ライフサイエンスコーパス: Porphyromonas gingivalis

1 of F. alocis are enhanced in coculture with Porphyromonas gingivalis.

2 roded titanium surfaces on the attachment of Porphyromonas gingivalis.

3 etween HIV-1 and an invasive oral bacterium, Porphyromonas gingivalis.

4 obulin (TG), Escherichia coli LPS, or intact Porphyromonas gingivalis.

5 ood mononuclear cells (PBMCs) in response to Porphyromonas gingivalis.

6 rally infected with the periodontal pathogen Porphyromonas gingivalis.

7 m johnsoniae and the nonmotile oral pathogen Porphyromonas gingivalis.

8 isease that is triggered by bacteria such as Porphyromonas gingivalis.

9 . pylori as well as Streptococcus mutans and Porphyromonas gingivalis.

10 y Actinobacillus actinomycetemcomitans), and Porphyromonas gingivalis.

11 t virulence determinant of the oral anaerobe Porphyromonas gingivalis.

12 itates colonization of the dental biofilm by Porphyromonas gingivalis.

13 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis.

14 m mice with DIO exposed to an oral pathogen, Porphyromonas gingivalis.

15 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis.

16 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis.

17 bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

18 clearance by opportunistic pathogens such as Porphyromonas gingivalis.

19 ontal disease, whose main infective agent is Porphyromonas gingivalis.

20 Mice were orally infected with Porphyromonas gingivalis 15 times over 2 weeks.

21 er actinomycetemcomitans (MT4/MSP: 42%/36%), Porphyromonas gingivalis (78%/66%), Tannerella forsythia

22 were as follows: Tannerella forsythia, 81%; Porphyromonas gingivalis, 78%; and Aggregatibacter (form

23 We challenged these primary cells with Porphyromonas gingivalis (a Gram-negative bacterium that

24 Culture supernatants of Porphyromonas gingivalis, a bacterium that secretes prot

25 sis of the genome-scale metabolic network of Porphyromonas gingivalis, a gram-negative anaerobe that

26 Porphyromonas gingivalis, a Gram-negative asaccharolytic

27 A major contributing pathogen is Porphyromonas gingivalis, a gram-negative bacterium reco

28 Porphyromonas gingivalis, a Gram-negative oral anaerobe,

29 In vivo challenge with live Porphyromonas gingivalis, a Gram-negative pathogenic bac

30 Porphyromonas gingivalis, a key periodontal pathogen, is

31 The peritoneal infection model using Porphyromonas gingivalis, a keystone pathogen for period

32 important role in eliciting inflammation to Porphyromonas gingivalis, a keystone pathogen in periodo

33 Porphyromonas gingivalis, a major etiologic agent of per

34 chronic inflammatory periodontal disease is Porphyromonas gingivalis, a non-motile, Gram-negative, r

35 bits production of a major adhesin, FimA, in Porphyromonas gingivalis, a primary periodontal pathogen

36 In contrast, infection with Porphyromonas gingivalis, a principal pathogen in chroni

37 nalysed in mice after oral administration of Porphyromonas gingivalis, a representative periodontopat

38 n 9 times with 10(9) colony-forming units of Porphyromonas gingivalis A7436 through an oral gavage mo

39 he chronic periodontitis-associated pathogen Porphyromonas gingivalis activates a Toll-like receptor

40 esions, we injected the periodontal pathogen Porphyromonas gingivalis adjacent to calvarial bone with

41 The susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

42 contamination with >/=3 specific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

43 The presence of periodontal pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

44 tes in diabetes showed a higher frequency of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

45 operties against three periodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemc

46 Levels of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

47 issue-degrading enzymes in the oral pathogen Porphyromonas gingivalis Although a number of subunits o

48 re Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis amounts in saliva and their ant

49 lipid A structures in the outer membrane of Porphyromonas gingivalis, an agent of human periodontal

50 Porphyromonas gingivalis, an opportunistic oral pathogen

51 Porphyromonas gingivalis,an anaerobic bacterium strongly

52 Porphyromonas gingivalis and Aggregatibacter actinomycet

53 he same biofilm plus the periodontopathogens Porphyromonas gingivalis and Aggregatibacter actinomycet

54 killing of periodontal pathogens, including Porphyromonas gingivalis and Aggregatibacter actinomycet

