ライフサイエンスコーパス: Pi

1 Pi association with tumor metabolism, buffer-mediated pr

2 Pi deficiency also triggered increased resistance to S.

3 Pi starvation reduced main root length and increased the

4 Pi was detected in all dental plaque samples but not in

5 Pi-deficient Arabidopsis (Arabidopsis thaliana) showed e

6 particular, recent measurements of the OD((2)Pi) product of the O((3)P)+D2 reaction have shown a clea

7 ellular Pi levels implies the existence of a Pi-sensing mechanism that signals to the body or individ

8 that the major phase 2 of force decay upon a Pi-jump does not reflect kinetics of the force-generatin

9 n status of downstream targets to accomplish Pi homeostasis under limited Pi supply.

10 y unbinding of the hydrolysis product (ADP + Pi) is followed by ATP uptake, which, in turn, leads to

11 th feedback by the energy state ([ATP]/[ADP][Pi ]) regulating the pathway.

12 ng in the rate being coupled to ([ATP]/[ADP][Pi ])(3) .

13 this Xpr1 deficiency significantly affected Pi efflux.

14 ;3, 1;7 and 1;8 to function as high affinity Pi transporters.

15 ily (MFS) protein, is the main high-affinity Pi transceptor in Saccharomyces cerevisiae Although tran

16 isiae pho2 mutant defective in high-affinity Pi transport rescues growth in low-Pi environments.

17 , plants have evolved high- and low-affinity Pi transporters and the ability to induce root architect

18 (<0.3 muM), as it was greatly enhanced after Pi addition to the incubation water.

19 ux and static concentrations of the alkaline Pi-pool and glycero-phosphocholine, suggesting the possi

20 in the Pi-to-ATP exchange flux, the alkaline Pi-pool, and glycero-phosphocholine concentrations betwe

21 d rate-limiting Pi release 20-fold, although Pi release continues to limit steady-state cycling in th

22 of Pm (odds ratio [OR] = 4.38, P = 0.02) and Pi (OR = 3.44, P = 0.049) and yielded moderate accuracy

23 ) to drive the synthesis of ATP from ADP and Pi.

24 Both the AM and Pi treatments corepressed iron homeostasis genes, result

25 f cytokinins were found in leaves of AM- and Pi-treated plants, whereas the level of ABA was increase

26 ns using phosphorylation assays from ATP and Pi Combining functional and structural data, we highligh

27 nt with the OER activity of Co-OEC in Bi and Pi.

28 gether, this work identifies RNA binding and Pi release as important biochemical transitions within t

29 and PiT2 form high-abundance homodimers and Pi-regulated low-abundance heterodimers.

30 etic measurements of ET, ATP hydrolysis, and Pi release during the presteady-state phase of electron

31 results, we propose that mycorrhization and Pi fertilization share cytokinin-mediated improved shoot

32 supports different mechanisms of protons and Pi accumulation in TME.

33 IRD-1 grown under phosphate (Pi) replete and Pi deplete conditions.

34 nse to the combination treatment of salt and Pi starvation within 330 Arabidopsis accessions could be

35 ors integrating responses to salt stress and Pi starvation.

36 els of extramitochondrial [ATP], [ADP], and [Pi].

37 In most cells, [ATP] and [Pi ] are much higher than [ADP] and change in [ADP] is p

38 plant's ability to forage for the available Pi in topsoil.

39 protein(s) participating in the basolateral Pi efflux remains unknown.

40 benthamiana, revealing that the link between Pi deficiency and enhanced herbivory resistance is conse

41 Correlation analysis between [Pi], pO2, pHe and tumor volumes reveal an association of

42 ded resulting in the release of bioavailable Pi.

43 nthesis, in part, may be caused by low blood Pi concentrations, which may explain some aspects of mus

44 old activation of 5-InsP7 kinase activity by Pi within the 0-5 mm range.

