ライフサイエンスコーパス: PE

1 PE + SU produced right ventricular hypokinesis, dilation

2 PE + SU rats exhibited sustained pulmonary hypertension

3 PE biodegradation is achieved through the combination of

4 PE dose-response for splanchnic flow and resistance were

5 PE episodes were documented 78 times, involving 50 of 72

6 PE is largely utilized in packaging, representing approx

7 PE signaling is significantly disrupted in individuals w

8 ty-one persons with NCC who experienced >/=1 PE episode were followed for a median of 10.6 years (ran

9 ) showed significantly altered lysoPC(18:1), PE(18:3/16:0), PC(20:1/18:3), DG(24:1/22:6/0:0), PS(18:2

10 filters were defined as VCFs placed after a PE.

11 We calculate a PE of 655 kJ/kg CO2, which is lower than that of the bes

12 Conclusion The odds of an acute PE finding in the ED when providers adhered to evidence

13 between the two groups, the odds of an acute PE finding were 51.3% lower when providers overrode aler

14 erts suggested d-dimer testing because acute PE is highly unlikely in these patients if d-dimer level

15 onsecutive patients were evaluated for acute PE.

16 placebo in normotensive patients with acute PE, right ventricular (RV) dysfunction on imaging, and a

17 Adjusted PE rates for the PTOS and NTDB showed significant initia

18 A neurons and increased addiction risk after PE.SIGNIFICANCE STATEMENT Prenatal ethanol exposure (PE)

19 e presence of circulating antibodies against PE is positively correlated with the symptoms of antipho

20 emical composition of coarse/ground bran and PE bran differed.

21 can be explained by the fact that coarse and PE bran hold more weakly bound water than ground bran, w

22 mobile water peak was noticed in coarse and PE bran, but not in ground bran.

23 Acute exposure to fibers and PE beads both showed a dose-dependent effect on survival

24 sequencing (PAS-Seq) of RNA from normal and PE human placentae to interrogate transcriptome-wide gen

25 udy we investigated the uptake of normal and PE STBEVs by primary human coronary artery endothelial c

26 architectonically defined areas, the PEc and PE.

27 ionwide temporal trends in VCF placement and PE occurrence from 2003 to 2015 using available data set

28 Temporal trends in VCF placement and PE rates, filter type (prophylactic or therapeutic), and

29 was found to be 1.5% and 3.1% after F&V and PE consumption, respectively.

30 and the mechanism by which circulating anti-PE antibodies access cellular PE remains unknown.

31 gs provide insight into the etiology of anti-PE autoimmunity and, because endosomes are of central im

32 with anti-PE autoimmunity, and purified anti-PE antibodies.

33 gest that the cellular vulnerability to anti-PE antibodies may be mediated by the binding of PE molec

34 ents, plasma samples from patients with anti-PE autoimmunity, and purified anti-PE antibodies.

35 ains an unusual family of abundant antigens (PE/PPEs).

36 Area PE received additional inputs from a posterior-dorsal pa

37 ral territories sending projections to areas PE and PEc partially overlapped zones previously shown t

38 interval 92%-100%) for classifying an eye as PE or PPE.

39 ch imaging technique for classifying eyes as PE or PPE, and misinterpretation rates of each imaging m

40 ATP attenuated PE-mediated vasoconstriction relative to adenosine (ADO)

41 ver, genetic depletion of Beclin1 attenuated PE-mediated/FAK-dependent initiation of myocyte hypertro

42 ubjects showed a reduced correlation between PE and activity within the ventromedial prefrontal corte

43 However, ATP maintained the ability to blunt PE-mediated vasoconstriction (PE-mediated DeltaFVC: KIR

44 enter T follicular helper cells, and blunted PE-specific germinal center B cell responses.

45 vity in visual regions, while stimulus-bound PEs correlated with gamma-band activity in posterior reg

46 In Trypanosoma brucei, PE synthesis has been shown to occur mainly via the Kenn

47 y for treating behavioral symptoms caused by PE.

48 nalyses have not adjusted for confounding by PE risk factors that are associated with season of conce

49 omparisons between the CA-PF, CAE-PE, and CA-PE groups.

50 xcept for comparisons between the CA-PF, CAE-PE, and CA-PE groups.

51 rculating anti-PE antibodies access cellular PE remains unknown.

