ライフサイエンスコーパス: PLA

1 PLA analysis of neurons in vitro and biochemical analysi

2 PLA generally has the lowest emissions when compared to

3 PLA(2) activity was found in conditioned media from both

4 PLA/emulsion interactions showed minor oil and aroma com

5 Furthermore, the frequency of IL-10+ PLA-specific B cells increased in allergic patients rece

6 among them several known phospholipase A(2) (PLA(2)) inhibitors.

7 The phospholipase A(2) (PLA(2)) superfamily consists of 16 groups and many subgr

8 nd that, among the three phospholipase A(2) (PLA(2))-regulated arachidonic acid (AA) metabolic pathwa

9 t of autotaxin (ATX) and phospholipase A(2) (PLA(2))enzymes.

10 lactic acid (PLA) diblock copolymer (NuBCP-9/PLA-PEG) or PEG-polypropylene glycol-PEG-modified PLA-te

11 G-modified PLA-tetrablock copolymer (NuBCP-9/PLA-PEG-PPG-PEG).

12 ssay for expression of LINC00518/PRAME and a PLA score with data on the predictive values of the info

13 e, through a genetic screen, we identified a PLA(2) Drosophila ortholog that specifically modulates r

14 d shelf life, the emulsions were stored in a PLA pack (tray and lid).

15 ration, and invasion of human EOC cells in a PLA(2)-dependent manner.

16 usually employed to screen phospholipase A (PLA) activities.

17 over that ACT has intrinsic phospholipase A (PLA) activity, and that such activity determines AC tran

18 ding at least 15 different phospholipases A (PLA).

19 nella pneumophila, such as phospholipases A (PLAs) and glycerophospholipid:cholesterol acyltransferas

20 e major bee venom allergen phospholipase A2 (PLA) were isolated from beekeepers who displayed toleran

21 Phospholipase A2 (PLA)-specific B cells were identified using dual-color s

22 We employed the pulsed laser ablation (PLA) technique to exfoliate GQDs from multi-wall carbon

23 almitis secondary to pyogenic liver abscess (PLA) is becoming a globally emerging infectious disease,

24 tatistical difference from poly lactic acid (PLA) and polycaprolactone (PCL).

25 ertain the effectiveness of polylactic acid (PLA) based packaging solution to store red fresh meat du

26 ylene glycol (PEG)-modified polylactic acid (PLA) diblock copolymer (NuBCP-9/PLA-PEG) or PEG-polyprop

27 mug/min), and lactide from polylactic acid (PLA) filaments (ranging from approximately 4 to approxim

28 aerosols, and wood-infused polylactic acid (PLA) filaments generated the smallest amount.

29 s new tracer is composed of polylactic acid (PLA) microspheres into which short strands of synthetic

30 biobased polymer families: polylactic acid (PLA), polyhydroxybutyrate (PHB) and bioethylene-based pl

31 printed from nonconductive polylactic acid (PLA, housing) and conductive polyethylene terephthalate

32 A) is an aryl analogue of poly(lactic acid) (PLA) and a biodegradable analogue of polystyrene.

33 ) of lactide (LA) to form poly(lactic acid) (PLA) at room temperature.

34 kaging materials based on poly(lactic acid) (PLA) due to its eco-friendly nature.

35 Poly(lactic acid) (PLA) is a biodegradable polymer prepared by the catalyze

36 LGA) core surrounded by a poly(lactic acid) (PLA) shell were fabricated via the precision particle fa

37 res were formulated using poly(lactic acid) (PLA) to release brimonidine at a constant rate for 35 da

38 hydrolytically degradable poly(lactic acid) (PLA) vs core-shell block copolymer micelles having PLA c

39 composite films based on poly (lactic acid) (PLA) were prepared by incorporating thymol, as the activ

40 ene terephthalate) (PET), poly(lactic acid) (PLA), and poly(tetrafluoroethylene) (PTFE), are analyzed

41 and the cartridge material (polylactic acid, PLA).

42 tion of nonlamellar lysophospholipids by ACT-PLA activity into the cell membrane would form, likely i

43 Regulation of ACT-PLA activity thus emerges as novel target for therapeuti

44 oreover, we show that elimination of the ACT-PLA activity abrogates ACT toxicity in macrophages, part

45 ecommends participatory learning and action (PLA) in women's groups to improve maternal and newborn h

46 iation at nanomolar concentrations activates PLA activity.

