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1 sistant to infection with the rodent malaria Plasmodium chabaudi.
2 wise lethal blood-stage malaria of the genus Plasmodium chabaudi.
3 ses in mice infected with erythrocytic-stage Plasmodium chabaudi.
4 re we show that after infection of mice with Plasmodium chabaudi, a c-Kit(hi) progenitor subset posit
5 els of beta S globin are well-protected from Plasmodium chabaudi adami and partially protected agains
7 ice were immunized with purified recombinant Plasmodium chabaudi AMA-1 (PcAMA-1) and/or the 42-kDa C-
9 socosms'' - i.e. mice infected with malaria (Plasmodium chabaudi) and nematodes (Nippostrongylus bras
10 latelets during the ascending parasitemia in Plasmodium chabaudi- and Plasmodium berghei-infected mic
12 sed the nonlethal malaria infection model of Plasmodium chabaudi AS in combination with the gastroint
16 erythropoiesis, A/J mice were infected with Plasmodium chabaudi AS, treated with intravenous ferric
17 his study, the roles of IFN-gamma and TNF in Plasmodium chabaudi AS-induced fetal loss in mice were d
18 ized (gdx) C57BL/6 mice were inoculated with Plasmodium chabaudi AS-infected erythrocytes, and the re
22 etitive suppression of an avirulent clone of Plasmodium chabaudi (AS) by a virulent clone (AJ) in imm
24 oplast genome of the rodent malaria parasite Plasmodium chabaudi chabaudi (Pcc) isolate CB was charac
30 ransmission of serially blood passaged (SBP) Plasmodium chabaudi chabaudi to interrogate regulation o
31 gmodontis) and noncerebral malaria (clone AS Plasmodium chabaudi chabaudi), we quantified disease sev
34 ed at peak parasitemia during infection with Plasmodium chabaudi Concentrations of the closely relate
36 with experimental data in mice infected with Plasmodium chabaudi, finding that dying mice trace a lar
37 ng early infection with the malaria parasite Plasmodium chabaudi, implying that another MYD88-depende
43 uppress the parasitemia of acute blood-stage Plasmodium chabaudi malaria by an antibody- or T-cell-de
45 We outline here the parallels between the Plasmodium chabaudi mouse model of malaria and human mal
48 viously that chronic exposure to blood-stage Plasmodium chabaudi offers the best protection from para
49 e now report that the time course shows that Plasmodium chabaudi parasitemia in NADPH oxidase KO (p47
51 s an important fitness-determining trait and Plasmodium chabaudi parasites adjust their sex allocatio
53 rasitic disease and lymphomagenesis, we used Plasmodium chabaudi (Pc) to produce chronic malaria infe
54 e have reported that isolated, permeabilized Plasmodium chabaudi, releases Ca(2+) upon addition of ex
56 ne expression profiles in mice infected with Plasmodium chabaudi revealed and validated similar respo
57 o real data from mice infected with a single Plasmodium chabaudi strain, we estimate that transmissio
59 for the merozoite surface protein (MSP)-1 of Plasmodium chabaudi to show that activated T cells gener
61 -term artificial selection for resistance in Plasmodium chabaudi, we tested resilience of WP against
62 zed red blood cells from the rodent parasite Plasmodium chabaudi with seco-cyclopropyl pyrrolo indole
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