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1 PP chelates Mn(III) produced by the enzyme and subsequen
2 PP DCs augment IgA production by integrin alphavbeta8-me
3 PP detectors process signals produced by each particle a
4 PP IgA class switching requires innate lymphoid cells, w
5 PP plays a multifaceted role, slowing the reaction by co
6 PP prepared with Irgafos 168 led to a significantly stro
7 PP requiring hospitalization significantly decreased in
8 PP TC development across juvenile, transition, and adult
9 PP TCs with extensive wall ingrowths are ubiquitous in m
10 PP type M cells are thought to be dependent on cytokines
11 PP was analyzed as a continuous and categorical (i.e., b
12 PP was associated with reduced risk of neutropenia-relat
13 PP, mean arterial pressure, cardiac output, SVR, and asc
14 PP, VmaxAo, SVC, and IVC could be measured in 78.5%, 78.
15 ropenia-related hospitalization for TC (2.0% PP; 7.1% no PP; adjusted odds ratio [AOR], 0.29; 95% CI,
17 deficits in different frequency bands in 25 PP-MS patients (12 M, mean age 50.9 +/- 10.5 years) and
18 ], 0.29; 95% CI, 0.22 to 0.39) and TCH (1.3% PP; 7.1% no PP; AOR, 0.19; 95% CI, 0.12 to 0.30), but no
19 ies are required for between 14 (pH) and 32 (PP) years to distinguish a climate change trend from nat
20 the presence of genes for both CPD and (6-4)PP photolyases, as well as genes for nucleotide excision
22 trate that Arabidopsis removes CPDs and (6-4)PPs by a dual-incision mechanism that is essentially ide
23 esolved UV damage maps of both CPDs and (6-4)PPs by HS-Damage-seq and compared them to the complement
24 -4)pyrimidine-pyrimidone photoproducts [(6-4)PPs] (at 5 and 20 min and 1, 2, and 4 h) in normal human
25 imidine-pyrimidone (6-4) photoproducts [(6-4)PPs] from UV-irradiated cellular and naked DNA revealed
26 imidine (6-4) pyrimidone photoproducts [(6-4)PPs] that must be repaired for the survival of the plant
27 ts covalently bound 4'-phosphopantetheine (4-PP)-conjugated acyl chain to support maximal cysteine de
28 an obligate subunit but also exploits the 4-PP-conjugated acyl chain to coordinate mitochondrial fat
29 6 no PP); 2,292 patients received TCH (1,444 PP; 848 no PP); and 1,638 patients received AC (857 PP;
30 e [ITT: Delta = -2.0% (95% CI: -7.6%, 3.6%); PP: -1.9% (95% CI: -5.3%, 1.4%)], weight gain [Delta = -
31 .19; 95% CI, 0.12 to 0.30), but not AC (4.7% PP; 3.8% no PP; AOR, 1.21; 95% CI, 0.75 to 1.93) regimen
34 A total of 4,815 patients received TC (2,849 PP; 1,966 no PP); 2,292 patients received TCH (1,444 PP;
36 1 PHQ-9 points [95% CI -1.3 to 1.5], p=0.89; PP: CBT 7.9 PHQ-9 points [7.3]; BA 7.8 [6.5], mean diffe
38 yorelaxant peptide (SMP) was identified as a PP/OK-type neuropeptide in the starfish Patiria pectinif
39 (CPET), which relies on data collected by a PP detector to reconstruct the 3D distribution of a radi
41 ctronic ground-state calculations indicate a PP multiple bond character in which the terminal phospho
44 4/pRB pathway may no longer be accurate; all PPs are potentially critical targets of CDK-cyclins in m
48 ders a selective defect in POLA2-binding and PP stimulation, indicating that these activities are cri
49 ication group (e.g., central systolic BP and PP root-mean-square-errors of 3.3 and 4.2 mm Hg versus 6
52 a significantly strong cytotoxic effect and PP prepared with Irganox 1076 induced a dose-dependant e
53 l cellular regulatory functions of InsPs and PP-InsPs (including telomere maintenance, phosphate sens
56 rresponding figures on day 42 in the ITT and PP populations were 90.48% and 91.34%, respectively.
