ライフサイエンスコーパス: PUD

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1 n of H. pylori genotype with esophagitis and PUD.

2 g Chinese H. pylori, most isolates from both PUD and gastritis patients were toxigenic (35/46 and 29/

3 h an increased risk of peptic ulcer disease (PUD) (P = 0.003).

4 biopsy specimens of 68 peptic ulcer disease (PUD) and 327 chronic gastritis (CG) patients with a posi

5 risk of uncomplicated peptic ulcer disease (PUD) in a cohort of new users of low-dose acetylsalicyli

6 agnosis and treatment, peptic ulcer disease (PUD) remains a common reason for hospitalization and ope

7 ines for management of peptic ulcer disease (PUD), trends in physician practice and outcomes related

8 ng (UGIB) secondary to peptic ulcer disease (PUD).

9 ils is associated with peptic ulcer disease (PUD).

10 nderlying vacA gene to peptic ulcer disease (PUD).

11 th a reduction in repeat hospitalization for PUD or subsequent mortality, whereas counseling about th

12 Hospitalizations for PUD decreased in the United States from 1993 to 2006, su

13 he national estimate of hospitalizations for PUD decreased significantly from 222,601 in 1993 to 156,

14 omparison to 1993, patients hospitalized for PUD in 2006 more frequently had endoscopic treatment to

15 rends of hospitalizations and operations for PUD in the United States (US) since 1993.

16 ease in risk of 1-year rehospitalization for PUD (adjusted OR, 0.47; 95% CI, 0.22-0.99) and risk of a

17 es, there has been a significant decrease in PUD mortality, a significant increase in the use of ther

18 polymorphic H. pylori genes are important in PUD.

19 ere to be associated with the development of PUD and was a characteristic more frequently identified

20 Individuals with a history of PUD were more likely to develop uncomplicated PUD than t

21 The inpatient mortality rate of PUD decreased from 3.8% to 2.7% during 1993 to 2006 (P <

22 hospitals with their first UGIB secondary to PUD from 2004-2010 were identified using administrative

23 increase the incidence of UGIB secondary to PUD.

24 UD were more likely to develop uncomplicated PUD than those without such a history (hazard ratio [HR]

25 er risk factors for developing uncomplicated PUD included smoking, stress, depression, anaemia and so

26 ase the risk of development of uncomplicated PUD in new users of low-dose ASA.

27 The crude incidence of uncomplicated PUD was 1.41 per 1000 person-years (95% confidence inter

28 -control analyses, the risk of uncomplicated PUD was associated with current use of non-steroidal ant

29 Incidence of uncomplicated PUD was calculated and a nested case-control analysis ad

30 idered to be incident cases of uncomplicated PUD.

31 of potential risk factors with uncomplicated PUD.

32 It was used to study hospitalizations with PUD as the principal diagnosis during 1993 to 2006, incl

33 mprovement program for elderly patients with PUD resulted in increased screening for H pylori and inc

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