47 time and proportional to the amount of added PLA.

48 e technique for measuring antibodies against PLA(2)R and the relationship between antibody titer and

49 ls specific for the major bee venom allergen PLA isolated from nonallergic beekeepers show increased

50 apamycin treatment showed markedly amplified PLA signals for IGFBP-1 and CSNK-2beta (approximately 18

51 12-lipoxygenase as well as lipoxygenases and PLA(2) largely blocked the DOR- or AA-induced GABA inhib

52 s in the negative ion mode and from PMMA and PLA in the positive ion mode.

53 hat the size of the hydrophobic PCL, PS, and PLA blocks are correlated with the PEG blob size.

54 reased at early reperfusion in both RIPC and PLA.

55 with a pegylated leptin receptor antagonist (PLA) attenuates cachexia in mice with CKD.

56 Here, we measured anti-PLA(2)R (aPLA(2)R) antibody titer and subclass in a well

57 gram to design propargyl-linked antifolates (PLAs) against trimethoprim-resistant dihydrofolate reduc

58 ped a class of propargyl-linked antifolates (PLAs) that exhibit potent inhibition of the enzyme and b

59 e the utility of the pigmented lesion assay (PLA) for LINC00518/PRAME expression in decisions to biop

60 We used a proximity ligation assay (PLA) and determined that the N protein was in close prox

61 flow that combines proximity ligation assay (PLA) and digital PCR.

62 With in situ proximity ligation assay (PLA) and supported by coimmunoprecipitation and colocati

63 tation and in situ proximity ligation assay (PLA) assays in CHO cells revealed the presence of cell-s

64 Proximity Ligation Assay (PLA) demonstrated that HP1a is in close proximity to DNA

65 rane isolation and proximity ligation assay (PLA) demonstrated the translocation of Hrs from the cyto

66 agnetic bead-based proximity ligation assay (PLA) in which one of the assay proximity probes was dire

67 Proximity ligation assay (PLA) indicated proximity between IGFBP-1 and CSNK-2beta

68 tu, we developed a proximity ligation assay (PLA) that combined peptide-modified, multiply-labelled t

69 ion and an in situ proximity ligation assay (PLA) that we developed to localize specific transient RN

70 oach that uses the proximity ligation assay (PLA) to identify the presentation of an MHC class II-res

71 a high-resolution proximity ligation assay (PLA) to monitor the conformation of the BCR and its inte

72 tructural, in situ proximity ligation assay (PLA), and biochemical approaches.

73 py (dSTORM), and a proximity ligation assay (PLA), we provide evidence illustrating that M2-1 is loca

74 We describe a proximity ligation assay (PLA)-based method of assessing association of DNA and RN

75 recently developed proximity-ligation assay (PLA).

76 in situ proximity-dependent ligation assay (PLA).

77 re analyzed using proximity ligation assays (PLA), immunofluorescence, and GTPase and kinase assays.

78 ies using in situ proximity ligation assays (PLA).

79 Akt, as shown by proximity ligation assays (PLAs).

80 01) proved essential for all PlaC-associated PLA, lysophospholipase A, and GCAT activities.

81 5 is highly selective for l-LA, only atactic PLA is obtained in the polymerization of racemic LA.

82 biodegradable polymer, PLA(1k)-b-PEG(10k)-b-PLA(1k).

83 In summary, PEG-b-PLA micelles and PLGA-b-PEG-b-PLGA sol-gels may safely e

84 , preclinical and clinical research on PEG-b-PLA micelles and PLGA-b-PEG-b-PLGA sol-gels that has foc

85 PEG-b-PLA micelles are a first-generation platform for the sys

86 These results suggest that 3-in-1 PEG-b-PLA micelles can concurrently deliver PTX, 17-AAG, and R

87 However, PEG-b-PLA micelles rapidly release PTX, resulting in widesprea

88 Notably, o(LA)8-PTX and o(LA)16-PTX in PEG-b-PLA micelles resisted backbiting chain end scission, bas

89 d, o(LA)n-PTX was more compatible with PEG-b-PLA micelles than PTX, increasing drug loading from 11 t