52 olymers can weld common grades of commercial PE and iPP together, depending on the molecular weights

53 Consequently, PE represents by itself more than half of all plastic wa

54 Our data indicate that Psd1-dependent PE made at mitochondria and the ER has separable cellula

55 sion partners, and we examined how different PE distributions between the two leaflets of the support

56 tabolization in the presence of polymyxin E (PE) and PB via pH-induced color change.

57 Pre-eclampsia (PE), which affects approximately 8% of first pregnancies

58 transient development of perilesional edema (PE) around >/=1 calcification (defined as 1 episode) occ

59 ases are associated with perilesional edema (PE), likely due to host inflammation.

60 FVF as a mechanism in the Prevalence Effect (PE) in visual search.

61 ) within distinct brain regions, with effort PEs expressed in dorsomedial prefrontal cortex and rewar

62 (0.867) when classifying findings as either PE positive or PE negative, but no significant differenc

63 thod for the fabrication of paper electrode (PE) using silver nanoparticles capped with octylamine (A

64 sulator, polyethylene (PE), and at electrode/PE interfaces.

65 were suspected of having pulmonary embolism (PE) and who underwent CT pulmonary angiography between J

66 While pulmonary embolism (PE) causes approximately 100 000-180 000 deaths per year

67 NLP) model in extracting pulmonary embolism (PE) findings from thoracic computed tomography (CT) repo

68 Pulmonary embolism (PE) is a serious and prevalent cause of vascular disease

69 hrombolytic treatment of pulmonary embolism (PE) is unknown.

70 lter (VCF) placement for pulmonary embolism (PE) prophylaxis in trauma is controversial.

71 y used for prevention of pulmonary embolism (PE).

72 wed by epiblast (EPI) or primitive endoderm (PE) specification within the ICM.

73 e new organism, from the primitive endoderm (PE), which will form the yolk sac, is a crucial developm

74 Endosomal PE binding led to functional changes in endothelial cell

75 The parasitic energy (PE) has been put forward as a holistic parameter that me

76 r volume [FTV], peak percentage enhancement [PE], peak signal enhancement ratio [SER], and washout fr

77 rs in coarse, ground, and pericarp-enriched (PE) wheat bran and refined flour was investigated using

78 e locus driving Flt1 upregulation in both EO-PE and LO-PE.

79 ignatures associated with early-onset PE (EO-PE; symptom onset < 34 weeks) and late-onset PE (LO-PE;

80 e polyadenylation associated with PE, the EO-PE and LO-PE cohorts do exhibit gene expression profiles

81 ardial cells (PECs) from the pro-epicardium (PE) and their subsequent translocation to the heart to f

82 sentation of reinforcement prediction error (PE) (the difference between received and expected reinfo

83 trial-by-trial S-C and S-R prediction error (PE) estimates in model-based behavioral and fMRI analyse

84 l intelligence: the reward prediction error (PE)-the difference between how rewarding an event is, an

85 series of predictions and prediction errors (PEs) were associated with dissociable neural responses:

86 ding of effort and reward prediction errors (PEs) within distinct brain regions, with effort PEs expr

87 We identified the loci espE, PE-PGRS56, Rv0197, Rv2813-2814c, and Rv2815-2816c as tar

88 egation of three-ring phenylene-ethynylenes (PEs).

89 ial treatment consisted of plasma exchanges (PE), high doses of calcineurin inhibitors, and steroids.

90 as VCFs placed before or without an existing PE, while therapeutic filters were defined as VCFs place

91 oups: (a) people with psychotic experiences (PE; n=20) at increased risk for psychotic disorders; (b)

92 FICANCE STATEMENT Prenatal ethanol exposure (PE) is among many adverse developmental factors known to

93 Prenatal ethanol exposure (PE) leads to increased addiction risk which could be med

94 dded, or the corresponding phenolic extract (PE).

95 suggest that there is a seasonal driver for PE, with effects that are not modified by parity, latitu

96 pretation rates of each imaging modality for PE and PPE.

97 focal adhesion kinase (FAK) is necessary for PE-stimulated autophagy suppression and subsequent initi

98 curvature of tN-Ras when substituting PC for PE, and when decreasing acyl chain length from 14 to 12

99 e to the endothelial dysfunction typical for PE.

100 nt (N = 10) or failed attempted weaning from PE (N = 3).

101 either the initial treatment or weaning from PE.

