ライフサイエンスコーパス: PHA

1 PHA induces accumulation of cyclin T1 mRNA and protein,

2 PHA was calculated using the non-laboratory Framingham C

3 PHA-665752 also exhibited >50-fold selectivity for c-Met

4 PHA-665752 was identified as a small molecule, ATP compe

5 PHA-665752 was identified as a small molecule, ATP-compe

6 PHA-L-labeled fibers were observed in the ipsilateral po

7 PHA-L-labeled trigemino- and spinothalamic (TSTT) termin

8 Systemic pseudohypoaldosteronism type 1 (PHA-1) is a severe salt-losing syndrome caused by loss-o

9 containing a pseudohypoaldosteronism type 1 (PHA-1)-causing missense point mutation.

10 LIN-15B, LIN-39, MAB-5, MDL-1, MEP-1, PES-1, PHA-4, PQM-1, SKN-1, and UNC-130) at diverse development

11 ted, HT of excess sludge with moderate (13%) PHA content can produce about 50 kg of alkenes per tonne

12 -3-yl]furo[2,3-c]pyridine-5-carboxamide (14, PHA-543,613), a novel agonist of the alpha7 neuronal nic

13 script cause pseudohypoaldosteronism type 2 (PHA II), characterized by hypertension and hyperkalemia.

14 The cocrystal structure of 3 (PHA-665752), bound to c-MET kinase domain, revealed a no

15 o three MET kinase inhibitors (JNJ-38877605, PHA-665752, crizotinib) and one antagonistic anti-MET an

16 uenza A virus (Flu) or phytohemagglutinin A (PHA), but had no effect on CD8(+) T cells.

17 Though much is known about PHA synthesis, little is known about inclusion structure

18 When peat humic acid (PHA) was added to a NOM-deficient sediment concentration

19 pment, extinguish PHA-4 and fail to activate PHA-4 target genes.

20 ive sediment but not in sediments with added PHA, suggesting that the native NOM and the PHA respond

21 In addition, PHA-665752 inhibited HGF-stimulated or constitutive phos

22 on alone, RF ablation combined with adjuvant PHA-665752 or semaxanib reduced distant tumor growth, pr

23 ze the thickness and coverage of an adsorbed PHA layer in a natural nanocolloid is also presented.

24 We examined predicted heart/vascular age (PHA) in six LMICs and the United States.

25 ls, but proliferated in response to CMV Ags, PHA, and third party cells.

26 ora kinase inhibitors (AZD1152, MK-0457, and PHA-739358), at low nanomolar concentrations.

27 of pha-4 function, suggesting that PEB-1 and PHA-4 have common functions in some tissues where they a

28 sponses to filarial Ags, nonparasite Ag, and PHA by PBMC compared with the low transmission village (

29 liferation stimulated by dendritic cells and PHA were 3.9 microM and 2.9 microM, respectively, that i

30 cted, other c-MET inhibitors, crizotinib and PHA-665752, suppressed the growth of c-MET-addicted canc

31 d T lymphocytes were stimulated with LPS and PHA, respectively.

32 the anterograde tracers BDA, neurobiotin and PHA-L in the host.

33 cetyl cysteine, as well as by plumericin and PHA-408, inhibitors of the NF-kappaB pathway.

34 10 responses to EBV lytic antigens, PPD, and PHA correlated inversely with malaria exposure regardles

35 id A, Ppg, and PHA; farming mothers: Ppg and PHA), influenced by maternal farming.

36 ation (nonfarming mothers: lipid A, Ppg, and PHA; farming mothers: Ppg and PHA), influenced by matern

37 s of the shared nervous system (URY, PQR and PHA) to produce mate-searching behavior.

38 ainst VGluT2, tyrosine hydroxylase (TH), and PHA-L.

39 reduction, phosphate release and uptake, and PHA dynamics for all systems, suggesting the validity an

40 ar to those observed in Pelger-Huet anomaly (PHA).

41 In two patients, the proper hepatic artery (PHA) was the first branch of the SMA and the gastroduode

42 ssion of G1-S cyclins but often as potent as PHA in inducing RNAP II cyclin/CDK complexes.

