ライフサイエンスコーパス: POP

1 POP amounts (mg/ha) stored in different tree components,

2 POP concentrations in the plastic did not differ signifi

3 POP formation was highest in shallow-fried potatoes with

4 POP is a vaginal problem, which is best treated vaginall

5 POP levels varied significantly among sites, more than 3

6 POP were analyzed using a GC-MS method.

7 POPs are suggested to interfere with the interaction bet

8 POPs with high TEFs and AhR affinity were associated wit

9 mily microRNAs, affect the activity of LIT-1/POP-1 cellular asymmetry machinery and APR-1 polarity du

10 he cellular polarizing activity of the LIT-1/POP-1 system as development proceeds.

11 the summed molar blood concentrations of 14 POPs were positively related to TAC.

12 rnal and paternal serum concentrations of 63 POPs, comprising five major classes of pollutants, with

13 new lanthipeptide flavipeptin, proved that a POP-type FlaP protease is responsible for leader removal

14 results highlight the advantages of using a POP platform to develop new catalysts which are otherwis

15 garine increased POP content in foods with a POP profile characterized by a higher ratio of epoxy- to

16 salmon tissue to differing degrees, acquire POPs from different dietary sources, or bioaccumulate or

17 All POPs were unexpectedly found to be active and selective

18 By combining correlations among POP concentrations, differences in tissue concentrations

19 espectively, were 0.27 and 0.22 for anatomic POP (treatment difference of 0.050; 95% CI, -0.161 to 0.

20 reatment or self-reported bulge; or anatomic POP failure requiring retreatment or Pelvic Organ Prolap

21 unravel relationships between age, BMI, and POP concentrations, informing efforts to understand pote

22 Here, we discuss our model of COP- and POP-mediated inflammasome regulation.

23 s in the blood of children with leukemia and POP levels in dust from their household vacuum cleaners.

24 umannii and Pseudomonas aeruginosa (SNAP and POP studies).

25 or age at Tanner stages 2 or higher (2+) and POPs quartiles (Q1-4), adjusting for confounders (race/e

26 elucidated, but the effects of most COPs and POPs on inflammasome assembly have not been investigated

27 oteins encoding only a CARD or PYD, COPs and POPs, respectively, are assumed to inhibit inflammasome

28 rs associated with exposure to pathogens and POPs.

29 anges in the rates of decline of atmospheric POP concentrations encoded into synthetic time series.

30 stigate the historical trends of atmospheric POPs in a region.

31 salmon has no crucial impact on the average POP intake for typical Latvians.

32 mpler and that spatial analysis of tree bark POPs concentrations can often pinpoint their sources.

33 hese findings support an association between POP exposure and the risk of T2D.

34 by the absence of clear dependencies between POP concentrations and most evaluated environmental and

35 as used to evaluate the relationship between POP levels in blood and dust, adjusting for child's age,

36 udies have investigated associations between POPs and LTL.

37 gical evidence of the putative disruption by POPs of the adipose tissue oxidative microenvironment.

38 ight be involved in substrate recognition by POPs.

39 Risk-based analysis of several carcinogenic POPs indicated that the fish with the highest levels of

40 ignificantly lower concentrations of certain POPs (trans-nonachlor, p,p'-DDE, and several PCBs) in th

41 s a source of children's exposure to certain POPs.

42 lipophilic and readily adsorbs and collects POPs from the atmosphere.

43 mechanisms for a combination of three common POPs using a systems biology approach, which may link PO

44 scuss various vaginal techniques for complex POP.

45 0.060 to 0.162), 0.48 and 0.34 for composite POP (treatment difference of 0.134; 95% CI, -0.096 to 0.

46 higher than levels observed for conventional POPs detected in the samples.

47 ponents, including posterior pharynx defect (POP-1)/TCF, APC related/adenomatosis polyposis coli (APR

48 Dietary POP levels differed significantly between treatments in

49 ing molecular mechanisms that link different POPs to common metabolic diseases.

50 the vertical distribution of truly dissolved POPs at two sites in the Atlantic Ocean.

51 lychlorinated biphenyls (PCBs), the dominant POP in beluga.

52 (biological pump), as key processes driving POPs plankton phase concentrations in the global oceans.

