ライフサイエンスコーパス: PPD

1 PPD and ManLAM specifically down-regulated CCR2 expressi

2 PPD and the protein cocktails tested induced strong DTH

3 PPD but not ManLAM specifically induced MCP-1/CCL2 produ

4 PPD correlated negatively with phenolics and carotenoids

5 PPD is an enzymatically mediated oxidative process with

6 PPD is an inhibitory feedback mechanism that prevents ex

7 PPD is the most common reason for revision with adjustab

8 PPD limits cassava's marketing possibilities in countrie

9 PPD proteins interact with KIX8 and KIX9, which act as a

10 PPD selectively delivered polyIC into PSMA-overexpressin

11 PPD-induced proliferative responses were evident by day

12 PPD-modified HSA triggered T-cell responses through a cl

13 PPD-specific CD4+ T cells proliferated and differentiate

14 PPD-specific immune recovery was lower, whereas levels o

15 PPD-specific T cell clones readily trafficked to the air

16 PPD/polyIC invokes antitumor immunity, but unlike many i

17 A total of 70 PPD-specific and 10 PPD-HSA-specific CD4+, CD8+, and CD4+CD8+, Th2-secreting

18 Lymphocytes from 10 PPD-allergic patients, but not tolerant/naive individual

19 tive (PPD)(-) (P = 0.0059) and 4 of 10 (40%) PPD(+) (P = 0.7233) control subjects, suggesting that re

20 A total of 70 PPD-specific and 10 PPD-HSA-specific CD4+, CD8+, and CD4

21 ) (P < 0.0001, Fisher's exact test), and 7/8 PPD-positive (PPD+) subjects (P = 0.21).

22 stinguishable from DTH responses driven by a PPD injection.

23 sequences and on the properties of Twi1p, a PPD protein required for both sequence elimination and s

24 rence risk of postpartum AD for women with a PPD hospital contact after first birth was 55.4 per 100

25 beta mRNA levels peaked as soon as 1 h after PPD stimulation and 4 h after M. tuberculosis H37Rv infe

26 Compared to a nonpathogen Ag, PPD and SEA bead elicited larger lesions with the former

27 artle response (SR) was also observed in all PPD rats relative to control rats.

28 c target-6, culture filtrate protein-10, and PPD as stimulatory antigens were undertaken, and cytokin

29 gene expression, scopoletin accumulation and PPD symptom development.

30 in biopsies at 8 and 48 h after allergen and PPD challenges, respectively, from 10 allergic rhinitics

31 levels had no significant impact on CAL and PPD improvements in teriparatide patients at 1 yr, but i

32 +) or perforin(+) Vgamma2Vdelta2 T cells and PPD-specific IFNgamma(+)alphabeta T cells.

33 nal effects on both long-term depression and PPD were eliminated by treatment with an mGluR1-selectiv

34 ET) was also observed during dye-doping, and PPD was detected with a limit of detection (LOD, 3sigma)

35 l corticotropin-releasing hormone (pCRH) and PPD incidence has been hypothesized, but empirical evide

36 ve individuals, were stimulated with PPD and PPD-modified HSA.

37 40 clones were stimulated with both PPD and PPD-modified HSA.

38 ymmetry of quantal modulation during PPF and PPD was demonstrated at the same single synapse at diffe

39 Many of the up-regulated and PPD-specific expressed sequence tags were predicted to p

40 cs to generate an extensive cassava root and PPD proteome.

41 IL-10 responses to EBV lytic antigens, PPD, and PHA correlated inversely with malaria exposure

42 measured showed similar improvement such as PPD reduction, CAL gain, IBD reduction and defect resolu

43 cyanide-induced oxidative stress as well as PPD control strategies involving inhibition of ROS produ

44 stablished a reliable method for image-based PPD symptom quantification and used label-free quantitat

45 Means were adjusted for baseline PPD, education, and cigarette pack-years, and time-depen

46 n total, 40 clones were stimulated with both PPD and PPD-modified HSA.

47 CL9, CXL10, CCL2, CCL17, and CCL22 mRNA, but PPD beads caused biased CXCL2 CXCL5, CCL3, and CCL4 expr

48 of the murine CES2 homologue, was induced by PPD.

49 because this cytokine was highly induced by PPD.

50 II (carcinogenic dye), which was oxidized by PPD doped in P-CNDs.

51 rgets of PPD2, being negatively regulated by PPDs and KIX8/9.