45 PvPht1;3 is induced by Pi deficiency and arsenate, and encodes a phosphate tran

46 es of AM plants differ from those induced by Pi fertilization is poorly understood.

47 ivities of PPIP5Ks are strongly inhibited by Pi (40-90% within the 0-1 mm range).

48 the induction of the JA signaling pathway by Pi deficiency was influenced by PHOSPHATE STARVATION RES

49 es and molecular events driving transport by Pi:H(+) symporters are unclear.

50 th rates are calculated using the calculated Pi values and are compared to those measured for plant a

51 ed to be the Na(+) /Pi symporters catalysing Pi uptake in chlorophytes, whereas PHOSPHATE TRANSPORTER

52 more complete picture of the proton-coupled Pi transport cycle of a transceptor.

53 ore proposed for buffer changes in cytosolic Pi concentration.

54 sh a buffer for transient drops in cytosolic Pi levels but that it can actively decrease or increase

55 se or increase the steady state of cytosolic Pi.

56 estingly, in the absence of sodium-dependent Pi transport activity, the PiT1-PiT2 heterodimerization

57 ophosphatemia as well as in sodium-dependent Pi transporter solute carrier family 34, member 1 (NaPi2

58 ement of the high-affinity, sodium-dependent Pi transporters PiT1 and PiT2 in mediating Pi signaling

59 ular system acts as the site of root-derived Pi stress perception, and the phloem serves to deliver a

60 , modern breeding programmes seek to develop Pi-efficient crops with sustainable yields under reduced

61 sted that S. aureus possesses three distinct Pi transporters: PstSCAB, PitA, and NptA.

62 ] starvation and subsequently recover during Pi replenishment.

63 analogs of the intermediate conformations E2.Pi.2K(+) and Na(+)-bound E1 approximately P.ADP suggest

64 how plant tissue-specific responses to early Pi stress at the transcription level and a predominant r

65 del organism for M. tuberculosis, encounters Pi stress during infection, which shows the relevance of

66 n and metabolite profiling revealed enhanced Pi starvation responses, such as up-regulation of multip

67 systems and to develop plants with enhanced Pi uptake and internal P-use efficiency (PUE).

68 e two ATP to two ADP and two Pi for each ET, Pi release, and dissociation of oxidized Fe protein-(ADP

69 ticity under the increasing supply of excess Pi.

70 urred in metal nutrients level due to excess Pi supply.

71 Here, we report that plants experiencing Pi deficiency induce the jasmonic acid (JA) pathway and

72 ization was still regulated by extracellular Pi levels.

73 ion was no longer regulated by extracellular Pi These observations suggested that Pi binding rather t

74 organisms to detect changes in extracellular Pi levels implies the existence of a Pi-sensing mechanis

75 In Saccharomyces cerevisiae, extracellular [Pi] is "sensed" by the inositol-hexakisphosphate kinase

76 dtii mutant lacking the transcription factor Pi Starvation Response1 (PSR1).

77 that the EXS domain of PHO1 is essential for Pi export activity and proper localization to the Golgi

78 ison of this conformation with the model for Pi release in PiPT revealed that Tyr(179) in Pho84 (Tyr(

79 t suggested that both PiTs are necessary for Pi signaling.

80 ion with disease status was most obvious for Pi (odds ratio [OR]: 15.1; 95% confidence interval [CI]:

81 Proteins responsible for Pi homeostasis are critical for all life.

82 Values for Pi are calculated for plant, algal, and fungal cells fro

83 ple strategies to integrate the signals from Pi deprivation and salinity were identified.

84 In comparison, gastric Pi loading elicited similar but delayed phosphaturia and

85 Intravenous but not gastric Pi loading in parathyroidectomized rats also led to high

86 the rearrangements in the ribosome-EF-Tu-GDP-Pi-Lys-tRNA(Lys) complex following GTP hydrolysis by EF-

87 lymerization by the presence of a GTP or GDP/Pi cap.

