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   1                                              PHI and VIP inhibited proliferation at concentrations as
     2                                              PHI could change according to the application time, beca
     3                                              PHI manifested with typical ARS in 202 (70%) and with at
     4                                              PHI scores were measured in men undergoing re-biopsy wit
     5                                              PHI-base is available at: http://www.phi-base.org/.     
     6                                              PHI-base is the first on-line resource devoted to the id
     7                                              PHI-base is therefore an invaluable resource for the dis
     8                                              PHI-base phenotypes were mapped via their associated gen
     9                                              PHI-BLAST is applied to the analysis of CED4-like cell d
    10                                              PHI-BLAST searches a protein database for other instance
  
    12     Samples from 14 healthy donors (HDs), 40 PHI patients, 17 Chronics, and 13 Elite controllers (ECs
    13 C-PS and (12)C-PS were also analyzed using a PHI TRIFT I time-of-flight mass spectrometer, with 15-ke
  
  
  
  
  
  
  
    21 se efforts, the pattern-hit initiated BLAST (PHI-BLAST) program described here takes as input both a 
  
  
    24  discovery of a new inhibitor protein called PHI-1 that is specific for type-1 protein phosphatase (P
    25 ll lines, and a second larger protein called PHI-2 was present in different muscles, especially cardi
  
  
    28 that contain radiologic images often contain PHI, and this may violate laws, including the U.S. Healt
  
  
  
  
  
    34     The Pathogen-Host Interactions database (PHI-base) catalogues experimentally verified pathogenici
    35     The pathogen-host interactions database (PHI-base) is available at www.phi-base.org PHI-base cont
  
  
  
    39 reservoir in patients initiating cART during PHI and to assess the impact of the timing of cART initi
    40 -lymphocyte (CTL) responses developed during PHI in comparison to CTL responses in chronic infection 
  
  
    43 ths of high-dose zidovudine initiated during PHI results in higher CD4 cell counts and lower PBMC cul
  
    45 ntacts may overestimate transmissions during PHI for real, dynamic sexual partnerships with varying (
  
    47 ion of gp41-induced membrane fusion by early PHI conformation and fusion arrest after folding to the 
    48 P may therefore be associated with the early PHI conformation and FP withdrawal with the final hairpi
  
    50 between level of cognitive decline following PHI and the possession of certain genetic markers that h
  
    52 at integrates phenotypic data for genes from PHI-base, an expertly curated catalog of genes with expe
    53  very low concentrations of glucocorticoids, PHI-induced stimulation of BFU-E progenitors thus repres
  
  
    56 roscopy and photothermal heterodyne imaging (PHI) show that the spatial distribution of the HS-bindin
  
  
  
  
  
    62 c MRI (mpMRI) and the Prostate Health Index (PHI) have shown promise in predicting a positive biopsy 
  
    64 an immunodeficiency virus (HIV)-1 infection (PHI) to receive zidovudine, 1000 mg daily, or placebo fo
    65 y infected cells in primary HIV-1 infection (PHI) would predict clinical progression and viral replic
  
  
  
  
    70 itiating HAART during primary HIV infection (PHI) or chronic HIV infection (CHI) using flow cytometry
    71 jects enrolled during primary HIV infection (PHI), 10 chronically infected subjects (chronic), and 7 
    72 ing highly infectious primary HIV infection (PHI), we developed a stochastic individual-based model t
  
    74 uman immunodeficiency virus (HIV) infection (PHI) yields a larger decrease in cell-associated HIV-DNA
    75 ly infected patients (primary HIV infection [PHI] group) who started therapy within 6 months of the o
  
    77 ctivation by a prolyl hydroxylase inhibitor (PHI) synergizes with glucocorticoids and enhances produc
    78  exposure to a small molecule PHD inhibitor (PHI) (2-(1-chloro-4-hydroxyisoquinoline-3-carboxamido) a
  
    80   Several HIF-prolyl hydroxylase inhibitors (PHIs) induced erythropoietin (epo) expression in vitro a
  
    82 s include an early pre-hairpin intermediate (PHI) characterized by extended coiled-coil structure in 
    83 e accessible in the prehairpin intermediate (PHI) state and block the formation of the six-helix bund
    84 refusion state to a prehairpin intermediate (PHI), while the second phase is marked by transition fro
    85 ce of fluopyram at the pre-harvest interval (PHI) of 7-21 d was between 0.0108 and 0.1603 mg/kg, and 
    86 degradation rates, the pre-harvest interval (PHI) values and the half-life values of the three pestic
  
  
    89 sidues dissipated with preharvest intervals (PHIs) of 33.5, 12 and 32 days at recommended dose with n
  
  
  
    93 d the level of peptide histidine isoleucine (PHI), a peptide coded for by the same mRNA that encodes 
    94 peptide (VIP), peptide histidine isoleucine (PHI), and pituitary adenylate cyclase-activating peptide
  
    96 ly after removal of plasma IgA from 12-month PHI samples: the magnitude of ADCC not only increased af
  
    98 sion, secretion, and distribution of AMF/NLK/PHI/MF in neoplastic and their normal counterpart cells.
    99 ochemical visualization has depicted AMF/NLK/PHI/MF to be localized into tubular-like vesicles, diffu
  
  
   102 80% of PHI enrollment samples and in 100% of PHI 12-month, chronic, and EC samples; it peaked after a
  
  
  
  
   107 31-45 to 15-23 events; a shorter duration of PHI and/or a lower testing frequency reduces the number 
   108 f the high infectivity but short duration of PHI, even short-term behavior change can significantly r
  
   110 ter HIV transmission, though a proportion of PHI patients demonstrate relatively reduced CSF HIV RNA 
  
  
  
   114  (PHI-base) is available at www.phi-base.org PHI-base contains expertly curated molecular and biologi
   115 vels of CSF inflammation lower than in other PHI participants but higher than in HIV-seronegative con
   116 onstrate potent regulatory actions of PACAP, PHI, and VIP on neuroblastoma cell proliferation which a
   117 ding experiments using 125I-labeled PACAP27, PHI, and VIP, demonstrated the presence of PACAP-preferr
  
  
   120 ized a NiV heptad repeat peptide to quantify PHI formation and the half-lives (t(1/2)) of the first a
   121 largely due to an enhanced dynamic response (PHId), and a more than twofold improvement in glucose to
  
  
   124 ring the responses of patients treated since PHI and showing a similar virological and immunological 
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
   143 ST search function is provided and a link to PHI-Canto, a tool for authors to directly curate their o
   144 psis, is structurally related to the tobacco PHI-1 gene, which is re-activated in cultured cells foll
  
  
  
  
  
  
  
  
   153 ators should know the conditions under which PHI may be accessed for research purposes (ie, with auth
  
   155 id (CSF) and blood from 96 participants with PHI and compared them with samples from neuroasymptomati
   156 ccurred in a large fraction of patients with PHI and were associated with substantial morbidity.     
   157 ression procedures, we found that those with PHI demonstrated a greater degree of cognitive decline o
   158  decline does occur in Vietnam veterans with PHI and it is apparently unrelated to dementia and is de
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