ライフサイエンスコーパス: P2 purinergic receptor

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1 ion and releases IL-33 through activation of P2 purinergic receptors.

2 cellular functions through the activation of P2 purinergic receptors.

3 and its occurrence led to the appearance of P2 purinergic receptors.

4 danger associated molecular pattern through P2 purinergic receptors.

5 her by autocrine activity of secreted ATP on P2 purinergic receptors.

6 eatment, G(q/11) antagonism, and blockade of P2 purinergic receptors.

7 tes ligand availability to a large family of P2 (purinergic) receptors.

8 In this study, the effect of P2 purinergic receptor activation on contractions and on

9 se of Ca2+ from internal stores initiated by P2 purinergic receptor activation.

10 gnaling by extracellular nucleotides through P2 purinergic receptors affects diverse macrophage funct

11 Unlike results with PMA, or with the P2 purinergic receptor agonist ATP, epinephrine-stimulat

12 Inhibition by P2 purinergic receptor agonists was comparable to that i

13 on and suggest a role for extracellular ATP, P2 purinergic receptors, and calcium-dependent ERK signa

14 se dependent, occurred predominantly through P2 purinergic receptors, and was observed even when cell

15 d hepatic JNK signaling, while infusion of a P2 purinergic receptor antagonist prior to partial hepat

16 or attenuated by the further addition of the P2 purinergic receptor antagonist pyridoxal phosphate-6-

17 appears to be specific for LPS, because the P2 purinergic receptor antagonists do not affect P388D(1

18 e results together with the observation that P2 purinergic receptors are present throughout the brain

19 n, in an autocrine or paracrine manner, with P2 purinergic receptors expressed on the cell surface.

20 exist between hCPC phenotypic properties and P2 purinergic receptor expression.

21 mobilization of intracellular calcium by the P2-purinergic receptor, ionomycin, or a direct activator

22 A23187, by physiologic concentrations of the P2 purinergic receptor ligand ATP, or by changes in extr

23 uggests that extracellular ATP signaling via P2 purinergic receptors may be involved in different ren

24 the involvement of extracellular ATP and its P2 purinergic receptor-mediated signaling in physiologic

25 These ionotropic and metabotropic P2 purinergic receptors modulate a variety of physiologi

26 by endoplasmic reticulum (ER) depletion and P2 purinergic receptor (P2R)/phospholipase C (PLC) inhib

27 E1 osteoblasts, where it associates with the P2 purinergic receptor P2rx7 to stimulate Mmp13 expressi

28 t the hypothesis that caudal commissural NTS P2 purinergic receptors play a role in the neurotransmis

29 of ATP from apoptotic cancer cells to engage P2 purinergic receptor signaling cascades in nearby leuk

30 other cell systems, ATP and UTP, acting via P2 purinergic receptors, stimulated cAMP production in M

31 n many cell types via activation of multiple P2-purinergic receptor subtypes.

32 atory responses by binding to plasmamembrane P2 purinergic receptors, which are widely expressed on c

33 ignals are partly dependent on activation of P2 purinergic receptors, which was judged from the effec

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