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1 ed or regulated during bud outgrowth in pea (Pisum sativum).
2 nd Tha4 and disulfide bond formation in pea (Pisum sativum).
3 s for their hormonal activity in garden pea (Pisum sativum).
4  coordinately regulate bud outgrowth in pea (Pisum sativum).
5  Arabidopsis (Arabidopsis thaliana) and pea (Pisum sativum).
6 d in the supernodulating nod3 mutant of pea (Pisum sativum).
7 lar, is phytotoxic to etiolated seedlings of Pisum sativum.
8 syltransferase, was cloned from root tips of Pisum sativum.
9 s type of learning occurs in the garden pea, Pisum sativum.
10 -hydroxymaackiain 3-O-methyltransferase from Pisum sativum.
11  part of the chloroplast import machinery in Pisum sativum.
12 istatic isoflavonoid produced by garden pea (Pisum sativum), a host for this fungus.
13  lectins Galanthus nivalis agglutinin (GNA), Pisum sativum agglutinin (PSA), and Lens culinaris agglu
14 ously selected for resistance to pea lectin (Pisum sativum agglutinin) and shown to behave dominantly
15 ectin, Griffonia simplicifolia lectin I, and Pisum sativum agglutinin.
16            In leguminous plants such as pea (Pisum sativum), alfalfa (Medicago sativa), barrel medic
17  against equine plasma amine oxidase (EPAO), Pisum sativum amine oxidase (PSAO), Pichia pastoris lysy
18 n of leaf complexity to different degrees in Pisum sativum and Lotus japonicus.
19 um meliloti, required for nodulation of pea (Pisum sativum) and alfalfa (Medicago sativum), respectiv
20 ng of endogenous Toc75 POTRA domains in pea (Pisum sativum) and Arabidopsis (Arabidopsis thaliana) co
21                                         Pea (Pisum sativum) and Arabidopsis contain similar, if not i
22 We characterized the class II smHSPs of pea (Pisum sativum) and compared them with class I smHSPs.
23 ologs from other legume species, namely pea (Pisum sativum) and Lotus japonicus, we show that this mo
24 rminal in some legume species, such as peas (Pisum sativum) and peanuts (Arachis hypogaea), likely du
25 cotyledon phenotypes, has been identified in Pisum sativum, Arabidopsis thaliana, and Festuca pratens
26 ke cells." Whereas root border cells of pea (Pisum sativum) are clearly involved in defense against f
27     The Tic20 protein was identified in pea (Pisum sativum) as a component of the chloroplast protein
28 hese complexes by the embryo, using the pea (Pisum sativum) as a model species.
29 ein fusions, and import assays with purified Pisum sativum chloroplasts, indicated chloroplastic loca
30 nd alpha-CT were imported into isolated pea (Pisum sativum) chloroplasts and became integrated into A
31        Dual import assays with purified pea (Pisum sativum) chloroplasts and mitochondria, and subcel
32 d AtBCCP2 was competent for import into pea (Pisum sativum) chloroplasts and processed to a 25-kD pol
33                      Import assays with pea (Pisum sativum) chloroplasts showed that PyrR and PyrD ar
34                  An in vitro assay with pea (Pisum sativum) chloroplasts was developed to conduct mut
35 vement across the thylakoid membrane of pea (Pisum sativum) chloroplasts was directly measured by sto
36 t across the inner envelope membrane of pea (Pisum sativum) chloroplasts was directly measured by sto
37   Upon incubation of the precursor with pea (Pisum sativum) chloroplasts, it was imported and process
38 been studied extensively using isolated pea (Pisum sativum) chloroplasts.
39 I integral membrane protein abundant in pea (Pisum sativum) clathrin-coated vesicles (CCVs) that bind
40 fected region of soybean (Glycine max), pea (Pisum sativum), clover (Trifolium pratense), and common
41 uncatula nodule root (noot) mutants and pea (Pisum sativum) cochleata (coch) mutants, which are chara
42                         An apyrase from pea (Pisum sativum) complements a yeast (Saccharomyces cerevi
43 bi5 mutants in a second legume species, pea (Pisum sativum), confirmed a role for ABI5 in the regulat
44      A protein of 80 kD from developing pea (Pisum sativum) cotyledons has previously been shown to e
45 r chaperones, Escherichia coli DnaK and pea (Pisum sativum) CSS1, was investigated in detail.
