ライフサイエンスコーパス: Porphyromonas

1 Treponema (34%), Mycoplasma (29%) and Porphyromonas (15%) were the most abundant genera associ

2 25% x 20%), Streptococcus (19.83% x 17.61%), Porphyromonas (3.63% x 1.42%, P = 0.03), Treponema (1.02

3 ncreased numbers of the following anaerobes: Porphyromonas (5.2 x 10(7) 16S rRNA gene copies/swab in

4 sequences in the genera Bacteroidetes [G-3], Porphyromonas, Abiotrophia, Filifactor, Peptostreptococc

5 These clusters are driven by Porphyromonas and Fusobacterium genera in chimpanzees an

6 II), Staphylococcus epidermidis (type III), Porphyromonas and Peptoniphilus species (type IV), and P

7 eolytic bacteria such as Peptostreptococcus, Porphyromonas and Prevotella.

8 d Finegoldia, Parvimonas, Peptoniphilus, and Porphyromonas and reduced Butyricicoccus and Faecalibact

9 Porphyromonas and Solobacterium showed significantly hig

10 and Alloprevotella, Leptotrichia, Neisseria, Porphyromonas, and Prevotella.

11 etes increases the levels of Capnocytophaga, Porphyromonas, and Pseudomonas, while Prevotella and Sel

12 onic acid generating bacteria from the genus Porphyromonas, and that this may explain adolescent/adul

13 In 1 index sample, routine culture found Porphyromonas endodontalis and Streptococcus intermedius

14 manifestation of endodontic infection where Porphyromonas endodontalis is frequently encountered.

15 tophaga granulosa, G. morbillorum, P. micra, Porphyromonas endodontalis, Streptococcus spp., and Tann

16 bacterium fastidiosum, Eubacterium saphenum, Porphyromonas endodontalis, Treponema medium, Synergiste

17 both multiple group testing and regression, Porphyromonas (FDR p-value = 0.02), Prevotella (FDR p-va

18 er actinomycetemcomitans (MT4/MSP: 42%/36%), Porphyromonas gingivalis (78%/66%), Tannerella forsythia

19 g a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingivalis JCVI SC001) usin

20 nts with chronic periodontitis and intraoral Porphyromonas gingivalis (P. gingivalis) and Tannerella

21 analogs was synthesized and their effects on Porphyromonas gingivalis (P. gingivalis) elicited inflam

22 dontal health by investigating its effect on Porphyromonas gingivalis (P. gingivalis), lipopolysaccha

23 Tannerella forsythia (T.f.), 2.5 x 10(4) for Porphyromonas gingivalis (P.g.), 5.3 x 10(3) for Prevote

24 Porphyromonas gingivalis (P.g.)-induced TNF-alpha can be

25 an and mouse macrophages exposed to LPS from Porphyromonas gingivalis (Pg LPS).

26 ucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and Tannerella forsythia,

27 nis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (Pg) biofilms elicited signific

28 Porphyromonas gingivalis (Pg) capsule enables evasion fr

29 LM group also presented greater reduction of Porphyromonas gingivalis (Pg) DNA counts at 6 months (P

30 This study compared the effect of Porphyromonas gingivalis (Pg) infection and experimental

31 Porphyromonas gingivalis (Pg) is a keystone pathogen in

32 Porphyromonas gingivalis (Pg) is a major periodontal pat

33 or TRAM, HGF and HPDLF were stimulated with Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS) o

34 n vitro study examines the effect of EGCG on Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS)-e

35 6 cells were used to determine the effect of Porphyromonas gingivalis (Pg) LPS on insulin secretion.

36 al administration of a periodontal pathogen, Porphyromonas gingivalis (Pg) to WT mice results in insu

37 The interaction of the periodontal pathogen Porphyromonas gingivalis (Pg) with commensal streptococc

38 Quantification of Tannerella forsythia (Tf), Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

39 Total amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

40 Aggregatibacter actinomycetemcomitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr)

41 ates biofilms, consisting of species such as Porphyromonas gingivalis (Pg), in the etiology of peri-i

42 were used for analysis of bacterial DNA for Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

43 y associated with the submucosal presence of Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

44 Quantitative analysis of Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

45 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

46 study investigated the role of GRHL2 in the Porphyromonas gingivalis (Pg)-induced impairment of epit

47 tis (RA), specifically regarding the role of Porphyromonas gingivalis (Pg).