57 d a greater diagnostic accuracy than IVC and PP, but its measurement requires transesophageal echocar
60 monal insulin+, glucagon+, somatostatin+ and PP+ cells and glucose-responsive synchronized calcium si
62 d inhibits neuronal activity with attenuated PP-DG synapse plasticity, leading to hippocampus-depende
63 esional skin from psoriasis patients before (PP) and after treatment (PT) with adalimumab and in norm
67 rformed to determine the association between PP and cardiovascular outcomes, including cardiovascular
68 fferential expression of 971 lncRNAs between PP and NN, 157 between PP and PT, and 377 between PT and
69 ing differentially expressed lncRNAs between PP and NN, we identified a molecular lncRNA signature th
70 ectively potentiate Y4R agonism initiated by PP was confirmed in mouse descending colon mucosa prepar
76 e to obtain for the first time with Corolase PP soy hydrolysates with potential antioxidant and ACE i
77 der two temperature conditions with Corolase PP was studied, assessing the impact of hydrolysis on po
80 to phenotypically characterize hPSC-derived PPs and distinguish these cells from non-PP populations
81 rovide a stability framework for data-driven PP-GLMs and shed new light on the stochastic dynamics of
86 lis var. Energo genotypes (polyploid Energo [PP-E]; 2n = 4x = 76) with variation in the green pixel-b
87 all samples were obtained by MAE as follows: PP (18.92+/-0.11), ML (15.19+/-0.11), HL (12.69+/-0.16),
90 0% in each group (for AP diet P < .0002; for PP diet P < .0002); decrease in FGF21 correlated with lo
92 isk 9.3 [95% confidence interval 3.3-26] for PP and 9.7 [95% confidence interval 3.2-29.6] for second
95 at implantation 31 years), 61 (76%) were for PP; of 68 females (median age at implantation 43 years),
99 precursor N-acetyl-D-galactosamine (GalNAc)-PP-Und (2 weeks) and (ii) enzymatic extension of the pre
101 and N-acetylperosamine (PerNAc) onto GalNAc-PP-Und to form the intact RU-PP-Und in a stepwise manner
103 ltration were significantly reduced in group PP relative to group PR, and the loss of collagen matrix
104 eater collagen deposition was noted in group PP relative to groups PR and PH at 4 and 14 days after s
111 Here, we review current understanding of how PPs foster B-cell encounters with antigen, how they favo
112 , discuss how they have helped to illuminate PP-InsP structure and function, and highlight opportunit
113 Sebaceous glands were markedly atrophic in PP versus nonlesional psoriatic skin (91% average reduct
115 results point to wall ingrowth deposition in PP TCs being under control of the genetic program regula
117 e of a widespread cognitive deterioration in PP-MS patients, with main involvement of visuo-spatial a
118 dule N5 genes were strongly downregulated in PP versus normal skin (fold change < 0.25, 44.4-fold) an
119 l activity underlie cognitive dysfunction in PP-MS, and that CLs possibly play a role in variability
120 lely of module N5 genes, whose expression in PP versus normal skin was inversely correlated with the
121 orphometry identified elongated follicles in PP versus nonlesional psoriatic skin (average 1.7 vs. 1.