90 st, when 3-in-1 and single drug-loaded PEG-b-PLA micelles were administrated at modest dose (PTX, 17-

91 To improve delivery of PTX by PEG-b-PLA micelles, monodisperse oligo(l-lactic acid), o(LA)8

92 er weekly IV injections at 20 mg/kg as PEG-b-PLA micelles, o(LA)8-PTX induced tumor regression in A54

93 nd o(LA)n-PTX improves PTX delivery by PEG-b-PLA micelles, providing a strong justification for clini

94 d higher drug loading densities inside PEG-b-PLA micelles.

95 e glycol)-block-poly(D,L-lactic acid) (PEG-b-PLA) micelles and poly(D,L-lactic-co-glycolic acid)-bloc

96 e glycol)-block-poly(d,l-lactic acid) (PEG-b-PLA) micelles are nanocarriers for poorly water-soluble

97 glycol)-block-poly(D, L-lactic acid) (PEG-b-PLA) micelles can serve as a safe delivery platform for

98 ylene glycol)-b-poly(D,L-lactic acid) (PEG-b-PLA) micelles.

99 ell-defined triblock copolymers (PLA-b-PPC-b-PLA).

100 ectively, our results show that Fab'-bearing PLA-PEG NPs are powerful and efficient nanosized tools f

101 the adjusted HRs for endophthalmitis between PLA patients suffering from diabetes and those in whom d

102 To assess interactions between PLA and food, an oil in-water model emulsion was formula

103 igations evaluating the relationship between PLA and endogenous endophthalmitis remain scarce.

104 n, ATK treatment significantly reduced brain PLA(2) activity.

105 ith BrU and testing their close proximity by PLA demonstrates that RNA synthesis in individual cells

106 (see picture; PPC=poly(propylene carbonate); PLA=polylactide).

107 cases of surgically resected WTs in Chinese PLA General Hospital and Beijing Shijitan Hospital of Ca

108 tum, in PD0 striatum we did identify a clear PLA signal for this pair of receptors.

109 cid and that is compatible with its combined PLA(1)/A(2) activity.

110 The influence of solvent composition, PLA conditioning of the cartridge with isopropanol, and

111 cid) poly (ethylene glycol) block copolymer (PLA-PEG), and that grafting of the Fab' portion of the F

112 s produces well-defined triblock copolymers (PLA-b-PPC-b-PLA).

113 (mean GHG savings up to 1.4 kg CO2e/kg corn PLA and 2.9 kg CO2e/kg switchgrass PLA).

114 es of linear PLA (<3%) in a sample of cyclic PLA that was supposedly pure according to other characte

115 (2)beta selective) or pyrrolidine (cytosolic PLA(2)alpha selective), markedly attenuated Ca(2+)-depen

116 findings where we failed to detect a D1R-D2R PLA signal in the adult striatum, in PD0 striatum we did

117 Furthermore, treatment with 7 mg/kg per day PLA attenuated the CKD-associated increase in the transc

118 Treatment with 7 mg/kg per day PLA stimulated appetite and weight gain, improved lean m

119 erization techniques to produce well-defined PLA-PbetaMdeltaVL-PLA triblock polymers, where PLA stand

120 ection of the catalytically inactive deleted PLA(2)IValpha mutant (PLA(2)IValpha(1-525)) and point mu

121 However, different PLA(2) types have unique three-dimensional structures an

122 Understanding how the different PLA(2) enzymes associate with phospholipid membranes, sp

123 This digital PLA method has femtomolar sensitivity, which enables the

124 At these doses, PLA did not influence serum leptin levels in mice.

125 desorbed by the various buffers used during PLA assays.

126 To assess equilibrium effects, PLA was conditioned in vapour contact with the aroma com

127 We examined PLA(2)G4A dependence of the processes of ovulation and f

128 se data unveil a new mechanism of action for PLA(2)IValpha, which demonstrates that the membrane bind

129 ltraviolet (UV) spectrophotometric assay for PLA, we have synthesized a specific glycerophosphatidylc

130 on catalyst in cyclohexane, a nonsolvent for PLA.

131 not the enzymatic activity, is required for PLA(2)IValpha modulation of FcR-mediated phagocytosis.