53 ablished cohort, quantifying levels of eight POPs and four groups of oxidative stress biomarkers in a

54 The temporal trends of new (or emerging) POPs in the atmosphere are often unclear because time se

55 e used to determine the state of equilibrium POPs reached in the passive samplers.

56 Contrary to expectation, POP concentrations did not demonstrate an associated dis

57 stimated probabilities of treatment failure (POP, SUI, UI) from parametric survival modeling for the

58 such, tree bark is an ideal sampler to find POPs sources globally, regionally, or locally.

59 For stream-resident fish, POP congener pattern was influenced by the presence of s

60 For POP concentrations in sediments, this trend could be att

61 tion after the placement of vaginal mesh for POP repair or SUI surgery.

62 women who underwent transvaginal repair for POP or SUI with mesh between January 1, 2008, and Decemb

63 se inhibitors that were highly selective for POP over a number of homologous proteins.

64 sis shows that plastic with low affinity for POPs such as polystyrene will have a marginal decreasing

65 ions, or plastics with a higher affinity for POPs.

66 d the TMF significance for cVMS, but not for POPs.

67 egetation is also an important reservoir for POPs.

68 The work provides promising results for POPs as an economical material for multiple environmenta

69 ne challenged with SAV as fry in freshwater (POP 1) and one challenged with SAV as post-smolts in sea

70 st resistance to PD in both populations (fry POP 1 h(2)~0.5; post-smolt POP 2 h(2)~0.4).

71 ork is needed to develop the next-generation POPs for practical applications.

72 The TCF homolog POP-1 binds a site in the egl-18 promoter in vitro, and

73 decreasing the concentrations of hydrophobic POPs in productive waters.

74 in both the sire- and dam-based analyses in POP 1, and genome-wide significance in a combined analys

75 -wide significance in a combined analysis in POP 2.

76 were significant interannual differences in POP concentrations in multiparous females' milk from fiv

77 were not consistent with trends observed in POP concentrations in fur seal milk, suggesting that cli

78 hose in the control group (mean reduction in POP-SS from baseline 3.77 [SD 5.62] vs 2.09 [5.39]; adju

79 A decreasing trend in POP levels toward the sea was observed.

80 portant to be able to predict variability in POP concentrations in changing environments.

81 erage concentrations as well as variation in POPs among individuals.

82 In conclusion, PS liquid margarine increased POP content in foods with a POP profile characterized by

83 total POP content by over 75% and individual POP content by over 50%.

84 ght important differences between individual POP members.

85 are limited, and the results for individual POPs are not entirely consistent across studies.

86 Of the four known POPs, only POP2 is reported to regulate NF-kappaB-depend

87 ed with metabolic diseases; three well-known POPs [2,3,7,8-tetrachlorodibenzodioxin (TCDD), 2,2 ,4,4

88 ower cVMS bioaccumulation compared to legacy POPs, and affected the TMF significance for cVMS, but no

89 FA) and persistent organic pollutant levels (POPs).

90 g a systems biology approach, which may link POP exposure to diseases.

91 As a storage compartment for lipophilic POPs, AT plays a critical role in the toxicokinetics of

92 The accumulation of lipophilic POPs will increase total body burden.

93 oncentrations were higher than most man-made POPs, suggesting that the source of most of these compou

94 le diet) rather than metabolites of man-made POPs.

95 Mean POP-SS at 1 year did not differ substantially between co

96 Mean POP-SS at 2 years were: standard 4.9 (SD 5.1) versus mes

97 The mean POP-SS score at 2 years was 3.2 (SD 3.4) in the interven

98 na from 12 locations worldwide, and measured POP levels using combined liquid or gas chromatography a

99 The noninvasive method for measuring POP concentrations in killer whales through scat employe

100 ontrol margarine resulted in a higher median POP content per food portion (1.35mg, range 0.08-13.20mg

101 onstrated that the extent of salmon-mediated POP transfer and uptake in Great Lakes tributaries is lo

102 tary sources, or bioaccumulate or metabolize POPs differently.

103 The resulting metallated POPs were then evaluated for catalytic alkyne hydrogenat

104 Three of the metallated POPs (V, Cr, Mn) are the first of their kind to be activ

105 l of the axial ligand afforded a microporous POP that is catalytically active in the methanolysis of

106 Currents seemed more important in moving POPs to deeper water masses than the biological pump.

107 ers, and the cause of endocrine and neuronal POP effects.

