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1 ge priming, in a process that exploits novel polycomb activities.
2 s, potentially accounting for enhancement of Polycomb activity, and suggesting that AEBP2 normally pl
3 ranscriptional modulator, which impacts both Polycomb and active gene transcription in mammalian cell
4 nd suggest a functional intersection between Polycomb and AR signaling in CRPC.
5 eukemias revealed that KDM2B cooperates with polycomb and trithorax complexes to regulate differentia
6 tic modifying genes, most prominently in the polycomb and trithorax families.
7 es) and variants in transcription, repressed Polycomb, and enhancer regions, as well as identify five
8 s, which target the 3' UTR of EZH2 and other Polycomb-associated transcripts.
9 ation, and the added repressive influence of polycomb at a subset of CG-dense cis-regulatory sequence
10            Here, we identified an additional Polycomb-binding lncRNA, COLDWRAP.
11 munication between PRC1 and PRC2, repressive Polycomb chromatin domains can erode, rendering target g
12 expression and function of the non-canonical polycomb complex 1 (PRC1) subunit RYBP.
13 Bayesian network analysis connects RBFox2 to Polycomb complex 2 (PRC2) and H3K27me3, and biochemical
14 X disrupts SWI/SNF complex inhibition of the polycomb complex 2 (PRC2) methyltransferase Enhancer of
15 amming and found that reducing levels of the polycomb complex gene Bmi1 significantly enhanced induct
16   In Arabidopsis AtVRN2 is a key member of a Polycomb complex involved in stable repression of Arabid
17                   One of the proteins of the Polycomb complex is the Ring finger protein 1 (RING1).
18                                          The Polycomb complex often binds to lncRNAs in eukaryotes, a
19                                   Though the polycomb complex proteins are thought to primarily regul
20                             We find that the polycomb complex proteins BMI1 and RING1A regulate the u
21                                          The polycomb complex proto-oncogene BMI1 [B lymphoma Mo-MLV
22 , which codes for a catalytic subunit of the polycomb complex.
23                                              Polycomb complexes are central to this memory, but many
24 is epigenetically silenced by the repressive Polycomb complexes in growing cells but is activated in
25 -directed chromatin recruitment of mammalian Polycomb complexes is a fundamental component of epigene
26                            Gene silencing by Polycomb complexes is central to eukaryotic development.
27 e repression, notably through recruitment of Polycomb complexes, and has long been considered essenti
28 s, suggesting cooperativity between Brg1 and Polycomb complexes.
29 for further analyses of the role of distinct Polycomb components in the control of gene expression pr
30 es, which involve specialization of distinct Polycomb components to deliver first metastable then lon
31 ed into three functionally distinct modules: Polycomb, Core, and Myc.
32 roliferation in the developing epidermis via Polycomb-dependent and -independent SUMO E3 ligase activ
33 rosophila and co-occupies PREs to antagonize Polycomb-dependent silencing.
34 lleles, as defined by differential levels of Polycomb-dependent trimethylation of histone H3 Lys27 (f
35  LIKE HETEROCHROMATIN PROTEIN 1 (LHP1) binds Polycomb-deposited H3K27me3 and is required for repressi
36  study identifies a novel mechanism by which Polycomb directs the developmental process by activating
37 blish precedence for H2AK119u1 in initiating Polycomb domain formation in a physiological context.
38 ions is critical for the formation of stable Polycomb domains at target gene loci.
39 mpeting forces, such as the self-assembly of polycomb domains within the nucleus.
40  links PRC1 and PRC2 in the establishment of Polycomb domains.
41  roles via altering nucleosome stability, at polycomb-enriched bivalent genes the same remodellers ac
42 mity assay, we find that BAF directly evicts Polycomb factors within minutes of its occupancy, thereb
43 stable epigenetic silencing through separate Polycomb factors, which spread across the locus after co
44 ite instability, IDH1 mutation, 19p gain and polycomb features, and backbone demethylation with chrom
45 ex combs (Asx), a regulator of trithorax and polycomb function in Drosophila.