88 TRANSPORTER 1 (PHT1) proteins are the H(+) /Pi symporters that carry out Pi uptake in angiosperms.

89 +/- 0.6 mmol/L, P < 0.001), whereas hepatic Pi and HCL were similar in patients when compared with C

90 tumor volumes reveal an association of high [Pi] with changes in tumor metabolism and supports differ

91 and compared them with those induced by high-Pi treatment in nonmycorrhized plants.

92 ntial is achieved by mitochondria at higher [Pi] for a given flux of respiration; (2) the time hierar

93 f Arabidopsis (Arabidopsis thaliana) and how Pi levels modulate responses of the root to salt stress.

94 However, Pi uptake in streptophyte algae and marine angiosperms r

95 go truncatula in conditions with no improved Pi status and compared them with those induced by high-P

96 biomass, and total and soluble P contents in Pi-deficient transgenic seedlings treated with phytate a

97 ed the functions of its different domains in Pi homeostasis and signaling.

98 Anion exchange in Pi is slow, taking days to equilibrate at room temperatu

99 a positive feedback on hypoxia formation in Pi-enriched coastal subsurface waters, as a higher OM de

100 lity of the plant and lead to an increase in Pi uptake.

101 nd plants, the molecular players involved in Pi sensing in mammals remain unknown.

102 up-regulation of multiple genes involved in Pi uptake and solubilization, accumulation of organic ac

103 eplete conditions, consistent with a role in Pi uptake from the environment.

104 aete nitellarum are higher in low-Pi than in Pi-replete conditions, consistent with a role in Pi upta

105 orce decay (phase 2) upon rapid increase in [Pi] is determined by sarcomere dynamics.

106 Rapid increases in [Pi] induced a biphasic force decay with an initial slow

107 alised with triheptanoin, that is, increased Pi/PCr ratio during brain activation compared to the rec

108 Tannerella forsythia, Prevotella intermedia (Pi), and Treponema denticola significantly more in group

109 onas gingivalis (Pg), Prevotella intermedia (Pi), Tannerella forsythia (Tf), and Fusobacterium nuclea

110 onas gingivalis (Pg), Prevotella intermedia (Pi), Treponema denticola (Td), and Escherichia coli usin

111 rmal tissues, were observed for interstitial Pi - the only parameter that also allowed for discrimina

112 e suggests that an intravenous or intestinal Pi bolus causes rapid phosphaturia through mechanisms re

113 ar phosphate levels plateaued, intracellular Pi content increased significantly (11%; P<0.001).

114 esults of this study show that intracellular Pi is the source of Pi removed during dialysis.

115 The intracellular Pi increase may reflect cellular stress induced by hemod

116 t 4 hours after intravenous and intragastric Pi loading in rats.

117 Intravenous Pi loading (0.5 mmol) caused a transient rise in plasma

118 oidectomized rats, however, only intravenous Pi loading caused phosphaturia, which was blunted and tr

119 ow loadings (0.2 mol %), and isoselectivity (Pi =0.92, THF, 258 K).

120 gation speed and rate constant of phase 2 (k+Pi(2)) had a similar [Pi]-dependence, indicating that th

121 1 after rapid [Pi]-increase (rate constant k+Pi(1)) and during the single-exponential force rise (rat

122 nically and Ca(2+)-induced force kinetics (k+Pi(1) approximately k-Pi approximately kTR approximately

123 The values of k+Pi(1) and k-Pi were similar to the rate constant of mech

124 The values of k+Pi(1) and k-Pi were similar to the rate constant of mechanically ind

125 uced force kinetics (k+Pi(1) approximately k-Pi approximately kTR approximately kACT).

126 ngle-exponential force rise (rate constant k-Pi) after rapid [Pi]-decrease.

127 ne, which PHOSPHO1 can hydrolyze to liberate Pi This hypothesis is supported by evidence that both en

128 acid (PyA) as sole P sources, and liberated Pi into the medium.

