ライフサイエンスコーパス: Panton-Valentine leukocidin

1 omosome mec (SCCmec) typing, and PCR for the Panton-Valentine leukocidin.

2 es encoding toxic shock syndrome toxin 1 and Panton-Valentine leukocidin.

3 mplexes, the junkyard regions as well as the Panton-Valentine leukocidin.

4 resistance, as well as toxic shock toxin and Panton-Valentine leukocidin.

5 A) to SEH, toxic shock syndrome toxin 1, and Panton-Valentine leukocidin.

6 but lacked all common toxin genes, including Panton-Valentine leukocidin.

7 were less diverse (DI = 0.566), positive for Panton-Valentine leukocidin (96.3%), and resistant to er

8 e properties of CA-MRSA, with an emphasis on Panton-Valentine leukocidin, alpha-hemolysin, and the re

9 n transcription and translation of genes for Panton-Valentine leukocidin, alpha-hemolysin, and toxic-

10 of nafcillin induced and prolonged mRNA for Panton-Valentine leukocidin, alpha-toxin, and toxic-shoc

11 trains did differ, however, in expression of Panton-Valentine leukocidin and in the degree of inflamm

12 strains express a number of toxins, such as Panton-Valentine leukocidin and LukAB, that have specifi

13 s aureus (MRSA) strain with genes coding for Panton-Valentine leukocidin and the arginine catabolic m

14 of the Rams' MRSA; all carried the gene for Panton-Valentine leukocidin and the gene complex for sta

15 The vaginal isolate was mecA, Panton-Valentine leukocidin, and staphylococcal enteroto

16 avoiding barotrauma to lungs made friable by Panton-Valentine leukocidin expressing S. aureus infecti

17 ere were four patients with sputum-confirmed Panton-Valentine leukocidin expressing S. aureus pneumon

18 available on the management of patients with Panton-Valentine leukocidin expressing S. aureus pneumon

19 we reviewed our experience and outcomes with Panton-Valentine Leukocidin expressing S. aureus pneumon

20 Panton-Valentine leukocidin expressing S. aureus pneumon

21 Panton-Valentine leukocidin expressing Staphylococcus au

22 teremia were less likely to be infected with Panton-Valentine leukocidin gene (pvl)-constitutive MRSA

23 sessed different exotoxin gene profiles (eg, Panton Valentine leukocidin genes) compared with health

24 ec (SCCmec) type IV, 145 (35.9%) carried the Panton-Valentine leukocidin genes (PVL+), and 162 (40.1%

25 eus isolates from Cape Verde showed that (i) Panton-Valentine leukocidin genes were present in 35% of

26 aphylococcal virulence factors, particularly Panton-Valentine leukocidin, is common in CA-MRSA, empha

27 aphylococcal bicomponent pore-forming toxins Panton-Valentine leukocidin LukSF-PV (PVL) and gamma-hem

28 Neither Panton-Valentine leukocidin nor protein A expression was

29 of CA-MRSA isolates tested were positive for Panton-Valentine leukocidin, of which 90% carried staphy

30 gel electrophoresis (PFGE), spa typing, and Panton-Valentine leukocidin PCR.

31 e of ST5, 2 carried arcA and opp3, and 1 was Panton-Valentine leukocidin positive (PVL(+)).

32 of the 63-wound isolates belonged to the CC8/Panton-Valentine leukocidin-positive (PVL(+)) group of S

33 assette chromosome mec (SCCmec) type IV, and Panton-Valentine leukocidin-positive clustered separatel

34 colonization rate of ciprofloxacin-sensitive Panton-Valentine leukocidin-positive methicillin-resista

35 Panton-Valentine leukocidin-positive USA300 strains caus

36 t two cases of invasive infections caused by Panton-Valentine leukocidin-positive, community-associat

37 The role of the Panton Valentine leukocidin (PVL) in CA-MRSA pathogenesi

38 al cassette (SCC)mec type IV, and 61.5% were Panton Valentine leukocidin (PVL) positive.

39 lpha-hemolysin (Hla), delta-hemolysin (Hld), Panton Valentine leukocidin (PVL), staphylococcal entero

40 The gamma-hemolysins (HlgAB and HlgCB) and Panton-Valentine leukocidin (PVL or LukSF) were shown to

41 aturity (P = 0.02) and isolates positive for Panton-Valentine leukocidin (PVL) (P = 0.008).

42 f potent staphylococcal exotoxins, including Panton-Valentine leukocidin (PVL) and alpha-hemolysin (H

43 ate isolates showed all strains were USA300, Panton-Valentine leukocidin (PVL) and arginine catabolic

44 reus protein A (spa) typing and detection of Panton-Valentine leukocidin (PVL) and scn genes.

45 ant Staphylococcus aureus strains expressing Panton-Valentine leukocidin (PVL) are associated with se

46 Staphylococcus aureus (MRSA) expressing the Panton-Valentine leukocidin (PVL) are rampant, but the c

47 s, the most prominent CA-MRSA strain encodes Panton-Valentine leukocidin (PVL) cytotoxin genes, belon

48 l cassette chromosome mec (SCCmec) types and Panton-Valentine leukocidin (PVL) gene carriage were com

49 The lukF/lukS Panton-Valentine leukocidin (PVL) genes did not directly

50 testing by broth microdilution, detection of Panton-Valentine leukocidin (PVL) genes, arginine catabo

51 nce typing (MLST), as well as assays for the Panton-Valentine leukocidin (PVL) genes, the protein A g

52 dulins, and acquisition of the phage-encoded Panton-Valentine leukocidin (PVL) genes.

