ライフサイエンスコーパス: PRRS

1 PRRS is one of the most important diseases in swine indu

2 rm that CD163 is the likely receptor for all PRRS viruses.

3 rus derived from VR-2332, the North American PRRS virus prototype.

4 ental viruses have been detected in clinical PRRS cases, raising the need for a better differential t

5 MLV vaccines are widely used to control PRRS; however, there have been serious concerns regardin

6 Current PRRS inactivated vaccine provides only a limited protect

7 ted under the conditions of this experiment, PRRS virus was shown to evolve continuously in infected

8 High-risk areas for PRRS were best-predicted by pig density and climate seas

9 hylodynamic models to generate risk maps for PRRS outbreaks and reconstructed the evolutionary histor

10 l immunity and leading to novel vaccines for PRRS prevention.

11 Furthermore, PRRS viruses were characterized by typical seasonality i

12 his study was to describe ongoing changes in PRRS virus during replication in pigs under experimental

13 B cell expansion in PRRS was not associated with preferential VH gene usage

14 ntal step in elucidating the role of nsp2 in PRRS pathogenesis and provides an important insight in f

15 ferent secondary bacterial diseases occur in PRRS virus (PRRSV)-infected pigs.

16 - 0.0%), 98-37120 (0.8% +/- 0.42%), Ingelvac PRRS MLV (0.9% +/- 0.46%), and negative controls (2.3% +

17 ted vaccines, Ingelvac PRRS MLV and Ingelvac PRRS ATP, derived from VR2332 and JA142, respectively, h

18 ccine strains Ingelvac PRRS MLV and Ingelvac PRRS ATP, respectively.

19 son with modified live virus (MLV) (Ingelvac PRRS MLV).

20 isolate 98-38803 is a derivative of Ingelvac PRRS MLV and that the isolate is pneumovirulent.

21 ates and the VR2332 parent virus of Ingelvac PRRS MLV, which were all highly related to Ingelvac PRRS

22 orm homoduplexes with the reference Ingelvac PRRS MLV.

23 ensed low-virulence vaccine strain, Ingelvac PRRS modified live virus (MLV), activated expression of

24 s infection by an avirulent strain, Ingelvac PRRS modified live virus (MLV), did not.

25 h the HMA reference vaccine strains Ingelvac PRRS MLV and Ingelvac PRRS ATP, respectively.

26 nificantly more severe lesions than Ingelvac PRRS MLV (0.7 +/- 0.17) and controls (0.7 +/- 0.15).

27 V, which were all highly related to Ingelvac PRRS MLV with </=2% nucleotide sequence divergence as de

28 Two attenuated vaccines, Ingelvac PRRS MLV and Ingelvac PRRS ATP, derived from VR2332 and

29 g field isolate was identified when Ingelvac PRRS ATP was used as the HMA reference except for its pa

30 roup 2 (n = 19) was inoculated with Ingelvac PRRS MLV vaccine, group 3 (n = 20) was inoculated with t

31 Compared with Ingelvac PRRS((R)) MLV strain, A2MC2-P90 elicits higher virus neu

32 ates to those of a modified live virus (MLV) PRRS vaccine and its parent strain.

33 uvant candidate for the development of novel PRRS inactivated vaccine.

34 ent may have contributed to the emergence of PRRS.

35 ed the relations between the spatial risk of PRRS outbreaks and its phylodynamic history in the U.S d

36 rcine reproductive and respiratory syndrome (PRRS) caused by the PRRS virus (PRRSV) is an important s

37 rcine reproductive and respiratory syndrome (PRRS) consistently elevates the frequency of disease and

38 rcine reproductive and respiratory syndrome (PRRS) has a heritable component, yet little is known abo

39 rcine reproductive and respiratory syndrome (PRRS) virus nonstructural protein 2 (nsp2) contains a cy

40 rcine reproductive and respiratory syndrome (PRRS) virus.

41 rcine reproductive and respiratory syndrome (PRRS) viruses are recognized as possessing a high degree

42 rcine reproductive and respiratory syndrome (PRRS).

43 ate immune evasion properties encoded by the PRRS viral genome.

44 nd respiratory syndrome (PRRS) caused by the PRRS virus (PRRSV) is an important swine disease worldwi

45 as reported to be present in only one of the PRRS modified live virus vaccine strains (Ingelvac) and

46 lop production animals that are resistant to PRRS, the costliest viral disease to ever face the swine

47 chitecture of gene expression in response to PRRS virus (PRRSV) infection.