129 s to accomplish Pi homeostasis under limited Pi supply.

130 RNA increases kcat and rate-limiting Pi release 20-fold, although Pi release continues to lim

131 In combination with salt, low Pi dampened the inhibiting effect of mild salt stress (7

132 grams primary root growth in response to low Pi availability.

133 ted by the observation that growth under low Pi conditions as well as chemical activation of the stri

134 -affinity Pi transport rescues growth in low-Pi environments.

135 have a long-root phenotype when grown in low-Pi media.

136 lga Coleochaete nitellarum are higher in low-Pi than in Pi-replete conditions, consistent with a role

137 showed little homology with other known low-Pi-responsive HAD superfamily members.

138 nd characterized a PHR2-regulated, novel low-Pi-responsive haloacid dehalogenase (HAD)-like hydrolase

139 genomic, and transcriptomic analysis of low-Pi insensitive mutants revealed that the genes previousl

140 he isolation and characterization of two low-Pi insensitive mutants (lpi5 and lpi6), which have a lon

141 ivated upon contact of the root tip with low-Pi media and induces premature cell differentiation and

142 ly translated sORFs, including five with low-Pi up-regulation that correlated with enhanced translati

143 addition, within the MDD group, gray matter Pi, a regulator of oxidative phosphorylation, correlated

144 ough the EXS domain by itself cannot mediate Pi export.

145 ulated PiT1-PiT2 heterodimerization mediates Pi sensing independently of Pi uptake.

146 t Pi transporters PiT1 and PiT2 in mediating Pi signaling in skeletal cells.

147 Pi uptake may be the key factor in mediating Pi signaling through the PiT proteins.

148 ncodes a mitochondrial inner membrane ATP-Mg/Pi carrier.

149 Mitochondrial ATP-Mg/Pi carriers import adenine nucleotides into the mitochon

150 gical function and link mitochondrial ATP-Mg/Pi transport to the development of skeletal and connecti

151 rectly related to cellular and mitochondrial Pi uptake in L6 and RC13 rodent myocytes and isolated mu

152 An excess supply of 20 mM Pi (P20) produces a shallow root system architecture (RS

153 equires Na(+) influx, suggesting that Na(+) /Pi symporters also function in some streptophytes.

154 We tested the hypothesis that Na(+) /Pi symporters exist in streptophytes.

155 ) proteins are hypothesized to be the Na(+) /Pi symporters catalysing Pi uptake in chlorophytes, wher

156 villi, and less abundant expression of Na(+)/Pi cotransporter 2, claudin-2, and aquaporin 1.

157 shoot growth and overexpression of numerous Pi deficiency-responsive genes, expression of only the E

158 s in which S. aureus can successfully obtain Pi Consistent with this idea, in a systemic mouse model

159 did not reduce the expression or activity of Pi transporters.

160 Analysis of Pi transport in primary cultures of proximal tubular cel

161 intestine in stimulating renal clearance of Pi.

162 that control pericellular concentrations of Pi and PPi include tissue-nonspecific alkaline phosphata

163 d increases in growth and total P content of Pi-deficient wild-type rice (Oryza sativa) seedlings.

164 consistent with the zero-order dependence of Pi on the OER current density; in contrast, Bi exchange

165 cription factor regulating the expression of Pi starvation-induced genes.

166 Specifically, imposition of Pi stress induces rapid and major changes in the mRNA po

167 ization mediates Pi sensing independently of Pi uptake.

168 these results indicate that a high level of Pi is crucial in stimulating the growth of bacterial str

169 basal ATPase activity from 21 to 38 nmol of Pi.mg(-1).min(-1), and ATPase activity was further stimu

170 ate (5-InsP7) as follows: during a period of Pi starvation, there is a decline in cellular [ATP]; the

171 The other phases of Pi-induced force kinetics that occur in the absence of "

172 esults identify XPR1 as a major regulator of Pi homeostasis and as a potential therapeutic target in

173 on of Pho84 was used to study the release of Pi A comparison of this conformation with the model for

174 in and Fe protein dissociation to release of Pi Because the Fe protein cannot interact with flavodoxi

175 rements, we demonstrated that the release of Pi is a critical step for transceptor signaling.