55 Porphyromonas gingivalis and especially Prevotella inter

56 Porphyromonas gingivalis and Filifactor alocis are fasti

57 derived from the common human oral bacterium Porphyromonas gingivalis and from bacteria commonly foun

58 ) on the virulence of a mixed infection with Porphyromonas gingivalis and Fusobacterium nucleatum in

59 odontitis was induced by oral inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in

60 Although not statistically significant, Porphyromonas gingivalis and Fusobacterium nucleatum occ

61 Periodontal pathogens such as Porphyromonas gingivalis and Fusobacterium nucleatum pro

62 that aPDT treatment can simultaneously kill Porphyromonas gingivalis and inactivate its virulence-as

63 Mice were orally infected with human Porphyromonas gingivalis and observed for 42 days.

64 mDC microbiome by 16S rDNA sequencing showed Porphyromonas gingivalis and other species, including (c

65 d oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevotella nigrescens induc

66 The Orange-Red cluster score (that included Porphyromonas gingivalis and Prevotella spp.) was positi

67 Interspecies signalling between Porphyromonas gingivalis and Streptococcus gordonii serv

68 d Treponema denticola, and the prevalence of Porphyromonas gingivalis and T. denticola associated sig

69 ted with periodontal pathology and number of Porphyromonas gingivalis and Tannerella forsythia (forme

70 tus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tannerella forsythia), and

71 es that gram-negative oral bacteria, such as Porphyromonas gingivalis and Tannerella forsythia, use d

72 The keystone pathogen Porphyromonas gingivalis and the accessory pathogen Stre

73 The periodontal pathogen Porphyromonas gingivalis and the endodontic species Ente

74 modes of mucosal vaccination with whole-cell Porphyromonas gingivalis and to test the role of various

75 how galactose-inhibitable coaggregation with Porphyromonas gingivalis and were defective in cell bind

76 presence of specific bacterial species (i.e. Porphyromonas gingivalis) and their effects in immune re

77 the proteases of Pseudomonas aeruginosa and Porphyromonas gingivalis, and enables its antimicrobial

78 uced the numbers of Streptococcus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, a

79 iodontitis is initiated by bacteria, such as Porphyromonas gingivalis, and is caused largely by host

80 nitiated by anaerobic oral pathogens such as Porphyromonas gingivalis, and it is characterized by hos

81 ously Actinobacillus actinomycetemcomitans), Porphyromonas gingivalis, and Prevotella intermedia.

82 ously Actinobacillus actinomycetemcomitans), Porphyromonas gingivalis, and Tannerella forsythia (prev

83 eptide showed 82% homology with enolase from Porphyromonas gingivalis, and the levels of antibodies t

84 or more overtly pathogenic organisms such as Porphyromonas gingivalis, and the two species assemble i

85 Anti-Porphyromonas gingivalis antibody testing was performed

86 hemistry, we have identified the presence of Porphyromonas gingivalis antigens in placental tissues.

87 ibular second premolars, followed by topical Porphyromonas gingivalis application (10(9) colony formi

88 riae proteins produced by the human pathogen Porphyromonas gingivalis are required for invasion of hu

89 , FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are responsible for adherence t

90 rial manipulation of neutrophil responses by Porphyromonas gingivalis as a mechanism that contributes

91 early and late colonizer pathogens, such as Porphyromonas gingivalis, as the biofilm ages and period

92 The oral pathogen Porphyromonas gingivalis, as well as its purified fimbri

93 gnaling in alveolar bone resorption, using a Porphyromonas gingivalis-associated ligature-induced per

94 gens (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Campylobacter rectus, and Tann

95 However, the periodontal pathogen Porphyromonas gingivalis can control C5aR activation by

96 en" where low-abundance microbial pathogens (Porphyromonas gingivalis) can orchestrate inflammatory d

97 the periodontitis-associated oral bacterium Porphyromonas gingivalis, can subvert host immunity to r

98 The oral pathogen Porphyromonas gingivalis causes inflammatory host respon

99 The gram-negative anaerobe Porphyromonas gingivalis colonizes the gingival crevice

100 in substrates of a novel secretion system of Porphyromonas gingivalis contain a conserved C-terminal

101 Porphyromonas gingivalis contains lipid A with structura

102 A modest effect of DHA + aspirin on Porphyromonas gingivalis counts was associated with 14%

103 zation (red complex-KCNK1, p = 3.4 x 10(-7); Porphyromonas gingivalis-DAB2IP, p = 1.0 x 10(-6)).

