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1 PHN occurred in 26 vaccinated men (6.0%) versus 25 unvac
2 PHN-associated VZV significantly increased sodium curren
3 PHN-associated VZV sodium current increases were therefo
6 s clear whether the vaccine protects against PHN among patients who develop HZ despite previous vacci
10 , physicians who strongly agreed that HZ and PHN cause significant burden were more likely to recomme
16 Vaccine efficacy against incident zoster and PHN has been demonstrated in clinical trials, but effect
18 dence rates and hazard ratios for zoster and PHN were determined in vaccinated and unvaccinated indiv
21 famciclovir and placebo groups who developed PHN; the impact of famciclovir treatment on the duration
22 the cinchona alkaloid-derived catalyst DHQD-PHN was clarified by catalyst conformation studies with
23 stereochemical model to rationalize how DHQD-PHN differentiates the two enantiotopic carbonyl groups
24 ree cinchona alkaloid catalysts, namely DHQD-PHN, DHQD-MEQ, and DHQD-CLB, based on calculations of ou
25 Thirty vaccinated women (4.2%) experienced PHN, compared with 75 unvaccinated women (10.4%), with a
27 as higher and better preserved over time for PHN and HZ-associated hospitalizations than for communit
29 tive in scenarios where PHN risk was higher, PHN duration longer, or antiviral shortening of PHN grea
35 CI) causes progressive hemorrhagic necrosis (PHN), a poorly understood pathological process character
37 nous nephropathy (passive Heymann nephritis (PHN)), complement C5b-9-induced proteinuria was associat
42 rrent treatments for postherpetic neuralgia (PHN) have led to the investigation of localised, non-sys
45 vaccination reduces postherpetic neuralgia (PHN) risk by reducing herpes zoster (HZ) occurrence, it
48 llness, incidence of postherpetic neuralgia (PHN), and incidence of HZ were assessed for the STPS pop
49 rpes zoster (HZ) and postherpetic neuralgia (PHN), intentions for recommending the HZ vaccine, and pe
50 litating pain called postherpetic neuralgia (PHN), which can last for months after the disappearance
51 painful condition of postherpetic neuralgia (PHN), which has been difficult to treat because the unde
52 t on the duration of postherpetic neuralgia (PHN), which was defined as pain persisting after rash he
56 group 1), zoster and postherpetic neuralgia (PHN; group 2), or no history of zoster (group 3) reveale
59 tude in the cell line when compared with non-PHN VZV, wild-type (Dumas) or vaccine VZV strains ((POka
63 decline of total nephrin on days 4 and 7 of PHN as well as a reduction in the actin-associated fract
64 y endothelium are critical to development of PHN and constitute a major target for therapy in SCI.
65 of famciclovir treatment on the duration of PHN, while controlling for significant covariates; and t
67 50.1%, vaccine efficacy for the incidence of PHN decreased from 66.5% to 60.1%, and vaccine efficacy
71 HZ to prevent PHN, and the intractability of PHN, the advent of the HZ vaccine appears to be a crucia
74 ignificant covariates; and the prevalence of PHN at monthly intervals from 30 to 180 days after enrol
78 imitation (P limitation) or during growth on PHN compared with their rates in the cultures with Pi we
80 imitation with those responding to growth on PHN, one can speculate which proteins are likely involve
85 tate growth in media containing phosphonate (PHN) as the sole P source was examined by two-dimensiona
86 ficulty of adequately treating HZ to prevent PHN, and the intractability of PHN, the advent of the HZ
92 NPY-evoked cardiovascular responses from the PHN by determining the rank order of potency of several
94 Y(1) receptor antagonist BIBP 3226 into the PHN prior to NPY completely blocked the cardiovascular r
97 considered cost effective in scenarios where PHN risk was higher, PHN duration longer, or antiviral s
98 nfection in vitro study to determine whether PHN-associated VZV isolates induce changes in sodium ion
102 ts with a history of zoster, with or without PHN (21 [67%] of 32 subjects in groups 1 and 2), than in
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