132 s, as well as assay conditions selective for PLA(2) subtypes, were optimized and validated.

133 to CIT was not significantly different from PLA at week 12 (45.9% vs 37.9%; RR, 1.21; 95% CI, 0.82-1

134 In parallel, fibroblasts from PLA(2)IValpha knock-out mice overexpressing FcgammaRIIA

135 tion, showed a sustained release of PAs from PLA-NPs.

136 and urbanization level, those suffering from PLA were found to have a greater likelihood of developin

137 -loaded polylactic acid-polyethylene glycol (PLA-PEG) nanoparticles with adjustable release rates wit

138 ene-based polylactic acid filament (graphene/PLA) has been 3D printed to fabricate a range of 3D disc

139 s core-shell block copolymer micelles having PLA cores revealed remarkable acceleration in the protei

140 as it may help to understand how homeostatic PLAs are regulated and how degradation and biosynthesis

141 arch could address the mechanisms behind how PLA improves survival, in order to adapt this method to

142 PEO brushes protruding from the hydrophobic PLA cores normal to the interface.

143 size in pigs to 16+/-11% versus 43+/-11% in PLA (% area at risk; mean+/-SD; P<0.05).

144 5+/-5% of ventricular mass versus 38+/-5% in PLA; P<0.05) and activated both RISK and SAFE.

145 ng sorption of additional aroma compounds in PLA.

146 pha C2 domain (which is directly involved in PLA(2)IValpha membrane binding), but not of PLA(2)IValph

147 s muscle of Piemontese beef were packaged in PLA trays closed with a lid made of PLA film and for com

148 s and in mammalian cells; Ca(2+)-independent PLA-beta (iPLAbeta) in particular has been implicated in

149 with both cytosolic and calcium-independent PLA(2) were found in human EOC ascites for the first tim

150 Interestingly, PLA-specific B cells showed increased CCR5 expression af

151 hich may protect the bacterium from internal PLA activity, but enzyme dissociation may allow its acti

152 al-color staining with fluorescently labeled PLA.

153 skin were stabilized with poly-d,l-lactide (PLA) polymer by the emulsion-evaporation method.

154 om a block copolymer of poly (D, L-lactide) (PLA) and monomethoxy polyethylene glycol (mPEG).

155 plied to mixtures of cyclic poly(L-lactide) (PLA) with increasing amounts of its linear topological i

156 selected diluents with a poly(d,l-lactide) (PLA), polydimethylsiloxane (PDMS), or polystyrene (PS) m

157 sensitive enough to detect traces of linear PLA (<3%) in a sample of cyclic PLA that was supposedly

158 Lp-PLA(2) has emerged as a potential therapeutic target in

159 sPLA(2) activity (-29.5% versus -19.2%), Lp-PLA(2) mass (-35.8% versus -6.2%), and Lp-PLA(2) activit

160 ipoprotein-associated phospholipase A(2) (Lp-PLA(2)) associated with high-density lipoprotein (HDL) (

161 ipoprotein-associated phospholipase A(2) (Lp-PLA(2)) is a member of the phospholipase A(2) superfamil

162 ipoprotein-associated phospholipase A(2) (Lp-PLA(2)) is a proinflammatory enzyme bound to low-density

163 ipoprotein-associated phospholipase A(2) (Lp-PLA(2)/PLA2G7) in human inflammation and coronary athero

164 lipoprotein-associated phospholipase A2 (Lp-PLA(2)) are enzyme biomarkers of increased cardiovascula

165 Lp-PLA(2) mass (-35.8% versus -6.2%), and Lp-PLA(2) activity (-24.3% versus 5.4%; P<0.001 for all).

166 sPLA(2) and Lp-PLA(2) mass and activity were measured in 2587 patients

167 Baseline levels of sPLA(2) and Lp-PLA(2) mass and activity were not associated with the pr

168 licated at MS4A4E and TMEM49 for baseline Lp-PLA(2) activity with genome-wide significant joint P val

169 re identified and replicated for baseline Lp-PLA(2) mass at CETP and for Lp-PLA(2) activity at the AP

170 ty acids and lyso-PS species generated by Lp-PLA(2) may represent important signals facilitating and

171 ghting the challenge in using circulating Lp-PLA(2) as a biomarker of Lp-PLA(2) actions in the vascul

172 Circulating Lp-PLA(2) did not increase during acute phase response in h

173 of variation in PLA2G7, the gene encoding Lp-PLA(2), with coronary artery calcification.

