ライフサイエンスコーパス: PLGF

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1 PLGF significantly increased BBB permeability in NP plas

2 PLGFs elicit diverse biological effects via the activati

3 the C-terminus of placental growth factor-2 (PLGF-2) was selected to enhance the activity of IFNalpha

4 Tumor-derived VEGF-A, PLGF-2, and VEGF-B augment pain sensitivity through sele

5 VEGF165 homodimers and PLGF/VEGF heterodimers stimulate tyrosine phosphorylatio

6 Our findings suggest that VEGF and PLGF enhance BBB permeability through different VEGFR pa

7 nd that circulating sFlt1 prevents VEGF- and PLGF-induced BBB permeability during pregnancy.

8 There was no association between PLGF change and OS in arm A.

9 (SIFalpha) by fusing the positively charged PLGF-2 peptide to the C-terminus of the human IFNalpha.

10 expression of the proinflammatory cytokines PLGF (OMIM 601121), TGFbeta (OMIM 190180), END1 (OMIM +1

11 actor (VEGF)/placenta-derived growth factor (PLGF) and soluble Fms-like tyrosine kinase-1 (sFLT-1, th

12 h factor (VEGF) and placental growth factor (PLGF) are increased in the maternal circulation during p

13 association between placental growth factor (PLGF) change and OS.

14 VEGF receptor-1 and placental growth factor (PLGF) in HEECs.

15 rete high levels of placental growth factor (PLGF), LLC cells produce high levels of VEGF, but low le

16 response to VEGF or placental growth factor (PLGF).

17 xpressed monomers of placenta growth factor (PLGF)129 and vascular endothelial growth factor (VEGF)16

18 rally occurring phospholipid growth factors (PLGFs).

19 (VEGF)165 can be re-folded in vitro to form PLGF/VEGF heterodimers.

20 ion of angiogenic growth factors (VEGF, HGF, PLGF), and SDF-1alpha receptor CXCR4.

21 ccurs in some women with low sFLT-1 and high PLGF levels.

22 However, PLGF/VEGF heterodimers display 20-50-fold less mitogenic

23 develop in all women with high sFLT-1 or low PLGF levels, and it also occurs in some women with low s

24 We also demonstrate the presence of natural PLGF/VEGF heterodimers in the conditioned media of vario

25 s was restored to LLC cells by expression of PLGF, strongly suggesting that the angiogenic phenotype

26 EGF-induced angiogenesis by the formation of PLGF/VEGF heterodimers in cells producing both factors.

27 oduce high levels of VEGF, but low levels of PLGF.

28 d Sp2-O-Ag14 cells suggests that a number of PLGF receptor subtypes remain unidentified.

29 e that pharmacologically distinct subsets of PLGF receptors exist that distinguish between cyclic-PA

30 ted ex vivo migration in response to VEGF or PLGF.

31 grate or activate AKT in response to VEGF or PLGF.

32 The purified recombinant PLGF/VEGF heterodimers and VEGF homodimers have potent m

33 These data indicate that PLGF may modulate VEGF-induced angiogenesis by the forma

34 mbilical vein endothelial cells reveals that PLGF/VEGF heterodimers and VEGF165 homodimers, but not P

35 All three PLGFs induced the formation of stress fibers in NIH3T3 f

36 on patterns were established among the three PLGFs by monitoring changes in intracellular Ca2+ in NIH

37 e for LPA and not activated by the other two PLGFs.

38 The effect of VEGF, PLGF, and VEGF-receptor (VEGFR) activation on BBB permea

39 While PLGF/VEGF heterodimers bind with high affinity to a solu

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