46  phenotype, normally linked to antagonism of Polycomb function.
47 estion of how noncoding RNAs are involved in Polycomb group (PcG) and Trithorax group (TrxG) regulati
48 upports a model in which CpG islands recruit Polycomb group (PcG) complexes; however, which subset of
49 ed by epigenetic regulators belonging to the Polycomb Group (PcG) protein family.
50 ere found most often in association with the polycomb group (PcG) protein Polycomb (Pc), a subunit of
51                                              Polycomb Group (PcG) proteins are epigenetic repressors
52                                              Polycomb group (PcG) proteins are responsible for mainta
53                                              Polycomb Group (PcG) proteins assemble a chromatin state
54  sheds light on how nuclear organization and Polycomb group (PcG) proteins contribute to epigenetical
55                                              Polycomb group (PcG) proteins function as chromatin-base
56                                          The Polycomb group (PcG) proteins modulate nucleosomes to ma
57   Regulatory decisions in Drosophila require Polycomb group (PcG) proteins to maintain the silent sta
58 regulation is the balanced activities of the Polycomb group (PcG) proteins within the PRC1 and PRC2 c
59 h INK4A and MIR31HG genomic regions and with Polycomb group (PcG) proteins, and that MIR31HG is requi
60 that regulate genome architecture, including Polycomb group (PcG) proteins, form subnuclear structure
61 s regulated gene silencing is carried out by Polycomb group (PcG) proteins, which must be correctly r
62 d Pol II and enriched with promoter-proximal Polycomb Group (PcG) proteins, yet lacking the classical
63 l modulator that maintains downregulation of Polycomb Group (PcG) target genes in lhp1 mutants, while
64 latory genes require stable silencing by the polycomb group (PcG) to prevent misexpression during dif
65  this screen, CAT7, regulates expression and polycomb group binding at the MNX1 locus during early ne
66      Here we report that Piwi interacts with Polycomb group complexes PRC1 and PRC2 in niche and germ
67 m chromatin and found candidates that impact polycomb group protein (PcG)-regulated gene expression i
68 ever, constitutive repression of CCN3 by the Polycomb group protein EZH2 disrupted this negative feed
69 ogenesis is to suppress transcription of the Polycomb group protein, EZH2, thereby de-repressing gene
70                                              Polycomb group proteins (PcG) are transcriptional repres
71                                              Polycomb group proteins and associated histone marks are
72                                          The Polycomb group proteins and the associated H3K27me3 hist
73                                          The Polycomb group proteins are transcriptional repressors t
74  miR-16 levels down-regulated BMI1 and other polycomb group proteins like RING1A, RING1B, EZH2 and al
75                                              Polycomb Group regulation in Arabidopsis (Arabidopsis th
76          Here, we show that the noncanonical Polycomb group RING finger 3/5 (PCGF3/5)-PRC1 complex in
77        Here, we show that the PRC1 component polycomb group ring finger 6 (Pcgf6) is required to main
78 using on promoter CpG sites that localize to Polycomb group target genes that are unmethylated in 11
79  effects on gene expression, particularly of polycomb group target genes.
80                                          The Polycomb group transcriptional repressor Bmi-1 often ove
81  mutations affect BAP1 interactions with the Polycomb group-like protein, ASXL2, using combinations o
82 alysis of Polycomblike revealed that loss of Polycomb group-mediated repression of the Hox gene Abdom
83 ansitions between trithorax-group (TrxG) and polycomb-group (PcG) chromatin states are vital for the
84                                              Polycomb-group (PcG) proteins function to ensure correct
85 -binding of histone deacetylases (HDACs) and Polycomb-group (PcG) proteins.
86                                              Polycomb-group proteins control many fundamental biologi
87 eversible chromatin interactions mediated by Polycomb-group proteins play an important role in these
88 tioning in Arabidopsis thaliana We show that Polycomb-group proteins repress nucellus degeneration be
89 ppression as an alternative to regulation by Polycomb-group proteins, which coordinate embryonic germ
90 rast, regions associated with the repressive Polycomb-Group showed low turnover in ESCs.