52 he most common complication of childbearing, PPD has a prevalence of 13%, but there are no widespread

53 The Protein pK(a) Database (PPD) v1.0 provides a compendium of protein residue-speci

54 Plasma Proteome Database (PPD) was initially described in the year 2005 as a part

55 t nadir (PTG), percentage platelet decrease (PPD) in counts, and platelet nadir (PN) counts were meas

56 additional gene candidates to further delay PPD.

57 e peroxidase in storage roots showed delayed PPD and reduced lipid peroxidation as well as decreased

58 Polyphenylene dendrimers (PPDs) represent a unique class of dendrimers based on th

59 Postpartum depression (PPD) affects approximately 10-18% of women in the genera

60 Postpartum depression (PPD) is common and has serious implications for the moth

61 ctively predictive of postpartum depression (PPD) when modeled in antenatal blood.

62 all women experience postpartum depression (PPD), which for many is their first psychiatric disorder

63 a prodromal state for postpartum depression (PPD), with severe PPB strongly associated with an elevat

64 ones recovered from paired-pulse depression (PPD) at rates similar to the recovery of exocytotic capa

65 e of GABA undergoes paired-pulse depression (PPD) that recovers in <1 min (single exponential time co

66 However, during paired-pulse depression (PPD), there was a significant decrease in unitary quanta

67 s well as increased paired-pulse depression (PPD).

68 indicate that prenatal protein deprivation (PPD) affects many aspects of adult brain function.

69 Probing pocket depth (PPD) was measured by calibrated examiners.

70 rushes and flossing on probing pocket depth (PPD), plaque indices, and bleeding on probing (BOP) meas

71 mm vs. 0.92 mm, p < 0.01) and probing depth (PPD) reduction (0.43 mm vs. 1.83 mm, p < 0.01) than vita

72 months was based upon probing pocket depths (PPD), clinical attachment levels (CAL), and whole-mouth

73 ccumulation for purified protein deritative (PPD)-specific T effector cells producing IFN-gamma durin

74 efore and after purified protein derivative (PPD) and mannosilated lipoarabinomannan (ManLAM) stimula

75 stimulated with purified protein derivative (PPD) and phytohemagglutinin (PHA).

76 obacteria bovis purified protein derivative (PPD) and Schistosoma mansoni soluble egg Ags (SEA).

77 incubation with purified protein derivative (PPD) in human immunodeficiency virus-negative patients w

78 canavalin A and purified protein derivative (PPD) in vitro.

79 canavalin A and purified protein derivative (PPD) in vitro.

80 cted tuberculin purified protein derivative (PPD) into the skin of immune individuals and isolated re

81 Purified protein derivative (PPD) is a widely used reagent for the diagnosis of Mycob

82 stimulated with purified protein derivative (PPD) or M. tuberculosis H37Ra or H37Rv, and the total RN

83 gous T-Regs and purified protein derivative (PPD) preprimed T-Reg-depleted effector cells.

84 reby tuberculin purified protein derivative (PPD) was injected into the skin and the local T cell res

85 s ionomycin, or purified protein derivative (PPD) was studied in vitro.

86 th 0 of 11 (0%) purified protein derivative (PPD)(-) (P = 0.0059) and 4 of 10 (40%) PPD(+) (P = 0.723

87 in testing with purified protein derivative (PPD), a complex mix of partly denatured mycobacterial an

88 glutinin (PHA), purified protein derivative (PPD), and T cell epitope peptides derived from merozoite

89 t tuberculosis, purified protein derivative (PPD), lacks specificity and sensitivity.

90 al injection of purified protein derivative (PPD), we investigated the effect of recall Ags to MTb up

91 atients but not purified protein derivative (PPD)-negative or PPD-positive healthy control subjects,

92 Purified protein derivative (PPD)-specific immune recovery was determined by interfer

93 -4 or -7/8, or purified protein derivative (PPD).

94 cific antigens (purified protein derivative [PPD] and ESAT-6/CFP10), followed by polychromatic flow c

95 osis infection (purified protein derivative [PPD] positive) and no infection (PPD negative).

96 evelopment of pigmented purpuric dermatosis (PPD).

97 of postharvest physiological deterioration (PPD) in cassava roots.

98 id post-harvest physiological deterioration (PPD) of its roots that occurs within 24-72 h of harvest,

99 pid postharvest physiological deterioration (PPD) of the root is a major constraint to commercial cas

100 id post-harvest physiological deterioration (PPD) response that can occur within 24-72 h of harvest.

