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1 esized and assayed for activity at the human P2Y6 receptor.
2 ptor is a species homologue of the mammalian P2Y6 receptor.
3 mologous to the avian p2y3 receptor than the P2Y6 receptor.
4  at the turkey p2y3 receptor than at the rat P2Y6 receptor.
5 ective, indicating the absence of functional P2Y6 receptors.
6 2 and P2Y4 receptors, and at recombinant rat P2Y6 receptors.
7 e that the signaling apparatus downstream of P2Y6 receptor activation is moderately saturable.
8 A being produced upon UDP stimulation of the P2Y6 receptor and that PA levels are tightly regulated.
9 uridine nucleotide-responsive receptors, the P2Y6 receptor and the P2Y4 receptor.
10 nderstanding of molecular recognition at the P2Y6 receptor and will be helpful in designing selective
11 te the P2Y14 receptor over the UDP-activated P2Y6 receptor, and these molecules stimulated phosphoryl
12 d by treatment of the cells with a selective P2Y6 receptor antagonist (MRS2578), which did not interf
13           The present study identifies mouse P2Y6 receptor as a regulator of cardiac development and
14  and MRS2578, an inhibitor of the purinergic P2Y6 receptor, blocked NET formation triggered by MSU cr
15 hairpin RNA-mediated knockdown of CysLT1R or P2Y6 receptors, but not of CysLT2R.
16 by homology screening with radiolabeled P2Y1-P2Y6 receptor cDNAs.
17                                              P2Y6 receptor could constitute a therapeutic target to r
18                We then observed that loss of P2Y6 receptor enhanced pathological cardiac hypertrophy
19 llowed the clear identification of the human P2Y6 receptor gene.
20  modeling of the uracil nucleotide activated P2Y6 receptor have been studied.
21 e stably expressed this receptor and the rat P2Y6 receptor in 1321N1 human astrocytoma cells.
22 ably favored by the upregulation of P2Y4 and P2Y6 receptors in airway epithelium during sensitization
23 ized tracheas showed slight fluorescence for P2Y6 receptors in epithelium and none for P2Y4 .
24           Moreover, the expression levels of P2Y6 receptors in GC tissues were correlated to tumor si
25  with a large increment in mRNA for P2Y4 and P2Y6 receptors in sensitized animals.
26 es by ATP and UTP but not by ADP or UDP; the P2Y6 receptor is activated most potently by UDP but weak
27 be helpful in designing selective and potent P2Y6 receptor ligands.
28 tive as either an agonist or antagonist in a P2Y6 receptor-mediated contractile response.
29 e-activated human P2Y4 receptor (P2Y4-R) and P2Y6 receptor (P2Y6-R) was studied.
30 between C5a and UDP, mediated by the C5a and P2Y6 receptors, required dual receptor occupancy, and af
31 ceptor that is selectively activated by UDP (P2Y6 receptor), selectively activated by UTP (P2Y4 recep
32  study focuses on profiling the PA pool upon P2Y6 receptor signaling manipulation to determine the ma
33 eam events resulting in PA production in the P2Y6 receptor signaling pathway.
34 ptor of C6 glioma cells or on P2Y2, P2Y4, or P2Y6 receptors stably expressed in 1321N1 human astrocyt
35  P2Y receptor with 65% identity to mammalian P2Y6 receptors, termed the p2y3 receptor, was recently c
36 human MC line LAD2 all express UDP-selective P2Y6 receptors that cooperate with CysLT1R to promote ce
37                         Ncleotides activated P2Y6 receptors to raise cytosolic Ca(2+) concentrations
38 ively, we conclude that nucleotides activate P2Y6 receptors to suppress GC growth through a novel SOC
39 r evidence that demonstrates the presence of P2Y6 receptor transcripts in rat pulmonary arterial smoo
40 expression of mRNA for P2Y1, P2Y2, P2Y4, and P2Y6 receptors was demonstrated in HaCaT cells and diffe
41 te for the first time that the expression of P2Y6 receptors was markedly down-regulated in human GC c
42  or was a species homologue of the mammalian P2Y6 receptor, we screened two different human genomic l
43                             Furthermore, the P2Y6 receptor, which displays a uridine nucleotide selec
44                            Thus, CysLT1R and P2Y6 receptors, which are coexpressed on many cell types

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