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1 d using generalized estimating equations for Poisson regression.
2 ence rate ratios (IRRs) were estimated using Poisson regression.
3 95% confidence intervals were calculated by Poisson regression.
4 ence risk ratios (PRRs) were estimated using Poisson regression.
5 2, with those in 1993-97 using multivariable Poisson regression.
6 Mortality rate ratios were estimated by Poisson regression.
7 ios adjusted for potential confounders using Poisson regression.
8 me periods were calculated using conditional Poisson regression.
9 95% confidence intervals) were estimated by Poisson regression.
10 rtality was estimated with spline fits using Poisson regression.
11 maternal death between the two surveys using Poisson regression.
12 one to donate were quantified using modified Poisson regression.
13 /mL by 15 months was assessed using modified Poisson regression.
14 standardized incidence were calculated using Poisson regression.
15 rent HPV infections per woman was studied by Poisson regression.
16 The risk of IPD was assessed using Poisson regression.
17 rand (Boostrix/Adacel), were estimated using Poisson regression.
18 os and 95% CIs were estimated using modified Poisson regression.
19 years and age-adjusted trends estimated from Poisson regression.
20 th disease occurrence were computed by using Poisson regression.
21 We calculated relative rates using Poisson regression.
22 age, sex, and co-morbidity using multilevel Poisson regression.
23 Trends over time were evaluated using Poisson regression.
24 est, and risk factors were investigated with Poisson regression.
25 d ARV discontinuations were identified using Poisson regression.
26 fferences by AIDS status and over time using Poisson regression.
27 s determined using multilevel, mixed-effects Poisson regression.
28 d factors associated with viral rebound with Poisson regression.
29 using relative and absolute risk models via Poisson regression.
30 ed incidence rates of WL were analyzed using Poisson regression.
31 gnosed asthma were computed using a modified Poisson regression.
32 h experiencing a serious adverse event using Poisson regression.
33 1 or PM2.5 were evaluated with a time-series Poisson regression.
34 g Fisher's exact test and bivariate modified Poisson regression.
35 mple) by treatment arm were calculated using Poisson regression.
36 r age, sex, race/ethnicity, and season using Poisson regression.
37 CLABSI incidence rates were compared using Poisson regression.
38 cidence rate ratios (IRRs) were estimated by Poisson regression.
39 ding meal using univariate and multivariable Poisson regressions.
40 idence intervals (CIs) were calculated using Poisson regression adjusted for age group, sex, race, an
41 tagonist users and nonusers, estimated using Poisson regression adjusted for age, calendar year, dise
43 ease and some follow-up at ages 35-74 years, Poisson regression (adjusted for age at risk, amount smo
44 idence intervals (CIs) were calculated using Poisson regression, adjusted for age, sex, co-morbidity,
45 ratio (IRR) was calculated using conditional Poisson regression, adjusted for possible confounders.
46 atios were estimated following multivariable Poisson regression, adjusting for age, sex, ethnicity, s
48 rates in women without celiac disease using Poisson regression, adjusting for sociodemographics, com
49 or machines or logistic regression (LR), and Poisson regression against traditional LR to predict 30-
50 Rates of ADHD diagnosis were derived using Poisson regression analyses after adjustments for potent
56 A time-series design, based on Joinpoint and Poisson regression analyses, was used to assess the chan
60 oup than in the placebo group according to a Poisson regression analysis (roflumilast 0.805 vs placeb
61 ce ratios (SIRs) and, for SCC, multivariable Poisson regression analysis of SIR ratios, adjusting for
64 econd, based on results from the first step, Poisson regression analysis was used to derive the final
65 22 calendar years, 14 geographic areas, and Poisson regression analysis was used to quantify the eff
74 concurrent medications were estimated using Poisson regression and inverse probability of treatment
79 We modelled incidence rate ratios with quasi-Poisson regression and we analysed parasite densities us
80 us non-PKD renal transplant recipients using Poisson regression, and we determined incidence rate rat
86 Longitudinal transition models with modified Poisson regression calculated adjusted relative risks an
87 Using time series analysis and multilevel poisson regression clustering to the hospital level, we
88 with the rate of invasive interval cancers (Poisson regression coefficient -0.084 [95% CI -0.13 to -
89 rming within each city were characterized as Poisson regression coefficients describing change in abu
91 Incidence rate ratios were estimated using Poisson regression, comparing rates of events in the 2-y
96 patients during 1997-2011 was estimated with Poisson regression for all TDR mutations and individuall
97 nce rate ratios (IRRs) were calculated using Poisson regression for DLBCL risk in relation to HLA mis
