ライフサイエンスコーパス: Porphyromonas

1 Treponema (34%), Mycoplasma (29%) and Porphyromonas (15%) were the most abundant genera associ

2 25% x 20%), Streptococcus (19.83% x 17.61%), Porphyromonas (3.63% x 1.42%, P = 0.03), Treponema (1.02

3 ncreased numbers of the following anaerobes: Porphyromonas (5.2 x 10(7) 16S rRNA gene copies/swab in

4 sequences in the genera Bacteroidetes [G-3], Porphyromonas, Abiotrophia, Filifactor, Peptostreptococc

5 II), Staphylococcus epidermidis (type III), Porphyromonas and Peptoniphilus species (type IV), and P

6 eolytic bacteria such as Peptostreptococcus, Porphyromonas and Prevotella.

7 Porphyromonas and Solobacterium showed significantly hig

8 and Alloprevotella, Leptotrichia, Neisseria, Porphyromonas, and Prevotella.

9 onic acid generating bacteria from the genus Porphyromonas, and that this may explain adolescent/adul

10 l variant of Lemierre's disease secondary to Porphyromonas asaccharolytica, a rarely described etiolo

11 ation led to a decrease in the prevalence of Porphyromonas endodontalis and Dialister pneumosintes at

12 We hypothesized that Porphyromonas endodontalis and Escherichia coli LPSs ind

13 In 1 index sample, routine culture found Porphyromonas endodontalis and Streptococcus intermedius

14 manifestation of endodontic infection where Porphyromonas endodontalis is frequently encountered.

15 ly reduced in GR included Bacteroidetes sp., Porphyromonas endodontalis, Porphyromonas gingivalis, Pr

16 tophaga granulosa, G. morbillorum, P. micra, Porphyromonas endodontalis, Streptococcus spp., and Tann

17 both multiple group testing and regression, Porphyromonas (FDR p-value = 0.02), Prevotella (FDR p-va

18 er actinomycetemcomitans (MT4/MSP: 42%/36%), Porphyromonas gingivalis (78%/66%), Tannerella forsythia

19 We challenged these primary cells with Porphyromonas gingivalis (a Gram-negative bacterium that

20 g a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingivalis JCVI SC001) usin

21 nts with chronic periodontitis and intraoral Porphyromonas gingivalis (P. gingivalis) and Tannerella

22 Porphyromonas gingivalis (P. gingivalis) is an opportuni

23 dontal health by investigating its effect on Porphyromonas gingivalis (P. gingivalis), lipopolysaccha

24 Porphyromonas gingivalis (P.g.)-induced TNF-alpha can be

25 ucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and Tannerella forsythia,

26 nis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (Pg) biofilms elicited signific

27 Porphyromonas gingivalis (Pg) capsule enables evasion fr

28 LM group also presented greater reduction of Porphyromonas gingivalis (Pg) DNA counts at 6 months (P

29 This study compared the effect of Porphyromonas gingivalis (Pg) infection and experimental

30 Porphyromonas gingivalis (Pg) is a keystone pathogen in

31 Porphyromonas gingivalis (Pg) is a major periodontal pat

32 or TRAM, HGF and HPDLF were stimulated with Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS) o

33 n vitro study examines the effect of EGCG on Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS)-e

34 6 cells were used to determine the effect of Porphyromonas gingivalis (Pg) LPS on insulin secretion.

35 Total amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

36 Aggregatibacter actinomycetemcomitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr)

37 ates biofilms, consisting of species such as Porphyromonas gingivalis (Pg), in the etiology of peri-i

38 y associated with the submucosal presence of Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

39 were used for analysis of bacterial DNA for Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

40 factors from periodontal pathogens, such as Porphyromonas gingivalis (Pg), stimulate the respiratory

41 Quantitative analysis of Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

42 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

43 HP1 monocytic cells following infection with Porphyromonas gingivalis (Pg).

44 tis (RA), specifically regarding the role of Porphyromonas gingivalis (Pg).

