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1                                              PSV and VICA/VCCA helped classify, respectively, 185 and
2                                              PSV patients' SF-36 scores, except for mental health, we
3                                              PSV was more common in males (23.5/million; 95% CI 17.3-
4                                              PSVs were directly purified from mature seeds by differe
5 r confirming alpha2,3-linked SA on GD1a as a PSV receptor.
6 ibuted to the cisternal ER as expected for a PSV-localized protein, but instead are targeted to the p
7 mately 80 proteins, and we have identified a PSV-specific GTP-binding protein that may be involved in
8                            The addition of a PSV threshold resulted in significant decrease in overal
9                Patients were randomized to a PSV protocol (n = 62), VSV protocol (n = 60), or no prot
10 ults suggest that as the LV transitions to a PSV, the tonoplast remodels before the large vacuole lum
11 inguishable with high specificity by using a PSV greater than 10 cm/sec and an RI greater than 0.65 a
12 hese internal membrane structures to yield a PSV morphology different from that of tomato or tobacco.
13 veloped a new method to detect low-abundance PSVs with improved sensitivity.
14                                     Aleurone PSVs contain zein-rich protein inclusions, a matrix, and
15  the ER, zeins are delivered to the aleurone PSVs in atypical prevacuolar compartments that seem to a
16 oninvasive mechanical ventilation algorithm (PSV-NIV+), neurally adjusted ventilatory assist without
17 oninvasive mechanical ventilation algorithm (PSV-NIV-), pressure support ventilation with a noninvasi
18  in neurally adjusted ventilatory assist and PSV-NIV+ than in PSV-NIV- (p < .05).
19 ystole was shortened, EDV was decreased, and PSV was increased.
20                                      ESA and PSV were significantly lower in the renal artery than in
21  significant association between farming and PSV has been identified for the first time.
22   Among patients having CXRs on both IMV and PSV breaths, 15 of 67 (22%) had their overall degree of
23 ology resembling an amalgamation of a LV and PSV.
24 nd 23 patients were entered into the PAV and PSV groups, respectively, and had similar diagnoses and
25 ction and metabolic signature observed in AP-PSV could be largely mitigated with the OP procedure.
26                            In five arteries, PSV ratios exceeded a threshold of 1.8 (suggesting > or
27 nal parameters, ICA-to-common carotid artery PSV ratio and ICA end-diastolic velocity may also be use
28 rface carbohydrates could not be utilized as PSV receptors for binding and infection.
29 gas exchange and avoid intubation as well as PSV and be more comfortable and tolerable for patients.
30 ficant inverse correlation was seen between %PSV and the percent change of intrastent volume (P<0.000
31 scribed family of proteins, the Brassicaceae PSV-embedded proteins (BPEPs), associated with 'crystall
32  These results demonstrate that Brassicaceae PSVs contain internalized membranes, and raise the possi
33                                          CCA PSV and EDV ranges averaged 23.1 cm/sec +/- 15.7 (SD) an
34                            Right-to-left CCA PSV ratios were abnormal in up to 26 patients (suggestin
35 enting there was a drop in the contralateral PSV and EDV of 60.3 cm/s (p = 0.005) and 15.1 cm/s (p =
36                                         CPAP(PSV) provides a straightforward and effective alternativ
37                                         CPAP(PSV) resulted in an alveolar-arterial partial pressure o
38 PaO(2)) was significantly higher during CPAP(PSV) compared with CPAP or IPPV (98+/-10, 61+/-27, and 7
39 (VO(2)) was significantly higher during CPAP(PSV) than with the other ventilation modes (P<0.05) and
40 t of cardiac output was 76+/-17% during CPAP(PSV), 61+/-21% during CPAP (P<0.01), and 54+/-13% during
41 n was recorded in 8 of 8 animals in the CPAP(PSV) group, in 6 of 8 in the CPAP group, and in 3 of 8 i
42 nced with pressure support ventilation (CPAP(PSV)) during CPR.
43 o ventricular fibrillation and CPR with CPAP(PSV), CPAP, or IPPV.