91 silenced in the T-cell lineage, and that the polycomb H3K27me3-repressive mark is focally diminished
92 nescence is epigenetically controlled by the polycomb histone methyltransferase enhancer of zeste hom
93 gene repression, highlighting a key role for Polycomb in Xist-dependent chromosome silencing.
94                  Here we define the Xist RNA Polycomb Interaction Domain (XR-PID), a 600 nt sequence
95                                              Polycomb-like (PCL) proteins, such as PHF1, MTF2 and PHF
96        We find that intragenic levels of the polycomb mark H3K27me3 anti-correlate with changes in tr
97 chromatin changes include initial repressive polycomb marking of genes, later manifesting abnormal DN
98 to find that actively transcribed genes with Polycomb marks have greater cell-to-cell variation in ex
99 enovirus E1A, modulate cellular responses to polycomb-mediated epigenetic methylations in human papil
100 NG 6 (REF6, also known as JMJ12) counteracts Polycomb-mediated gene silencing by removing methyl grou
101 hanism of an H3K27 demethylase to counteract Polycomb-mediated gene silencing that regulates plant de
102 ghlights that H3K9me3 and, most importantly, polycomb-mediated H3K27me3 chromatin pathways can secure
103           These data extend the knowledge of polycomb-mediated regulation of gene expression to endot
104  MADS box transcription factor relieves this Polycomb-mediated repression and therefore allows nucell
105 spread enhancer activation and opposition of Polycomb-mediated repression at bivalent promoters.
106             In conclusion, MDM2 supports the Polycomb-mediated repression of lineage-specific genes,
107            Brg1 is also required to maintain Polycomb-mediated repression of non-mesodermal developme
108 on through pharmacological inhibition of the polycomb-mediated repression system.
109 hed by increased CG dinucleotide density and polycomb-mediated repression, manifested by the addition
110 ation was not dependent upon the presence of polycomb-mediated repression.
111 tone H3 at lysine 27 (H3K27ac), which blocks Polycomb-mediated trimethylation of H3K27 (H3K27me3).
112                                          The Polycomb module represses developmental genes, while the
113 zes at chromatin with members of the Myc and Polycomb module.
114    To identify novel factors involved in the Polycomb pathway in plants, we performed a forward genet
115  epigenomic pathway in which reversal of the Polycomb pathway of repressive histone methylation is re
116 iation with the polycomb group (PcG) protein Polycomb (Pc), a subunit of polycomb repressive complex
117  development is subject to regulation by the Polycomb (Pc), Trithorax (Trx), and Compass chromatin mo
118                                          The Polycomb PRC1 plays essential roles in development and d
119 ically, VEC exerts this effect by inhibiting polycomb protein activity on the specific gene promoters
120              Disruption of gene silencing by Polycomb protein complexes leads to homeotic transformat
121 rate that a specific interaction between the polycomb protein EZH2 and RNA made from B2 SINE retrotra
122  this issue, Zovoilis et al. report that the polycomb protein EZH2, upon heat shock, facilitates tran
123                                              Polycomb proteins are critical chromatin modifiers that
124                                              Polycomb proteins are involved in specific transcription
125 e ribosomal serine/threonine kinase S6K1 and Polycomb proteins at genomic regulatory regions to repre
126                                              Polycomb proteins interact with each other to form chrom
127 , we identified expression of BMI1 and other Polycomb proteins not previously identified in olfactory
128 n, in this study, we discover that these two polycomb proteins regulate the transcription of active g
129 remodeling and carcinogenesis, together with Polycomb proteins.