174 r baseline Lp-PLA(2) mass at CETP and for Lp-PLA(2) activity at the APOC1-APOE and PLA2G7 loci.

175 s a predictor of cardiac death, while HDL-Lp-PLA(2) is associated with lower risk for cardiac death i

176 In contrast, HDL-Lp-PLA(2) mass and activity were associated with lower risk

177 Total plasma Lp-PLA(2) and HDL-Lp-PLA(2) mass and activity, lipids, and C-reactive protein

178 with high-density lipoprotein (HDL) (HDL-Lp-PLA(2)) in patients with stable coronary artery disease

179 tions with rosuvastatin-induced change in Lp-PLA(2) activity were observed in ABCG2 and LPA, likely b

180 associated with change in statin-induced Lp-PLA(2) activity at genome-wide significance but were sub

181 These data link Lp-PLA(2) to atherosclerosis in humans while highlighting t

182 nd C-reactive protein, blood and monocyte Lp-PLA(2) messenger ribonucleic acid decreased transiently,

183 g circulating Lp-PLA(2) as a biomarker of Lp-PLA(2) actions in the vasculature.

184 novel influences on circulating levels of Lp-PLA(2) activity.

185 We examined inflammatory regulation of Lp-PLA(2) during experimental endotoxemia in humans, probed

186 dotoxemia in humans, probed the source of Lp-PLA(2) in human leukocytes under inflammatory conditions

187 sparse regarding genetic determinants of Lp-PLA(2) mass and activity, and no prior data are availabl

188 The ability of Lp-PLA(2) to hydrolyze peroxidized species of phosphatidyls

189 Two measures of Lp-PLA(2), mass and activity, are associated with increased

190 monocytes, are major leukocyte sources of Lp-PLA(2).

191 Total plasma Lp-PLA(2) and HDL-Lp-PLA(2) mass and activity, lipids, and

192 Total plasma Lp-PLA(2) is a predictor of cardiac death, while HDL-Lp-PLA

193 ic acid decreased transiently, and plasma Lp-PLA(2) mass declined modestly during endotoxemia.

194 Data supporting Lp-PLA(2) are indirect and confounded by species difference

195 We found that Lp-PLA(2) catalyzed the hydrolysis of both nontruncated and

196 In vitro, Lp-PLA(2) expression increased dramatically during human mo

197 onfounded by species differences; whether Lp-PLA(2) is causal in coronary heart disease remains in qu

198 e nucleotide polymorphisms in PLA2G7 with Lp-PLA(2) activity or mass, numerous PLA2G7 single nucleoti

199 EG) or PEG-polypropylene glycol-PEG-modified PLA-tetrablock copolymer (NuBCP-9/PLA-PEG-PPG-PEG).

200 Moreover, PLA demonstrated colocalization between the class II epi

201 ment of SPIONs loaded in the core of an mPEG-PLA micelle coated with cationic polymers provides effic

202 cally inactive deleted PLA(2)IValpha mutant (PLA(2)IValpha(1-525)) and point mutant (PLA(2)IValpha-S2

203 ant (PLA(2)IValpha(1-525)) and point mutant (PLA(2)IValpha-S228C) also promotes recovery of this impa

204 h polylactide-stabilized gold nanoparticles (PLA-AuNPs) and methylene blue (MB) was employed as the r

205 Direct imaging of the resulting nanoporous PLA was obtained by scanning electron microscopy.

206 ughput PLA assay could be used to screen new PLA and/or PLA inhibitors present in various biological

207 s of GPLs by some secretory (nonhomeostatic) PLAs.

208 The noninvasive PLA enables dermatologists to significantly improve biop

209 Using this new route, a series of nonracemic PLA inhibitors was prepared and shown to possess potent

210 lighting the superior qualities of the novel PLA-PEG-PPG-PEG tetrablock copolymer formulation as a to

211 Following the action of PLA at the oil-water interface, alpha-eleostearic acid i

212 A close spatiotemporal association of PLA(2)G4A with PTGS2 was found in mouse and rat preovula

213 The contribution of PLA(2) activities to ovarian tumorigenesis was investiga

214 tate influence the hydrolytic degradation of PLA films.

