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1 ed by reactive blue 2, an antagonist of some P2 purinoceptors.
2 G protein-coupled receptor with homology to P2 purinoceptors.
3 e cerebral vessels through the activation of P2 purinoceptors.
4 cellular functions through the activation of P2-purinoceptors.
6 microinjection of suramin (50 nl, 0.02 M), a P2 purinoceptor antagonist, into the retrofacial area in
7 nal purinoceptors in nociception, the potent P2-purinoceptor antagonist reactive red 2 was studied in
8 was abolished by apamin (0.3 microM) and the P2-purinoceptor antagonist suramin (100 microM); the oth
10 the effects of P2X purinoceptor agonists and P2 purinoceptor antagonists on mesenteric afferent nerve
16 red 2 exerts antinociception by blockade of P2-purinoceptors in the spinal cord and, hence, support
17 s and fibroblasts and that the activation of P2 purinoceptors is involved in the regulation of DNA sy
18 These results support the hypothesis that P2 purinoceptors may be involved in mediating autoregula
19 ays and molecular mechanisms responsible for P2-purinoceptor-mediated chloride (Cl(-)) currents (I(Cl
23 l ATP, via interactions with apical membrane P2-purinoceptors, regulates the balance of active ion se
24 indicated that P2Y3 is a previously unknown P2 purinoceptor subtype with a preference for nucleoside
27 ry efficiency before and after saturation of P2 purinoceptors with acute intra-arterial administratio
28 activates extracellular ATP, and by blocking P2 purinoceptors with pyridoxal phosphate-6-azo(benzene-
29 e abolished following the desensitization of P2-purinoceptors with alpha, beta-methylene ATP (m-ATP;
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