46 of aggregated proteins from tobacco and pea (Pisum sativum cv "Little Marvel") chloroplasts showed th
47  is localized to developing vacuoles in pea (Pisum sativum cv Extra Early Alaska).
48 romoter fragment of the Blec4 gene from pea (Pisum sativum cv. Alaska) and demonstrate that it is cap
49 t phenotype was introgressed into a standard Pisum sativum cv. Birte to provide near-isogenic lines w
50 full-length CPCT cDNA (designated PCT2) from Pisum sativum cv. Feltham First using an Arabidopsis pro
51 sponds to a native protein of 159 kD in pea (Pisum sativum), designated Toc159.
52 ha and beta subunits of the ch-cpn60 of pea (Pisum sativum), expressed them individually in Escherich
53 ansport chain, we overexpressed a minor pea (Pisum sativum) Fd isoform (PsFd1) in tobacco (Nicotiana
54 sgenic tobacco (Nicotiana tabacum), the pea (Pisum sativum) Ferredoxin 1 (Fed-1) mRNA dissociates fro
55 ther t-loops exist in the higher order plant Pisum sativum (garden pea).
56 -1,4-D-glucanase (EGase, EC 3.2.1.4) of pea (Pisum sativum) has been cloned and characterized.
57      The inheritance of flower color in pea (Pisum sativum) has been studied for more than a century,
58                                         Pea (Pisum sativum) has served as a model for development of
59 olymorphic PDR1 retrotransposon insertion in Pisum sativum have been developed.
60 eral ramosus (rms) branching mutants in pea (Pisum sativum) have SL defects, perturbed xylem CK level
61             The function of PsBRC1, the pea (Pisum sativum) homolog of the maize (Zea mays) TEOSINTE
62 tured firefly luciferase (Luc) bound to pea (Pisum sativum) Hsp18.1 as a model to define the minimum
63 t Aux/IAA proteins from Arabidopsis and pea (Pisum sativum) interact in vitro with recombinant phytoc
64                                              Pisum sativum is of great economic and nutritional inter
65     Here, we show that this dwarfism in pea (Pisum sativum) is not attributable to the strong branchi
66 eversibly glycosylated polypeptide from pea (Pisum sativum) is thought to have a role in the biosynth
67 solated a class 1 knox cDNA from pea, Pskn1 (Pisum sativum knotted1) and examined its expression patt
68 ravitropic curvature response of intact pea (Pisum sativum L. cv Alaska) epicotyls was examined by ge
69 were extracted from 7 day old etiolated pea (Pisum sativum L. cv Alaska) epicotyls with 3 molar LiCl.
70                                         Pea (Pisum sativum L. cv Alcan) endocarp tissue challenged wi
71 cDNA has been cloned and sequenced from pea (Pisum sativum L. cv. Birte).
72  was imported and processed by isolated pea (Pisum sativum L. Laxton's Progress No. 9) chloroplasts a
73 was investigated in etiolated pea seedlings (Pisum sativum L. var Alaska) by excising the cotyledons.
74 ation zone of the epicotyl of etiolated pea (Pisum sativum L. var Alaska) seedlings, there is little
75                                    When pea (Pisum sativum L. var Onward) seedlings are treated with
76      GalAT activity in homogenates from pea (Pisum sativum L. var. Alaska) stem internodes co-localiz
77 5% of the SMM was extrachloroplastic in pea (Pisum sativum L.) and spinach (Spinacia oleracea L.), wh
78          A cDNA fragment coding for the pea (Pisum sativum L.) chloroplastic glyceraldehyde-3-P dehyd
79 phatase was purified from the stroma of Pea (Pisum sativum L.) chloroplasts that is capable of dephos
80 t across the inner-envelope membrane of pea (Pisum sativum L.) chloroplasts was directly measured by
81                             We show for pea (Pisum sativum L.) epicotyl sections that this hypothesis
82                      The lip1 mutant of pea (Pisum sativum L.) exhibits a de-etiolated phenotype.
83 tely 80% identical with Arabidopsis and pea (Pisum sativum L.) homologs.
