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1 licles on the face, neck, chest, and back by Propionibacterium acnes.
2 e immune response to the commensal bacterium Propionibacterium acnes.
3 antimicrobial activity against S. aureus and Propionibacterium acnes.
4 es, is caused by multiple factors, including Propionibacterium acnes.
5 46% vs 80%; 3) the most frequent isolate was Propionibacterium acnes (11/26) vs coagulase-negative St
6 iome at the strain level and genome level of Propionibacterium acnes, a dominant skin commensal, betw
7 mechanism by which S. aureus may commandeer Propionibacterium acnes, a key member of the human skin
8 In this issue, Kistowska et al. confirm that Propionibacterium acnes activates inflammasomes leading
10 asts, 6 nonfermenters, and 1 isolate each of Propionibacterium acnes and coagulase-negative staphyloc
11 olleagues present strain-based resolution of Propionibacterium acnes and its association with the com
12 the presence of the Gram-positive bacterium Propionibacterium acnes and its potential association wi
15 s associated with formation of resistance in Propionibacterium acnes and other bacteria, with clinica
16 ndependent growth, requiring helpers such as Propionibacterium acnes and Prevotella intermedia for st
21 Bradyrhizobium, Anaerococcus, Peptoniphilus, Propionibacterium acnes, Dorea, and Ruminococcus and red
22 rial pathogens including Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both
23 resulting from the immune response targeting Propionibacterium acnes has a significant role in its pa
26 e primed 12 days in advance with heat-killed Propionibacterium acnes, IL-18BP-Fc prevents LPS-induced
27 ase-negative staphylococci in 6.0% (27/448), Propionibacterium acnes in 4.7% (21/448), and Pseudomona
28 ica charantia Linn. var. abbreviata Ser.) on Propionibacterium acnes-induced inflammation and to iden
29 us studies showed that TLR9 and TLR2 mediate Propionibacterium acnes-induced sensitization to lipopol
47 1), Propionibacterium species (n = 15), and Propionibacterium acnes (n = 19) isolates; all of these
48 stigated the in vitro susceptibilities of 23 Propionibacterium acnes ophthalmic isolates to ertapenem
50 S (20 microg/mouse) 7 days after heat-killed Propionibacterium acnes (P. acnes) injection into wild-t
51 monstrate that fermentation of glycerol with Propionibacterium acnes (P. acnes), a skin commensal bac
52 ne treatment as a natural antibiotic against Propionibacterium acnes (P. acnes), which promotes folli
53 e investigated the roles of TLR2 and TLR4 in Propionibacterium acnes (P. acnes)-primed, LPS-induced l
56 overed, including Escherichia coli phage T3, Propionibacterium acnes phage PA6, and Streptococcus mit
57 on of early childhood acne, the emergence of Propionibacterium acnes resistance, and the rare but ser
58 teen of the species or phylotypes, including Propionibacterium acnes, Staphylococcus spp., and the op
59 observed the decline of an early-colonizing Propionibacterium acnes strain similar to SK137 and the
60 ny countries reporting that more than 50% of Propionibacterium acnes strains are resistant to topical
61 es of the human skin microbiome suggest that Propionibacterium acnes strains may contribute different
64 s-wide comparative analysis of 90 genomes of Propionibacterium acnes that represent the known diversi
65 rect antimicrobial activity in vitro against Propionibacterium acnes, the bacterium linked to the pat
72 ct to sequence multiple clinical isolates of Propionibacterium acnes, we have produced a draft genome
73 teria such as Staphylococcus epidermidis and Propionibacterium acnes, were identified in bacteriologi
74 ulture showed antimicrobial activity against Propionibacterium acnes, whereas the enhanced antimicrob
76 t contributes to the pathogenesis of acne is Propionibacterium acnes; yet, the molecular mechanism by
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