176 lectron transfer, ATP hydrolysis, release of Pi, and dissociation of the oxidized, MgADP-bound Fe pro

177 ce kinetics neither enable the separation of Pi-release from the rate-limiting transition f into forc

178 show that intracellular Pi is the source of Pi removed during dialysis.

179 onless coefficients (groups of variables, or Pi parameters).

180 serum Pi content and muscle VATP After oral Pi repletion and normalization of serum Pi levels, muscl

181 s can only acquire inorganic orthophosphate (Pi), meaning global crop production is frequently limite

182 convert phosphite (Phi) into orthophosphate (Pi) offers an alternative selectable marker gene as demo

183 s are the H(+) /Pi symporters that carry out Pi uptake in angiosperms.

184 orylation could be rescued by overexpressing Pi transport-deficient PiT mutants.

185 nts can absorb only the inorganic form of P (Pi), a large portion of soil P (organic and inorganic P

186 The periodontal pathogens Pg, Pi, Tf, and Fn are associated with peri-implantitis.

187 sis (Arabidopsis thaliana) PHOSPHATE1 (PHO1) Pi exporter and defined the functions of its different d

188 available for the effect of excess phosphate Pi on plants.

189 ganic phosphate (PPi) to inorganic phosphate Pi, driving biosynthetic reactions; they are essential f

190 Phosphate (Pi) homeostasis is regulated by renal, intestinal, and e

191 In borate (Bi) and phosphate (Pi) buffers, anions must be displaced to allow water to

192 heir response to nitrogen (N) and phosphate (Pi) supplementation.

193 Extracellular phosphate (Pi) can act as a signaling molecule that directly alters

194 heir ability to release inorganic phosphate (Pi ) and mediate lead bioprecipitation during growth on

195 ses pyrophosphate (PPi)/inorganic phosphate (Pi) (acetyl phosphate + Pi [Formula: see text] acetate +

196 Inorganic phosphate (Pi) accumulation within matrix vesicles (MVs) is a funda

197 levels of extracellular inorganic phosphate (Pi) and pyrophosphate (PPi).

198 Here, we describe how inorganic phosphate (Pi) availability affects the root system architecture (R

199 lyP synthesis transfers inorganic phosphate (Pi) from the cytosol into the acidocalcisome- and lysoso

200 s of ATP and release of inorganic phosphate (Pi) from the nucleotide cleft of actin.

201 uisition, which has two inorganic phosphate (Pi) importers, genomic analysis suggested that S. aureus

202 lular and intracellular inorganic phosphate (Pi) levels is critical to most biochemical and physiolog

203 tration of interstitial inorganic phosphate (Pi) may provide unique insights into biological processe

204 Two inorganic phosphate (Pi) uptake mechanisms operate in streptophytes and chlor

205 Phosphocreatine and inorganic phosphate (Pi) varied in opposite directions across gray matter and

206 flux (VATP) and plasma inorganic phosphate (Pi) were reduced by 50% in mice with diet-induced hypoph

207 osphocreatine (PCr) and inorganic phosphate (Pi) within the occipital cortex during (activation) and

208 Hepatic gammaATP, inorganic phosphate (Pi), and triglyceride concentrations (hepatocellular lip

209 Low phosphate (Pi) availability constrains plant development and seed p

210 e screened under low and 'normal' phosphate (Pi) supply using a 'pouch and wick' system, and had been

211 Depletion of finite global rock phosphate (Pi) reserves will impose major limitations on future agr

212 t levels increased in response to phosphate (Pi) deficiency and arsenate exposure.