102 tipotency, failing to form TE and generating PE on their surface.

103 on, in the case of the glycerophospholipids (PE, PS, and PC), via ester bond cleavage.

104 at age 20 years in 123 participants who had PEs and 124 individuals serving as controls.

105 ups including only patients unlikely to have PE led to similar results.

106 d-dimer testing in patients unlikely to have PE.

107 tter pathophysiological understanding of how PE transitions to CTEPH in human treatments.

108 However, PE is generally sequestered inside a normal resting cell

109 substrate competence to generate hydroperoxy-PE.

110 Inadequate reduction of hydroperoxy-PE due to insufficiency or dysfunction of a selenoperoxi

111 aptic plasticity is fundamentally altered in PE animals.

112 nd endoplasmic reticulum stress described in PE, as well as endothelial reprogramming that may underl

113 The NIS showed an initial increase in PE rates followed by a period of stagnation.

114 To identify the pathways involved in PE consumption, we used in the present study a combinati

115 In contrast, eCB-LTD in PE animals was not rescued by increasing presynaptic act

116 Interestingly, eCB-LTD in PE animals was rescued by metabotropic glutamate recepto

117 aptic activity, which actually led to LTP in PE animals, whereas LTD was still observed in controls.

118 Uptake of PCBs in PE after 7, 14, 28, and 56 days under different vibratio

119 ascularization and cellular proliferation in PE that was distinctly absent in the PE + SU group.

120 ease was regulated by tonic eCB signaling in PE animals.

121 tes, suggesting that current goals influence PE and prediction signalling.

122 Interestingly, PE, but not F&V, significantly decreased the speed and e

123 rhages rival overall harms from intermediate PE.

124 tb1 expression leads to differentiation into PE and a decrease in EPI, as a result of the modulation

125 lcium phosphate (ACP) were incorporated into PE and PEHB at 40% filler level.

126 matic hydrocarbons (PAHs) from sediment into PE.

127 Strikingly, cells that lack PE methylation accumulate SAM, which leads to hypermethy

128 vage of pro-LC3 and the de-lipidation of LC3-PE from autophagosomes, both executed by the same cystei

129 rfaces (PF) and from enamel carious lesions (PE) and dentin carious lesions (PD) were collected.

130 vert their fate into an alternative lineage (PE towards EPI and vice versa), until peri-implantation

131 synthesis of the copolymer, a cross-linkable PE.

132 ylation associated with PE, the EO-PE and LO-PE cohorts do exhibit gene expression profiles distinct

133 iving Flt1 upregulation in both EO-PE and LO-PE.

134 ptom onset < 34 weeks) and late-onset PE (LO-PE; symptom onset > 34 weeks) cohorts.

135 5% CI: 1.17 to 2.48); nonsignificantly lower PE-related mortality (OR: 0.51; 95% CI: 0.25 to 1.05); a

136 MOF, 1 [Ni-(4PyC)2.DMF], that has the lowest PE for postcombustion CO2 capture reported to date.

137 -directed therapy for submassive and massive PE and gives representative examples that reflect modern

138 tionale for stratification, imaging, massive PE management and outcomes, submassive PE management and

139 ptor antagonist in polystyrene microspheres (PE) + tyrosine kinase inhibitor SU5416 (SU) group.

140 In adjusted models, PE risk was related to season, with higher risk in sprin

141 antibodies may be mediated by the binding of PE molecules in the membrane of the early endosome.

142 liarizes the reader with these categories of PE.

143 sothermal and nonisothermal decomposition of PE in NOx-doped atmosphere.

144 tion, and thermogravimetric decomposition of PE under controlled atmospheres, with NOx concentration

145 We report the fast bio-degradation of PE by larvae of the wax moth Galleria mellonella, produc

146 l, fail to induce a clear-cut degradation of PE samples, showing that bottlenecks to reach optimal bi

147 btained during the electrical degradation of PE.

148 Four different distributions of PE in the planar bilayer mimicking the plasma membrane w

149 In this study, we investigated the effect of PE on endocannabinoid-mediated long-term depression (eCB

150 Small effects of PE addition were detected suggesting slightly increased

151 oncentration, largely removed all effects of PE, shows that PE did not act as a measurable vector of

152 es the number of EPI cells at the expense of PE.