43 Typhi-infected autologous PHA-activated PBMC as target cells.

44 converting most (>70% w/w) of the bacterial PHA stored inside microbial cells into alkene/CO2 gas mi

45 EPHA) was defined as the differences between PHA and chronological age >5 years.

46 ed extensive regulatory interactions between PHA-4, SKN-1, and miRNAs and points to two aging-associa

47 um was indicated by the relationship between PHA content and growth capabilities of the genetically m

48 xybutyrate] (PHB), a renewable biodegradable PHA polymer with potential commercial applications in pl

49 is also found in T cells upon activation by PHA.

50 regulators and cell signaling events, and by PHA-1, an essential cytoplasmic protein of unknown funct

51 genes and activation of pharyngeal genes by PHA-4.

52 proliferation and IL-2 production induced by PHA, concanavalin A (conA), and anti-TCR MAb.

53 esizing neurons appeared to be innervated by PHA-L-containing axons.

54 tetraethylammonium), decynium-22, carnitine, PHA (p-aminohippurate), alanine, or inosine.

55 llin 3 (Cul3)--mutations of which all caused PHA-II phenotypes.

56 In contrast, PHA/IL-2, which is widely used to prime cells for HIV in

57 We cultured PHA-activated human T lymphocytes in varying concentrati

58 e expression, mediated by pharynx defective (PHA)-4/FoxA in combination with additional, largely unid

59 examined under a light microscope to detect PHA-L-labeled fibers.

60 d those genes to determine which were direct PHA-4 targets.

61 The dominant PHA-II mutation in KLHL3 impaired claudin-8 binding, ubi

62 lectins that bind Gal, including L-PHA and E-PHA.

63 the PhaC2 protein is not the most efficient PHA polymerase biocatalyst.

64 lity, even though both of these genes encode PHA polymerases that are more efficient enzymes.

65 s established that each gene product encodes PHA polymerase.

66 High excess PHA (HEPHA) was defined as the differences between PHA a

67 subsequently arrest development, extinguish PHA-4 and fail to activate PHA-4 target genes.

68 209 binds the pan-pharyngeal Forkhead factor PHA-4 in vitro and responds to ectopic pha-4 expression

69 gans foregut development, the pioneer factor PHA-4/FoxA binds promoters and recruits RNA polymerase I

70 e forkhead box A (FOXA) transcription factor PHA-4 and that autophagy is required to extend longevity

71 orhabditis elegans, the transcription factor PHA-4 has an essential role in the embryonic development

72 s regulated by the FoxA transcription factor PHA-4.

73 R) pathway and the Foxa transcription factor PHA-4.

74 ed miRNAs connected to transcription factors PHA-4/FOXA and SKN-1/Nrf, which are both necessary for D

75 Decreasing MCL-1 levels with flavopiridol, PHA 767491, or shRNA restored sensitivity to ABT-737 res

76 r the control group (median cpm: 107,049 for PHA, 2,111 for Flu, and 2,068 for VLP).

77 [median counts per minute (cpm): 72,849 for PHA, 1,241 for Flu, and 727 for VLP] than for the contro

78 ewly discovered, adult-specific function for PHA-4 in the regulation of diet-restriction-mediated lon

79 igh homology, but TFO71 has unique genes for PHA synthesis, gene regulation and granule management.

80 the tracer, simultaneous immunolabeling for PHA-L and proTRH peptides was performed and mapped in di

81 more efficient and economical processes for PHA production, isolation, purification and improvement

82 Compared to previous protocols for PHA injection in amphibians, this method induced up to 2

83 ium-chain-length synthase are unable to form PHA(MCL)s when grown in the presence of fatty acids.

84 ma up-regulation in Tempus purified RNA from PHA stimulated cells while only IL2 was up-regulated usi

85 rynx) cells in response to the selector gene PHA-4/FoxA.

86 e TBX-38 and express the organ selector gene PHA-4/FoxA.