101 pid postharvest physiological deterioration (PPD) that occurs within 72 h following harvest.

102 rom postharvest physiological deterioration (PPD).

103 Sixteen women developed PPD symptoms.

104 o control (mean percentage point difference [PPD]: DEV vs control, -2.2 [90% CI, -8.0 to 3.49], P = .

105 These responses consistently displayed PPD.

106 e cell types in the posterior pars distalis (PPD), and fewer melanotropes in the posterior region of

107 This disynaptic PPD is independent of mGluR-mediated plasticity and depl

108 PAZ/PIWI domain (PPD) proteins carrying small RNAs (sRNAs) function in ge

109 AtTIFY4B has three domains (PPD, TIFY and CCT_2) conserved between homologs from dif

110 Pentyl PABC-Doxaz (PPD) is a Doxaz carbamate prodrug that is hydrolyzed by

111 Pentyl PABC-Doxaz (PPD) is a prodrug of Doxaz that is activated by carboxyl

112 We designed a nonviral vector, PEI-PEG-DUPA (PPD), comprising polyethylenimine-polyethyleneglycol (PE

113 gnificantly higher in the non-atopics during PPD-induced responses (P = 0.003).

114 Degradation of starch granules during PPD was also detected.

115 lvement in the discoloration observed during PPD.

116 owed significant abundance regulation during PPD.

117 in several genotypes of cassava roots during PPD.

118 ease progressive pseudorheumatoid dysplasia (PPD) (Online Mendelian Inheritance in Man database numbe

119 rder progressive pseudorheumatoid dysplasia (PPD).

120 with Progressive Pseudorheumatoid Dysplasia (PPD).

121 ed with greater P-gp expression using either PPD (median expression, resistant 1.89 vs. sensitive 0.9

122 n 10% reduction in proliferation with either PPD or PHA at 2 hours compared with 0 hours.

123 cumulative numbers of teeth with PPD events (PPD > 3 mm) at each dental examination and the slopes as

124 nerable groups, such as women who experience PPD as a first psychiatric episode.

125 cells by glutamatergic neurons that express PPD, and these are likely to correspond to local neurons

126 Finally, PPD-induced proliferative responses of pleural cells fro

127 Following PPD challenge CD3(+)STAT4(+)cells and CD3(+)T-bet(+)cell

128 ystems were significantly shrunken following PPD/polyIC treatment, in all cases.

129 terase (hiCE) as the agent of activation for PPD.

130 There were 132 cases for PPD requiring surgical intervention before September 201

131 antidepressants and/or hospital contact for PPD within 6 months after childbirth.

132 oximately 100-fold reduction in the IC50 for PPD compared to cells lacking the carboxylesterase.

133 These data reveal a mechanism for PPD in cassava based on cyanide-induced oxidative stress

134 and treatment of pregnant women at risk for PPD.

135 on reprogramming exists in those at risk for PPD.

136 trongly associated with an elevated risk for PPD.

137 t subsequently required revision surgery for PPD were assessed and compared with those that did not.

138 e data identify Cys as the single target for PPD-HSA binding, and show that PPD protein adducts are a

139 itive and specific early diagnostic test for PPD symptoms.

140 Transfer of DCs from PPD Ag-challenged lungs conferred a Th1 anamnestic cytok

141 icantly impaired in MTb-stimulated PBMC from PPD-anergic donors.

142 xocytotic capacitance changes recovered from PPD at similar rates in both cell types.

143 c and postsynaptic measures of recovery from PPD suggests that 80-90% of the depression at these syna

144 rizing stimuli promoted faster recovery from PPD.

145 rcadian gene expression rhythms and identify PPD as an ELF3 co-ortholog, termed ELF3b Genetic interac

146 Drug-induced PPD is distinct from idiopathic PPD, and it is important to consider isotretinoin as a p

147 The delay in PPD in transgenic plants was also observed under field c

148 iatal NMDA receptor binding was increased in PPD females.

149 cantly enriched the proteomic information in PPD.

150 reviously identified as dominant proteins in PPD were tested for the capacity to induce delayed-type

151 howed that the observed greater reduction in PPD and BOP in persons using interdental brushing than i

152 MTb recall Ags inhibits HIV-1 replication in PPD-anergic individuals.

153 accharides and may play an important role in PPD delay.