104 echanism by which the opportunistic pathogen Porphyromonas gingivalis dampens innate immune responses

105 eomic responses to the periodontal pathogen, Porphyromonas gingivalis, donor-matched human blood mono

106 We show here that Porphyromonas gingivalis Dps (PgDps), previously describ

107 complex), produced by the keystone pathogen Porphyromonas gingivalis, dramatically increased their a

108 0068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gingivalis DSM20709) were placed in a seri

109 periodontitis caused by oral infection with Porphyromonas gingivalis enhances articular bone loss.

110 s infection with the anaerobic oral pathogen Porphyromonas gingivalis exerts a regulatory effect on a

111 The oral mucosal pathogen Porphyromonas gingivalis expresses at least two adhesins

112 Porphyromonas gingivalis FeoB1 is a ferrous iron transpo

113 occus mutans and an unencapsulated strain of Porphyromonas gingivalis following as few as 100 pulses

114 ndard chow diet were given an oral gavage of Porphyromonas gingivalis for 15 wk.

115 Porphyromonas gingivalis has been implicated in the etio

116 Porphyromonas gingivalis has orthologs for each of these

117 virulence factor of the periodontal pathogen Porphyromonas gingivalis, has been shown to induce prote

118 The oral microbiome, and specifically Porphyromonas gingivalis, has long been implicated.

119 of Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis have been shown to induce diffe

120 Studies with one of these bacteria, Porphyromonas gingivalis, have revealed that it contains

121 dy, we hypothesized that histatin 5 binds to Porphyromonas gingivalis hemagglutinin B (HagB) and atte

122 ed/attenuated the CXCL1 and TNF responses to Porphyromonas gingivalis hemagglutinin B (HagB).

123 of IL-6 by whole blood cells stimulated with Porphyromonas gingivalis HmuY protein.

124 onin, and immunoglobulin G (IgG) antibody to Porphyromonas gingivalis in control patients or patients

125 the mechanism of action of ROS stimulated by Porphyromonas gingivalis in gingival epithelial cells.

126 ion, immunoglobulin (Ig)G subclasses against Porphyromonas gingivalis in individuals with pre-RA and

127 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis in patients with chronic period

128 ne the impact of RvE1 on the phagocytosis of Porphyromonas gingivalis in T2D.

129 The persistence of Porphyromonas gingivalis in the inflammatory environment

130 tory stimulus (lipopolysaccharide [LPS] from Porphyromonas gingivalis) in a manner consistent with th

131 Prevotella intermedia and Porphyromonas gingivalis increased in the control group.

132 s, Veillonella sp., and the middle colonizer Porphyromonas gingivalis, indicating specificity among t

133 acid (FA) levels on alveolar bone loss in a Porphyromonas gingivalis-induced model of periodontal di

134 wild-type (WT) controls in a murine model of Porphyromonas gingivalis-induced periodontitis and repor

135 the regeneration of alveolar bone following Porphyromonas gingivalis-induced periodontitis in rats.

136 s demonstrated in distinct models, including Porphyromonas gingivalis-induced periodontitis, ligature

137 se (DIO) mice and improves the resolution of Porphyromonas gingivalis-induced periodontitis.

138 Specifically, the oral/systemic pathogen Porphyromonas gingivalis induces cross-talk between CXCR

139 etiological agent of chronic periodontitis, Porphyromonas gingivalis, infect blood myeloid dendritic

140 ression was higher in periodontal tissues of Porphyromonas gingivalis-infected mice as compared with