130 tion according to which, in the naive state, polycomb recessive complex 2 repressed the IRAK-M promot
131 27 (H3K27) methyltransferase and part of the polycomb recessive complex 2, enhancer of Zeste 2, resul
132    Our findings overturn existing models for Polycomb recruitment by Xist RNA and establish precedenc
133        Our findings define a key pathway for Polycomb recruitment by Xist RNA, providing important in
134                                              Polycomb recruitment is initiated by the PCGF3/5-PRC1 co
135  Deletion of XR-PID abolishes Xist-dependent Polycomb recruitment, in turn abrogating Xist-mediated g
136 king XR-PID is sufficient for Xist-dependent Polycomb recruitment.
137 , inducing both repression and activation of polycomb-regulated loci.
138 h as super-enhancers, bivalent promoters and Polycomb repressed regions, and identify additional patt
139 ucleosome-Secured Regions (NSRs) occupied by polycomb-repressed chromatin played a role in repressing
140 ptionally active and inactive chromatin, the Polycomb-repressed domains are characterized by a high d
141                                              Polycomb-repressed domains show the densest packing and
142      Moreover, compared to inactive domains, Polycomb-repressed domains spatially exclude neighbourin
143 rs in the dorsal ectoderm is associated with Polycomb-repressed H3K27me3 chromatin.
144                                              Polycomb-repressed Hox domains are shown by the same app
145 s into transcriptionally active, inactive or Polycomb-repressed states, and observed distinct chromat
146 uction of de novo superenhancers antagonizes Polycomb repression and superimposes aberrant stem-epith
147            Our findings identify reversal of polycomb repression as a key step in gene activation aft
148 Enhancer of Zeste [E(z)], a component of the Polycomb repression complex 2 (PRC2), is required to mai
149 rikingly insensitive to OGA RNAi at sites of polycomb repression such as the Hox and NK homeobox gene
150 ylation convergently impacted Wnt signaling, Polycomb repression, innate immune cell interactions and
151                                              Polycomb Repressive Complex (PRC) 2 catalyzes the H3K27m
152                        The dependency on the polycomb repressive complex (PRC2) and EZH2 represents o
153              BMI1 is a core component of the polycomb repressive complex 1 (PRC1) and emerging data s
154                  In multicellular organisms, Polycomb Repressive Complex 1 (PRC1) and PRC2 repress ta
155 egration site 1 (BMI1) is a component of the polycomb repressive complex 1 (PRC1) complex that is ove
156 e of these sites are binding sites for other Polycomb repressive complex 1 (PRC1) components, others
157                   The essential functions of polycomb repressive complex 1 (PRC1) in development and
158                                          The polycomb repressive complex 1 (PRC1) is a multi-subunit
159 tone H2AK119 E3 ubiquitin ligase activity of Polycomb repressive complex 1 (PRC1) is defined by the c
160                                          The polycomb repressive complex 1 (PRC1), containing the cor
161           CBX2, a component of the mammalian Polycomb repressive complex 1 (PRC1), contains a compact
162 We identified UbE2E1 as a novel component of Polycomb repressive complex 1 (PRC1), the E3 ligase comp
163                                  Subunits of polycomb repressive complex 1 (PRC1), the major histone
164                   BMI1 is a component of the Polycomb Repressive Complex 1 (PRC1), which plays a key
165 fferent CBX proteins associate with the core Polycomb repressive complex 1 (PRC1).
166 up (PcG) protein Polycomb (Pc), a subunit of polycomb repressive complex 1 (PRC1).
167 corresponded to locations bound by canonical polycomb repressive complex 1 (PRC1).
168 that Cbx4/2, Rybp, and Ring1B [components of polycomb repressive complex 1 (PRC1)] are predominantly
169 K27 methylation leads to a redistribution of polycomb repressive complex 1 and de-repression of its t
170 ation is linked to the silencing of Scml2, a Polycomb Repressive Complex 1 protein that drives loss o
171 that recognizes CpG islands and recruits the polycomb repressive complex 1.