154 Incident PPD occurred in 5.1% and 1.3% of women and men, respecti

155 Drug-induced PPD is distinct from idiopathic PPD, and it is important

156 t with other described cases of drug-induced PPD.

157 first reported case of isotretinoin-induced PPD.

158 derivative [PPD] positive) and no infection (PPD negative).

159 in pull-down assays to identify interacting PPD products.

160 Since many PPD-expressing dorsal horn neurons respond to noxious st

161 ndation did not achieve noninferiority (mean PPD: DEV vs control, 0.9 [90% CI, -4.9 to 6.7], P = .23

162 rmal weight) were associated with worse mean PPD (p < .005), percentage of PPD > 4 mm (p = .01), but

163 e, we demonstrate that in two murine models, PPD was effective at slowing tumor growth and demonstrat

164 > 0.14-0.39 or > 0.39 cm/yr experienced more PPD events than men in the lowest tertile (</= 0.14 cm/y

165 lb during follow-up) had significantly more PPD events than men in the lowest tertile of weight gain

166 uberculosis purified protein derivative (Mtb PPD) in UK adolescents, but not in Malawian adolescents.

167 We hypothesized that Mtb PPD-induced IFN-gamma after BCG vaccination would be sim

168 UK infants had an IFN-gamma response to Mtb PPD, compared to 41 (53%) of 78 of Malawian infants, in

169 were recognized by T-cells from in-vitro Mtb-PPD and ESAT6/CFP10-positive donors by proliferation and

170 , 1/24 purified-protein-derivative-negative (PPD-) (P < 0.0001, Fisher's exact test), and 7/8 PPD-pos

171 ate Tetrahymena thermophila encodes numerous PPD proteins exclusively of the Piwi clade.

172 of 106 Da, corresponding to the addition of PPD and not to the secondary products of self conjugatio

173 h prevented BB formation, but not binding of PPD to cells and serum, did not prevent p-phenylenediami

174 Recently, the molecular composition of PPD was defined, with hundreds of mycobacterial protein

175 torage and were correlated with the delay of PPD.

176 major functional role in the development of PPD symptoms, rather than merely paralleling symptom dev

177 The potent antitumor effects of PPD/polyIC should spur its development for clinical use.

178 However, the molecular identity of PPD has so far remained elusive.

179 biochemical events during the initiation of PPD is a rapid burst of reactive oxygen species (ROS) ac

180 s to define the nature of the interaction of PPD with the protein and the antigenic determinant that

181 grouped samples according to their levels of PPD and chemical compositions.

182 between pea ELF3 genes suggest that loss of PPD function does not affect flowering time in the prese

183 te at the anterior margin in mouse models of PPD.

184 ation was associated with a delayed onset of PPD by 14 to 21 d after harvest of greenhouse-grown plan

185 ith worse mean PPD (p < .005), percentage of PPD > 4 mm (p = .01), but not with FMBS (p > .05) or CAL

186 these specific products drive the potency of PPD remains in question.

187 resulted in a cross-sectional prediction of PPD status with an AUC of 0.81 (95% CI: 0.68-0.93, p=0.0

188 is sensitivity enable accurate prediction of PPD.

189 25 weeks' GA, pCRH was a strong predictor of PPD symptoms (R(2) = 0.21; beta = 0.46 [P < .001]), an e

190 Presentation of PPD to several clones was dependent on protein complex f

191 The proportions of PPD-activated IFN-gamma- and TNF-alpha-producing CD4(+)

192 Younger women are at higher risk of PPD after LAGB surgery than men and women older than 50

193 Tissue fluid obtained from the site of PPD challenge in the skin inhibited the induction of the

194 Main Outcome Measure Symptoms of PPD were assessed at a mean (SD) of 8.7 (2.94) weeks aft

195 nstrated that higher doses of ESAT-6 than of PPD were needed to induce substantial delayed-type hyper

196 trategies that allow for the modification of PPDs at the core, scaffold, and surface to introduce num

197 w is aimed to demonstrate the versatility of PPDs through their site-specific chemical functionalizat

198 tic glutamate receptors had little effect on PPD.

199 nd the effects of changing hormone levels on PPD biomarker loci.

200 CD62L MFI on PPD- and PWM-stimulated gammadelta T-cell receptor-posit

201 vitro stimulation with either Antigen-mix or PPD jhonin, than PBMC from MAP316F vaccinated animals.