87 human ENaC that contained subunits harboring PHA-1-causing substitutions within an absolutely conserv

88 phosphate-accumulating organisms (PAO), have PHA metabolism-related genes with high homology, but TFO

89 characterizations indicate that heteromeric PHA polymerases composed of mixtures of different PhaC p

90 vealed by the occurrence of three homologous PHA polymerase genes (phaC1, phaC2, and phaC3).

91 However, PHAs need to have tunable hydrophilicity, chemical funct

92 romosomes (and human orthologs) PHA1 (HSA1), PHA 2 (HSA3), PHA4 (HSA6), PHA11 (HSA12), PHA13 (HSA2),

93 13 (HSA2), PHA16 (HSA17), and PHA17 (HSA13) (PHA, P. hamadryas; HSA, Homo sapiens).

94 tations confirm the molecular basis of human PHA and provide a small animal model for determination o

95 We recently found that human PHA is caused by mutations in the gene (LBR) encoding la

96 produce fluorinated poly(hydroxyalkanoate) (PHA) bioplastics with fluorine substitutions ranging fro

97 n syndrome--pseudohypoaldosteronism type II (PHA-II)--features hypertension, hyperkalemia, and metabo

98 KS-WNK1 (kidney-specific, KS) is altered in PHA II is not known.

99 ted whole PBMC lysate by Western blot and in PHA-activated CD56 and CD19 subsets by immunohistochemis

100 MDA-7/IL-24 was detected in PHA- and LPS-stimulated whole PBMC lysate by Western blo

101 PRP4 is expressed in PHA-stimulated human T lymphocytes from days 1 and 7 wit

102 Hyperkalemia in PHA II patients with PHA II mutations may be caused, at

103 , and interferon-gamma (IFN-gamma) levels in PHA-stimulated peripheral blood mononuclear cell superna

104 xplanation for the collecting duct's role in PHA-II.

105 Voltage sensitivity in PHA-1 mutants stems from the disruption of critical stru

106 roscopy determined the presence of VGluT2 in PHA-L- or WGA-positive terminals.

107 activity analyses suggest a role for yfcX in PHA monomer unit formation in recombinant E. coli fadB m

108 Pb exposure increased PHA response in both seasons, and decreased T-independen

109 -6 knockout mice (n = 24) or c-met inhibitor PHA 665752 (n = 15), to elucidate the key factors facili

110 e (AMPPNP) or the pyrrolo-pyrazole inhibitor PHA-680626 at 2.4 and 2.1 A resolution, respectively.

111 arily susceptible to the selective inhibitor PHA-665752.

112 was combined with either a c-Met inhibitor (PHA-665752) or VEGF receptor inhibitor (semaxanib) and c

113 line, SNU638, and two related MET inhibitors PHA-665752 and PF-2341066.

114 Specifically, transcripts for several known PHA-inducible genes, including IFNgamma, IL13, IL2, IL3,

115 l transformations were observed: at 255 mg/L PHA aggregation of the nanocolloid was actually enhanced

116 lloid was actually enhanced, but at 380 mg/L PHA disaggregation and colloidal stability were promoted

117 antification of p24 in phytohemagglutinin-L (PHA)-stimulated CD4(+) T cells from individuals under ef

118 of other lectins that bind Gal, including L-PHA and E-PHA.

119 of activated T cells by Concanavalin A or L-PHA was also reduced in Fng tKO mice.

120 labeled tissue, we found that double-labeled PHA-L (+)/VGluT2 (+) axon terminals formed synaptic cont

121 ated by a non-LPS stimulus, the plant lectin PHA.

122 ted with Phaseolus vulgaris leucoagglutinin (PHA-L) and labeled dextrans.