108 Lipid-normalized POPs concentrations were elevated in amphipods with lipi

109 end testosterone as a sensitive biomarker of POP exposure and that seasonal patterns are investigated

110 sed design and synthesis of a novel class of POP inhibitors based on hexahydroisoindoles.

111 The long-term decline of POP concentrations in the Southern Ocean seawater since

112 Eight male foxes were given a diet of POP-contaminated minke whale blubber, whereas their eigh

113 To explore the nature and extent of POP biotransport by salmon, we compared 58 PCB and 6 PBD

114 Predictive models of POP content versus storage time were established.

115 Here, we present an exhaustive sampling of POP with a known inhibitor, Z-pro-prolinal.

116 The combined use of POP mesh and SUI mesh sling was associated with the high

117 can act as a vector for the assimilation of POPs into marine organisms.

118 Bioaccumulation of POPs increased with age, with the exception of presumed

119 the effects of plastic on bioaccumulation of POPs.

120 t serum concentrations of several classes of POPs (polybrominated diphenyl ethers [PBDEs], polychlori

121 Four classes of POPs and seven halogenated phenols were detected in at l

122 rall mass, concentration, and composition of POPs in yellowfin and examined the differences in levels

123 ation of lipids resulted in concentration of POPs in the remaining lipid pool.

124 ociations between maternal concentrations of POPs and offspring allergic sensitization at 20 years of

125 ons, differences in tissue concentrations of POPs between plastic ingestion subgroups, fugacity calcu

126 Concentrations of POPs detected in the North Atlantic near the surface (e.

127 Maternal concentrations of POPs were, however, positively associated with offspring

128 oup (n=14) had the highest concentrations of POPs, which might suggest that mobilization of contamina

129 10-2013) were analyzed for concentrations of POPs.

130 less recipients of atmospheric deposition of POPs that have undergone long-range atmospheric transpor

131 esults generally reflect the distribution of POPs in densely populated and industrialized countries l

132 The spatial distribution of POPs indicated that the major sources in the region are

133 We assessed the combined effect of POPs on oxidative stress/glutathione system biomarkers u

134 the Beaufort Sea to evaluate the effects of POPs on the levels of 13 health-related gene transcripts

135 t of predictive biomarkers of the effects of POPs, which could translate into disease prevention and

136 o understand potential obesogenic effects of POPs.

137 n field measurements of air-soil exchange of POPs show that there is a close coupling of the polychlo

138 (SO-MUM), to estimate emissions and fate of POPs in an urban area on a 5 x 5 km(2) cell resolution.

139 our understanding of the large-scale fate of POPs in the Arctic and may also provide a means to test

140 ween the structural and chemical features of POPs and their adsorption capacities is discussed, mainl

141 The largest diffusive fluxes of POPs were calculated at station 8C, the site where the h

142 tations and demonstrates the introduction of POPs recently listed under the Stockholm Convention into

143 stically significant reductions in levels of POPs that have had control actions enacted to restrict o

144 are occupationally exposed to high levels of POPs.

145 We assessed the underlying mechanisms of POPs that have been associated with metabolic diseases;

146 commonly used to evaluate the performance of POPs for carbon capture, including CO2 capacity, enthalp

147 first seasonally resolved emission rates of POPs, which were determined with our combined measuremen

148 back-calculate atmospheric emission rates of POPs.

149 UNEP global convention for the regulation of POPs.

150 rrent and future remobilization and sinks of POPs are a strong function of the close coupling of clim

151 tterns indicated that salmon are a source of POPs to brook trout in stream reaches receiving salmon s

152 ighlight the relevance of ongoing sources of POPs, even decades after regulations aiming to reduce or

153 s buffering function, AT is also a target of POPs and may mediate part of their metabolic effects.

