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1 n the absence of factor B, and by purinergic P2 receptor antagonists.
2 (2+)]i oscillations, which were abolished by P2 receptor antagonists.
3 reduced or abolished by prior application of P2 receptor antagonists.
4 dently reduced in the presence of ATPases or P2 receptor antagonists.
5 l CO2/H+ sensitivity by an amount similar to P2 receptor antagonists.
6 ng expansion, and were reversibly blocked by P2 receptor antagonists.
8 nical strain and was completely blocked by a P2 receptor antagonist and by inhibition of p38/mitogen-
9 terms of insensitivity to blockade by known P2 receptor antagonists and the ineffectiveness of adeno
14 sly we have shown that injection of PPADS, a P2 receptor antagonist, into the arterial supply of skel
19 L67156 and were depressed in the presence of P2 receptor antagonists PPADS (10 microm) and suramin (5
21 ATP receptors in the VLM (microinjection of P2 receptor antagonist pyridoxal-5'-phosphate-6-azopheny
22 and hP2X(2b) receptors were sensitive to the P2 receptor antagonist pyridoxal-5-phosphate-6-azophenyl
23 agal neurons which were blocked by the broad P2 receptor antagonist pyridoxal-phosphate-6-azophenyl-2
24 agal neurons which were blocked by the broad P2 receptor antagonist pyridoxal-phosphate-6-azophenyl-2
26 gent of ADP degradation), suramin (a general P2 receptor antagonist), pyridoxal 5'-phosphonucleotide
27 ve antagonist, MRS2179, and the nonselective P2 receptor antagonists, pyridoxal phosphate 6-azophenyl
28 lphabetam-ATP were blocked completely by the P2 receptor antagonist pyridoxalphosphate-6-azophenyl-2'
30 sion of GFAP and MMP-9 and a purinergic ATP (P2) receptor antagonist reduction in calcium response id
35 ntagonist oxidized ATP (oATP) but not by the P2 receptor antagonist suramin; both ATP and BzATP incre
36 s blocked by ionophoretic application of the P2 receptor antagonists suramin (0.02 M) and pyridoxal-p
40 ist sulfonylphenyl theophylline, but not the P2-receptor antagonist suramin, antagonized the effect o
41 ed by apyrase inactivation of nucleotidases, P2 receptor antagonists, tetrodotoxin (TTX), or piroxica
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