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1 ng this pathway has therapeutic potential in Peutz-Jeghers syndrome.
2 tumor suppressor that is causally linked to Peutz-Jeghers syndrome.
3 tal cancer, juvenile polyposis syndrome, and Peutz-Jeghers syndrome.
4 ex, the PTEN-related hamartoma syndromes and Peutz-Jeghers syndrome.
5 bute to tumour formation in individuals with Peutz-Jeghers syndrome.
6 wth arrest may explain the manifestations of Peutz-Jeghers syndrome.
7 creased risk for cancer in patients with the Peutz-Jeghers syndrome.
8 a hereditary intestinal polyposis condition, Peutz-Jeghers syndrome.
9 the LKB1 tumor suppressor gene result in the Peutz-Jeghers syndrome, an autosomal dominant condition
10 1 is a tumor suppressor gene responsible for Peutz-Jeghers syndrome, an inherited cancer disorder ass
11 18.5 (95% CI, 8.5 to 35.2) in women with the Peutz-Jeghers syndrome and 6.2 (CI, 2.5 to 12.8) in men
12 m-line mutations at LKB1/STK11 result in the Peutz-Jeghers syndrome and an increased risk of cancer,
13 ase B1 (LKB1) is mutationally inactivated in Peutz-Jeghers syndrome and in a variety of cancers inclu
14 11, STK11) is a tumor suppressor mutated in Peutz-Jeghers syndrome and in a variety of sporadic canc
15 The human homologue, LKB1, is mutated in Peutz-Jeghers syndrome and is regulated by prenylation a
16 remonitory stigmata such as pigmentations in Peutz-Jeghers syndrome and the phenotypic features of fa
17 inical syndromes such as tuberous sclerosis, Peutz-Jeghers syndrome, and Cowden's disease, which are
20 nase 11 (STK11, also called LKB1) results in Peutz-Jeghers syndrome, characterized by intestinal hama
21 opathic intestinal pseudoobstruction and the Peutz-Jeghers syndrome; defining the role of gut barrier
22 LKB1 (STK11) are found in most patients with Peutz-Jeghers syndrome; however the function of LKB1 is
25 , hereditary nonpolyposis colorectal cancer, Peutz-Jeghers syndrome, juvenile polyposis syndrome, and
26 similarity to a human kinase associated with Peutz-Jeghers Syndrome, LKB1 (STK11), and a Xenopus egg
29 main syndromes associated with these tumors: Peutz-Jeghers syndrome (PJS) caused mainly by mutations
30 Germ-line mutations of the LKB1 gene cause Peutz-Jeghers syndrome (PJS) characterized by mucocutane
37 B1 lead to gastrointestinal tumorigenesis in Peutz-Jeghers syndrome (PJS) patients and mouse models;
39 B1 (also known as STK11) are associated with Peutz-Jeghers syndrome (PJS), a disorder with predisposi
40 KB1, a tumor-suppressor gene responsible for Peutz-Jeghers syndrome (PJS), plays a role in the pathog
41 ions in LKB1 (STK11) are associated with the Peutz-Jeghers syndrome (PJS), which includes aberrant mu
42 m line LKB1 loss-of-function mutations cause Peutz-Jeghers syndrome (PJS), which is characterized by
45 endoscopic techniques (e.g., enteroscopy) in Peutz-Jeghers Syndrome to prevent intussusception have b
46 1 (STK11) serine-threonine kinase gene cause Peutz-Jeghers syndrome, which is associated with inherit
47 line-inactivating mutation of Lkb1 leads to Peutz-Jeghers syndrome, which is characterized by benign
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