123 l tracer Phaseolus vulgaris leucoagglutinin (PHA-L) and performed double-label immunofluorescence wit

124 e tracer Phaseolus vulgaris leucoagglutinin (PHA-L) and the retrograde tracer FluoroGold in specific

125 beled by Phaseolus vulgaris leucoagglutinin (PHA-L) injections, whereas tyrosine hydroxylase (TH) was

126 ction of Phaseolus vulgaris leucoagglutinin (PHA-L) or an adeno-associated virus encoding wheat germ

127 e tracer Phaseolus vulgaris leucoagglutinin (PHA-L) was injected into one inferior colliculus of 10 a

128 First, Phaseolus vulgaris-leucoagglutinin (PHA-L) was injected in the ventrolateral PAG in Sprague-

129 BDA) and Phaseolus vulgaris-leucoagglutinin (PHA-L), into four subdivisions of OFC.

130 tracers Phaseolus vulgaris-leucoagglutinin (PHA-L; for outputs) and cholera toxin B subunit (CTB; fo

131 nsport of Phaseolus vulgaris leucoagglutinin(PHA-L) or carbocyanine dyes, we characterize the POm tha

132 odium loss, in the ENaC-mediated salt-losing PHA-1 phenotype.

133 mcl-PHAs were then isolated from the cells and demonstrated

134 ies to conventional carbohydrate-derived mcl-PHAs, which have applications as bioplastics.

135 dium chain-length polyhydroxyalkanoates (mcl-PHAs).

136 further demonstration of their utility, mcl-PHAs were catalytically converted to both chemical precu

137 kept on a regular-salt diet, thus mimicking PHA-1.

138 Given the conservation of miRNAs, PHA-4, and SKN-1 across phylogeny, these interactions ar

139 rt that treatment of PBLs with two mitogens, PHA and PMA, results in accumulation of cyclin T1 via di

140 tronger responses to ConA (373+/-174 ng/mL), PHA (498+/-196 ng/mL) and EBV (152+/-179 ng/mL), when co

141 VL or to HVL patients (ConA 185+/-114 ng/mL, PHA 318+/-173 ng/mL, and EBV 33+/-42 ng/mL).

142 te to feed aerobic bacteria and produce more PHA.

143 o stimuli that mimic TCR activation, namely, PHA and PMA.

144 Simultaneously, most of non-PHA biomass was converted into water-soluble compounds (

145 enzenes) and higher order products observed (PHA, PHQ, and LMW oxo- and dicarboxylic acids).

146 Approximately 95% of PHA-L-labeled terminals from the central lateral, midlin

147 Furthermore, the relative affinity of PHA-4 for different TRTTKRY (R = A/G, K = T/G, Y = T/C)

148 The accumulation of significant amount of PHA inside aerobic microbial cells occurs when a surplus

149 ion of recombinant human TL1A to cultures of PHA-stimulated lamina propria mononuclear from CD patien

150 In in vivo studies, a single dose of PHA-665752 inhibited c-Met phosphorylation in tumor xeno

151 nt with the predicted immunologic effects of PHA stimulation.

152 n and, most recently, protein engineering of PHA biosynthetic enzymes.

153 hromatin opening, is an important feature of PHA-4 pioneer factor activity.

154 Herein, hydrothermal treatment (HT) of PHA-containing sludge at 300 and 375 degrees C was demon

155 etabolic alkalosis, an exact mirror image of PHA-II.

156 , isolation, purification and improvement of PHA material properties.

157 rial antigens, and resulted in inhibition of PHA-induced proliferation.

158 Swelling induced by single injection of PHA or killed bacteria was not significantly affected by

159 swelling caused by a secondary injection of PHA, was significantly reduced by B. dendrobatidis super

160 Stereotaxic injections of PHA-L were targeted to the mid-dorsal and mid-ventral po

161 Our current knowledge of PHA production and utilization in vitro and in vivo as w

162 ctural analysis reveals that the majority of PHA neurons contain glutamate.

163 ctivity was detected in the vast majority of PHA-L- or WGA-positive terminals forming asymmetric syna

164 To determine if the major surface protein of PHA inclusions, PhaP, is involved in the structure of th

165 Finally, the physiological role of PHA accumulation in enhancing the fitness of R. rubrum w

166 The results indicated a substantial role of PHA consumption in N2O accumulation due to the relativel

167 to describe N2O dynamics and the key role of PHA consumption on N2O accumulation during the denitrify

168 The role of PHA-4 in lifespan determination is specific for dietary

169 In this work, the selection of PHA storing bacteria was integrated with the side stream

170 ine conditions accomplished the selection of PHA storing biomass and nitrogen removal via nitrite.