141 Porphyromonas gingivalis initially colonizes the oral ca

142 Porphyromonas gingivalis is a consensus periodontal path

143 Porphyromonas gingivalis is a Gram-negative bacterium th

144 Porphyromonas gingivalis is a Gram-negative obligate ana

145 Porphyromonas gingivalis is a Gram-negative obligate ana

146 Porphyromonas gingivalis is a Gram-negative obligately a

147 The SerB protein of Porphyromonas gingivalis is a HAD family serine phosphat

148 The oral bacterium Porphyromonas gingivalis is a key etiological agent of h

149 Porphyromonas gingivalis is a keystone pathogen that con

150 Porphyromonas gingivalis is a keystone periodontal patho

151 Porphyromonas gingivalis is a major pathogen in periodon

152 Porphyromonas gingivalis is a member of the human oral m

153 Porphyromonas gingivalis is a pathogen in severe periodo

154 Porphyromonas gingivalis is a peptide-fermenting asaccha

155 Porphyromonas gingivalis is a predominant pathogen that

156 Porphyromonas gingivalis is a primary etiological agent

157 Porphyromonas gingivalis is able to invade and modulate

158 Porphyromonas gingivalis is an established pathogen in p

159 hereas the phylogenetically related pathogen Porphyromonas gingivalis is associated with the chronic

160 Porphyromonas gingivalis is considered a keystone pathog

161 Porphyromonas gingivalis is etiologically associated wit

162 The periodontal pathogen Porphyromonas gingivalis is implicated in certain system

163 Porphyromonas gingivalis is implicated in the etiology o

164 Porphyromonas gingivalis is intimately associated with p

165 rthermore, the mechanism of TLR induction by Porphyromonas gingivalis is investigated in human gingiv

166 Porphyromonas gingivalis is one of the major causative a

167 Porphyromonas gingivalis is one of the oral microorganis

168 Porphyromonas gingivalis is present in dental plaque as

169 By far, Porphyromonas gingivalis is the best-studied periodontal

170 Porphyromonas gingivalis is the major causative agent of

171 of the features of the periodontal pathogen Porphyromonas gingivalis is the presence of complex iron

172 st response to lipopolysaccharide (LPS) from Porphyromonas gingivalis is unusual inasmuch as differen

173 virulence of periodontal pathogens, such as Porphyromonas gingivalis, is expressed in the context of

174 umerous chronic infectious agents, including Porphyromonas gingivalis, is shown to drive-differentiat

175 Priming PDL cells with Porphyromonas gingivalis, its lipopolysaccharide (LPS),

176 In monoinfection, heat-killed Porphyromonas gingivalis led to augmented levels of IL-1

177 Porphyromonas gingivalis lipid A heterogeneity modulates

178 Porphyromonas gingivalis lipid A is heterogeneous with r

179 olated from C57BL/6J mice were cultured with Porphyromonas gingivalis lipopolysaccharide (LPS) and cy

180 with three consecutive palatal injections of Porphyromonas gingivalis lipopolysaccharide (LPS) at 48-

181 roducts (AGE) in the presence and absence of Porphyromonas gingivalis lipopolysaccharide (LPS) on IL-

182 man monocytic cell line, we demonstrate that Porphyromonas gingivalis lipopolysaccharide (LPS), which

183 ived IFN-gamma in constitutively released or Porphyromonas gingivalis lipopolysaccharide (PgLPS)-stim

184 was induced via silk ligature placement with Porphyromonas gingivalis lipopolysaccharide injection in

185 e biopsies and in a preclinical rat model of Porphyromonas gingivalis lipopolysaccharide-induced expe

186 ls upon stimulation by heat-killed wild-type Porphyromonas gingivalis, live P. gingivalis protease-de

187 a [TNF-alpha]), bacterial virulence factors (Porphyromonas gingivalis LPS) or a combination in a biom

188 In this study we report that Porphyromonas gingivalis LPS, a TLR2 agonist, fails to i

189 y be caused by periodontal bacteria, such as Porphyromonas gingivalis Mast cells are sentinels at muc

190 We postulated that the periodontal pathogen, Porphyromonas gingivalis may suppress the inflammasome a

191 The fimbriae of Porphyromonas gingivalis mediate critical roles in host

192 Recent reports indicate that Porphyromonas gingivalis mediates alveolar bone loss or

193 Gram-negative, anaerobic periodontopathogen Porphyromonas gingivalis must withstand nitrosative stre

194 he present study is to analyze the effect of Porphyromonas gingivalis on differentiation of primary o

195 (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human macrophages.