213 velopmental response of plants to phosphate (Pi) deficiency and is thought to enhance a plant's abili

214 The response of shoots to phosphate (Pi) deficiency implicates long-distance communication be

215 ic analysis of BIRD-1 grown under phosphate (Pi) replete and Pi deplete conditions.

216 s in [ATP], [ADP], and inorganic phosphate ([Pi]) were characterized over a range of steady-state lev

217 inorganic phosphate (Pi) (acetyl phosphate + Pi [Formula: see text] acetate + PPi).

218 ses in concentration of inorganic phosphate [Pi] were investigated in calcium-activated cardiac myofi

219 itional benefit (mostly inorganic phosphate [Pi]), leading to improved growth, and nonnutritional ben

220 ifted from replete to deficient phosphorous (Pi) nutrition.

221 availability of soluble reactive phosphorus (Pi) in the surface water (<0.3 muM), as it was greatly e

222 of regulation of oxidative phosphorylation, [Pi] does not modulate the activity of complex III.

223 sinks, presumably to coordinate whole-plant Pi homeostasis.

224 (0.5 mmol) caused a transient rise in plasma Pi levels and creatinine clearance and an increase in ph

225 pophosphatemic groups after restoring plasma Pi concentrations.

226 For PPIP5K2, Pi sensing by InsP8 is amplified by a 2-fold activation

227 ed as the principal enzyme acting to produce Pi PHOSPHO1 is a dual-specific phosphocholine/phosphoeth

228 Of note, when two putative Pi-binding residues, Ser-128 (in PiT1) and Ser-113 (in P

229 force rise (rate constant k-Pi) after rapid [Pi]-decrease.

230 ve" was observed during phase 1 after rapid [Pi]-increase (rate constant k+Pi(1)) and during the sing

231 based on a 3D-printed mainframe, a Raspberry Pi computer, and high-definition camera system as well a

232 3D printing technology and rely on Raspberry Pi microcomputers and Arduino boards to provide affordab

233 ion of any two systems significantly reduced Pi accumulation and growth in divergent environments.

234 crops with sustainable yields under reduced Pi fertilizer inputs.

235 that ALS3 cooperates with LPR1/2 to regulate Pi deficiency-induced remodeling of root architecture by

236 The importance of Tyr(179) in regulating Pi release was supported by site-directed mutagenesis an

237 al tubule in mice resulted in impaired renal Pi reabsorption.

238 o decreased expression and activity of renal Pi transporters after 4 hours.

239 ms requiring PTH and downregulation of renal Pi transporters but does not support a role of the intes

240 edlings treated with phytate as a restricted Pi source.

241 d force development (kACT) measured at same [Pi].

242 LC34A3 who had a 50% reduction in both serum Pi content and muscle VATP After oral Pi repletion and n

243 oral Pi repletion and normalization of serum Pi levels, muscle VATP completely normalized in the pati

244 le the Arabidopsis pho1 mutant has low shoot Pi and shows all the hallmarks associated with Pi defici

245 mprovement of shoot growth despite low shoot Pi.

246 y evolutionarily conserved and significantly Pi-regulated lncRNAs.

247 constant of phase 2 (k+Pi(2)) had a similar [Pi]-dependence, indicating that the kinetics of the majo

248 It is shown that a single Pi parameter controls the wall stress relaxation rate.

249 Hitherto, such Pi sensing has not been documented in metazoans.

250 ations suggested that Pi binding rather than Pi uptake may be the key factor in mediating Pi signalin

251 From our experiments, we conclude that Pi starvation interferes with salt responses mainly at t

252 ken together, these results demonstrate that Pi-regulated PiT1-PiT2 heterodimerization mediates Pi se

253 Altogether, these observations indicate that Pi uptake by S. aureus differs from established models a

254 Model simulations indicate that Pi-induced force kinetics neither enable the separation

255 These observations suggest that Pi acquisition via NptA is particularly important for th

256 ellular Pi These observations suggested that Pi binding rather than Pi uptake may be the key factor i

257 The Pi binding kinetics are too slow to establish a pre-equi

258 The Pi deprivation response of lateral roots appeared to be

259 The Pi parameter represents the ratio of plastic and elastic

260 l hydrophilic SPX domain does not affect the Pi export capacity of the truncated PHO1 in N. benthamia

261 ing step" occurs before, along, or after the Pi-release.