171 The results showed that the selection of PHA storing biomass was successful in both configuration

172 cent progress in the synthetic strategies of PHA-based water soluble polymers, including the function

173 ) was employed to elucidate the structure of PHA inclusions at the nanoscale level, including the cha

174 inobutyric acid are also found in subsets of PHA neurons, and fibers immunoreactive for these substan

175 Suppression of PHA-induced T-cell proliferation by dexamethasone was as

176 The use of PHA may offer a useful avenue to communicate CVD risk.

177 oups and the block/graft copolymerization of PHAs with hydrophilic components in various polymeric ar

178 polymers, including the functionalisation of PHAs with polar functional groups and the block/graft co

179 The versatility of PHAs has made them good candidates for the study of thei

180 in accumulated PhaP in a manner dependent on PHA production, and the phaC deletion strain accumulated

181 nd the regulation of these miRNAs depends on PHA-4 and SKN-1.

182 polyphosphate and four-step anoxic growth on PHA using nitrate, nitrite, nitric oxide (NO), and N2O c

183 sistant 1.89 vs. sensitive 0.96, P =0.02) or PHA (1.66 vs. 0.96, respectively, P =0.02).

184 DTH responses induced by killed bacteria or PHA in the presence of B. dendrobatidis supernatants.

185 duction in response to lipopolysaccharide or PHA was lower in the salmon group (all P </= 0.045).

186 eduction in proliferation with either PPD or PHA at 2 hours compared with 0 hours.

187 ulated with lipid A, peptidoglycan (Ppg), or PHA.

188 ter stimulation with lipid A, peptidoglycan, PHA, house dust mite (Der p 1), or Der p 1 plus lipid A.

189 In combination with phytohaemagglutin (PHA) lymphocyte-conditioned medium, the number and size

190 out prior incubation in phytohaemagglutinin (PHA) - a process commonly used in immune population expe

191 Phytohemagglutinin (PHA), purified protein derivative (PPD), and T cell epit

192 ein derivative (PPD) and phytohemagglutinin (PHA).

193 oncanavalin A (ConA) and phytohemagglutinin (PHA; 190+/-86 ng/mL, 328+/-163 ng/mL) and detectable EBV

194 eptide loaded autologous phytohemagglutinin (PHA) blasts.

195 3/4 and NS5) and control phytohemagglutinin (PHA) was monitored prospectively and was correlated with

196 MMG eliminated phytohemagglutinin (PHA)-stimulated proliferation of PBMCs.

197 In phytohemagglutinin (PHA)-stimulated CD8-depleted peripheral blood mononuclea

198 MV-Edm was restricted in phytohemagglutinin (PHA)-stimulated peripheral blood lymphocytes (PBLs) but

199 ature dendritic cells or phytohemagglutinin (PHA) but did not induce apoptosis.

200 concanavalin A (ConA) or phytohemagglutinin (PHA) stimulated canine peripheral blood mononuclear cell

201 on of killed bacteria or phytohemagglutinin (PHA).

202 4 (TLR4), and suppressed phytohemagglutinin (PHA)-mediated proliferation of normal human T lymphocyte

203 tor-alpha in response to phytohemagglutinin (PHA) and of IL-2 in response to Dermatophagoides pterony

204 oliferative responses to phytohemagglutinin (PHA), influenza virus (Flu), and HPV16 virus-like partic

205 ne or IL-2 combined with phytohemagglutinin (PHA) in CD8-depleted PBMCs.

206 Plant lectins such as phytohemagluttinin (PHA) can activate the T cell receptor (TCR) and other ce

207 responsible for mediating TNF-alpha and PMA/PHA-induced expression.

208 regulation of the MIP-1alpha promoter by PMA/PHA stimulation in Jurkat T-cells.

209 the proximal RUNX site is essential for PMA/PHA-stimulated activation of the MIP-1alpha promoter.