44                                    Detecting PSVs and determining their origins, either by DNA mutati
45 an Arabidopsis thaliana mutant that disrupts PSV trafficking identified TERMINAL FLOWER 1 (TFL1), a s
46 tically significant differences in MFV, EDV, PSV and RI (all p > 0.05).
47                                As expected, %PSV directly correlated with %VV (P<0.0001, r=0.935), wi
48                                 Seventy-five PSV cases and 273 controls (220 nonvasculitis, 19 second
49 5% confidence interval [CI]: 0.95, 1.00) for PSV and 0.86 (95% CI: 0.78, 0.95; P = .011) for RI.
50 ide, the difference averaged 1.0 +/- 1.3 for PSV ratios and 2.7 +/- 6.9 for EDV ratios, depending on
51  weaning was not significantly different for PSV (1.6 days), VSV (1.8 days), and no protocol (2.0 day
52 e rates were not significantly different for PSV (15%), VSV (24%), and no protocol (17%) (P =.44).
53 ACH homologs acted in a cascading manner for PSV trafficking.
54 l surface GD1a ganglioside could be used for PSV binding and infection as a receptor.
55                We biochemically fractionated PSVs from Brassica napus and defined a crystalloid-like
56 rity (NHA) who were >15 years of age and had PSV first diagnosed between January 1, 1988 and December
57                           On the other hand, PSVs had no binding affinity for synthetic histo-blood g
58  or technical factors raise concern that ICA PSV may not be representative of the extent of disease.
59  is visible; (iii) 50%-69% stenosis when ICA PSV is 125-230 cm/sec and plaque is visible; (iv) > or =
60  or =70% stenosis to near occlusion when ICA PSV is greater than 230 cm/sec and visible plaque and lu
61 A should be diagnosed as (i) normal when ICA PSV is less than 125 cm/sec and no plaque or intimal thi
62 ning is visible; (ii) <50% stenosis when ICA PSV is less than 125 cm/sec and plaque or intimal thicke
63 aspects of QOL are significantly impaired in PSV.
64  decreased hydrolysis of storage proteins in PSV.
65 as receptors, we examined the role of SAs in PSV binding and infection.
66 sted ventilatory assist and PSV-NIV+ than in PSV-NIV- (p < .05).
67      Thereafter, patients were ventilated in PSV and NAVA under continuous rocuronium infusion for 2
68 logy to pathways in yeast or mammals, nor is PSV ultrastructure known in Arabidopsis vegetative tissu
69                  Diabetic patients had lower PSV and higher RI within CRA and TPCA (P</=0.05).
70                                         Mean PSV and VICA/VCCA increased with stenosis level (P < .01
71                                         Mean PSV, VICA/VCCA, and SDs were calculated for each categor
72 ts, 36 (38.7%) had proven appendicitis (mean PSV, 19.7 cm/sec; mean RI, 0.69) and 57 patients (61.2%)
73  0.69) and 57 patients (61.2%) did not (mean PSV, 7.1 cm/sec, P < .0001; mean RI, 0.50, P < .0001).
74 n the central retinal artery (CRA), the mean PSV decreased 16% (P = 0.0137), and the mean EDV decreas
75 significant differences observed in the mean PSV, EDV, or RI at 24 hours or 1 month after treatment.
76 tion of five mutation and 139 mistranslation PSVs.
77                             A porcine model (PSV) was utilized to validate these metabolomic changes
78 ying stenoses as 69% or less or 70% or more, PSV and VICA/VCCA were correct in 90.6% and 90.3% of ves
79 ture of the cruciferin holocomplex, a native PSV fraction was analyzed by single particle electron mi
80 s proposed to determine whether the observed PSVs originate from mutation or mistranslation by charac
81 sured 6-8 mm, the discriminatory criteria of PSV greater than 10 cm/sec and RI greater than 0.65 yiel
82            Chosen discriminatory criteria of PSV greater than 10 cm/sec and RI greater than 0.65 yiel
83              The overall annual incidence of PSV among NHA residents was 19.8/million (95% confidence
84 ur study population, the annual incidence of PSV is slowly increasing with time and the incidence is
85 nd POB(N) were measured at various levels of PSV, ranging from 5 to 25 cm H(2)O.