100 years of calendar time were estimated using Poisson regression incidence rate ratios (IRRs), with su
101 hospitalization for HF was estimated with a Poisson regression model adjusting for comorbidity and c
103 phils >/=300 cells per muL), analysed with a Poisson regression model corrected for overdispersion wi
108 We used a generalized estimating equation Poisson regression model to examine the effect of each s
109 rom the Swedish Family Cancer Database and a Poisson regression model was applied to estimate relativ
114 significant overdispersion (invalidating the Poisson regression model) and residual autocorrelation (
127 uated this outcome as a discrete variable in Poisson regression models and as a categorical variable
128 cific associations using confounder-adjusted Poisson regression models and pooled the city-specific e
134 P. vivax parasite prevalence, and multilevel Poisson regression models showed that such differences w
135 ere is a well-established association) using Poisson regression models that controlled for shared sea
137 HIV-infected and -uninfected children using Poisson regression models that incorporated HIV prevalen
139 ated, followed by bivariate and multivariate Poisson regression models to assess the relationship bet
145 and pandemic) and RSV infection by applying Poisson regression models to monthly all-respiratory and
152 nt discharge data, multistate and log-linear Poisson regression models were used to calculate hospita
160 ed according to a multi-component scale, and Poisson regression models were used to examine associati
163 edding (VL > 40 copies/mL) were estimated by Poisson regression models with generalized estimating eq
183 tiveness was estimated by using multivariate Poisson regression models; effectiveness was allowed to
186 ents with the CC or CT genotype (2.4-fold by Poisson regression [P<.0001] and 2.7-fold based on mean
188 risk factor associations were determined by Poisson regression (plaque presence), negative binominal
189 motherapy for non-Hodgkin lymphoma (n = 158; Poisson regression Ptrend < .001), declined for ovarian
190 (CIMT) at baseline (2004) and used modified Poisson regression (robust error variance) to estimate p
192 5 years was investigated using multivariate Poisson regressions stratified according to initial Body
193 een cART regimens and KS using multivariable Poisson regression, stratified or adjusted for timing ar
196 After adjusting for confounding factors in Poisson regression, the relationship between linezolid u
197 als (CI) were estimated using random effects Poisson regression to account for clustering within gene
204 omist-drawn blood cultures was modeled using Poisson regression to compare the 12-month intervention
207 te of the first offered appointment; we used Poisson regression to compare the proportion of women wh
208 idence rate ratios (IRRs) were calculated by Poisson regression to determine differences in GW rates
213 or decompensations, excluding HCC) and used Poisson regression to estimate incidence rate ratios.
218 infection with the general population, used Poisson regression to evaluate anal cancer incidence amo
219 in number of deaths and place of death, and Poisson regression to evaluate factors associated with c
220 onfidence intervals were calculated by using Poisson regression to evaluate lifetime use of 48 pestic
222 infants using propensity scores, and we used Poisson regression to examine the effect of postnatal CM
226 We performed a time-series analysis using Poisson regression to relate monthly CFP call incidence
227 rental educational and employment status, by Poisson regression, to compare individuals with and with
234 eometric mean reproductive hormones, whereas Poisson regression was used to assess risk of sporadic a
235 l intake and hormone concentrations, whereas Poisson regression was used to assess RR of cycle-averag
242 accounting for the competing risk of death; Poisson regression was used to compare rates of NCD occu
255 d-lag nonlinear modeling integrated in quasi-Poisson regression was used to examine the exposure-lag-
275 Incidence rate ratios were calculated using Poisson regressions while adjusting for sociodemographic
277 isorder outcome were estimated by log linear Poisson regression with adjustments for the calendar per
280 and were related to SGA risk with the use of Poisson regression with confounder adjustment; linear sp
281 bic-restricted splines and multivariable log-Poisson regression with empirical standard errors were u
282 HIV incidence estimated using multivariable Poisson regression with generalized estimating equations
283 nce intervals were estimated from log-linked Poisson regression with generalized estimating equations
287 months after each of these were analysed by Poisson regression with invasive interval cancer screen
291 owth and obesity were assessed by linear and Poisson regression with robust standard errors, adjustin
293 012 and 2015 for several risk factors, using Poisson regression with robust variance and a bootstrap-
300 ces (IRDs) of condyloma were estimated using Poisson regression with vaccine dose as a time-dependent
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