210 zed polyacrylamide, polyhydroxamicalkanoate (PHA), which mimics the performance of the acetylcholines

211 Polyhydroxyalkanoate (PHA) inclusions are polymeric storage inclusions formed

212 uptake, glycolysis and polyhydroxyalkanoate (PHA) synthesis were conserved in all these Accumulibacte

213 dation of glycogen and polyhydroxyalkanoate (PHA), which function as energy and carbon storage compou

214 M effects on bacterial polyhydroxyalkanoate (PHA), specifically polyhydroxybutyrate (PHB), biosynthes

215 torage polymers (e.g., Polyhydroxyalkanoate (PHA)) could be produced and consumed dynamically.

216 or medium-chain-length polyhydroxyalkanoate (PHA(MCL)) formation from fatty acids in an E. coli fadB

217 o produce a variety of polyhydroxyalkanoate (PHA) biopolymers with desirable structures and material

218 review then focuses on polyhydroxyalkanoate (PHA) synthases that generate polyoxoesters.

219 Polyhydroxyalkanoates (PHA) are a key constituent of excess sludge produced by

220 Polyhydroxyalkanoates (PHAs) are excellent candidate biomaterials due to their

221 Polyhydroxyalkanoates (PHAs) are polyoxoesters that are produced by diverse bac

222 Polyhydroxyalkanoates (PHAs) from activated sludge and renewable organic materi

223 Production of polyhydroxyalkanoates (PHAs) has been investigated for more than eighty years b

224 n the biosynthesis of polyhydroxyalkanoates (PHAs) in the Rhodospirillum rubrum genome revealed by th

225 t-based production of polyhydroxyalkanoates (PHAs), silk, elastin, collagen, and cyanophycin with an

226 cellular free iron and polyhydroxylakanoate (PHA), a bacterial energy storage polymer.

227 death in a significant population of primary PHA-activated T cells (72%) and lymphoid tumor cell line

228 r cells, using flow cytometry, without prior PHA treatment.

229 C2 contributes the major capacity to produce PHA, even though the PhaC2 protein is not the most effic

230 The properties associated with the produced PHA suggest that they are suitable for thermoplastic pro

231 accumulating in cells that are not producing PHA.

232 les of PhaR in regulating PhaP and promoting PHA production are presented.

233 Pseudohypoaldosteronism (PHA) types I and II are curious genetic disorders that s

234 gulated genes were related to N2O reduction, PHA synthesis and acetyl-CoA formation.

235 er transcription factor and master regulator PHA-4/FoxA, followed by the cytoskeletal regulator and k

236 gulated by miR-228, whereas miR-71 represses PHA-4.

237 gh a dietary restriction mechanism requiring PHA-4.

238 f mono- and polyhydroxylated aromatic rings (PHA) and chromophoric mono- and polyhydroxylated quinone

239 escue diet reinstated the symptoms of severe PHA-1 syndrome and significantly reduced NCC phosphoryla

240 In addition, water soluble PHA monomer production will be briefly introduced, with

241 These chemically modified water soluble PHAs have significant impact on materials engineering an

242 The applications of water soluble PHAs in controlled drug release, cancer therapy, DNA/siR

243 roposed biomimetic biosensor, denoted as SPE/PHA/mPEG, represents a significant advance in the field,

244 At a later developmental stage, PHA-4 promotes chromatin opening.

245 an be highly induced by the T cell-stimulant PHA, suggesting it is a particularly important TNF-alpha

246 In cellular studies, PHA-665752 potently inhibited HGF-stimulated and constit

247 tive CD4 T cell lines, as well as short-term PHA-activated CD4 T cells, can express NKG2C, the activa

248 We studied induced immunity by testing PHA and humoral responses.

249 We also found that PHA and anti-CD3 Abs induce the expression of both the c

250 We found that PHA-L- or WGA-positive terminals from tagged VTA cells m

251 henotypic evidence to support the model that PHA-1, a novel protein, and UBC-18, a ubiquitin-conjugat

252 In turn, we show that PHA-4 and SKN-1 are negatively regulated by miR-228, whe

253 We show that PHA-4 directly activates mRNA expression of a broad coho

254 Our data suggest that PHA-4 and DAF-12 endow the pharynx with transcriptional

255 Our data suggest that PHA-4 directly activates most or all pharyngeal genes.

256 ic pha-4 expression in vivo, suggesting that PHA-4 directly initiates ceh-22 expression through de209

257 The PHA bears functional groups inserted along its backbone

258 The PHA selection degree was evaluated by the volatile fatty

259 The PHA turnovers play important roles in nitrous oxide (N2O

260 based interfering species did not affect the PHA performance, which endorsed its superior behavior.