48 by ligature; 3) group G-Pg: oral gavage with Porphyromonas gingivalis (Pg); 4) group G-PgFn: oral gav

49 AW264.7 and human monocyte THP-1 to LPS from Porphyromonas gingivalis (PgLPS), an oral microbe implic

50 n 9 times with 10(9) colony-forming units of Porphyromonas gingivalis A7436 through an oral gavage mo

51 he chronic periodontitis-associated pathogen Porphyromonas gingivalis activates a Toll-like receptor

52 issue-degrading enzymes in the oral pathogen Porphyromonas gingivalis Although a number of subunits o

53 re Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis amounts in saliva and their ant

54 killing of periodontal pathogens, including Porphyromonas gingivalis and Aggregatibacter actinomycet

55 Porphyromonas gingivalis and Aggregatibacter actinomycet

56 iovascular disease, such as the link between Porphyromonas gingivalis and atherosclerosis.

57 54, are produced by the periodontal pathogen Porphyromonas gingivalis and can be detected in lipid ex

58 Porphyromonas gingivalis and especially Prevotella inter

59 Porphyromonas gingivalis and Filifactor alocis are fasti

60 ring inhibition of the periodontal pathogens Porphyromonas gingivalis and Fusobacterium nucleatum gro

61 odontitis was induced by oral inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in

62 ) on the virulence of a mixed infection with Porphyromonas gingivalis and Fusobacterium nucleatum in

63 Although not statistically significant, Porphyromonas gingivalis and Fusobacterium nucleatum occ

64 Periodontal pathogens such as Porphyromonas gingivalis and Fusobacterium nucleatum pro

65 ribosomal RNA analysis of keystone pathogen Porphyromonas gingivalis and its consortium members Fuso

66 se in 2 animal models through inoculation of Porphyromonas gingivalis and ligature around the murine

67 Mice were orally infected with human Porphyromonas gingivalis and observed for 42 days.

68 mDC microbiome by 16S rDNA sequencing showed Porphyromonas gingivalis and other species, including (c

69 inulin and whether LS inhibits the growth of Porphyromonas gingivalis and Prevotella intermedia.

70 d oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevotella nigrescens induc

71 The Orange-Red cluster score (that included Porphyromonas gingivalis and Prevotella spp.) was positi

72 d Treponema denticola, and the prevalence of Porphyromonas gingivalis and T. denticola associated sig

73 ted with periodontal pathology and number of Porphyromonas gingivalis and Tannerella forsythia (forme

74 tus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tannerella forsythia), and

75 es that gram-negative oral bacteria, such as Porphyromonas gingivalis and Tannerella forsythia, use d

76 The keystone pathogen Porphyromonas gingivalis and the accessory pathogen Stre

77 The periodontal pathogen Porphyromonas gingivalis and the endodontic species Ente

78 modes of mucosal vaccination with whole-cell Porphyromonas gingivalis and to test the role of various

79 The recolonization of Porphyromonas gingivalis and Treponema denticola was red

80 how galactose-inhibitable coaggregation with Porphyromonas gingivalis and were defective in cell bind

81 Anti-Porphyromonas gingivalis antibody testing was performed

82 , FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are responsible for adherence t

83 rial manipulation of neutrophil responses by Porphyromonas gingivalis as a mechanism that contributes

84 rated that antibodies raised in mice against Porphyromonas gingivalis caused fetal loss in a mouse pr

85 The gram-negative anaerobe Porphyromonas gingivalis colonizes the gingival crevice

86 in substrates of a novel secretion system of Porphyromonas gingivalis contain a conserved C-terminal

87 Porphyromonas gingivalis contains lipid A with structura

88 A modest effect of DHA + aspirin on Porphyromonas gingivalis counts was associated with 14%

89 echanism by which the opportunistic pathogen Porphyromonas gingivalis dampens innate immune responses

90 ignificantly increased in the RA group, only Porphyromonas gingivalis displayed significant correlati

91 human gingival epithelial (HGEp) cells with Porphyromonas gingivalis disrupts barrier function by in

92 We show here that Porphyromonas gingivalis Dps (PgDps), previously describ

93 0068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gingivalis DSM20709) were placed in a seri

94 periodontitis caused by oral infection with Porphyromonas gingivalis enhances articular bone loss.

95 that infection with the periodontal pathogen Porphyromonas gingivalis enhances the activity of Janus

96 ndard chow diet were given an oral gavage of Porphyromonas gingivalis for 15 wk.