172 ation blocks nucleation of plant homeodomain-Polycomb repressive complex 2 (PHD-PRC2) and indicates a
173         In hepatocytes, EpCAM is silenced by polycomb repressive complex 2 (PRC2) and ZNF198/LSD1/Co-
174 ermethylated phenotype, mainly occupying the polycomb repressive complex 2 (PRC2) binding sites in hu
175                                              Polycomb repressive complex 2 (PRC2) catalyzes histone H
176                                              Polycomb Repressive Complex 2 (PRC2) catalyzes the trime
177 ds to the transient recruitment of EZH2, the polycomb repressive complex 2 (PRC2) component responsib
178 referential dependency on genes encoding the polycomb repressive complex 2 (PRC2) components EZH2, EE
179                                              Polycomb repressive complex 2 (PRC2) controls maintenanc
180                                              Polycomb repressive complex 2 (PRC2) has been shown to p
181                                              Polycomb repressive complex 2 (PRC2) is a histone methyl
182                                              Polycomb repressive complex 2 (PRC2) is a key chromatin
183                                              Polycomb repressive complex 2 (PRC2) is an essential reg
184                                              Polycomb repressive complex 2 (PRC2) is responsible for
185                                          The Polycomb repressive complex 2 (PRC2) mainly mediates tra
186                                              Polycomb repressive complex 2 (PRC2) mediates gene silen
187                                              Polycomb repressive complex 2 (PRC2) methylates lysine 2
188                                              Polycomb repressive complex 2 (PRC2) modifies chromatin
189                                              Polycomb repressive complex 2 (PRC2) participates in tra
190 lycomb response elements (PREs), that direct Polycomb repressive complex 2 (PRC2) placement at develo
191 lly considered to promote transcription, and Polycomb Repressive Complex 2 (PRC2) places K27me3, whic
192  repressive complex 2 in cell fate decisions.Polycomb repressive complex 2 (PRC2) plays an essential
193                                          The polycomb repressive complex 2 (PRC2) regulates different
194                                              Polycomb repressive complex 2 (PRC2) silences gene expre
195 , but there is a clear lack of the essential Polycomb Repressive Complex 2 (PRC2) subunit EED in the
196  enzymes that catalyze H3K27me3, namely, the polycomb repressive complex 2 (PRC2) subunits enhancer o
197 omologue 2 or 1) is the catalytic subunit of polycomb repressive complex 2 (PRC2) that catalyzes meth
198 histone methyltransferase is a member of the polycomb repressive complex 2 (PRC2) that is highly expr
199  represses SMN2 expression by recruiting the Polycomb Repressive Complex 2 (PRC2) to its locus.
200 ctivity-based communication between PRC1 and Polycomb repressive complex 2 (PRC2) which is required f
201 y activating specific genes, silenced by the Polycomb repressive complex 2 (PRC2), and identify the c
202 etermined to have physical interactions with Polycomb repressive complex 2 (PRC2), and systematically
203 homologue 2 (EZH2), the catalytic subunit of Polycomb Repressive Complex 2 (PRC2), contributes to mai
204 MLL1 and H3K4me2, H3K27me3 levels, a mark of Polycomb repressive complex 2 (PRC2), increase at these
205 e 12 homologue (SUZ12), which is a domain of Polycomb repressive complex 2 (PRC2), promoting the meth
206 er of zeste homolog 2 (EZH2), a component of polycomb repressive complex 2 (PRC2), to inhibit c-Met e
207 issociation of EZH2 and SUZ12, components of polycomb repressive complex 2 (PRC2), transcriptional ac
208                            Here we show that polycomb repressive complex 2 (PRC2), which supports neu
209            Here, we show that, in mESCs, the Polycomb repressive complex 2 (PRC2)-associated protein
210 ZH2 exerts its oncogenic function is through polycomb repressive complex 2 (PRC2)-mediated gene repre
211  accompanied by a reduction in the levels of polycomb repressive complex 2 (PRC2)-mediated H3K27 trim