45 by ligature; 3) group G-Pg: oral gavage with Porphyromonas gingivalis (Pg); 4) group G-PgFn: oral gav

46 AW264.7 and human monocyte THP-1 to LPS from Porphyromonas gingivalis (PgLPS), an oral microbe implic

47 Mice were orally infected with Porphyromonas gingivalis 15 times over 2 weeks.

48 n 9 times with 10(9) colony-forming units of Porphyromonas gingivalis A7436 through an oral gavage mo

49 he chronic periodontitis-associated pathogen Porphyromonas gingivalis activates a Toll-like receptor

50 issue-degrading enzymes in the oral pathogen Porphyromonas gingivalis Although a number of subunits o

51 re Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis amounts in saliva and their ant

52 Porphyromonas gingivalis and Aggregatibacter actinomycet

53 he same biofilm plus the periodontopathogens Porphyromonas gingivalis and Aggregatibacter actinomycet

54 killing of periodontal pathogens, including Porphyromonas gingivalis and Aggregatibacter actinomycet

55 Porphyromonas gingivalis and especially Prevotella inter

56 Porphyromonas gingivalis and Filifactor alocis are fasti

57 derived from the common human oral bacterium Porphyromonas gingivalis and from bacteria commonly foun

58 ) on the virulence of a mixed infection with Porphyromonas gingivalis and Fusobacterium nucleatum in

59 odontitis was induced by oral inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in

60 Although not statistically significant, Porphyromonas gingivalis and Fusobacterium nucleatum occ

61 Periodontal pathogens such as Porphyromonas gingivalis and Fusobacterium nucleatum pro

62 that aPDT treatment can simultaneously kill Porphyromonas gingivalis and inactivate its virulence-as

63 Mice were orally infected with human Porphyromonas gingivalis and observed for 42 days.

64 mDC microbiome by 16S rDNA sequencing showed Porphyromonas gingivalis and other species, including (c

65 d oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevotella nigrescens induc

66 The Orange-Red cluster score (that included Porphyromonas gingivalis and Prevotella spp.) was positi

67 Interspecies signalling between Porphyromonas gingivalis and Streptococcus gordonii serv

68 d Treponema denticola, and the prevalence of Porphyromonas gingivalis and T. denticola associated sig

69 ted with periodontal pathology and number of Porphyromonas gingivalis and Tannerella forsythia (forme

70 tus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tannerella forsythia), and

71 es that gram-negative oral bacteria, such as Porphyromonas gingivalis and Tannerella forsythia, use d

72 The keystone pathogen Porphyromonas gingivalis and the accessory pathogen Stre

73 The periodontal pathogen Porphyromonas gingivalis and the endodontic species Ente

74 modes of mucosal vaccination with whole-cell Porphyromonas gingivalis and to test the role of various

75 how galactose-inhibitable coaggregation with Porphyromonas gingivalis and were defective in cell bind

76 Anti-Porphyromonas gingivalis antibody testing was performed

77 hemistry, we have identified the presence of Porphyromonas gingivalis antigens in placental tissues.

78 ibular second premolars, followed by topical Porphyromonas gingivalis application (10(9) colony formi

79 riae proteins produced by the human pathogen Porphyromonas gingivalis are required for invasion of hu

80 , FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are responsible for adherence t

81 rial manipulation of neutrophil responses by Porphyromonas gingivalis as a mechanism that contributes

82 However, the periodontal pathogen Porphyromonas gingivalis can control C5aR activation by

83 The oral pathogen Porphyromonas gingivalis causes inflammatory host respon

84 The gram-negative anaerobe Porphyromonas gingivalis colonizes the gingival crevice

85 in substrates of a novel secretion system of Porphyromonas gingivalis contain a conserved C-terminal

86 Porphyromonas gingivalis contains lipid A with structura

87 A modest effect of DHA + aspirin on Porphyromonas gingivalis counts was associated with 14%

88 echanism by which the opportunistic pathogen Porphyromonas gingivalis dampens innate immune responses

89 We show here that Porphyromonas gingivalis Dps (PgDps), previously describ

90 0068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gingivalis DSM20709) were placed in a seri

91 periodontitis caused by oral infection with Porphyromonas gingivalis enhances articular bone loss.

92 s infection with the anaerobic oral pathogen Porphyromonas gingivalis exerts a regulatory effect on a

93 The oral mucosal pathogen Porphyromonas gingivalis expresses at least two adhesins

94 Porphyromonas gingivalis FeoB1 is a ferrous iron transpo

95 ndard chow diet were given an oral gavage of Porphyromonas gingivalis for 15 wk.