202 urified protein derivative (PPD)-negative or PPD-positive healthy control subjects, demonstrating its

203 h this picture, removal of either the Ram or PPD binding sites, separately, modestly reduces Notch ac

204 e axons was counted in paraphenylenediamine (PPD)-stained sections and compared between EphB mutants

205 f rodents stained with paraphenylenediamine (PPD).

206 Here, we characterized PEAPOD (PPD) proteins, the only transcriptional regulators known

207 of the regioisomeric pairs MME/MEM, PPE/PEP, PPD/PDP, EEP/EPE and DDP/DPD (M=14:0, P=16:0, E=20:5, D=

208 In contrast, IFN- gamma responses to PHA, PPD, and Plasmodium falciparum MSP-1 peptides did not si

209 Accordingly, kix8 kix9 mutants phenocopied PPD loss-of-function producing larger leaves resulting f

210 an electrodeposited poly-m-phenylenediamine (PPD) layer and an R. gracilis D-amino acid oxidase (RgDA

211 was to determine whether p-phenylenediamine (PPD) and/or Bandrowski's base (BB) stimulate T cells fro

212 Exposure to p-phenylenediamine (PPD) is associated with the development of T-cell-mediat

213 ocytes after exposure to p-phenylenediamine (PPD).

214 A fourth pea locus, PHOTOPERIOD (PPD), also contributes to the photoperiod response in a

215 Preaxial polydactyly (PPD) is a common limb-associated birth defect characteri

216 nterior limb bud and a preaxial polydactyly (PPD) skeletal phenotype.

217 echanism that leads to preaxial polydactyly (PPD).

218 utine practice, all 875 adults with positive PPD skin test results at pre-employment examinations per

219 or TB screening of individuals with positive PPD skin test results.

220 a from purified protein derivative-positive (PPD+) healthy subjects confirms its expression only duri

221 Fisher's exact test), and 7/8 PPD-positive (PPD+) subjects (P = 0.21).

222 high-risk women, we prospectively predicted PPD status in an independent N=51 women using first trim

223 at the HP1BP3 and TTC9B genes that predicted PPD with an area under the receiver operator characteris

224 of amphetamine to increase preprodynorphin (PPD), preprotachykinin (PPT), preproenkephalin (PPE), an

225 ugh the dynorphin precursor preprodynorphin (PPD) was only present in 4-7% of VGLUT2 boutons in lamin

226 d faster recovery of spinal preprodynorphin (PPD) mRNA than did the adults during complete Freund's a

227 inotecan, a clinical CES2-activated prodrug, PPD produced no visible gastrointestinal damage.

228 en synthetic achievements that have produced PPDs with a range of polarities that break the hydrophob

229 eveloped via introduction of protoporphyrin (PPD, a photosensitizer) which has great doping affinity

230 , CRY2, ELF4, GHD7, VGT1, HY1/SE5, TOC1/PRR7/PPD-1, PIF3, ZCN8, and ZCN19.

231 dose, or red marrow radiation dose and PTG, PPD, PN, and 1/PN.

232 usted marrow doses were correlated with PTG, PPD, PN, and 1/PN.

233 test response differed for the two reagents; PPD showed maximal response at 72 h, but the response to

234 release with NMDA receptors, did not reduce PPD.

235 hione peroxidase as a candidate for reducing PPD.

236 Pocket Probing Depth reduction (PPD), Clinical Attachment Level (CAL) gain and radiograp

237 In addition both loci showed PPD-specific trajectories with age, possibly mediated by

238 roups demonstrated statistically significant PPD reduction, CAL gain and radiographic bone gain.

239 localization sequences, in a domain we term PPD (potential phosphorylated domain).

240 in, PINK1, and DJ-1 formed a complex (termed PPD complex) to promote ubiquitination and degradation o

241 llen allergen) and a cutaneous model of Th1 (PPD) responses in man.