86                    Decreased mobilization of PSV reserves is accompanied by reductions in the free am
87                            The prevalence of PSV in this cohort was estimated on December 31, 1997.
88 lation by characterizing the distribution of PSVs.
89 osynthesis is fully functional, the roles of PSVs in adult vegetative tissues are not understood.
90                                    Fifty-one PSV patients completed questionnaires assessing QOL (Sho
91                                      Optimal PSV cutoff values were less than 67 cm/sec and 39 cm/sec
92 d using the Respironics Vision ventilator or PSV using a Puritan-Bennett 7200ae critical care ventila
93 w lumen diameters and blood flow parameters (PSV, EDV, PI, RI).
94 endicitis exhibit significantly higher point PSV and point RI values than do patients without appendi
95                                 Poststenotic PSV was mildly dependent on end-organ vascular resistanc
96 d-organ vascular resistance and poststenotic PSV.
97 reported variability from using poststenotic PSV to detect hemodynamically significant renal arterial
98                   We show that cPrG prevents PSV acidification in aleurone layers and prevents synthe
99   We used papillomavirus (PV) pseudoviruses (PSVs) as a model vaccine and a gene delivery vector to a
100   Taking a biochemical approach, we purified PSVs from different developmental stages.
101      We found that oral immunization with PV PSV induced minimal mucosal and systemic Abs and CTLs sp
102 rovided a basis for clinical trials using PV PSVs encoding IL-2 for vaccination of the elderly.
103        When aged mice were immunized with PV PSVs encoding human IL-2, specific Th cells were generat
104 e, but not other gangliosides, could restore PSV binding and infection, further confirming alpha2,3-l
105                     The porcine sapelovirus (PSV) is known to cause enteritis, pneumonia, polioenceph
106     The receptor(s) for porcine sapelovirus (PSV), which causes diarrhea, pneumonia, polioencephalomy
107 y useful tool for consideration when setting PSV to unload the respiratory muscles.
108                      The presence of similar PSVs also containing prolamins and large systems of intr
109 e large vacuole lumen is replaced by smaller PSVs.
110 erior ciliary (TPCA) arteries, the systolic (PSV), end-diastolic and mean blood flow velocities as we
111 l and phospholipase C), we demonstrated that PSV could recognize alpha2,3-linked SA on glycolipids as
112 lood group antigens (HBGAs), suggesting that PSVs could not use HBGAs as receptors.
113                                          The PSV matrix contains glycoproteins that are trafficked th
114                                          The PSV preparations are ideal for a global proteome analysi
115 results suggest a developmental role for the PSV in vegetative tissues.
116 o the PSV and its spatial arrangement in the PSV.
117 systolic velocity (PSV) and the ratio of the PSV in the ICA to that in the ipsilateral common carotid
118 and storage within peripheral regions of the PSV.
119 ctron microscopic observations show that the PSV preparations are homogenous, with the soluble spore
120            However, the receptor(s) that the PSV utilizes to enter host cells remains largely unknown
121 fficient transport of alpha-globulins to the PSV and its spatial arrangement in the PSV.
122                              En route to the PSV, the proteins co-localize in large (>200 nm) vesicle
123  defective in trafficking of proteins to the PSV.
124 ant species, such as tomato and tobacco, the PSV contains two types of microscopically visible intra-
125 ak systolic velocity (PSV) compared with the PSV immediately upstream.
126 ophagic compartments before merging with the PSV.
127 nt of alpha-globulin and glutelin within the PSV, with the accompanying presence of numerous small al
128                                          The PSVs are not clathrin-coated and do not contain the SpiA
129                                          The PSVs contain approximately 80 proteins, and we have iden
130     In seeds of the family Brassicaceae, the PSVs lack visible crystalloids and have many small globo
131                       During development the PSVs increase in size and density concomitant with an in
132 l and systemic Abs and CTLs specific for the PSVs in aged mice compared with young adult mice.
133 localized the BPEPs to structures within the PSVs, whose appearance was consistent with a diffuse net
134                        Focal 2- to 2.9-times PSV elevation was associated with 75% or greater stenosi
135 cuoles for membrane proteins: a direct ER to PSV pathway, and a separate pathway via the Golgi to the
136       However, this trafficking mechanism to PSV is poorly understood.
137 2 with a defect in seed protein transport to PSV.