196 e direct effects of the periodontal pathogen Porphyromonas gingivalis on osteoclast differentiation a

197 esponsiveness of whole blood stimulated with Porphyromonas gingivalis or Escherichia coli LPS were mo

198 experimental periodontitis induced by either Porphyromonas gingivalis or ligature, gamma-proteobacter

199 lex anaerobic periodontal pathogens (such as Porphyromonas gingivalis or Treponema denticola) provide

200 g a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingivalis JCVI SC001) usin

201 nts with chronic periodontitis and intraoral Porphyromonas gingivalis (P. gingivalis) and Tannerella

202 Porphyromonas gingivalis (P. gingivalis) is an opportuni

203 dontal health by investigating its effect on Porphyromonas gingivalis (P. gingivalis), lipopolysaccha

204 Porphyromonas gingivalis (P.g.)-induced TNF-alpha can be

205 tudy is to determine whether the presence of Porphyromonas gingivalis peptidylarginine deiminase (PPA

206 ucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and Tannerella forsythia,

207 nis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (Pg) biofilms elicited signific

208 Porphyromonas gingivalis (Pg) capsule enables evasion fr

209 LM group also presented greater reduction of Porphyromonas gingivalis (Pg) DNA counts at 6 months (P

210 This study compared the effect of Porphyromonas gingivalis (Pg) infection and experimental

211 Porphyromonas gingivalis (Pg) is a keystone pathogen in

212 Porphyromonas gingivalis (Pg) is a major periodontal pat

213 or TRAM, HGF and HPDLF were stimulated with Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS) o

214 n vitro study examines the effect of EGCG on Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS)-e

215 6 cells were used to determine the effect of Porphyromonas gingivalis (Pg) LPS on insulin secretion.

216 Total amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

217 Aggregatibacter actinomycetemcomitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr)

218 ates biofilms, consisting of species such as Porphyromonas gingivalis (Pg), in the etiology of peri-i

219 y associated with the submucosal presence of Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

220 were used for analysis of bacterial DNA for Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

221 factors from periodontal pathogens, such as Porphyromonas gingivalis (Pg), stimulate the respiratory

222 Quantitative analysis of Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

223 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

224 HP1 monocytic cells following infection with Porphyromonas gingivalis (Pg).

225 tis (RA), specifically regarding the role of Porphyromonas gingivalis (Pg).

226 by ligature; 3) group G-Pg: oral gavage with Porphyromonas gingivalis (Pg); 4) group G-PgFn: oral gav

227 AW264.7 and human monocyte THP-1 to LPS from Porphyromonas gingivalis (PgLPS), an oral microbe implic

228 Recent results suggest that Porphyromonas gingivalis PorN, which is similar in seque

229 SprF exhibits some similarity to Porphyromonas gingivalis PorP, which is required for sec

230 SprE is similar in sequence to Porphyromonas gingivalis PorW, which is required for sec

231 Porphyromonas gingivalis, Prevotella intermedia, and Fus

232 Porphyromonas gingivalis, Prevotella intermedia, and T.

233 owth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotella intermedia, Fusobac

234 are anaerobic and include organisms such as Porphyromonas gingivalis, Prevotella intermedia, Fusobac

235 his adipokine and the presence and levels of Porphyromonas gingivalis, Prevotella intermedia, Prevote

236 mpylobacter rectus, Fusobacterium nucleatum, Porphyromonas gingivalis, Prevotella intermedia, Tannere

237 cteroidetes sp., Porphyromonas endodontalis, Porphyromonas gingivalis, Prevotella spp., Tannerella fo

238 Porphyromonas gingivalis produces unusual sphingolipids

239 c periodontitis, the Gram-negative bacterium Porphyromonas gingivalis, produces a vast arsenal of vir

240 The Porphyromonas gingivalis recombinant VimA can interact w

241 by Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis, respectively.