96 Porphyromonas gingivalis has been implicated in the etio

97 Porphyromonas gingivalis has orthologs for each of these

98 of Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis have been shown to induce diffe

99 dy, we hypothesized that histatin 5 binds to Porphyromonas gingivalis hemagglutinin B (HagB) and atte

100 ed/attenuated the CXCL1 and TNF responses to Porphyromonas gingivalis hemagglutinin B (HagB).

101 of IL-6 by whole blood cells stimulated with Porphyromonas gingivalis HmuY protein.

102 the mechanism of action of ROS stimulated by Porphyromonas gingivalis in gingival epithelial cells.

103 ion, immunoglobulin (Ig)G subclasses against Porphyromonas gingivalis in individuals with pre-RA and

104 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis in patients with chronic period

105 ne the impact of RvE1 on the phagocytosis of Porphyromonas gingivalis in T2D.

106 Prevotella intermedia and Porphyromonas gingivalis increased in the control group.

107 Porphyromonas gingivalis is a Gram-negative obligate ana

108 Porphyromonas gingivalis is a Gram-negative obligate ana

109 Porphyromonas gingivalis is a Gram-negative obligately a

110 The oral bacterium Porphyromonas gingivalis is a key etiological agent of h

111 Porphyromonas gingivalis is a keystone pathogen that con

112 Porphyromonas gingivalis is a keystone periodontal patho

113 Porphyromonas gingivalis is a major pathogen in periodon

114 Porphyromonas gingivalis is a member of the human oral m

115 Porphyromonas gingivalis is a pathogen in severe periodo

116 Porphyromonas gingivalis is a peptide-fermenting asaccha

117 Porphyromonas gingivalis is a predominant pathogen that

118 Porphyromonas gingivalis is a primary etiological agent

119 Porphyromonas gingivalis is able to invade and modulate

120 Porphyromonas gingivalis is an established pathogen in p

121 hereas the phylogenetically related pathogen Porphyromonas gingivalis is associated with the chronic

122 Porphyromonas gingivalis is considered a keystone pathog

123 Porphyromonas gingivalis is etiologically associated wit

124 Porphyromonas gingivalis is implicated in the etiology o

125 Porphyromonas gingivalis is intimately associated with p

126 rthermore, the mechanism of TLR induction by Porphyromonas gingivalis is investigated in human gingiv

127 Porphyromonas gingivalis is one of the major causative a

128 Porphyromonas gingivalis is one of the oral microorganis

129 Porphyromonas gingivalis is present in dental plaque as

130 By far, Porphyromonas gingivalis is the best-studied periodontal

131 Porphyromonas gingivalis is the major causative agent of

132 st response to lipopolysaccharide (LPS) from Porphyromonas gingivalis is unusual inasmuch as differen

133 In monoinfection, heat-killed Porphyromonas gingivalis led to augmented levels of IL-1

134 Porphyromonas gingivalis lipid A heterogeneity modulates

135 olated from C57BL/6J mice were cultured with Porphyromonas gingivalis lipopolysaccharide (LPS) and cy

136 with three consecutive palatal injections of Porphyromonas gingivalis lipopolysaccharide (LPS) at 48-

137 roducts (AGE) in the presence and absence of Porphyromonas gingivalis lipopolysaccharide (LPS) on IL-

138 man monocytic cell line, we demonstrate that Porphyromonas gingivalis lipopolysaccharide (LPS), which

139 ived IFN-gamma in constitutively released or Porphyromonas gingivalis lipopolysaccharide (PgLPS)-stim

140 was induced via silk ligature placement with Porphyromonas gingivalis lipopolysaccharide injection in

141 a [TNF-alpha]), bacterial virulence factors (Porphyromonas gingivalis LPS) or a combination in a biom

142 In this study we report that Porphyromonas gingivalis LPS, a TLR2 agonist, fails to i

143 y be caused by periodontal bacteria, such as Porphyromonas gingivalis Mast cells are sentinels at muc

144 We postulated that the periodontal pathogen, Porphyromonas gingivalis may suppress the inflammasome a

145 The fimbriae of Porphyromonas gingivalis mediate critical roles in host

146 Recent reports indicate that Porphyromonas gingivalis mediates alveolar bone loss or

147 Gram-negative, anaerobic periodontopathogen Porphyromonas gingivalis must withstand nitrosative stre

148 he present study is to analyze the effect of Porphyromonas gingivalis on differentiation of primary o

149 (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human macrophages.