154 imatic influences on air-to-milk transfer of POPs needs to be accounted for when using contamination

155 Transfer of POPs through mobilization of endogenous lipid stores dur

156 sfer factors highlight that cold-trapping of POPs in pastures is mostly due to increased gas-phase de

157 Observed increasing trends of POPs in the tree-ring samples were representative for th

158 This study broadens the understanding of POPs in the endangered Southern Resident killer whale po

159 act as a passive sampler than as a vector of POPs, thus reflecting the POP profiles of simultaneously

160 s; namely, 1,3-dipalmitoyl-2-oleoylglycerol (POP) (8.6-17.7%), 1-palmitoyl-2-oleoyl-3-stearoyl-glycer

161 Prolyl oligopeptidase (POP) is a large 80 kDa protease, which cleaves oligopept

162 ing a gene encoding a prolyl oligopeptidase (POP).

163 that future assessment of climate impact on POP fate and distribution should take into consideration

164 vides tunable control of surface features on POPs, thereby affording control over bulk material prope

165 an simultaneously also enhance the opposing, POP-1 activity, suggesting a role in modulating the pote

166 Other POPs were not significantly associated with diabetes.

167 ons with diabetes after adjustment for other POPs were strongest with the more volatile, non-dioxin-l

168 Thus, unlike other POPs thought to diminish innate protection, POP2 reduces

169 s, were strongly associated with the overall POP load.

170 iety of drugs and pollutants, in particular, POPs.

171 We prospectively examined plasma POP concentrations in relation to incident T2D and summa

172 ominated their persistent organic pollutant (POP) burdens.

173 ations between persistent organic pollutant (POP) concentrations and body mass index (BMI).

174 BDE-209 in the persistent organic pollutant (POP) convention as well as the need for strategies to re

175 nships between persistent organic pollutant (POP) levels in the blood of children with leukemia and P

176 -PBDEs) can be persistent organic pollutant (POP) metabolites or natural marine compounds.

177 is to modulate persistent organic pollutant (POP) toxicity through several mechanisms.

178 ling (EPS) of persistent organic pollutants (POP) in fish tissue has been hitherto limited to applica

179 re exposed to persistent organic pollutants (POP) originating primarily from southern latitudes.

180 anthropogenic persistent organic pollutants (POP), but nevertheless recipients of atmospheric deposit

181 rations of 18 persistent organic pollutants (POPs) and 9 toxic elements in blood, as well as total an

182 ations of 164 persistent organic pollutants (POPs) and halogenated phenols in 53 Hawaiian green turtl

183 tions between persistent organic pollutants (POPs) and metabolic diseases, but testable hypotheses re

184 ata regarding persistent organic pollutants (POPs) and risk of type 2 diabetes (T2D) are limited, and

185 tions between persistent organic pollutants (POPs) and type 2 diabetes.

186 cumulation of persistent organic pollutants (POPs) are critical for hazard and ecosystem health asses

187 Persistent organic pollutants (POPs) are developmental toxicants, but the impact of bot

188 Persistent organic pollutants (POPs) are ubiquitous contaminants of environmental and h

189 y exposure to persistent organic pollutants (POPs) for 22 months.

190 e analysis of persistent organic pollutants (POPs) has the potential to characterize sources, sinks,

191 reservoirs of persistent organic pollutants (POPs) have accumulated in soils and snow/ice in polar re

192 exposures to persistent organic pollutants (POPs) have been associated with asthma medication use an

193 Concern about persistent organic pollutants (POPs) in Californians prompted the state's biomonitoring

194 the level of Persistent Organic Pollutants (POPs) in feed products.

195 thesized that persistent organic pollutants (POPs) in microplastic may pose a risk to aquatic organis

196 as to examine persistent organic pollutants (POPs) in relation to pubertal onset.

197 entrations of persistent organic pollutants (POPs) in the atmosphere have been monitored at multiple

198 stribution of persistent organic pollutants (POPs) in the deep ocean.

199 e a source of persistent organic pollutants (POPs) in the human diet.

200 ative role of persistent organic pollutants (POPs) in the induction of oxidative damage in cell struc

201 entrations of persistent organic pollutants (POPs) in the surface sediments and near-bottom water.

202 Exposure to persistent organic pollutants (POPs) is known to increase risk of diabetes.

203 l exposure to persistent organic pollutants (POPs) is limited by costs and logistics of follow-up.