242 show that intraoral inoculation of mice with Porphyromonas gingivalis resulted in infection, alveolar

243 Porphyromonas gingivalis secretes a serine phosphatase e

244 A key pathogen, Porphyromonas gingivalis, secretes gingipains, cysteine

245 Porphyromonas gingivalis showed the greatest reduction i

246 sms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Camp

247 Bacterial loads of Porphyromonas gingivalis, T. forsythia, Parvimonas micra

248 of pathogens related to periodontal disease (Porphyromonas gingivalis, Tannerella forsythensis, Trepo

249 tinomycetemcomitans), Prevotella intermedia, Porphyromonas gingivalis, Tannerella forsythia (previous

250 Those included Treponema denticola, Porphyromonas gingivalis, Tannerella forsythia (previous

251 alysis identified twenty-one OTUs, including Porphyromonas gingivalis, Tannerella forsythia and Filif

252 Levels of Porphyromonas gingivalis, Tannerella forsythia, Aggregat

253 ding on probing and levels of microorganisms Porphyromonas gingivalis, Tannerella forsythia, and Camp

254 ase) in GCF and subgingival plaque levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

255 revealed that periodontal pathogens, such as Porphyromonas gingivalis, Tannerella forsythia, and Prev

256 lifactor alocis, Fretibacterium fastidiosum, Porphyromonas gingivalis, Tannerella forsythia, and Sele

257 d for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

258 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

259 using immunofluorescence for the presence of Porphyromonas gingivalis, Tannerella forsythia, Fusobact

260 n for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Fusobact

261 of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

262 T1 decreased the counts of Porphyromonas gingivalis, Tannerella forsythia, Prevotel

263 Eikenella corrodens, Campylobacter concisus, Porphyromonas gingivalis, Tannerella forsythia, Treponem

264 ctus, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

265 rial, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

266 ts of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

267 ases and are more likely to be infected with Porphyromonas gingivalis than non-smokers.

268 ce factor secreted by the periodontopathogen Porphyromonas gingivalis that attacks host vasculature a

269 cell surface anionic polysaccharide (APS) in Porphyromonas gingivalis that is required for cell integ

270 which activity was induced by infection with Porphyromonas gingivalis The expression of several miRNA

271 Porphyromonas gingivalis, the causative agent of adult p

272 In the periodontal pathogen Porphyromonas gingivalis, the CTD is cleaved off by PorU

273 biosis and preponderance of bacteria such as Porphyromonas gingivalis, the main etiological agent of

274 have evaluated the periopathogenic roles of Porphyromonas gingivalis, the oral microbiome, and mecha

275 ntly reported that the oral mucosal pathogen Porphyromonas gingivalis, through its 67-kDa Mfa1 (minor

276 ptococcus gordonii SspB (AgI/II) is bound by Porphyromonas gingivalis, thus promoting oral colonizati

277 The adaptation of Porphyromonas gingivalis to H2O2-induced stress while in

278 y, the abundance of the periodontal pathogen Porphyromonas gingivalis trended with higher risk of ESC

279 IgG levels to several species, including Porphyromonas gingivalis, Treponema denticola, and Campy

280 ealing of abutments, rats were infected with Porphyromonas gingivalis, Treponema denticola, and Tanne

281 Porphyromonas gingivalis, Treponema denticola, and Tanne

282 moderate evidence supporting association of Porphyromonas gingivalis, Treponema denticola, and Tanne

283 ed with polybacterial inoculum consisting of Porphyromonas gingivalis, Treponema denticola, and Tanne

284 For Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, and Tanne

285 ously Actinobacillus actinomycetemcomitans), Porphyromonas gingivalis, Treponema denticola, Tannerell

286 uding Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, Tannerell

287 the ability of a polymicrobial consortium of Porphyromonas gingivalis, Treponema denticola, Tannerell

288 Porphyromonas gingivalis, Treponema denticola, Tannerell

289 ic Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis viability.

290 The Porphyromonas gingivalis VimA protein has multifunctiona

291 Mice were infected with the oral pathogen Porphyromonas gingivalis W50 (P. gingivalis) in the maxi

292 ranscriptome analysis for a microbial genome Porphyromonas gingivalis W83 can be viewed at http://bio

293 y been used to identify 115 genes induced in Porphyromonas gingivalis W83 during human infection.

294 f F. alocis and its ability to interact with Porphyromonas gingivalis W83, several clinical isolates

295 An inferior frequency detection of Porphyromonas gingivalis was observed in the PDT protoco

296 ll-characterized human periodontal pathogen, Porphyromonas gingivalis We found that oral mucosal LCs

297 In unadjusted models, high titers to Porphyromonas gingivalis were most strongly associated w

298 Porphyromonas gingivalis, which can invade endothelial c

299 virulence factors of the periodontopathogen Porphyromonas gingivalis, which causes chronic periodont

300 ns Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis with extended NO-release kineti