212 G1, was previously known for its function in polycomb repressive complex 2 (PRC2)-mediated transcript
213 ML, via transcription repression mediated by Polycomb repressive complex 2 (PRC2).
214                                              Polycomb repressive complex 2 (PRC2-EZH2) methylates his
215                                              Polycomb repressive complex 2 and the epigenetic mark th
216                                              Polycomb repressive complex 2 deficiency impaired clonal
217               Here, we show that a subset of Polycomb repressive complex 2 factors nucleate silencing
218 suggested that in the crystal structure of a polycomb repressive complex 2 from Chaetomium thermophil
219 the crystal structure of the protein complex polycomb repressive complex 2 from Chaetomium thermophil
220              Here we report that knockout of polycomb repressive complex 2 genes in human embryonic s
221 age- and pluripotent state-specific roles of polycomb repressive complex 2 in cell fate decisions.Pol
222 opmental transcription factor genes bound by Polycomb repressive complex 2 in human embryonic stem ce
223  the roles of the distinct components of the Polycomb repressive complex 2 in the control of skin dev
224                                 In contrast, polycomb repressive complex 2 is dispensable for pluripo
225 androgen receptor signaling and induction of Polycomb Repressive Complex 2 signaling.
226 atalytic activity of the enhancer of zeste 2 polycomb repressive complex 2 subunit (EZH2), forming re
227 the conditional ablations of three essential Polycomb repressive complex 2 subunits (EED, Suz12, or E
228 e homolog 2 (EZH2), the catalytic subunit of polycomb repressive complex 2 that mediates histone H3 l
229  KSHV genome and contributed to tethering of polycomb repressive complex 2 through physical interacti
230  of H3K27 methyltransferases or of the PRC2 (Polycomb Repressive Complex 2) by pharmaceutical inhibit
231 of zeste homolog 2, the catalytic subunit of polycomb repressive complex 2, and MEF2C-dependent gene
232  lncOL1 interacts with Suz12, a component of polycomb repressive complex 2, to promote oligodendrocyt
233 d Ezh1/2 function largely as subunits of the Polycomb repressive complex 2, which is important in the
234 RC2)-associated protein EPOP (Elongin BC and Polycomb Repressive Complex 2-associated protein; a.k.a.
235                         We further show that polycomb repressive complex 2-deficient mouse embryonic
236    These results demonstrate a dual role for polycomb repressive complex 2-mediated epigenetic silenc
237 without invoking the increased enrichment of Polycomb Repressive Complex 2.
238  including homeobox genes and targets of the Polycomb repressive complex 2.
239                    Interestingly, targets of polycomb repressive complex in stem cells were mostly af
240                It is likely that the role of Polycomb repressive complex is to dampen expression of t
241     EZH2 is a component of the multi-subunit polycomb repressive complex PRC2, and the down-regulatio
242  and beta-catenin, which contribute to PRC2 (polycomb repressive complex-2) binding to promoter regio
243  SMN expression by recruiting the epigenetic Polycomb repressive complex-2.
244                                 We find that Polycomb repressive complex-active genes have greater ce
245 to S-nitrosylate RING1A, a key member of the polycomb repressive complex.
246 with LIKE HETEROCHROMATIN PROTEIN1 (LHP1), a Polycomb Repressive Complex1 (PRC1) subunit.
247                                   We studied POLYCOMB REPRESSIVE COMPLEX2 (PRC2) in Brassicaceae.
248  zeste 12 homolog (SUZ12), a core subunit of Polycomb repressive complex2 (PRC2), undergoes proteasom
249                                              Polycomb repressive complexes (PRC) are frequently impli
250                       The opposition between Polycomb repressive complexes (PRCs) and BAF (mSWI/SNF)
251                                              Polycomb repressive complexes (PRCs) are important histo
252                                              Polycomb repressive complexes (PRCs) are key to normal d
253 mplexes, are known to oppose the activity of Polycomb repressive complexes (PRCs).
254 m chromatin-associated repressive complexes (Polycomb repressive complexes 1 and 2) leading to chroma
255 r regulation of pluripotency and development.Polycomb repressive complexes modify histones but it is
256                                          The Polycomb repressive complexes PRC1 and PRC2 are key medi
257                           Recruitment of the Polycomb repressive complexes PRC1 and PRC2 by Xist RNA
258                                          The Polycomb repressive complexes PRC1 and PRC2 play a centr
259      This is followed by dissociation of the polycomb repressive complexes, including the H3K27 methy
260 iated, at least in part, by cooperation with Polycomb repressive complexes.