122 es, and suggest that loss of visible hair in PP skin may result from abnormal sebaceous gland functio
125 ided therapy achieved a greater reduction in PP, which may have contributed to reduce the risk of reb
126 decreased differentiation gene signature in PP/PN-KCs that had not been identified by pre-genomic st
128 s suggest that inflammatory cytokine-induced PP type M cells may be a useful correlate of chronic int
130 phate, UDP-glucose, and Delta(2)-isopentenyl-PP play similar roles in using group transfer chemistry
132 y monolayer KC cultures grown from lesional (PP) and non-lesional (PN) biopsies of psoriasis patients
133 P with greater accuracy than GTFs in the low PP amplification group (e.g., central systolic BP and PP
137 cell proliferation and invasion, whereas Lox-PP acts as tumor suppressor because of its Ras recision
138 cal persistence of MAC-LD was defined as MAC-PP exceeding 1 year, in contrast with the negative-conve
140 , and positive AFS had an OR of 17.7 for MAC-PP, and those with >/=2 of the factors had a 4.5-fold in
141 f persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioration in MAC-
145 tients with MAC-LD, 75 (60%) were in the MAC-PP group; these patients had a higher proportion of radi
146 stant N-glycans originating from Man5GlcNAc2-PP-dolichol transferred by TbSTT3A, and endoglycosidase
147 ed to translocate the glycolipid Man5GlcNAc2-PP-dolichol (needed to synthesize N-glycan precursors) a
150 Point process generalized linear models (PP-GLMs) provide an important statistical framework for
153 ted hospitalization for TC (2.0% PP; 7.1% no PP; adjusted odds ratio [AOR], 0.29; 95% CI, 0.22 to 0.3
154 CI, 0.22 to 0.39) and TCH (1.3% PP; 7.1% no PP; AOR, 0.19; 95% CI, 0.12 to 0.30), but not AC (4.7% P
157 ,292 patients received TCH (1,444 PP; 848 no PP); and 1,638 patients received AC (857 PP; 781 no PP)
158 815 patients received TC (2,849 PP; 1,966 no PP); 2,292 patients received TCH (1,444 PP; 848 no PP);
162 stent with this conclusion, the abundance of PP TCs with extensive wall ingrowths varied across roset
163 contrasts with the myoexcitatory actions of PP/OK-type neuropeptides in protostomian invertebrates.
169 FB, by driving differentiation and egress of PP Tfh cells into systemic sites, boosted systemic Tfh c
172 nthetic CO2 uptake was detected in leaves of PP-E plants with increased chlorophyll and carotenoid co
178 bitory-excitatory transition in the roles of PP/OK-type neuropeptides as regulators of muscle activit
182 control of GPEs, this is the first report of PPs content for Bonarda and Aspirant Bouchet GPE, so the
184 s in biological media.1-3 The versatility of PPs stem from innovative synthetic strategies that have
185 of the V1a -AVP receptor partial agonist on PP was associated with an improvement in urine volume an
187 AP (rich in meat and dairy foods; n = 18) or PP (mainly legume protein; n = 19) without calorie restr
188 C. neoformans: ipk1Delta produced no IP6 or PP-IP5/IP7 and, in contrast to ipk1Deltakcs1Delta, accum
189 lag period between induction of total PP or PP/Trp53 KO and melanoma development indicates that addi
193 ll ingrowth deposition in phloem parenchyma (PP) transfer cells (TCs) in leaf veins of Arabidopsis (A
194 ficantly increased numbers of Peyer's patch (PP) phenotype M cells were induced at the peak of inflam
195 s model, we demonstrated that Peyer's patch (PP) Tfh cells were essential for gut commensal segmented
200 resentation ofDrosophilaexpression patterns, PP are comparable to a fate map generated experimentally
201 ), 4-tert-butylphenol (TBP), 4-pentylphenol (PP), 4-hexylphenol (HP), 4-tert-octylphenol (TOP), 4-n-o
206 ic or nonbacteremic patients with pneumonia (PP) caused by A. baumannii (13 from the unicenter and 23
207 ed via aqueous precipitation polymerization (PP) of di(ethylene glycol) methyl ether methacrylate (ME
209 its cognate ligand, pancreatic polypeptide (PP), are involved in the regulation of energy expenditur
213 ibes the printing of an inert polypropylene (PP) architecture with the concurrent printing of soft ma
214 cs such as polyethylene (PE), polypropylene (PP), polystyrene (PS), and polyethylene terephthalate (P
216 (ArPPLN1a-e) derived from the SMP precursor (PP-like neuropeptide precursor 1; ArPPLNP1) in the starf
217 stemic administration of the heme precursors PP-IX or ALA + DFO into zebrafish larvae provides a new