97 Additionally, we compare a nearly complete Porphyromonas gingivalis genome to previously published

98 Encapsulation of Porphyromonas gingivalis has been demonstrated as respon

99 Porphyromonas gingivalis has orthologs for each of these

100 of Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis have been shown to induce diffe

101 dy, we hypothesized that histatin 5 binds to Porphyromonas gingivalis hemagglutinin B (HagB) and atte

102 ed/attenuated the CXCL1 and TNF responses to Porphyromonas gingivalis hemagglutinin B (HagB).

103 of IL-6 by whole blood cells stimulated with Porphyromonas gingivalis HmuY protein.

104 the mechanism of action of ROS stimulated by Porphyromonas gingivalis in gingival epithelial cells.

105 ion, immunoglobulin (Ig)G subclasses against Porphyromonas gingivalis in individuals with pre-RA and

106 ne the impact of RvE1 on the phagocytosis of Porphyromonas gingivalis in T2D.

107 Prevotella intermedia and Porphyromonas gingivalis increased in the control group.

108 wild-type and Msx2 knock-in Swiss mice using Porphyromonas gingivalis infected ligatures.

109 Porphyromonas gingivalis is a Gram-negative obligate ana

110 Porphyromonas gingivalis is a Gram-negative obligately a

111 The oral bacterium Porphyromonas gingivalis is a key etiological agent of h

112 vity by the Gram-negative bacterial pathogen Porphyromonas gingivalis is a key event in the initiatio

113 Porphyromonas gingivalis is a keystone pathogen of perio

114 Porphyromonas gingivalis is a keystone pathogen that con

115 Porphyromonas gingivalis is a keystone pathogen, and its

116 Porphyromonas gingivalis is a keystone periodontal patho

117 Porphyromonas gingivalis is a major pathogen in periodon

118 Porphyromonas gingivalis is a member of the dysbiotic or

119 Porphyromonas gingivalis is a member of the human oral m

120 Porphyromonas gingivalis is a peptide-fermenting asaccha

121 Porphyromonas gingivalis is a periodontal pathogen impli

122 Porphyromonas gingivalis is a predominant pathogen that

123 Porphyromonas gingivalis is a primary etiological agent

124 Porphyromonas gingivalis is able to invade and modulate

125 Porphyromonas gingivalis is an established pathogen in p

126 Overabundance of Porphyromonas gingivalis is associated with shorter surv

127 Porphyromonas gingivalis is considered a keystone pathog

128 Porphyromonas gingivalis is considered a keystone pathog

129 Porphyromonas gingivalis is intimately associated with p

130 rthermore, the mechanism of TLR induction by Porphyromonas gingivalis is investigated in human gingiv

131 Porphyromonas gingivalis is one of the oral microorganis

132 Porphyromonas gingivalis is strongly implicated in perio

133 Porphyromonas gingivalis is the major causative agent of

134 st response to lipopolysaccharide (LPS) from Porphyromonas gingivalis is unusual inasmuch as differen

135 In monoinfection, heat-killed Porphyromonas gingivalis led to augmented levels of IL-1

136 Porphyromonas gingivalis lipid A heterogeneity modulates

137 olated from C57BL/6J mice were cultured with Porphyromonas gingivalis lipopolysaccharide (LPS) and cy

138 ation and inflammatory gene expression using Porphyromonas gingivalis lipopolysaccharide (LPS) as a p

139 roducts (AGE) in the presence and absence of Porphyromonas gingivalis lipopolysaccharide (LPS) on IL-

140 erichia coli lipopolysaccharide (Ec-LPS) and Porphyromonas gingivalis lipopolysaccharide (Pg-LPS) sti

141 resence and absence of ultrapure or standard Porphyromonas gingivalis lipopolysaccharide (PgLPS), Pam

142 ived IFN-gamma in constitutively released or Porphyromonas gingivalis lipopolysaccharide (PgLPS)-stim

143 was induced via silk ligature placement with Porphyromonas gingivalis lipopolysaccharide injection in

144 a [TNF-alpha]), bacterial virulence factors (Porphyromonas gingivalis LPS) or a combination in a biom

145 y be caused by periodontal bacteria, such as Porphyromonas gingivalis Mast cells are sentinels at muc

146 ain colonization by the periodontal pathogen Porphyromonas gingivalis may link these two inflammatory

147 Recent reports indicate that Porphyromonas gingivalis mediates alveolar bone loss or

148 Although the periodontal pathogen Porphyromonas gingivalis must withstand high levels of n

149 Gram-negative, anaerobic periodontopathogen Porphyromonas gingivalis must withstand nitrosative stre

150 he present study is to analyze the effect of Porphyromonas gingivalis on differentiation of primary o

151 (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human macrophages.