138                      Trafficking pathways to PSVs and lytic vacuoles appear to be distinct.
139 owever, it is unclear whether trafficking to PSVs has any analogy to pathways in yeast or mammals, no
140                                        Total PSV, subgroups (47 Wegener's granulomatosis [WG], 12 mic
141                          Although plants use PSV proteins during germination, before photosynthesis i
142                 The protein storage vacuole (PSV) becomes acidified rapidly when aleurone cells are t
143                 The protein storage vacuole (PSV) is a specialized organelle in plant seeds that accu
144  trafficking to the protein storage vacuole (PSV) is a specialized process in seed plants.
145 ith glutelin in the protein storage vacuole (PSV).
146       Membranes of protein storage vacuoles (PSV) are marked by the presence of alpha-tonoplast intri
147 ge proteins inside protein storage vacuoles (PSVs) instead of the ER.
148 ins in specialized protein storage vacuoles (PSVs) within seeds and vegetative tissues.
149  finally stored in protein storage vacuoles (PSVs).
150 were identified as protein storage vacuoles (PSVs).
151                   Protein sequence variants (PSVs) are a type of product-related variant in which err
152 isms (SNPs) or paralogous sequence variants (PSVs) separated by several kilobases.
153 n children with primary systemic vasculitis (PSV).
154 ned pre-procedural peak systolic velocities (PSV) and end-diastolic velocities (EDV) in the contralat
155   The range of CCA peak systolic velocities (PSVs) and end diastolic velocities (EDVs) and velocity r
156              (c) ICA peak systolic velocity (PSV) and presence of plaque on gray-scale and/or color D
157  non-angle-corrected peak systolic velocity (PSV) and resistive index (RI) values were compared betwe
158 olic velocity (EDV), peak systolic velocity (PSV) and resistive index (RI).
159                  ICA peak systolic velocity (PSV) and the ratio of the PSV in the ICA to that in the
160  higher elevation of peak systolic velocity (PSV) compared with the PSV immediately upstream.
161         The range of peak systolic velocity (PSV) measurement (maximum minus minimum) averaged 20 cm/
162 ms) with and without peak systolic velocity (PSV) thresholds (determined with receiver operating char
163  acceleration (ESA), peak systolic velocity (PSV), end diastolic velocity (EDV), and waveform morphol
164 nges observed in the peak systolic velocity (PSV), end diastolic velocity (EDV), or resistive index (
165  8 ml/kg under pressure support ventilation (PSV) and under sedation.
166 re severe with pressure support ventilation (PSV) breaths than with intermittent mandatory ventilatio
167                Pressure support ventilation (PSV) is almost universally employed in the management of
168  applied using pressure support ventilation (PSV).
169  coordinate fusion of the prespore vesicles (PSVs) with the plasma membrane at the terminal stage of
170 intima outside the stent (peri-stent volume, PSV) and volume of neointima within the stent (intrasten
171 ins in a clinically selected population when PSV criteria are used.
172 th cANCA 3.3 [1.0-10.8]), drug allergy (with PSV 3.6 [1.8-7.0], with WG 4.0 [1.8-8.7], and with cANCA
173  during working lifetime was associated with PSV (2.2 [1.2-3.8]) and with WG (2.7 [1.3-5.7]).
174 index year was significantly associated with PSV (OR 2.3 [95% CI 1.2-4.6]), with WG (2.7 [1.2-5.8]),
175                          Fifty children with PSV, 17 children with nonvasculitic inflammatory disease
176                                Compared with PSV delivered with the Puritan-Bennett 7200ae, PAV is as
177 lvent exposure during working lifetime (with PSV 2.7 [1.1-6.6], with WG 3.4 [1.3-8.9], and with cANCA
178 A 4.7 [1.9-11.7]), and allergy overall (with PSV 2.2 [1.2-3.9], with WG 2.7 [1.4-5.7]).
179 onal silica exposure in the index year (with PSV 3.0 [1.0-8.4], with CSS 5.6 [1.3-23.5], and with ANC
180 nal solvent exposure in the index year (with PSV 3.4 [0.9-12.5], with WG 4.8 [1.2-19.8], and with cla

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