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1 13.4%) of 134 for FluA, 25 (64.1%) of 39 for PIV1, 8 (88.9%) of 9 for PIV2, 17 (30.1%) of 55 for PIV3
4 ndicates that comprehensive understanding of PIV1 evolution requires consideration of both anagenetic
5 ituting the full-length HN and F proteins of PIV1 for those of PIV3 in the attenuated cp45 PIV3 vacci
6 s are smaller for PIV1s from humans than for PIV1 from a chimpanzee for the pol, gag, and env glycopr
7 virus, designated rPIV3-1, that encodes the PIV1 HN and F glycoproteins in the background of the wt
8 ant wild-type (wt) PIV3 as the recipient for PIV1 HN and F, engineered so that each PIV1 open reading
10 the L polymerase protein; and (iii) a murine PIV1 (MPIV1) attenuated by a mutation in the accessory C
11 t for PIV1 HN and F, engineered so that each PIV1 open reading frame is flanked by the existing PIV3
15 umovirus (HMPV), parainfluenza virus 1 to 3 (PIV1, PIV2, and PIV3), and adenovirus (AdV) infections.
16 luA), parainfluenza virus types 1, 2, and 3 (PIV1, PIV2, and PIV3), human metapneumovirus (MPV), and
17 ossible to rapidly develop a live attenuated PIV1 vaccine by the staged introduction of known, charac
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