153 PE), >/=37 weeks, is the most common form of PE and the most difficult to predict.

154 that short-term exposure to various forms of PE, displaying different molecular weight distributions

155 Thus, common grades of PE and iPP do not adhere or blend, creating challenges f

156 t the performance gains by implementation of PE-UVPD for peptide analysis in an ion trap mass spectro

157 Both moderate and high levels of PE persistently reduced low-frequency stimulation-induce

158 s: the presence, chronicity, and location of PE.

159 phosphatidylcholine (PC), the methylation of PE facilitates the turnover of SAM for the synthesis of

160 more likely to lead to misinterpretation of PE as PPE, which could potentially result in failure to

161 likelihood (30%-70%) of misinterpretation of PE as PPE.

162 etailing trends in VCF use and occurrence of PE over time.

163 ss, optimal treatment for many phenotypes of PE remains uncertain.

164 therapeutic), and established predictors of PE (obesity, pregnancy, cancer, deep vein thrombosis, ma

165 say for selectively monitoring production of PE in reactions using a maltose-binding protein fusion w

166 have limited utility in influencing rates of PE.

167 ted day of the year of conception on risk of PE for births from the Medical Birth Registry of Norway

168 C filters versus none in patients at risk of PE.

169 ms have been shown to grow on the surface of PE materials, among which are the species of the Rhodoco

170 The number and timing of PE episodes in individuals with calcified NCC are variab

171 s of recycled PE reflects different types of PE in the plastic, as measurements of intrinsic viscosit

172 ic midbrain regions expressing both types of PE.

173 ation signatures associated with early-onset PE (EO-PE; symptom onset < 34 weeks) and late-onset PE (

174 PE; symptom onset < 34 weeks) and late-onset PE (LO-PE; symptom onset > 34 weeks) cohorts.

175 h could be fruitfully targeted for optimized PE consumption by R. ruber.

176 lassifying findings as either PE positive or PE negative, but no significant difference in sensitivit

177 rrectly classifying pediatric eyes as PPE or PE is FA.

178 strategy, termed precursor ejection UVPD or PE-UVPD, allows the ion trap to be filled with more ions

179 All other PE distributions decreased fusion efficiency, altered po

180 r classifying pediatric eyes as papilledema (PE) or pseudopapilledema (PPE).

181 hese microorganisms to use, at least partly, PE as a potent carbon source.

182 PC/PE binds between alphaTM2, 4, 6, and 9 and accelerates t

183 tures, the underlying mechanism of PS and PC/PE effects emerges.

184 sms, specific interactions of both PS and PC/PE have been inferred.

185 Main outcomes included subsequent PE, PE-related mortality, all-cause mortality, and subsequen

186 to intra-arterial infusion of phenylephrine (PE; alpha1 -agonist) during ATP or control vasodilatator

187 responsive to treatment with phenylephrine (PE) (10(-5) m) and ACh (10(-5) m).

188 arteriolar constriction with phenylephrine (PE) (10(-5) m) induced a significant increase of N-cadhe

189 lation of cardiomyocytes with phenylephrine (PE), a well known hypertrophic agonist, suppresses autop

190 Phosphatidylethanolamine (PE) and phosphatidylserine (PS) are ubiquitously express

191 Phosphatidylethanolamine (PE) is a major phospholipid species with important roles

192 Phosphatidylethanolamine (PE), a cone-shaped phospholipid, is predominantly locate

193 ylcholine (PC) and phosphatidylethanolamine (PE)) and enhance the signals from the low-abundance CLs

194 nking proteins and phosphatidylethanolamine (PE).

195 1, which generates phosphatidylethanolamine (PE).

196 )C-TrEnDi-modified phosphatidylethanolamine (PE), phosphatidylserine (PS), phosphatidylcholine (PC),

197 dylcholine (PC) or phosphatidylethanolamine (PE), known to stimulate Na,K-ATPase activity.

198 tidylcholine (PC), phosphatidylethanolamine (PE), and cholesterol were created by extrusion.

199 of a phospholipid, phosphatidylethanolamine (PE), is a major consumer of SAM.

200 olism that produce phosphatidylethanolamine (PE) in bacteria, protists, plants, and animals.

