ライフサイエンスコーパス: PFCP

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します

1 PFCP showed an excellent solubility and possessed interf

2 of distinction between polycythemia vera and PFCP.

3 The peptidic fraction of carotenoproteins (PFCP) contained 80.8 +/- 0.21% protein, 2.74 +/- 0.3% li

4 s of internalization in truncated EpoRs from PFCP patients contribute to Epo hypersensitivity and pro

5 Mutated EpoRs mimicking those from PFCP patients cannot bind p85, co-localize with epsin-1,

6 ignaling and erythroid hyperproliferation in PFCP.

7 milarly to previously described mutations in PFCP subjects.

8 The antioxidant activities of PFCP at different concentrations were evaluated using va

9 The antioxidant activity of PFCP, based on their protection of supercoiled DNA stran

10 ts were examined to confirm the diagnosis of PFCP.

11 ls were hypersensitive to EPO (a hallmark of PFCP) suggesting the presence of the disease at the leve

12 amily with autosomal dominant inheritance of PFCP in which four subjects were affected in three gener

13 The amino acid profile of PFCP showed a high percentage of essential amino acids,

14 rogenitors from that seen in this particular PFCP family.

15 rimary familial and congenital polycythemia (PFCP or familial erythrocytosis) is a rare proliferative

16 rimary familial and congenital polycythemia (PFCP) patients lacking the 3 important tyrosines do not

17 rimary familial and congenital polycythemia (PFCP).

18 rimary familial and congenital polycythemia (PFCP).

19 rimary familial and congenital polycythemia (PFCP; also known as familial erythrocytosis) is characte

20 We conclude that PFCP is a disorder that appears to be associated in some

21 Therefore, PFCP had a high nutritional value and could be used as a

22 Restoring p85 binding to PFCP receptors rescues Epo-induced epsin-1 co-localizati

23 EPOR mutations (6 out of 8) associated with PFCP result in an absence of the C-terminal negative reg

24 xamined the segregation of the mutation with PFCP in the family and found that a child in the third g

25 (EPOR) mutations were found in subjects with PFCP; most of these mutations resulted in the truncation

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。