152 ly reduced in both groups at 3 and 6 months, Porphyromonas gingivalis only in the test group.

153 esponsiveness of whole blood stimulated with Porphyromonas gingivalis or Escherichia coli LPS were mo

154 experimental periodontitis induced by either Porphyromonas gingivalis or ligature, gamma-proteobacter

155 tudy is to determine whether the presence of Porphyromonas gingivalis peptidylarginine deiminase (PPA

156 of periodontitis and the anaerobic bacterium Porphyromonas gingivalis plays a key role in driving chr

157 show that intraoral inoculation of mice with Porphyromonas gingivalis resulted in infection, alveolar

158 enotype was also observed following LPS from Porphyromonas gingivalis stimulation during osteogenic d

159 The Porphyromonas gingivalis strain ATCC 33277 (33277) and 3

160 ases and are more likely to be infected with Porphyromonas gingivalis than non-smokers.

161 ce factor secreted by the periodontopathogen Porphyromonas gingivalis that attacks host vasculature a

162 which activity was induced by infection with Porphyromonas gingivalis The expression of several miRNA

163 The adaptation of Porphyromonas gingivalis to H2O2-induced stress while in

164 biofilm formation promoted the tolerance of Porphyromonas gingivalis to oxidative stress under micro

165 SCs were seeded for 24 h and challenged with Porphyromonas gingivalis total protein extract (PgPE) (0

166 y, the abundance of the periodontal pathogen Porphyromonas gingivalis trended with higher risk of ESC

167 r 30 seconds and 1 minute, reducing, mainly, Porphyromonas gingivalis viability, with 2.78 and 1.7 lo

168 ic Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis viability.

169 The Porphyromonas gingivalis VimA protein has multifunctiona

170 Mice were infected with the oral pathogen Porphyromonas gingivalis W50 (P. gingivalis) in the maxi

171 Porphyromonas gingivalis W83 biofilm was cultured on tit

172 An inferior frequency detection of Porphyromonas gingivalis was observed in the PDT protoco

173 nd the growth of Streptococcus sanguinis and Porphyromonas gingivalis was significantly reduced in th

174 ll-characterized human periodontal pathogen, Porphyromonas gingivalis We found that oral mucosal LCs

175 ns Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis with extended NO-release kineti

176 We documented earlier that Porphyromonas gingivalis(Pg), keystone oral-pathobiont,

177 presence of specific bacterial species (i.e. Porphyromonas gingivalis) and their effects in immune re

178 en" where low-abundance microbial pathogens (Porphyromonas gingivalis) can orchestrate inflammatory d

179 tory stimulus (lipopolysaccharide [LPS] from Porphyromonas gingivalis) in a manner consistent with th

180 Culture supernatants of Porphyromonas gingivalis, a bacterium that secretes prot

181 Porphyromonas gingivalis, a Gram-negative asaccharolytic

182 Porphyromonas gingivalis, a key periodontal pathogen, is

183 erase Chain Reaction (qPCR) for detection of Porphyromonas gingivalis, a keystone pathogen and to ass

184 The peritoneal infection model using Porphyromonas gingivalis, a keystone pathogen for period

185 important role in eliciting inflammation to Porphyromonas gingivalis, a keystone pathogen in periodo

186 rted between RA and periodontal disease, and Porphyromonas gingivalis, a known driver of periodontiti

187 Porphyromonas gingivalis, a major etiologic agent of per

188 In contrast, infection with Porphyromonas gingivalis, a principal pathogen in chroni

189 nalysed in mice after oral administration of Porphyromonas gingivalis, a representative periodontopat

190 operties against three periodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemc

191 Levels of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

192 The susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

193 contamination with >/=3 specific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

194 Porphyromonas gingivalis, an asaccharolytic member of th

195 eases are critical virulence determinants of Porphyromonas gingivalis, an emerging Alzheimer's diseas

196 the proteases of Pseudomonas aeruginosa and Porphyromonas gingivalis, and enables its antimicrobial

197 uced the numbers of Streptococcus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, a

198 iodontitis is initiated by bacteria, such as Porphyromonas gingivalis, and is caused largely by host

199 tory responses to a keystone oral bacterium, Porphyromonas gingivalis, and restrains periodontal infl