201 sis indicated that phosphatidylethanolamine (PE) and phosphatidylcholine (PC) were the major lipid co

202 3 is conjugated to phosphatidylethanolamine (PE) on the inner and outer membrane of autophagosomes to

203 idylcholine (PC), phosphatidylethanolamines (PE), and phosphatidylinositol (PI), in newly fertilized

204 f polyunsaturated phosphatidylethanolamines (PE) by 15-lipoxygenases (15-LO) that normally use free p

205 phosphatidic acid (PA), phosphoethanolamine (PE), phosphatidylserine (PS), phosphatidylglycerol (PG),

206 Polyelectrolyte (PE) wrapping of colloidal nanoparticles (NPs) is a stand

207 Polyethylene (PE) and isotactic polypropylene (iPP) constitute nearly

208 Polyethylene (PE) and polypropylene (PP) represent approximately 92% o

209 Polyethylene (PE), one of the most prominent synthetic polymers used w

210 roplastic polyester fibers and polyethylene (PE) beads on freshwater zooplankton Ceriodaphnia dubia.

211 Common plastics such as polyethylene (PE), polypropylene (PP), polystyrene (PS), and polyethyl

212 sing recycled plastics such as polyethylene (PE).

213 pical and pervasive insulator, polyethylene (PE), and at electrode/PE interfaces.

214 fore, we studied the effect of polyethylene (PE) microplastic in sediment on PCB uptake by Arenicola

215 Periodic vibration of polyethylene (PE) passive samplers during exposure has been previously

216 nlight and rain degradation of polyethylene (PE) with organic and inorganic pigments.

217 language processing tool identified positive PE diagnoses, with subsegmental and/or indeterminate dia

218 CT pulmonary angiography (number of positive PE diagnoses/total number of CT pulmonary angiographic e

219 Preeclampsia (PE) is a dangerous and unpredictable pregnancy complicat

220 In preeclampsia (PE), cytotrophoblast (CTB) invasion of the uterus and sp

221 Maternal symptoms of preeclampsia (PE) are primarily driven by excess anti-angiogenic facto

222 pregnancy associated syndrome preeclampsia (PE), there is increased release of placental syncytiotro

223 wed a reduced correlation between punishment PEs, but not reward PEs, and activity within the left an

224 efficiency of NOx by fragments of pyrolyzing PE.

225 kJ/mol to 268 kJ/mol, for neat and recycled PE, respectively, and the so-called compensation effect

226 observed delay in thermal events of recycled PE reflects different types of PE in the plastic, as mea

227 rinsic viscosity indicate that, the recycled PE comprises longer linear chains.

228 in light through photoisomerization of N-ret-PE (N-retinylidene-phosphatidylethanolamine.

229 in dorsomedial prefrontal cortex and reward PEs in ventral striatum.

230 ation between punishment PEs, but not reward PEs, and activity within the left and right lentiform nu

231 For the highest-risk PE, presenting as shock or arrest, emergent systemic thr

232 prognosis of patients with intermediate-risk PE and the effect of thrombolytic treatment on the persi

233 unctional limitation after intermediate-risk PE, but CTEPH is infrequent.

234 mbolectomy is reasonable, while for low-risk PE, anticoagulation alone is often chosen.

235 d for VVS compared with control after saline+PE, but enhanced after L-NMMA (P<0.001).

236 Shams received volume-matched saline; PE and SU groups received only microspheres or SU5416, r

237 rfacial compatibilization of phase-separated PE and iPP with tetrablock copolymers enables morphologi

238 ts with liposome bound Atg18 yielded several PE cross-linked peptides.

239 ous pregnancy that was complicated by severe PE (sPE).

240 We studied the effects of severe PE (sPE) on the smooth chorion portion of the fetal memb

241 nosine (ADO) and sodium nitroprusside (SNP) (PE-mediated DeltaFVC: ATP: -16 +/- 2; ADO: -38 +/- 6; SN

242 a toxin B subunit-Alexa 647 and streptavidin-PE/Cy5, to membranes containing different amounts of lig

243 ssive PE management and outcomes, submassive PE management and outcomes, and future directions.

244 d to patients who have massive or submassive PEs.