204 Several persistent organic pollutants (POPs) like polycyclic aromatic hydrocarbons (PAHs), poly

205 asurements of persistent organic pollutants (POPs) near the North American Great Lakes-measurements t

206 vironmentally persistent organic pollutants (POPs) occurs through the diet, the host gastrointestinal

207 ions of total persistent organic pollutants (POPs) ranged from 9750 to 156,000 ng g(-1) lipid weight,

208 Exposure to persistent organic pollutants (POPs) such as dioxins, furans, and polychlorinated biphe

209 ment can sorb persistent organic pollutants (POPs) that can potentially be assimilated by organisms m

210 ng sources of persistent organic pollutants (POPs) to the atmosphere can sometimes be difficult.

211 contemporary persistent organic pollutants (POPs) to the local environment.

212 MF for legacy persistent organic pollutants (POPs) were analyzed for comparison.

213 own potential persistent organic pollutants (POPs) were evaluated for occurrence within the samples a

214 entrations of persistent organic pollutants (POPs), and evaluate risk factors associated with exposur

215 Persistent organic pollutants (POPs), contaminants that may bioaccumulate in upper trop

216 amples for 50 persistent organic pollutants (POPs), including 21 PCB congeners, 8 PBDE congeners, and

217 emissions of persistent organic pollutants (POPs), including polycyclic aromatic hydrocarbons, polyc

218 Persistent organic pollutants (POPs), specifically PCBs, PBDEs, and DDTs, in the marine

219 can transport persistent organic pollutants (POPs), such as polychlorinated biphenyls (PCBs) and poly

220 ndustrial-use Persistent Organic Pollutants (POPs), such as polychlorinated biphenyls (PCBs), have be

221 ary source of persistent organic pollutants (POPs).

222 t and fate of persistent organic pollutants (POPs).

223 cumulation of persistent organic pollutants (POPs).

224 ns of several persistent organic pollutants (POPs: PCBs, PBDEs, OCPs) in aquatic species from the Sch

225 ated into a catechol porous organic polymer (POP) and characterized using ATR-IR and XAS analysis.

226 incorporated into a porous organic polymer (POP) using a cobalt-catalyzed acetylene trimerization st

227 thiol functionalized porous organic polymer (POP) using simple free radical polymerization techniques

228 new porphyrin-based porous organic polymer (POP) with BET surface area ranging from 780 to 880 m(2)/

229 developed Programmable Order Polymerization (POP), Microfluidic Combinatorial Assembly of DNA (M-CAD)

230 The emergence of porous organic polymers (POPs) has provided great opportunities for new applicati

231 terials, especially porous organic polymers (POPs), as one type of the most promising candidates for

232 apacity among known porous organic polymers (POPs).

233 The Pregnancy Outcome Prediction (POP) study was a prospective cohort study of nulliparous

234 bility to reliably back-extrapolate prenatal POP levels from levels 9 years after delivery, with Supe

235 Plant sterol (PS) oxidation products (POP) derived from sitosterol and campesterol were measur

236 aldehyde and phytosterol oxidation products (POPs) were evaluated as oxidative markers.

237 roduction of phytosterol oxidation products (POPs).

238 ic and oleic acid) and triglyceride profile (POP, SOS and POS) to cocoa butter.

239 aft, has been used in pelvic organ prolapse (POP) repair and stress urinary incontinence (SUI) to aug

240 the United States for pelvic organ prolapse (POP).

241 y in the treatment of pelvic organ prolapse (POP); however, the robotic industry and decreased reside

242 ting the role of PYRIN domain-only proteins (POPs) and the related CARD-only proteins (COPs) in regul

243 Pyrin domain-only proteins (POPs) are recently evolved, primate-specific proteins de

244 s a critical and in-depth analysis of recent POP research as it pertains to carbon capture.

245 incomplete equilibration, and the resulting POP concentrations, determined by each material, agreed

246 The resulting POP is able to remove aqueous and airborne mercury with

247 .e. the Pelvic Organ Prolapse Symptom Score [POP-SS]) and condition-specific (ie, prolapse-related) q

248 in the pelvic organ prolapse symptom score [POP-SS]) at 12 months than those in the control group (m

249 mptoms (Pelvic Organ Prolapse Symptom Score [POP-SS]) at 2 years.