200 early and late colonizer pathogens, such as Porphyromonas gingivalis, as the biofilm ages and period

201 , in particular, Fusobacterium nucleatum and Porphyromonas gingivalis, based on results from epidemio

202 gens (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Campylobacter rectus, and Tann

203 the periodontitis-associated oral bacterium Porphyromonas gingivalis, can subvert host immunity to r

204 complex), produced by the keystone pathogen Porphyromonas gingivalis, dramatically increased their a

205 ., Peptoniphilaceae [G-1] bacterium HMT 113, Porphyromonas gingivalis, Fretibacterium fastidiosum, Fi

206 taxa implicated in periodontitis, including Porphyromonas gingivalis, Fretibacterium fastidiosum, Fi

207 latter consisted of five microbial species (Porphyromonas gingivalis, Fusobacterium nucleatum, Actin

208 ntal pathogens such as Streptococcus mutans, Porphyromonas gingivalis, Fusobacterium nucleatum, and P

209 Single and multi-species biofilms of Porphyromonas gingivalis, Fusobacterium nucleatum, and S

210 , traditional periodontal pathogens, such as Porphyromonas gingivalis, Fusobacterium nucleatum, and T

211 The oral microbiome, and specifically Porphyromonas gingivalis, has long been implicated.

212 Studies with one of these bacteria, Porphyromonas gingivalis, have revealed that it contains

213 etiological agent of chronic periodontitis, Porphyromonas gingivalis, infect blood myeloid dendritic

214 The Gram-negative anaerobe, Porphyromonas gingivalis, is a key periodontal pathogen,

215 virulence of periodontal pathogens, such as Porphyromonas gingivalis, is expressed in the context of

216 umerous chronic infectious agents, including Porphyromonas gingivalis, is shown to drive-differentiat

217 Porphyromonas gingivalis, like other bacteria belonging

218 ls upon stimulation by heat-killed wild-type Porphyromonas gingivalis, live P. gingivalis protease-de

219 Porphyromonas gingivalis, Prevotella intermedia, and T.

220 owth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotella intermedia, Fusobac

221 are anaerobic and include organisms such as Porphyromonas gingivalis, Prevotella intermedia, Fusobac

222 his adipokine and the presence and levels of Porphyromonas gingivalis, Prevotella intermedia, Prevote

223 is of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Prevotella intermedia, Tannere

224 c periodontitis, the Gram-negative bacterium Porphyromonas gingivalis, produces a vast arsenal of vir

225 by Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis, respectively.

226 A key pathogen, Porphyromonas gingivalis, secretes gingipains, cysteine

227 sms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Camp

228 Bacterial loads of Porphyromonas gingivalis, T. forsythia, Parvimonas micra

229 of pathogens related to periodontal disease (Porphyromonas gingivalis, Tannerella forsythensis, Trepo

230 alysis identified twenty-one OTUs, including Porphyromonas gingivalis, Tannerella forsythia and Filif

231 Levels of Porphyromonas gingivalis, Tannerella forsythia, Aggregat

232 ding on probing and levels of microorganisms Porphyromonas gingivalis, Tannerella forsythia, and Camp

233 es were collected to determine the levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

234 ls of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Fuso

235 ase) in GCF and subgingival plaque levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

236 revealed that periodontal pathogens, such as Porphyromonas gingivalis, Tannerella forsythia, and Prev

237 lifactor alocis, Fretibacterium fastidiosum, Porphyromonas gingivalis, Tannerella forsythia, and Sele

238 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

239 using immunofluorescence for the presence of Porphyromonas gingivalis, Tannerella forsythia, Fusobact

240 n for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Fusobact

241 of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

242 eria (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

243 T1 decreased the counts of Porphyromonas gingivalis, Tannerella forsythia, Prevotel

244 rial, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

245 ts of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

246 eria (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

247 Eikenella corrodens, Campylobacter concisus, Porphyromonas gingivalis, Tannerella forsythia, Treponem

248 ctus, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

249 Porphyromonas gingivalis, the causative agent of adult p

250 In the periodontal pathogen Porphyromonas gingivalis, the CTD is cleaved off by PorU

251 biosis and preponderance of bacteria such as Porphyromonas gingivalis, the main etiological agent of

252 have evaluated the periopathogenic roles of Porphyromonas gingivalis, the oral microbiome, and mecha

253 gainst invader pathobiotic bacteria, such as Porphyromonas gingivalis, through small danger molecule