261 ysine 119 (H2AK119Ub1) as a component of the Polycomb repressive deubiquitination (PR-DUB) complex.
262                            Here we show that Polycomb-repressive complex 1 (PRC1) directs timely acti
263 mplex (referred to as the DOT1L complex) and polycomb-repressive complex 1 (PRC1).
264 x (CBX)-containing proteins CBX4 and CBX6 of polycomb-repressive complex 1 through the removal of K48
265 rotease 26 in cellular reprogramming through polycomb-repressive complex 1.The ubiquitin-proteasome s
266 Cdkn2a locus and directly interacts with the Polycomb-repressive complex 2 (PRC2) to enable H3K27me3-
267 omolog 2 (EZH2), an enzymatic subunit of the polycomb-repressive complex 2 and the main writer of chr
268                                          The Polycomb-repressive complexes PRC1 and PRC2 play a key r
269 ation and gene silencing, as a member of the polycomb repressor 2 (PRC2) complex.
270 es also responded to the inactivation of the Polycomb Repressor Complex 2 (PRC2) and its catalytic co
271                                              Polycomb repressor complex 2 (PRC2) places the histone m
272                 Jarid2 is a component of the Polycomb Repressor complex 2 (PRC2), which is responsibl
273 pressed H3K27Me3 and effectively reactivated polycomb repressor complex 2 (PRC2)-silenced tumor suppr
274 ctly interacts with the catalytic subunit of polycomb repressor complex 2 (PRC2).
275            ZIC2 interacts with and maintains polycomb repressor complex 2 at the K-Rta promoter.
276 2, a histone methyl transferase subunit of a Polycomb repressor complex, is recurrently mutated in se
277                        Here we show that the polycomb repressor protein Bmi1 marks a population of ca
278                                              Polycomb response elements (PREs) are specific DNA seque
279  we define short genomic fragments, known as Polycomb response elements (PREs), that direct Polycomb
280 b is required for targeting PRC complexes to Polycomb Response Elements (PREs).
281 ted to DNA via cis-acting elements known as "Polycomb response elements" (PREs).
282  TBP-bound promoters along with promoters of polycomb-silenced genes apparently lacking TBP.
283 ver, the mechanisms underlying opposition to Polycomb silencing are poorly understood.
284                                              Polycomb silencing at FLC thus has mechanistically disti
285 rminants that specify the genomic targets of Polycomb silencing complexes are still unclear.
286 results indicate that vernalization-mediated Polycomb silencing is coordinated by lncRNAs in a cooper
287                                              Polycomb silencing of Arabidopsis FLOWERING LOCUS C (FLC
288       Here we demonstrate that ENL overcomes polycomb silencing through recruitment of PAF1 via the c
289 unity to study initiation and maintenance of Polycomb silencing.
290    In this issue, Mardaryev et al. show that Polycomb subunit Cbx4 acts downstream of transcriptional
291 identify important methylated genes (such as polycomb target genes and genes involved in transcriptio
292 27me3 and is required for repression of many Polycomb target genes in Arabidopsis Here we show that L
293 RNA binding of PRC2, and their enrichment at Polycomb target genes provides a means for RNA-mediated
294 's developmental trajectory or identity as a polycomb target.
295 pathway, and key driver analyses showed that Polycomb targets contribute significantly to the non-gen
296 ng astrocytes identifies changes at multiple polycomb targets, including the promoter of Foxo3 In pat
297 se regions and alleviates repression of some polycomb telomeric genes.
298 tes, and an lncRNA, COLDAIR, associates with Polycomb to mediate silencing of the floral repressor FL
299                      These observations link polycomb with dynamic changes in transcription and stall
300  complex recruitment is sufficient to oppose Polycomb within minutes, leading to activation of bivale

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