53 isolates were tested for the presence of the Panton-Valentine leukocidin (PVL) genes.

54 ons (SSTI) are associated with SCCmec IV and Panton-Valentine leukocidin (PVL) genes.

55 The role of Panton-Valentine leukocidin (PVL) in determining the sev

56 sThe role of Panton-Valentine leukocidin (PVL) in Staphylococcus aure

57 The role of Panton-Valentine leukocidin (PVL) in Staphylococcus aure

58 The Panton-Valentine leukocidin (PVL) is a cytotoxin express

59 lly, many workers have hypothesized that the Panton-Valentine leukocidin (PVL) is a key virulence det

60 The Staphylococcus aureus Panton-Valentine leukocidin (PVL) is a pore-forming toxi

61 Panton-Valentine leukocidin (PVL) is a two-component cyt

62 The S. aureus toxin Panton-Valentine leukocidin (PVL) is most likely causati

63 CA-MRSA) can harbor a bacteriophage encoding Panton-Valentine leukocidin (PVL) lysogenized into its c

64 Antimicrobial susceptibility patterns, Panton-Valentine leukocidin (PVL) occurrence, and staphy

65 The impact of Panton-Valentine leukocidin (PVL) on the outcome in Stap

66 Panton-Valentine leukocidin (PVL) production by methicil

67 All strains were subjected to Panton-Valentine leukocidin (PVL) screening, and SCCmec,

68 USA300 secretes Panton-Valentine leukocidin (PVL) toxin, which is associ

69 Panton-Valentine leukocidin (PVL) was present in 21.9%,

70 The genes lukS-PV and lukF-PV encoding the Panton-Valentine leukocidin (PVL) were present in all CA

71 Panton-Valentine leukocidin (PVL), a bacteriophage encod

72 Panton-Valentine leukocidin (PVL), a pore-forming cytoto

73 SA300, like other CA-MRSA strains, expresses Panton-Valentine leukocidin (PVL), a pore-forming toxin

74 of LukGH in vivo were compared with those of Panton-Valentine leukocidin (PVL), a well-characterized

75 f staphylococci, detection of genes encoding Panton-Valentine leukocidin (PVL), and antimicrobial res

76 he arginine catabolic mobile element (ACME), Panton-Valentine leukocidin (PVL), and other toxins that

77 e chromosome (SCC)mec type IV, the genes for Panton-Valentine leukocidin (PVL), and the enterotoxin Q

78 Most SSTI CA-MRSA strains produce Panton-Valentine leukocidin (PVL), but its contribution

79 for disease and whether a virulence factor, Panton-Valentine leukocidin (PVL), could account for the

80 Panton-Valentine leukocidin (PVL), encoded by the lukSF-

81 A single toxin, Panton-Valentine leukocidin (PVL), has been linked by ep

82 Exchangeable virulence factors, such as Panton-Valentine leukocidin (PVL), have been proposed to

83 are pore-forming cytolytic toxins, including Panton-Valentine leukocidin (PVL), leukotoxin GH (LukGH;

84 some mec type IV [SCCmecIV]) and carried the Panton-Valentine leukocidin (pvl), lukD, and lukE genes,

85 essively acquired the USA300 characteristics Panton-Valentine leukocidin (PVL), SCCmec IVa, the argin

86 filed gel electrophoresis (PFGE) and PCR for Panton-Valentine leukocidin (PVL), the arginine cataboli

87 resence of genes mecA and mupA and those for Panton-Valentine leukocidin (PVL), USA300, and USA400.

88 on of the lukF-PV gene, encoding part of the Panton-Valentine leukocidin (PVL), was observed in the c

89 tis caused by CA-MRSA LAC(WT) USA300 and its Panton-Valentine leukocidin (PVL)- and alpha-hemolysin (

90 of methicillin resistance, SCCmec type, and Panton-Valentine leukocidin (PVL)-producing genes on an

91 cluding alpha toxin, secreted proteases, and Panton-Valentine leukocidin (PVL).

92 ciated with strains harboring genes encoding Panton-Valentine leukocidin (PVL).

93 ing agr, saeRS, sarA, alpha-toxin (hla), and Panton-Valentine leukocidin (pvl).

94 ec but lacked genes for the virulence factor Panton-Valentine leukocidin (PVL).

95 ococcal exotoxins, alpha-hemolysin (Hla) and Panton-Valentine leukocidin (PVL; LukF-PV/LukS-PV subuni

96 phoresis clonal type, toxin genes (e.g., for Panton-Valentine leukocidin [PVL]), and staphylococcal c

97 occal cassette chromosome mec (SCCmec) type, Panton-Valentine leukocidin status, and multilocus seque

98 assays were used to detect mecA, mecC, vanA, Panton-Valentine Leukocidin toxin (PVL), and toxic shock

99 SCCmec type IV and the Panton-Valentine leukocidin toxin gene were detected in

100 All USA300 isolates were positive for the Panton-Valentine leukocidin toxin genes and agr I.

101 maximally expressed 4 h after infection, and Panton-Valentine leukocidin was maximally expressed 72 h

102 ce genes encoding secreted toxins, including Panton-Valentine leukocidin, were highly expressed durin

103 xin; however, carriage of the genes encoding Panton-Valentine leukocidin, while common among MRSA of