250 alyze a total of 748 time series of selected POPs in the atmosphere to determine if there are statist

251 In this study, concentrations of selected POPs were measured in ambient air in Zurich, Switzerland

252 tudinal study of puberty in girls with serum POPs measurements (to our knowledge) reveals a delay in

253 xamined the differences in levels of several POP congeners of potential relevance to human health.

254 ernal and paternal concentrations of several POPs were associated with statistically significant diff

255 172 and 195) serum concentrations of several POPs were statistically associated with lower birth weig

256 steady state bioaccumulation models simplify POP bioaccumulation dynamics, assuming that pollutant up

257 populations (fry POP 1 h(2)~0.5; post-smolt POP 2 h(2)~0.4).

258 Some POPs, i.e., hydroxylated metabolites of polychlorinated

259 but different major TAGs (PPO-, PSO-, SSO-, POP- and SOS-rich blends) were evaluated.

260 mospheric transport potential of the studied POPs.

261 thout mesh but concurrent sling use for SUI (POP sling group), and sling for SUI alone (SUI sling gro

262 We created three summed POP metrics based on toxic equivalency factor (TEF), a p

263 Symptomatic POP failure requiring retreatment or self-reported bulge

264 161 to 0.271), 0.29 and 0.24 for symptomatic POP (treatment difference of 0.049; 95% CI, -0.060 to 0.

265 ltidimensional scaling (NMDS), we found that POP congener patterns of Pacific salmon varied among reg

266 Our results indicate that POP exposure may enhance alternative pathways to the glu

267 rdens), contrasting traditional beliefs that POPs do not reach the deep ocean.

268 Taken together, these data suggest that POPs are not a major cofactor in causing the onset of FP

269 th 10% and 20% water-containing butanol, the POP emission factors were decreased by amounts in the ra

270 lmars from Norwegian waters and compared the POP concentrations in their liver and muscle tissue with

271 , thus resulting in a great reduction in the POP emissions.

272 ue to the annual lactational transfer of the POP burden from mother to pup.

273 r and fall, while mean concentrations of the POP chlordane (SigmaCHL) were lower for land-based bears

274 Supercritical CO2 processing of the POP dramatically increased the pore size and volume, all

275 act the influence of warming by reducing the POP fugacity in soil.

276 han as a vector of POPs, thus reflecting the POP profiles of simultaneously ingested prey.

277 w, representative recent developments in the POPs-based catalysts with hierarchically porous structur

278 tion to identify common mechanisms for these POPs within two or fewer interaction steps downstream of

279 ational estimates of contamination for these POPs.

280 that the atmospheric concentrations of these POPs are generally decreasing, consistent with the overa

281 are needed to confirm the causality of these POPs, which will shed new light on the pathogenesis of d

282 ar support for reducing inventories of these POPs.

283 ies into underlying mechanisms by which this POP mixture could produce or exacerbate metabolic diseas

284 explore associations between adipose tissue POP concentrations and the in situ oxidative microenviro

285 Due to POP's size and to the buried nature of its active site,

286 l gut microbiota are likely to be exposed to POPs.

287 Our data indicate that prenatal exposure to POPs appears to be associated with airway obstruction bu

288 sing the level and risk of human exposure to POPs through ingestion of wild fish.

289 e associations between prenatal exposures to POPs and allergic sensitization and lung function in 20-

290 Total POP contents followed the order MPS>>FJPS>MFJPS.

291 These models explain total POP content by over 75% and individual POP content by ov

292 vaginal mesh plus sling group), transvaginal POP repair with mesh and no concurrent sling use (vagina

293 sling use (vaginal mesh group), transvaginal POP repair without mesh but concurrent sling use for SUI

294 ups based on the amount of mesh transvaginal POP repair surgery with mesh and concurrent sling use (v

295 chemicals with properties similar to typical POPs.

296 Finally, a concise outlook for utilizing POPs for carbon capture is discussed, noting areas in wh

297 lenged with SAV as post-smolts in sea water (POP 2).

298 ptional activation through interactions with POP-1/Tcf.

299 regulated in vaginal tissues from women with POP.

300 the literature on the interaction of AT with POPs to provide a comprehensive model for this additiona

301 ongest positive correlation of diabetes with POPs occurred with organochlorine compounds, such as tra