150 e direct effects of the periodontal pathogen Porphyromonas gingivalis on osteoclast differentiation a

151 esponsiveness of whole blood stimulated with Porphyromonas gingivalis or Escherichia coli LPS were mo

152 experimental periodontitis induced by either Porphyromonas gingivalis or ligature, gamma-proteobacter

153 lex anaerobic periodontal pathogens (such as Porphyromonas gingivalis or Treponema denticola) provide

154 tudy is to determine whether the presence of Porphyromonas gingivalis peptidylarginine deiminase (PPA

155 Recent results suggest that Porphyromonas gingivalis PorN, which is similar in seque

156 SprF exhibits some similarity to Porphyromonas gingivalis PorP, which is required for sec

157 SprE is similar in sequence to Porphyromonas gingivalis PorW, which is required for sec

158 Porphyromonas gingivalis produces unusual sphingolipids

159 The Porphyromonas gingivalis recombinant VimA can interact w

160 show that intraoral inoculation of mice with Porphyromonas gingivalis resulted in infection, alveolar

161 Porphyromonas gingivalis secretes a serine phosphatase e

162 Porphyromonas gingivalis showed the greatest reduction i

163 ases and are more likely to be infected with Porphyromonas gingivalis than non-smokers.

164 ce factor secreted by the periodontopathogen Porphyromonas gingivalis that attacks host vasculature a

165 which activity was induced by infection with Porphyromonas gingivalis The expression of several miRNA

166 The adaptation of Porphyromonas gingivalis to H2O2-induced stress while in

167 y, the abundance of the periodontal pathogen Porphyromonas gingivalis trended with higher risk of ESC

168 ic Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis viability.

169 The Porphyromonas gingivalis VimA protein has multifunctiona

170 Mice were infected with the oral pathogen Porphyromonas gingivalis W50 (P. gingivalis) in the maxi

171 ranscriptome analysis for a microbial genome Porphyromonas gingivalis W83 can be viewed at http://bio

172 y been used to identify 115 genes induced in Porphyromonas gingivalis W83 during human infection.

173 f F. alocis and its ability to interact with Porphyromonas gingivalis W83, several clinical isolates

174 An inferior frequency detection of Porphyromonas gingivalis was observed in the PDT protoco

175 ll-characterized human periodontal pathogen, Porphyromonas gingivalis We found that oral mucosal LCs

176 In unadjusted models, high titers to Porphyromonas gingivalis were most strongly associated w

177 ns Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis with extended NO-release kineti

178 presence of specific bacterial species (i.e. Porphyromonas gingivalis) and their effects in immune re

179 en" where low-abundance microbial pathogens (Porphyromonas gingivalis) can orchestrate inflammatory d

180 tory stimulus (lipopolysaccharide [LPS] from Porphyromonas gingivalis) in a manner consistent with th

181 were as follows: Tannerella forsythia, 81%; Porphyromonas gingivalis, 78%; and Aggregatibacter (form

182 Culture supernatants of Porphyromonas gingivalis, a bacterium that secretes prot

183 Porphyromonas gingivalis, a Gram-negative asaccharolytic

184 Porphyromonas gingivalis, a Gram-negative oral anaerobe,

185 In vivo challenge with live Porphyromonas gingivalis, a Gram-negative pathogenic bac

186 Porphyromonas gingivalis, a key periodontal pathogen, is

187 The peritoneal infection model using Porphyromonas gingivalis, a keystone pathogen for period

188 important role in eliciting inflammation to Porphyromonas gingivalis, a keystone pathogen in periodo

189 Porphyromonas gingivalis, a major etiologic agent of per

190 chronic inflammatory periodontal disease is Porphyromonas gingivalis, a non-motile, Gram-negative, r

191 In contrast, infection with Porphyromonas gingivalis, a principal pathogen in chroni

192 nalysed in mice after oral administration of Porphyromonas gingivalis, a representative periodontopat

193 The susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

194 contamination with >/=3 specific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

195 The presence of periodontal pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

196 operties against three periodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemc

197 Levels of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

198 lipid A structures in the outer membrane of Porphyromonas gingivalis, an agent of human periodontal

199 the proteases of Pseudomonas aeruginosa and Porphyromonas gingivalis, and enables its antimicrobial

200 uced the numbers of Streptococcus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, a

201 iodontitis is initiated by bacteria, such as Porphyromonas gingivalis, and is caused largely by host

202 ously Actinobacillus actinomycetemcomitans), Porphyromonas gingivalis, and Prevotella intermedia.