254 IgG levels to several species, including Porphyromonas gingivalis, Treponema denticola, and Campy

255 moderate evidence supporting association of Porphyromonas gingivalis, Treponema denticola, and Tanne

256 ed with polybacterial inoculum consisting of Porphyromonas gingivalis, Treponema denticola, and Tanne

257 For Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, and Tanne

258 drugs against red complex pathogens, namely, Porphyromonas gingivalis, Treponema denticola, and Tanne

259 ealing of abutments, rats were infected with Porphyromonas gingivalis, Treponema denticola, and Tanne

260 uding Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, Tannerell

261 the ability of a polymicrobial consortium of Porphyromonas gingivalis, Treponema denticola, Tannerell

262 Porphyromonas gingivalis, Treponema denticola, Tannerell

263 periodontal pathogens Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, Tannerell

264 virulence factors of the periodontopathogen Porphyromonas gingivalis, which causes chronic periodont

265 Porphyromonas gingivalis,an anaerobic bacterium strongly

266 gnaling in alveolar bone resorption, using a Porphyromonas gingivalis-associated ligature-induced per

267 zation (red complex-KCNK1, p = 3.4 x 10(-7); Porphyromonas gingivalis-DAB2IP, p = 1.0 x 10(-6)).

268 ved exaggerated proinflammatory responses to Porphyromonas gingivalis-derived lipopolysaccharide in c

269 acid (FA) levels on alveolar bone loss in a Porphyromonas gingivalis-induced model of periodontal di

270 wild-type (WT) controls in a murine model of Porphyromonas gingivalis-induced periodontitis and repor

271 s demonstrated in distinct models, including Porphyromonas gingivalis-induced periodontitis, ligature

272 ression was higher in periodontal tissues of Porphyromonas gingivalis-infected mice as compared with

273 bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

274 clearance by opportunistic pathogens such as Porphyromonas gingivalis.

275 ontal disease, whose main infective agent is Porphyromonas gingivalis.

276 of F. alocis are enhanced in coculture with Porphyromonas gingivalis.

277 roded titanium surfaces on the attachment of Porphyromonas gingivalis.

278 etween HIV-1 and an invasive oral bacterium, Porphyromonas gingivalis.

279 obulin (TG), Escherichia coli LPS, or intact Porphyromonas gingivalis.

280 ood mononuclear cells (PBMCs) in response to Porphyromonas gingivalis.

281 rally infected with the periodontal pathogen Porphyromonas gingivalis.

282 m johnsoniae and the nonmotile oral pathogen Porphyromonas gingivalis.

283 f LPS purified from the periodontal pathogen Porphyromonas gingivalis.

284 eriodontal disease is an anaerobic bacterium-Porphyromonas gingivalis.

285 he groups, including Veillonella HOT 780 and Porphyromonas HOT 284, which were 4.6- and 9-fold higher

286 Seven genera, Capnocytophaga, Slackia, Porphyromonas, Kingella, Peptostreptococcaceae, Lactobac

287 Among them, Campylobacter, Bacteroides, Porphyromonas, Lachnospiraceae unclassified, Prevotella,

288 en nonsmokers and smokers in species such as Porphyromonas, Neisseria, and Gemella, but lung bacteria

289 teeth were associated with higher levels of Porphyromonas (p < 0.05) and Prevotella.

290 of Gemella species, Granulicatella elegans, Porphyromonas pasteri, Prevotella nanceiensis and Strept

291 , P. gingivalis secretes a PAD, termed PPAD (Porphyromonas peptidylarginine deiminase), which is gene

292 -negative anaerobic rod taxa, Prevotella and Porphyromonas, predominated, contrasting with a reduced

293 ranscriptome amplification and sequencing of Porphyromonas somerae, a microbe of interest in endometr

294 e Haemophilus, Neisseria, Fusobacterium, and Porphyromonas species and the Sphingomonodaceae family a

295 dent manner, mainly due to overenrichment of Porphyromonas species.

296 ance and reduced Streptococcus, Gemella, and Porphyromonas taxa relative abundance in patients with n

297 n were also genus-associated, including with Porphyromonas that correlated to disease scores and sali

298 Y1-CN subjects had higher levels of salivary Porphyromonas; their gut microbiota had increased abunda

299 Porphyromonas was the most abundant genus in all disease

300 C mucosa, Capnocytophaga, Fusobacterium, and Porphyromonas, were at low levels in marijuana users, wh