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1 is a severe and often fatal complication of Plasmodium falciparum infection.
2 production were found in women with current Plasmodium falciparum infection.
3 antibodies associated with the prevention of Plasmodium falciparum infection.
4 tion is central to the pathogenesis of acute Plasmodium falciparum infection.
5 are important in the control of blood stage Plasmodium falciparum infection.
6 ral malaria (CM) is a severe complication of Plasmodium falciparum infection.
7 gulated in vaccinees who were protected from Plasmodium falciparum infection.
8 is a common and often fatal complication of Plasmodium falciparum infection.
9 th infections may increase susceptibility to Plasmodium falciparum infection.
10 regarding fulminant disease associated with Plasmodium falciparum infection.
11 group affects host susceptibility to severe Plasmodium falciparum infection.
12 l malaria (HCM) is a serious complication of Plasmodium falciparum infection.
13 which targets the pre-erythrocytic stage of Plasmodium falciparum infection.
14 c, detection measures the real prevalence of Plasmodium falciparum infections.
15 chieving parasite densities similar to fatal Plasmodium falciparum infections.
16 malaria (CM) is a leading cause of death in Plasmodium falciparum infections.
17 ter the report of three cases of complicated Plasmodium falciparum infection acquired by transfusion,
20 l malaria is the most severe complication of Plasmodium falciparum infection and accounts for a large
21 ys leading to pathology and morbidity during Plasmodium falciparum infection and has been termed a ma
22 nd without inflammation and with and without Plasmodium falciparum infection and to assess the effect
23 Ap) represents a potential strategy to clear Plasmodium falciparum infections and reduce the human pa
24 from an Antarctic lake, plastids from mixed Plasmodium falciparum infections, and virulent Neisseria
26 ceptibility of the mosquito to bacterial and Plasmodium falciparum infection, but not to infection wi
27 ral malaria (CM) is a deadly complication of Plasmodium falciparum infection, but specific interactio
38 ccine development against the blood-stage of Plasmodium falciparum infection has focused on recombina
39 apacity of antibodies induced during natural Plasmodium falciparum infection has not been evaluated.
40 The widespread use of chloroquine to treat Plasmodium falciparum infections has resulted in the sel
41 chloroquine (CQ) as first-line treatment of Plasmodium falciparum infections has typically led to th
42 udy found that severely anemic children with Plasmodium falciparum infection have low plasma TNF leve
43 nonymous single-nucleotide polymorphisms and Plasmodium falciparum infection identified three natural
45 REP1) gene are significantly associated with Plasmodium falciparum infection in Anopheles gambiae, an
47 obium was associated with protection against Plasmodium falciparum infection in children who were 4 t
48 describe a mechanistic within-host model of Plasmodium falciparum infection in humans and pathogen t
50 previously identified to be associated with Plasmodium falciparum infection in natural Anopheles gam
53 c test and microscopy - for the detection of Plasmodium falciparum infections in endemic populations
54 a prevention primarily address prevention of Plasmodium falciparum infections in short-term travelers
55 acteristics of clinical illness accompanying Plasmodium falciparum infection induced by controlled ex
59 Severe malarial anemia (SMA) resulting from Plasmodium falciparum infection is one of the leading ca
63 bpopulation of individuals with asymptomatic Plasmodium falciparum infection may play a significant r
65 bral malaria (CM) is a major complication of Plasmodium falciparum infection, particularly in childre
66 33%), 172 (20%), 333 (43%), and 17 (2%) with Plasmodium falciparum infection, Plasmodium vivax infect
73 ral malaria (CM) is a severe complication of Plasmodium falciparum infection responsible for thousand
74 ia (CM), a severe and fatal form of clinical Plasmodium falciparum infection, results from a damaging
77 eted antihelminth therapy and its effects on Plasmodium falciparum infection status, hemoglobin level
78 t sickle-cell heterozygotes have less severe Plasmodium falciparum infections than do children with n
79 ew axis of mosquito resistance to monoclonal Plasmodium falciparum infections that includes the AP-1
82 We therefore studied spatial distribution of Plasmodium falciparum infections to compare simulated ef
84 homozygous primiparae, the odds of placental Plasmodium falciparum infection were reduced by 64%.
85 ebral malaria is a dangerous complication of Plasmodium falciparum infection, which takes a devastati
86 herapies are the first line of treatment for Plasmodium falciparum infections worldwide, but artemisi
87 mechanisms of naturally acquired immunity to Plasmodium falciparum infections would be highly valuabl
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