219 VXB significantly decreased portal pressure (PP) in both models of cirrhosis without changes in porta
221 the hemodynamic response of portal pressure (PP) to drug therapy accurately stratifies the risk of va
224 4; each 1-mm Hg increment in pulse pressure (PP) was associated with 1.6% (95% CI: 0.7-2.5%), 0.8% (9
225 re wave pulsatility (central pulse pressure [PP], reflected pressure wave, and reservoir pressure) at
226 medication use, and mean arterial pressure, PP quartile was still associated with all outcomes excep
227 follow-up 8.5 years) after ICD for primary (PP) and secondary prophylaxis in males and females, dete
228 e continuous data, e.g. particle-processing (PP) detectors, has the potential to eliminate null funct
229 Exogenously infused or endogenously produced PP-IX accumulates preferentially in the liver of zebrafi
233 on of DCF and other pharmaceutical products (PPs) with a KF Freundlich constant of 1.7 L g(-1) and no
234 applications of GP2.Pancreatic progenitors (PPs) can be derived from human pluripotent stem cells in
235 PDX1(+)/NKX6-1(+) pancreatic progenitors (PPs) give rise to endocrine cells both in vitro and in v
238 eveloped to distinguish between the proposed PP-InsP signal transduction mechanisms: protein binding,
239 zed for methylparaben (MP), n-propylparaben (PP), ethylparaben (EP), butylparaben (BP), isobutylparab
240 igh in animal protein (AP) vs plant protein (PP), which differ in levels of methionine and BCAAs, in
242 Intention-to-treat (ITT) and per-protocol (PP) analyses showed noninferiority of SMS-RUTF compared
246 8 in intent-to-treat (ITT) and per-protocol (PP) populations was 92.86% and 92.46% and 99.25% and 99.
250 horylation of IP3 to inositol pyrophosphate, PP-IP5/IP7, which is essential for virulence of Cryptoco
258 precursors: an SMP-type precursor (A. rubens PP-like neuropeptide precursor 1; ArPPLNP1) and a second
259 s in primary-progressive multiple sclerosis (PP-MS) and to explore how they are affected by the prese
260 orrelation networks were constructed for six PP that span along the embryonic anterior-posterior axis
261 nscriptome study of lesional psoriatic skin (PP) versus normal skin, we found a coexpressed gene modu
262 on GPE showed the highest content of studied PPs (9804.2 mug g(-1)GPE) followed by Bonarda GPE (7302.
263 the solubility and photostability of studied PPs (by 2 to 17-fold and 2 to 44-fold, respectively).
264 international cohort of high-risk subjects, PP, a readily available hemodynamic parameter, is associ
265 espite passing common goodness-of-fit tests, PP-GLMs estimated from data are often unstable, leading
266 ranscriptome sequence data has revealed that PP/OK-type neuropeptides also occur in a deuterostomian
267 ll mutants except ipk1Delta, suggesting that PP-IP4 partially compensates for absent PP-IP5/IP7 in ip
270 we observed a strong correlation between the PP-stimulatory activity of STN1 variants and their abili
274 iority of hydromorphone was confirmed in the PP analysis (-1.44; 90% CI, -3.22 to 0.27) for street he
275 up, whereas 135 (61%) were assessable in the PP population in the BA group compared with 151 (69%) in
278 is of the putative mutation signature of the PP;Trp53 KO melanomas suggests that melanocytes are prim
279 Our findings not only demonstrate that the PP-InsPs provide a central line of communication between
284 ingly, melanocyte-specific loss of all three PPs facilitated melanoma development (median age of onse
288 The lag period between induction of total PP or PP/Trp53 KO and melanoma development indicates tha
289 om the starfish Asterias rubens revealed two PP/OK-type precursors: an SMP-type precursor (A. rubens
291 her approach, and pulse pressure variations (PP) were recorded with the patient in the semirecumbent
292 itated by the total synthesis of the various PP-InsP isomers and by the development of new methods th
293 ver the state of the art research on various PPs related to the relationship between their structure
295 e aim of this study was to determine whether PP is associated with major adverse cardiovascular outco
296 usceptibility, and outcomes of children with PP 4 years before and 4 years after the introduction of
297 adult leaves, an observation consistent with PP TC development reflecting vegetative phase change (VP
300 We identified patients </=18 years with PP at 8 children's hospitals in the United States (2006-
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