245 ng IVC filters had lower risk for subsequent PE (OR: 0.50; 95% confidence interval [CI]: 0.33 to 0.75

246 Main outcomes included subsequent PE, PE-related mortality, all-cause mortality, and subse

247 ters appear to reduce the risk of subsequent PE, increase the risk for DVT, and have no significant e

248 We synthesized PE/iPP multiblock copolymers using an isoselective alken

249 presented here were conducted with synthetic PE-binding agents, plasma samples from patients with ant

250 Fibers showed greater adverse effects than PE beads.

251 Biodynamic model analysis confirmed that PE ingestion contributed marginally to bioaccumulation.

252 We found that PE estimates derived from S-C and S-R associations accou

253 ing between 4 and 10 weeks old.We found that PE led to increased amphetamine self-administration.

254 c studies for impaired eCB-LTD revealed that PE downregulated CB1 receptor function.

255 Our results show that PE persistently alters eCB signaling and impairs eCB-LTD

256 Previous studies have shown that PE enhances excitatory synaptic strength by facilitating

257 argely removed all effects of PE, shows that PE did not act as a measurable vector of PCBs.

258 amate receptor I activation, suggesting that PE did not impair the synthesis/release of eCBs.

259 The PE detection by fluorescence (lambdaex = 403 nm, lambdae

260 The PE encapsulation in the liposomes was responsible for ch

261 The PE was prepared by direct writing onto the photo paper u

262 The PE/AgNPs-OA electrode is found to be economic, facile an

263 The PE/AgNPs-OA exhibited a wide linear calibration range fr

264 The PE/AgNPs-OA was used as a working electrode in cyclic vo

265 istent with increased striatal dopamine, the PE group exhibited better behavioral learning (ie, faste

266 tion in PE that was distinctly absent in the PE + SU group.

267 (ArF-ArH) interactions cause twisting in the PE backbone, interrupting intramolecular conjugation as

268 tion of specific target genes, including the PE associated gene fms related tyrosine kinase 1 (FLT1).

269 flect the known functional properties of the PE and PEc, with the former containing neurons that are

270 y demonstrating that physical contact of the PE with the myocardium constitutes a third mechanism for

271 d 28-41, in the same order, depending on the PE conversion in the experimental interval of between 5

272 a larger FRN to punishment than reward, the PE group showed similar FRNs to punishment and reward, w

273 The projection zones to the PE and PEc overlapped extensively, and consisted of mult

274 re plentiful and extremely homologous, these PE/PPEs are very challenging to study, and it is difficu

275 VTA DA neurons, which in turn contributes to PE-induced increase in addiction risk.

276 munized mice with peptide Ag 2W1S coupled to PE in CFA and treated with the selective A2aR agonist CG

277 th no deformities observed after exposure to PE beads.

278 pathway, one of the three routes leading to PE synthesis in eukaryotes, while PS synthesis has not b

279 Together, PE leads to impaired eCB-LTD at the excitatory synapses

280 thochromic shift observed upon transitioning PEs from solution to solid.

281 a precipitous decline of VCF use in trauma, PE rates remained unchanged during this period.

282 Treating PE requires appropriate risk stratification as a first s

283 nd radiological course of persons undergoing PE episodes.

284 ility to blunt PE-mediated vasoconstriction (PE-mediated DeltaFVC: KIR blockade alone: -6 +/- 5%; com

285 he yolk in addition to the blastodisc, while PE and PI species are mostly absent in the yolk.

286 es upregulated across all transcriptome-wide PE analyses to date (microarray, RNA-Seq and PAS-Seq) ar

287 With PE exclusively in the opposite leaflet, resolution of po

288 With PE in the leaflet facing the DCVs, overall fusion was mo

289 Mortality associated with PE hospitalizations is declining, regardless of IVCF use

290 alternative polyadenylation associated with PE, the EO-PE and LO-PE cohorts do exhibit gene expressi

291 These effects combined with PE-induced anti-Hebbian long-term potentiation reported

292 FA of eyes with PE showed leakage of the optic nerve, whereas eyes with

293 Five children (10 eyes) with PE, 11 children (19 eyes) with PPE owing to suspected bu

294 Normotensive patients with PE but with indicia of right heart dysfunction (by bioma

295 Some intermediate-risk patients with PE may require urgent stabilization, and approximately 1

296 brane becomes ruffled, as was also seen with PE STBEVs.

297 ne of isolated VSMCs, whereas treatment with PE (10(-5) m) or sodium nitroprusside (10(-5) m) induced

298 Treatment with PE (10(-5) m) or sodium nitroprusside (10(-5) m) induced

299 rdiovascular disease reported for women with PE later in life.

300 Without PE methylation, particular sites on histones then become