84 lin (GA) controlling stem elongation in pea (Pisum sativum L.) is GA1, which is formed from GA20 by 3
85                     Stem elongation in peas (Pisum sativum L.) is under partial control by gibberelli
86 P21 complex purified from heat-stressed pea (Pisum sativum L.) leaves contained no proteins other tha
87 ase (ACCase) have been characterized in pea (Pisum sativum L.) leaves; a heteromeric chloroplast enzy
88                                       A pea (Pisum sativum L.) nuclear enzyme with protein tyrosine p
89 op genetics and processing in two pea lines (Pisum sativum L.) on starch digestion kinetics.
90                                         Pea (Pisum sativum L.) ornithine transcarbamylase (OTC) antis
91                               A cDNA of pea (Pisum sativum L.) RbcS 3A, encoding a small subunit prot
92 lene biosynthesis in stems of etiolated pea (Pisum sativum L.) seedlings by rapidly increasing the ex
93  Treatment of 5- to 6-day-old etiolated pea (Pisum sativum L.) seedlings with indole-3-acetic acid (I
94                                      In pea (Pisum sativum L.) seeds, GA 2-oxidation is controlled by
95 on on pods of specific genetic lines of pea (Pisum sativum L.) stimulates cell division at the sites
96                 Among a set of genes in pea (Pisum sativum L.) that were induced under drought-stress
97     An artificial gene encoding PBGS of pea (Pisum sativum L.) was designed to overcome previous prob
98                                      In pea (Pisum sativum L.), source to sink partitioning of amino
99 ox and GA 3beta-hy transcript levels in pea (Pisum sativum L.).
100 ivity in nuclei purified from etiolated pea (Pisum sativum, L.) plumules is present in a single enzym
101 ariations were then cross-compared with pea (Pisum sativum), leading to the identification of candida
102            Subcellular fractionation of pea (Pisum sativum) leaf protoplasts indicated that 30% of ly
103                         The vacuoles of pea (Pisum sativum) leaves and red beet (Beta vulgaris) stora
104                         Herein, we show that Pisum sativum LecRLK (PsLecRLK) localized in plasma memb
105                                   Three pea (Pisum sativum) loci controlling photoperiod sensitivity,
106        Dual-import assays with purified pea (Pisum sativum) mitochondria and chloroplasts, and subcel
107 hread formation, as a null SL-deficient pea (Pisum sativum) mutant forms significantly fewer infectio
108 ula ortholog of the Lotus japonicus and pea (Pisum sativum) NIN gene.
109  to Arabidopsis (Arabidopsis thaliana), pea (Pisum sativum), or tomato (Lycopersicon esculentum) tiss
110 rries a nonsense mutation in the single pea (Pisum sativum) ortholog of the ethylene signaling gene E
111                                     Two pea (Pisum sativum) P450 cDNAs (CYP73A9v1, encoding trans-cin
112 phenylpropanoid biosynthetic pathway of pea (Pisum sativum), partial cDNAs representing four distinct
113 ave isolated a DESD-box containing cDNA from Pisum sativum (Pea) and named it as PDH45.
114                The complex was purified from Pisum sativum (pea) chloroplast envelopes by native gel
115  bacteriophages were isolated from leaves of Pisum sativum, Phaseolus vulgaris, Lycopersicon esculent
116 rmation of individual transcription units in Pisum sativum plant nucleoli using a novel labelling tec
117 ify the role of GA3ox expression during pea (Pisum sativum) plant growth and development, we generate
118                          In this study, pea (Pisum sativum) plants overexpressing AMINO ACID PERMEASE
119 ortant for the metabolism and growth of pea (Pisum sativum) plants.
120         Our data indicate that moss and pea (Pisum sativum) plastid transport systems are functionall
121 a nucleolar protein was selected from a pea (Pisum sativum) plumule library, cloned, and sequenced.
122 t) DC3000 hrcC(-) and to the nonadapted pea (Pisum sativum) powdery mildew Erysiphe pisi However, PLC
123 r a pea seed nuclear DNA-binding protein (of Pisum sativum) present at the cell expansion phase.