261 atty acid (VFA) uptake rate (-qVFAs) and the PHA production rate (qPHA), which were 239 +/- 74 and 89

262 PHA, suggesting that the native NOM and the PHA respond differently to changes in ionic strength.

263 expression was approximately 40% of both the PHA-stimulated CD4+ and CD8+ T cell subsets, and virtual

264 L-7, as compared with those activated by the PHA/IL-2 treatment.

265 n previously reported, and in two cases, the PHA arose from the SMA.

266 gsae comparison, CompareProspector found the PHA-4 motif and the UNC-86 motif.

267 nd phaC1, the PHA polymerase situated in the PHA biosynthetic operon, plays a minor role in this capa

268 The monomers in the PHA polymer produced by these strains establish that Pha

269 strains that express different levels of the PHA polymer.

270 -37/ZTF-12 as an additional component of the PHA-1 network regulating pharyngeal development.

271 tributor to PHA productivity, and phaC1, the PHA polymerase situated in the PHA biosynthetic operon,

272 trograde tracing study demonstrates that the PHA receives input from multiple, diverse neuron populat

273 Experimental immunostimulation using the PHA (phytohaemagglutinin assay) challenge technique did,

274 te occurred in the same reactor in which the PHA selection process occurred, while in configuration 2

275 Thereby, PHA was immobilized on screen printed electrodes (SPE) t

276 st, phaC3 is an insignificant contributor to PHA productivity, and phaC1, the PHA polymerase situated

277 of testing were as follows: proliferation to PHA, 77%; maternal engraftment, 35%; and genotype, 79% (

278 n mutants were characterized with respect to PHA production and growth capabilities on acetate or hex

279 trating CD4(+)CD25(-) T cells in response to PHA stimulation.

280 b mesylate, they proliferated in response to PHA, demonstrating that inhibition is reversible.

281 uL) also had markedly decreased responses to PHA (typical SCIDs).

282 diversity, and 3) proliferative responses to PHA and streptococcal superantigen, streptococcal pyroge

283 In vitro responses to PHA were recorded in 88 patients, of whom 68 had a genet

284 ns of flow cytometry), in vitro responses to PHA, and TREC levels, all measured at presentation, were

285 In contrast, IFN- gamma responses to PHA, PPD, and Plasmodium falciparum MSP-1 peptides did n

286 us present with a blood phenotype similar to PHA, and develop other phenotypic abnormalities, includi

287 e relatively low N2O reduction rate by using PHA during denitrifying phosphorus removal.

288 HIV-1 viral infectivity both in vitro using PHA plus IL-2 activated PBL and in vivo using the human

289 similarly to vincristine treatment, whereas PHA-665752 or crizotinib treatment markedly induced G(0)

290 maximal proliferative response compared with PHA-activated stationary cultures.

291 ddition, the voltage dependence of ENaC with PHA-1 substitutions is akin to that which results from s

292 at de209 also binds factors functioning with PHA-4 to specifically activate ceh-22 expression in phar

293 We propose that PEB-1 functions with PHA-4 to activate target gene expression in cells in whi

294 By interacting with PHA-4 and SKN-1, miRNAs transduce the effect of dietary-

295 Stimulation of human T lymphocytes with PHA resulted in a strong downregulation of 5-LO mRNA exp

296 Hyperkalemia in PHA II patients with PHA II mutations may be caused, at least partially, by i

297 V-1 expression, PBMC were prestimulated with PHA, and then CD4+ T cells were purified, pretreated wit

298 n head kidney leukocytes by stimulation with PHA or poly(I:C) and in kidney and spleen of fish inject

299 Treatment with PHA-665752 triggers massive apoptosis in 5 of 5 gastric

300 D14(+)/CD16(-)) compared to controls without PHA.