203 or more overtly pathogenic organisms such as Porphyromonas gingivalis, and the two species assemble i

204 early and late colonizer pathogens, such as Porphyromonas gingivalis, as the biofilm ages and period

205 The oral pathogen Porphyromonas gingivalis, as well as its purified fimbri

206 gens (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Campylobacter rectus, and Tann

207 the periodontitis-associated oral bacterium Porphyromonas gingivalis, can subvert host immunity to r

208 eomic responses to the periodontal pathogen, Porphyromonas gingivalis, donor-matched human blood mono

209 complex), produced by the keystone pathogen Porphyromonas gingivalis, dramatically increased their a

210 virulence factor of the periodontal pathogen Porphyromonas gingivalis, has been shown to induce prote

211 The oral microbiome, and specifically Porphyromonas gingivalis, has long been implicated.

212 Studies with one of these bacteria, Porphyromonas gingivalis, have revealed that it contains

213 s, Veillonella sp., and the middle colonizer Porphyromonas gingivalis, indicating specificity among t

214 etiological agent of chronic periodontitis, Porphyromonas gingivalis, infect blood myeloid dendritic

215 virulence of periodontal pathogens, such as Porphyromonas gingivalis, is expressed in the context of

216 umerous chronic infectious agents, including Porphyromonas gingivalis, is shown to drive-differentiat

217 ls upon stimulation by heat-killed wild-type Porphyromonas gingivalis, live P. gingivalis protease-de

218 Porphyromonas gingivalis, Prevotella intermedia, and Fus

219 Porphyromonas gingivalis, Prevotella intermedia, and T.

220 owth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotella intermedia, Fusobac

221 are anaerobic and include organisms such as Porphyromonas gingivalis, Prevotella intermedia, Fusobac

222 his adipokine and the presence and levels of Porphyromonas gingivalis, Prevotella intermedia, Prevote

223 mpylobacter rectus, Fusobacterium nucleatum, Porphyromonas gingivalis, Prevotella intermedia, Tannere

224 cteroidetes sp., Porphyromonas endodontalis, Porphyromonas gingivalis, Prevotella spp., Tannerella fo

225 c periodontitis, the Gram-negative bacterium Porphyromonas gingivalis, produces a vast arsenal of vir

226 by Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis, respectively.

227 A key pathogen, Porphyromonas gingivalis, secretes gingipains, cysteine

228 sms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Camp

229 Bacterial loads of Porphyromonas gingivalis, T. forsythia, Parvimonas micra

230 of pathogens related to periodontal disease (Porphyromonas gingivalis, Tannerella forsythensis, Trepo

231 tinomycetemcomitans), Prevotella intermedia, Porphyromonas gingivalis, Tannerella forsythia (previous

232 alysis identified twenty-one OTUs, including Porphyromonas gingivalis, Tannerella forsythia and Filif

233 Levels of Porphyromonas gingivalis, Tannerella forsythia, Aggregat

234 ding on probing and levels of microorganisms Porphyromonas gingivalis, Tannerella forsythia, and Camp

235 ase) in GCF and subgingival plaque levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

236 revealed that periodontal pathogens, such as Porphyromonas gingivalis, Tannerella forsythia, and Prev

237 lifactor alocis, Fretibacterium fastidiosum, Porphyromonas gingivalis, Tannerella forsythia, and Sele

238 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

239 d for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

240 using immunofluorescence for the presence of Porphyromonas gingivalis, Tannerella forsythia, Fusobact

241 n for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Fusobact

242 of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

243 T1 decreased the counts of Porphyromonas gingivalis, Tannerella forsythia, Prevotel

244 ts of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

245 Eikenella corrodens, Campylobacter concisus, Porphyromonas gingivalis, Tannerella forsythia, Treponem

246 ctus, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

247 rial, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

248 Porphyromonas gingivalis, the causative agent of adult p

249 In the periodontal pathogen Porphyromonas gingivalis, the CTD is cleaved off by PorU

250 biosis and preponderance of bacteria such as Porphyromonas gingivalis, the main etiological agent of

251 have evaluated the periopathogenic roles of Porphyromonas gingivalis, the oral microbiome, and mecha

