syringae, affecting the expression of 569 genes at ei
155 side plant cells to promote the virulence of P. syringae pv. tomato strain DC3000 (PstDC3000) on Arab
158 culation with P. syringae DC3000(avrRpm1) or P. syringae DC3000(avrRpt2) induces differential decreas
163 produced by immunization with Shewanella or P. syringae cells bound to B. anthracis spores but not t
164 common N-terminal characteristic from other P. syringae type III secreted substrates increased HopS2
165 between BAK1 and HopF2 and between two other P. syringae effectors, AvrPto and AvrPtoB, was confirmed
167 or repertoire of the sequenced bean pathogen P. syringae pv. syringae (Psy) B728a using bioinformatic
168 lso enhanced the growth of the host pathogen P. syringae pv tabaci by increasing nutrient efflux into
169 enes in the repertoire of the model pathogen P. syringae pv. tomato DC3000 were deleted to produce po
170 rains closely related to the tomato pathogen P. syringae pv. tomato (Pto), including strains isolated
172 Pseudomonas syringae sustains but pathogenic P. syringae suppresses early MAMP (microbe-associated mo
174 ae 508 the genomic region that in pathogenic P. syringae strains contains the hrp-hrc cluster coding
175 d are sufficient to transform non-pathogenic P. syringae strains into virulent pathogens in immunodef
176 ot affect plant susceptibility to pathogenic P. syringae bacteria; conversely, expression of the cons
177 ere highly susceptible to non-host pathogens P. syringae pv. syringae and P. syringae pv. tabaci.
178 with a phylogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a strong degree
179 toB acts as a virulence protein by promoting P. syringae pv. tomato growth and enhancing symptoms ass
180 es to disease resistance in response to Pto (P. syringae pathovars tomato) DC3000(avrB), but not agai
182 (localized to chloroplasts) greatly reduces P. syringae-induced PCD, suggesting a pro-PCD role for m
183 Repression of auxin signaling restricts P. syringae growth, implicating auxin in disease suscept
184 induce systemic susceptibility to secondary P. syringae infection in the host plant Arabidopsis thal
186 type III effector suites from two sequenced P. syringae pathovars and show that type III effector pr
188 cens was used to test the ability of several P. syringae pv. tomato DC3000 effectors for their abilit
189 ype plants; however, responses to A. solani, P. syringae, or M. sexta were similar to the wild-type p
192 s from the closely related pathogenic strain P. syringae pv. syringae B728a, but none were detected.
197 ical evidence supporting the hypothesis that P. syringae pv. syringae B728a produces both of these si
212 n somewhat differently than YscP because the P. syringae T3SS pilus likely varies in length due to di
214 st that chloroplast Hsp70 is targeted by the P. syringae HopI1 effector to promote bacterial virulenc
215 use it is poorly secreted in cultures by the P. syringae Hrp system, was translocated into plant cell
218 es against P. syringae and X. campestris The P. syringae T3SE HopZ1a is an acetyltransferase that ace
222 peron from Photorhabdus luminescens into the P. syringae chromosome under the control of a constituti
223 I effector genes, which are orthologs of the P. syringae effector genes hopAA1-1 and hopM1, as well a
227 Por(1_6) helps to define an expansion of the P. syringae pan-genome, a corresponding contraction of t
229 Here, the functional significance of the P. syringae T3SS substrate compositional patterns was te
230 two distinct levels in the regulation of the P. syringae TTSS: regulation of assembly of the secreton
231 anslationally modified after delivery of the P. syringae type III effectors AvrRpm1, AvrB, or AvrRpt2
232 structural and regulatory components of the P. syringae type III secretion system (T3SS), essential
235 nduced resistance to H. arabidopsidis and to P. syringae pv tomato whereas jasmonic acid is essential
239 for the superior osmoprotection conferred to P. syringae by choline over glycine betaine when these c
244 uced accumulation of Pip in leaves distal to P. syringae inoculation, they display a considerable sys
245 tibility of WRKY33-over-expressing plants to P. syringae is associated with reduced expression of the
249 nea) mediates species-specific resistance to P. syringae expressing the avirulence protein AvrB, simi
250 Arabidopsis increased disease resistance to P. syringae Expression of CRK28 in Nicotiana benthamiana
253 ana benthamiana did not confer resistance to P. syringae pv. tabaci (Pta) expressing avrPto or avrPto
254 vely, of the variance of basal resistance to P. syringae pv. tomato DC3000 in the Col-0 x Fl-1 F(2) p
255 expression of WRKY18 enhanced resistance to P. syringae, its coexpression with WRKY40 or WRKY60 made
259 s overexpressing IOS1 were more resistant to P. syringae and demonstrated a primed PTI response.
260 mutant were substantially more resistant to P. syringae but more susceptible to B. cinerea than wild
261 reduced in the pbs3-1 mutant in response to P. syringae (avrRpt2) infection, free SA was elevated.
264 SA signalling responses; e.g. in response to P. syringae, PRR2 induces the production of SA and the a
266 f three aspects of A. thaliana's response to P. syringae: symptom severity, bacterial population size
268 we analyzed Arabidopsis defense responses to P. syringae through differential coexpression analysis.
271 ins result in an increased susceptibility to P. syringae, whereas overexpression of these genes alter
277 staining effect was suppressed by wild-type P. syringae pv. tabaci and P. fluorescens heterologously
281 n mutant was less responsive to BTH and upon P. syringae infection had reduced SA levels and increase
282 ica effector protein ATR13 was delivered via P. syringae by fusing the ATR13 gene with the avrRpm1 ty
286 and srfr3, that were susceptible to virulent P. syringae pv. tomato strain DC3000, but resistant to D
287 exhibits enhanced susceptibility to virulent P. syringae strains, suggesting it may impact basal dise
288 WIN2 or WIN3 confers resistance to virulent P. syringae, which is consistent with these proteins bei
289 iation in growth of the universally virulent P. syringae pv. maculicola ES4326 among more than 100 Ar
291 d to defense soon after initial contact with P. syringae, but these proteins were not secreted in the
292 fers a benefit when plants are infected with P. syringae carrying avrPphB2 but also incurs a large co
293 e was observed in irAOX plants infected with P. syringae, which correlated with higher levels of sali
296 reased significantly when co-inoculated with P. syringae pv. tomato but not when co-inoculated with a
299 ion in N. caerulescens, but inoculation with P. syringae did not elicit the defensive oxidative burst
300 susceptibility upon surface inoculation with P. syringae, wider stomatal apertures, and enhanced plas
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