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1 bit but not rodent members of the cytochrome P450 3A.
2 pha, albumin, cytokeratin 18, and cytochrome P450 3A.
3 ifampin and phenobarbital, which also induce p450 3A activity, have been reported to significantly de
4 inhibitors of the hepatic enzyme, cytochrome P450 3A, and the drug transporter, P-glycoprotein, which
6 However, MG132 has been reported to suppress P450s 3A as a result of impaired nuclear factor-kappaB a
8 icardipine-treated rats will form cytochrome P450 3A (CYP3A) aggregates when incubated at 37 degrees
9 -carbonitrile (PCN), which induce cytochrome P450 3A (CYP3A) expression and protect the body from har
15 matic increase in the activity of cytochrome P450 3A (CYP3A), which catalyzes side-chain hydroxylatio
18 ole, but not sulfaphenazole, suggesting that P450 3A enzymes are responsible for this activity in the
19 quinidine (QIN), substrates specific to cyt P450 3A enzymes, were used to demonstrate applicability
20 gnane X receptor (hPXR) activates cytochrome P450-3A expression in response to a wide variety of xeno
22 itazone is not metabolized by the cytochrome p450 3A isozyme family, it is a potential inducer of thi
23 monophosphate (NMP), anticipating cytochrome P450 3A-mediated oxidative cleavage to the NMP in hepato
25 al induction of the gene encoding cytochrome P450 3A oxygenase (CYP3A) causes a prominent class of da
27 rial cytochromes P450 can be used to predict P450 3A residues that contribute to regiospecific steroi
28 vir is an inhibitor of the enzyme cytochrome P450 3A, responsible for the metabolism of both cyclospo
30 idazolam, two clinically relevant cytochrome P450 3A substrates, after cardiac arrest and to investig
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