262 s between the RNA's phosphate groups and the Pi electrons of the peptide bonds in PNA.

263 nd that deletion of PiT1 or PiT2 blunted the Pi-dependent ERK1/2-mediated phosphorylation and subsequ

264 At higher salt concentrations, the Pi deprivation response prevailed over the salt stress o

265 action have shown a clear preference for the Pi(A') Lambda-doublet states, in apparent contradiction

266 c (31)P-MRS also revealed differences in the Pi-to-ATP exchange flux, the alkaline Pi-pool, and glyce

267 Pho84 (Tyr(150) in PiPT) is not part of the Pi binding site.

268 e EXS domain indicates an attenuation of the Pi signaling cascade and the up-regulation of genes invo

269 sults demonstrate that the propensity of the Pi(A') state is a consequence of the different mechanism

270 ots of wild-type plants and in shoots of the Pi-deficient mutant pho1 The kinetics of the induction o

271 due to a lack of detailed information on the Pi release step in PiPT.

272 od agreement and supports the claim that the Pi parameter is central to expansive growth rate of wall

273 It is found that the Pi values for each cell species are large and very diffe

274 n of the converter domain in response to the Pi release.

275 ere not sufficient to convert this enzyme to Pi/PPi utilization.

276 Arabidopsis mutant, hps10 (hypersensitive to Pi starvation 10), which is morphologically normal under

277 s and cause root growth to be insensitive to Pi deficiency.

278 lthough the long-term responses of plants to Pi stress are well documented, the early signalling even

279 eviously not associated with the response to Pi depletion, were also identified.

280 es of each Pseudomonas strain in response to Pi depletion.

281 tiation of meristematic cells in response to Pi deprivation.

282 kpoint in the root developmental response to Pi starvation in Arabidopsis thaliana Our results also s

283 s that regulate the primary root response to Pi-limiting conditions remain largely unknown.

284 hydrolysis of the two ATP to two ADP and two Pi for each ET, Pi release, and dissociation of oxidized

285 We conclude that land plants inherited two Pi uptake mechanisms - mediated by the PTB and PHT1 prot

286 Under Pi deficiency, als3 accumulates higher levels of Fe(3+)

287 Under Pi deficiency, the ratio of phospholipids to galactolipi

288 n 10), which is morphologically normal under Pi sufficient condition but shows increased inhibition o

289 d enhanced production of lateral roots under Pi deficiency.

290 with significantly altered translation under Pi-limited nutrition.

291 evels of both 5-InsP7 and ATP decrease upon [Pi] starvation and subsequently recover during Pi replen

292 Urinary Pi is freely filtered at the kidney glomerulus and is re

293 When Pi is scarce, modifications of root system architecture

294 inal, and endocrine mechanisms through which Pi intake stimulates parathyroid hormone (PTH) and fibro

295 e as substrates, and enzymatic activity with Pi/PPi was negatively impacted.

296 and shows all the hallmarks associated with Pi deficiency, including poor shoot growth and overexpre

297 of the pho1 mutant typically associated with Pi deficiency, such as high shoot anthocyanin levels and

298 Of the many genes associated with Pi deficiency, this review will focus on those that hold

299 rate in the subsurface water, enriched with Pi (0.4-1.2 muM), was twice as high as that in the surfa

300 ix ATP hydrolysis mass action ratio [ADP] x [Pi]/[ATP] provides feedback to the substrate dehydrogena