124 us analysis of the MAP kinase homologue from Pisum sativum (PsMAPK) revealed a potential MAP kinase m
125 and characterization of recombinant p68 from Pisum sativum (Psp68) is presented.
126 e required for plant life in that a UGT from Pisum sativum (PsUGT1) controls plant development by opp
127 curonosyltransferase-encoding gene from pea (Pisum sativum; PsUGT1).
128 d IV of the AUX/IAA protein PsIAA4 from pea (Pisum sativum) revealed a globular ubiquitin-like beta-g
129 iptomic and metabolomic analyses of the pea (Pisum sativum) rhizosphere, a suite of bioreporters has
130 took a systematic, detailed analysis of pea (Pisum sativum) root tip cell walls.
131                                         Pea (Pisum sativum) root tips incubated with [(32)P]dCTP duri
132  the production of root border cells by pea (Pisum sativum) root tips.
133                               Yet, when pea (Pisum sativum) roots are inoculated with the pea pathoge
134 rrying genes encoding soybean lectin or pea (Pisum sativum) seed lectin (PSL) were inoculated with Br
135 c activity found within the germinating pea (Pisum sativum) seed, 4 days from the initiation of imbib
136 eolin mRNA using plumules of dark-grown pea (Pisum sativum) seedlings.
137  complex (mt-PDC) by using intact green pea (Pisum sativum) seedlings.
138   Thermal profiles of viable, aged, and dead Pisum sativum seeds were recorded, and image analysis of
139                   Here, we show that in pea (Pisum sativum) seeds, 4-chloroindole-3-acetic acid is sy
140                    However, in maturing pea (Pisum sativum) seeds, the level of the chlorinated auxin
141 from endoplasmic reticula of developing pea (Pisum sativum) seeds.
142 eat (Triticum aestivum); but the dicots pea (Pisum sativum), soybean (Glycine max Merr.), and spinach
143 ids, Percoll-isolated chloroplasts from pea (Pisum sativum) synthesized FAD from imported riboflavin.
144 n nodulation in Medicago truncatula and pea (Pisum sativum) that form indeterminate nodules.
145 t at the LATE BLOOMER2 (LATE2) locus in pea (Pisum sativum) that is late-flowering with a reduced res
146                                      In pea (Pisum sativum), the protein-lysine methyltransferase (Ps
147                                      In pea (Pisum sativum), the reverse reaction, phenylpyruvate to
148 tematic cells exist within root tips of pea (Pisum sativum): the root apical meristem, which gives ri
149 m of 45Ca2+ transport into the lumen of pea (Pisum sativum) thylakoid membranes using silicone-oil ce
150                        We purified from pea (Pisum sativum) tissue an approximately 40 kDa reversibly
151 dopsis thaliana, Lycopersicon esculentum and Pisum sativum tissues.
152 s trend is found in the Fabaceae, where pea (Pisum sativum) uses UNIFOLIATA, an ortholog of the flora
153 n the Toc159 GTPase domain (Toc159G) of pea (Pisum sativum) using cleavage by bound preproteins conju
154  Rhizobium leguminosarum 3841, a symbiont of Pisum sativum, using chemical methods, mass spectrometry
155 opersicon lycopersicum), and sugar snap pea (Pisum sativum var. macrocarpon) from an industrially imp
156 profile from the oil of harvested field pea (Pisum sativum) varieties as part of a research project t
157 ith legumes belonging to the galegoid clade (Pisum sativum, Vicia faba and Vicia hirsuta).
158         The STERILE NODES (SN) locus in pea (Pisum sativum) was one of the first photoperiod response
159       Nectria haematococca infection of pea (Pisum sativum) was used as a model to explore possible m
160                         In contrast to peas (Pisum sativum), where mitochondrial lipoamide dehydrogen
161     We describe here the FRO1 gene from pea (Pisum sativum), which encodes an Fe(III)-chelate reducta
162  mutants at the Crispoid (Crd) locus in pea (Pisum sativum), which have altered auxin homeostasis and
163 ll wall of the root cap in two species: pea (Pisum sativum), which makes border cells, and Brassica n
164 nt steps of ABA biosynthesis as well as pea (Pisum sativum) wilty and tomato (Solanum lycopersicum) f

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