215 sions were performed to assess the effect of PLA on the hazard of developing endogenous endophthalmit

216 sociation was observed with 4 other forms of PLA(2).

217 tance to better understand the hydrolysis of PLA driven by water molecules either in liquid or in vap

218 Moreover, pharmacological inhibition of PLA(2)IValpha by pyrrophenone reduces particle internali

219 kaged in PLA trays closed with a lid made of PLA film and for comparison purposed in a conventional r

220 The quantitative measurement of PLA(2) activities in ascites and tissues, as well as ass

221 This work focuses on the modifications of PLA induced by water when simulating contact with semi-d

222 PLA(2)IValpha membrane binding), but not of PLA(2)IValpha-D43N (which cannot bind to membranes), res

223 Purification in gas phase of PLA mixtures was established based on SY curves obtained

224 Here, the role of PLA(2)IValpha in Fc receptor (FcR)-mediated phagocytosis

225 om plasma as well as culture supernatants of PLA-specific cells were measured by ELISA.

226 ed with wild-type cells, and transfection of PLA(2)IValpha fully recovers this impaired function.

227 Different molecular weights of PLA were used to form the shell layer for each formulati

228 assay could be used to screen new PLA and/or PLA inhibitors present in various biological samples.

229 Plasma sampled after RIPC or PLA, respectively, was transferred to isolated bioassay

230 CKD and Sham mice received vehicle or PLA (2 or 7 mg/kg per day).

231 ssembly of poly(butadiene)-poly(lactide) (PB-PLA) diblock copolymers followed by selective degradatio

232 es to produce well-defined PLA-PbetaMdeltaVL-PLA triblock polymers, where PLA stands for poly(lactide

233 than both free dendrimers and the empty PEG-PLA NPs.

234 ly(ethylene glycol)-b-poly(D,L-lactide) (PEG-PLA) NPs (~70nm).

235 leen, similar to the in vivo behavior of PEG-PLA NPs.

236 sensitive polymeric delivery system (PLA-PEG-PLA) loaded with chitosan-zinc-insulin complex was desig

237 The A-type phospholipases (PLAs) are key players in glycerophospholipid (GPL) homeo

238 to groups prescribed CIT (n = 101), placebo (PLA; n = 99), CGT with CIT (n = 99), and CGT with PLA (n

239 s hindlimb ischemia/reperfusion) or placebo (PLA) before 60/180 minutes coronary occlusion/reperfusio

240 , propionate (HP), butyrate (HB) or placebo (PLA) were rectally administered during fasting and postp

241 howed increased frequencies of plasmablasts, PLA-specific memory B cells, and IL-10-secreting CD73(-)

242 Polylactide (PLA) is the leading bioderived polymer produced commerci

243 Polylactide (PLA), a biobased polymer, might prove suitable as eco-fr

244 Conventional polyesters such as polylactide (PLA) or its copolymer, polylactide-co-glycolide (PLGA),

245 ush block copolymers containing polylactide (PLA) and poly(ethylene oxide) (PEO) side chains were syn

246 unctionalized polystyrene (PS), polylactide (PLA), or polydimethylsiloxane (PDMS) macromonomer mediat

247 t propylene oxide (PO) to yield polylactide (PLA) terminated by the -OCHMeCH2Cl group.

248 g and therapy) with a biodegradable polymer, PLA(1k)-b-PEG(10k)-b-PLA(1k).

249 r method, an array of monodisperse polymers (PLA(x)-ran-DME(1-x))n bearing variable grafting densitie

250 ysis suggests that women's groups practising PLA improve key behaviours on the pathway to neonatal mo

251 Overall, women's groups practising PLA improved behaviours during and after home deliveries

252 ence factor that exhibits the most prominent PLA activity in L. pneumophila.