242 culosis that are of greater specificity than PPD, early-secreted antigenic target 6-kDa protein (ESAT

243 We believe that PPD will facilitate both clinical and basic research by

244 We tested the hypothesis that PPD in rats would alter prepulse inhibition (PPI), which

245 Overall, our studies indicate that PPD is selectively hydrolyzed to the active metabolite b

246 ithout cyanide complementation, we show that PPD is cyanide dependent, presumably resulting from a cy

247 Mass spectrometry was employed to show that PPD oxidation products bind irreversibly to cysteine (Cy

248 le target for PPD-HSA binding, and show that PPD protein adducts are antigenic determinants in patien

249 These results suggest that PPD causes age- and sex-dependent decreases in PPI and i

250 the growing body of evidence suggesting that PPD is mediated by differential gene expression and epig

251 The PPD serves as a general protein pK(a) archive and as a s

252 significant change in expression during the PPD response we have used cDNA microarray technology to

253 Here, we show that the PPD locus also has a role in maintenance of diurnal and

254 We found that the PPD phenotype observed in Etv mutants is suppressed by a

255 ults demonstrate for the first time that the PPD response can be mimicked at the molecular level with

256 icantly increased HIV-1 replication in these PPD-anergic donors, indicating that an immunosuppressive

257 Furthermore, the addition of IFN-alpha to PPD-stimulated CD4+ T cells directly inhibited telomeras

258 eration of skin-derived responder T cells to PPD challenge.

259 gic amacrine cells is a major contributor to PPD.

260 de of delayed-type hypersensitivity (DTH) to PPD in the skin.

261 Female rats exposed to PPD had reduced levels of PPI at PND 56, but not PND 35,

262 h respond poorly and express little hiCE, to PPD together with hiCE resulted in a dramatic decrease i

263 asticity may be of etiological importance to PPD.

264 We hypothesized that predisposition to PPD risk is due to an altered sensitivity to estrogen-me

265 wer levels of IL-2 and TNF-alpha relative to PPD-responsive donors in response to PPD stimulation.

266 ction by CD4(+) T lymphocytes in response to PPD in BAL fluid is a promising new diagnostic test for

267 ly, we demonstrate that cellular response to PPD may be predicted with good accuracy using CES2 expre

268 tive to PPD-responsive donors in response to PPD stimulation.

269 Responses to PPD were significantly higher in IRIS patients compared

270 amming events in the hippocampus and risk to PPD using a cross-species translational design.

271 ental (ORI) cultivar was more susceptible to PPD.

272 rkers for sensitivity of a specific tumor to PPD treatment.

273 e onset of the oxidative burst that triggers PPD.

274 in derivative of Mycobacterium tuberculosis (PPD), as expected.

275 t the following URL: http://www.jenner.ac.uk/PPD.

276 obacterial protein representatives making up PPD.

277 me in the presence of functional HR, whereas PPD can compensate only partially for the lack of HR The

278 an increase of release probability, whereas PPD may be caused by decreases in both release probabili

279 We found that while PPD-specific CD4(+) and CD8(+) T effector clones employe

280 hocytes from allergic individuals alone with PPD represents an important discrimination between aller

281 DNA methylation associated with PPD risk correlated significantly with E2-induced DNA me

282 up to 50%, when T-Regs were cocultured with PPD-primed CD4(+) effector T cells.

283 , 100%; optimal cutoff, 3 mm), compared with PPD (sensitivity, 86%; specificity, 90%; optimal cutoff,

284 total protein amounts to be correlated with PPD.

285 ukin (IL)-1, IL-6, and IL-12 expression with PPD challenge.

286 polydactyly in four unrelated families with PPD and in the Hx mouse mutant.

287 tributes to cartilage failure in humans with PPD.

288 no acid substitutions found in patients with PPD had reduced activity in these assays.

289 ological abnormalities seen in patients with PPD.

290 tes from allergic patients proliferated with PPD and BB (n=8).

291 gic patients were stimulated separately with PPD and BB, while clones from volunteers proliferated wi

292 rant/naive individuals, were stimulated with PPD and PPD-modified HSA.

293 ed the mean cumulative numbers of teeth with PPD events (PPD > 3 mm) at each dental examination and t

294 The mean age of those with PPD was 39.9 +/- 9.25 compared with 43.9 +/- 11.0 for th

295 rgic patients proliferated when treated with PPD and Bandrowski's base (BB) and secreted IL-1alpha, -

296 ; however, 12% of clones were triggered with PPD directly.

297 rtum AD was markedly higher among women with PPD hospital contact after first birth compared to women

298 r year of birth and mother's age, women with PPD hospital contact after first birth had a 46.4 times

299 that the trajectories of pCRH in women with PPD symptoms are significantly accelerated from 23 to 26

300 each subject were incubated with or without PPD, and the proportions of CD4(+) T lymphocytes produci