252 ntly reported that the oral mucosal pathogen Porphyromonas gingivalis, through its 67-kDa Mfa1 (minor

253 ptococcus gordonii SspB (AgI/II) is bound by Porphyromonas gingivalis, thus promoting oral colonizati

254 IgG levels to several species, including Porphyromonas gingivalis, Treponema denticola, and Campy

255 For Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, and Tanne

256 ealing of abutments, rats were infected with Porphyromonas gingivalis, Treponema denticola, and Tanne

257 Porphyromonas gingivalis, Treponema denticola, and Tanne

258 moderate evidence supporting association of Porphyromonas gingivalis, Treponema denticola, and Tanne

259 ed with polybacterial inoculum consisting of Porphyromonas gingivalis, Treponema denticola, and Tanne

260 the ability of a polymicrobial consortium of Porphyromonas gingivalis, Treponema denticola, Tannerell

261 Porphyromonas gingivalis, Treponema denticola, Tannerell

262 uding Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, Tannerell

263 Porphyromonas gingivalis, which can invade endothelial c

264 virulence factors of the periodontopathogen Porphyromonas gingivalis, which causes chronic periodont

265 Porphyromonas gingivalis,an anaerobic bacterium strongly

266 gnaling in alveolar bone resorption, using a Porphyromonas gingivalis-associated ligature-induced per

267 zation (red complex-KCNK1, p = 3.4 x 10(-7); Porphyromonas gingivalis-DAB2IP, p = 1.0 x 10(-6)).

268 acid (FA) levels on alveolar bone loss in a Porphyromonas gingivalis-induced model of periodontal di

269 wild-type (WT) controls in a murine model of Porphyromonas gingivalis-induced periodontitis and repor

270 the regeneration of alveolar bone following Porphyromonas gingivalis-induced periodontitis in rats.

271 s demonstrated in distinct models, including Porphyromonas gingivalis-induced periodontitis, ligature

272 se (DIO) mice and improves the resolution of Porphyromonas gingivalis-induced periodontitis.

273 ression was higher in periodontal tissues of Porphyromonas gingivalis-infected mice as compared with

274 ontal disease, whose main infective agent is Porphyromonas gingivalis.

275 of F. alocis are enhanced in coculture with Porphyromonas gingivalis.

276 roded titanium surfaces on the attachment of Porphyromonas gingivalis.

277 etween HIV-1 and an invasive oral bacterium, Porphyromonas gingivalis.

278 obulin (TG), Escherichia coli LPS, or intact Porphyromonas gingivalis.

279 ood mononuclear cells (PBMCs) in response to Porphyromonas gingivalis.

280 rally infected with the periodontal pathogen Porphyromonas gingivalis.

281 m johnsoniae and the nonmotile oral pathogen Porphyromonas gingivalis.

282 isease that is triggered by bacteria such as Porphyromonas gingivalis.

283 . pylori as well as Streptococcus mutans and Porphyromonas gingivalis.

284 y Actinobacillus actinomycetemcomitans), and Porphyromonas gingivalis.

285 t virulence determinant of the oral anaerobe Porphyromonas gingivalis.

286 itates colonization of the dental biofilm by Porphyromonas gingivalis.

287 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis.

288 m mice with DIO exposed to an oral pathogen, Porphyromonas gingivalis.

289 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis.

290 bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

291 clearance by opportunistic pathogens such as Porphyromonas gingivalis.

292 he groups, including Veillonella HOT 780 and Porphyromonas HOT 284, which were 4.6- and 9-fold higher

293 Seven genera, Capnocytophaga, Slackia, Porphyromonas, Kingella, Peptostreptococcaceae, Lactobac

294 en nonsmokers and smokers in species such as Porphyromonas, Neisseria, and Gemella, but lung bacteria

295 , P. gingivalis secretes a PAD, termed PPAD (Porphyromonas peptidylarginine deiminase), which is gene

296 -negative anaerobic rod taxa, Prevotella and Porphyromonas, predominated, contrasting with a reduced

297 e Haemophilus, Neisseria, Fusobacterium, and Porphyromonas species and the Sphingomonodaceae family a

298 ance and reduced Streptococcus, Gemella, and Porphyromonas taxa relative abundance in patients with n

299 n were also genus-associated, including with Porphyromonas that correlated to disease scores and sali

300 Porphyromonas was the most abundant genus in all disease