253 The phospholipase A(2) receptor (PLA(2)R) is the major target antigen in idiopathic membr

254 the respective plant inhibit the respective PLA, a negative feedback that prevents continuous overex

255 In situ PLA and immunohistochemistry on Nog mutants confirmed in

256 Co-elution, competition, and in situ PLA experiments using full-length and truncated recombin

257 In situ PLA showed that the IDH1R132H epitope colocalizes with M

258 analysis and parameterization of the in situ PLA signals in over 1 million cells cultured on four ind

259 binding by pairs of antibodies using in situ PLA, compared to assays where each antibody preparation

260 ontrol over tacticity to produce stereoblock PLA, from rac-lactide improves thermal properties but is

261 e/kg corn PLA and 2.9 kg CO2e/kg switchgrass PLA).

262 Thermosensitive polymeric delivery system (PLA-PEG-PLA) loaded with chitosan-zinc-insulin complex w

263 cytosis was investigated, demonstrating that PLA(2)IValpha is selectively activated upon FcR-mediated

264 Collectively, our work implies that PLA(2) activity is a potential marker and therapeutic ta

265 Collectively, our data suggest that PLA may be a useful tool to acquire information on wheth

266 Our data strongly suggest that PLA(2)G4A amplifies hCG induction of PTGS2 and colocaliz

267 The PLA activity can be measured continuously by recording t

268 The PLA signals between Mcm10 and HP1a are specifically obse

269 controlled by both PEG chain length and the PLA molecular weight, permitting time-release over susta

270 The addition of thymol decreased the PLA glass transition temperature, as the result of the p

271 four cysteine residues are important for the PLA/GCAT activities as well as their oxidized state, and

272 After incorporating the PLA into their decision as to whether to biopsy a pigmen

273 Three-dimensional rendering of the PLA images validated that IGFBP-1 and CSNK-2beta interac

274 Finally, transfection of the PLA(2)IValpha C2 domain (which is directly involved in P

275 report the synthesis of 12 variations of the PLA-poly(ethylene glycol) (PEG) based precision-polyeste

276 These findings suggest the PLA(2)-AA-12-lipoxygenase pathway as a primary signaling

277 e and also in immature rats treated with the PLA(2)G4A inhibitor, archidonyl trifluoromethyl ketone.

278 This continuous high-throughput PLA assay could be used to screen new PLA and/or PLA inh

279 a true-living nature, which gives access to PLA materials of unprecedented microstructures.

280 though the individual regions contributed to PLA activity, were not essential for protein tetrameriza

281 unbiased small molecule screens and point to PLA(2) as a possible therapeutic target to treat FXTAS.

282 ever, one of the main limitations related to PLA is its reactivity with water.

283 iderations, and application to understanding PLA(2)/membrane interactions are addressed.

284 By using PLA packaging combination it was possible to maintain an

285 fasting free glycerol concentrations versus PLA (P < 0.05).

286 From a practical point of view, PLA packaging is very well suited for semi-dry foods, bu

287 Participants' response to CGT with PLA vs PLA (82.5% vs 54.8%; relative risk [RR], 1.51; 95% CI, 1

288 A-PbetaMdeltaVL-PLA triblock polymers, where PLA stands for poly(lactide).

289 ncreased from 32.1% to 56.9% (P < .001) with PLA data.

290 mitis was found in 106 patients (0.84%) with PLA and 42 comparison patients (0.07%).

291 n = 99), CGT with CIT (n = 99), and CGT with PLA (n = 96).

292 Participants' response to CGT with PLA vs PLA (82.5% vs 54.8%; relative risk [RR], 1.51; 95

293 mprove CGT outcome (CGT with CIT vs CGT with PLA: 83.7% vs 82.5%; RR, 1.01; 95% CI, 0.88-1.17; P = .8

294 added to treatment (CGT with CIT vs CGT with PLA: model-based adjusted mean [standard error] differen

295 ture increased after HA and HP compared with PLA (P < 0.05).

296 was significantly higher among patients with PLA compared with matched controls irrespective of diabe

297 endogenous endophthalmitis in patients with PLA compared with unaffected individuals by using a nati

298 In total, 12 727 patients with PLA were included in the study group and 63 635 matched

299 gmented lesions, first without and then with PLA gene expression information and were asked whether t

300 sensitivity and specificity with vs without PLA data.

301 ipose phospholipase A(2) (AdPLA or Group XVI PLA(2)) plays an important role in the onset of obesity