ライフサイエンスコーパス: Proteus

1 completely sequenced plasmid from the genus Proteus.

2 multicellular migration, most strikingly in Proteus.

3 We have developed Proteus, a web-based, context-specific tool for building

4 up of bacteria, including species of Vibrio, Proteus and Caulobacter that use the flagellum as a surf

5 letely understood, is far more palpable than Proteus and is (in most cases) much more readily subdued

6 ification systems, the flanking sequences in Proteus and Salmonella are completely different.

7 . coli and B. subtilis colonies, swarming by Proteus and Serratia, and spatially organized interspeci

8 on-UTI bacteria, Staphylococcus, Klebsiella, Proteus and Shigella.

9 fied TonB homologs in Shigella, Citrobacter, Proteus, and Kluyvera species.

10 Europe's obligate cave-dwelling amphibian Proteus anguinus inhabits subterranean waters of the nor

11 Toolkits in Proteus are context-independent representations of biolo

12 acteria), present in the xD strain of Amoeba proteus as required cell components, synthesize and expo

13 n range, we established a likely presence of Proteus at seven new sites, extending its range to Monte

14 Proteus combines these choices into a system of ODEs, wh

15 n one of these we found both black and white Proteus eDNA together.

16 Proteus FlgN has leucine zipper-like motifs arranged on

17 Proteus flgN is arranged in an operon with the class III

18 obes to discriminate the rare black morph of Proteus from the closely related white morph, we detecte

19 Proteus, implemented in C#, and a prototype toolkit for

20 t of these two cytotoxins is critical during Proteus infection.

21 -bp PCR product hybridized strongly with all Proteus isolates (n = 9) and 25% of 355 Escherichia coli

22 The expression of mannose-resistant/Proteus-like (MR/P) fimbria is phase variable because of

23 The mannose-resistant, Proteus-like (MR/P) fimbria, responsible for mannose-res

24 The mannose-resistant, Proteus-like (MR/P) fimbriae and flagellum are among vir

25 ene cluster, which encodes mannose-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indic

26 mrp gene cluster encoding mannose-resistant Proteus-like (MR/P) fimbriae of uropathogenic Proteus mi

27 ract infections, expresses mannose-resistant Proteus-like (MR/P) fimbriae whose expression is phase v

28 lled bacteria or purified mannose-resistant, Proteus-like (MR/P) fimbriae, a surface antigen expresse

29 pregulated in vivo encoded mannose-resistant Proteus-like (MR/P) fimbriae, urease, iron uptake system

30 tract infection, expresses mannose-resistant/Proteus-like (MR/P) fimbriae.

31 e were examined for PTEN mutations, only the Proteus-like patient was found to harbour a germline R33

32 Thus, PTEN may be involved in Proteus-like syndrome with its implications for cancer d

33 ley-Ruvalcaba syndrome, Proteus syndrome and Proteus-like syndrome, collectively classified as PTEN h

34 ey-Ruvalcaba syndrome, Proteus syndrome, and Proteus-like syndrome.

35 Five had Proteus syndrome and one, a Proteus-like syndrome.

36 as broadened to include Proteus syndrome and Proteus-like syndromes.

37 (4.4%), Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and

38 ae (n = 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Steno

39 ifferentiation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by

40 Proteus mirabilis alternates between motile and adherent

41 have shown previously using hyperflagellated Proteus mirabilis and a motile but non-swarming flgN tra

42 significant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia c

43 of Gram-negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates

44 We focused on Lrp from Proteus mirabilis and E. coli, orthologs with 98% identi

45 The closely related Proteus mirabilis and Enterobacterlaceae plasmid-encoded

46 of a urease-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was as

47 during UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition

48 operon as a major assimilatory checkpoint in Proteus mirabilis and other Gram-negative bacteria and e

49 The Proteus mirabilis and plasmid-encoded urease loci contai

50 The urease-positive species Proteus mirabilis and Providencia stuartii are two of th

51 zation by urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occ

52 s II promoter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed det

53 cherichia coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by i

54 segregate from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcomp

55 ted urinary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology i

56 Fimbriae of the human uropathogen Proteus mirabilis are the only characterized surface pro

57 Using Proteus mirabilis as a model, we investigate the role of

58 ol for application of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a se

59 One of the six predicted Proteus mirabilis autotransporters (ATs), ORF c2341, is

60 onstructed from the 50 %-identical TEM-1 and Proteus mirabilis beta-lactamases.

61 become encrusted and blocked by crystalline Proteus mirabilis biofilms.

62 Proteus mirabilis can rapidly utilize choline to enhance

63 Proteus mirabilis causes complicated urinary tract infec

64 fundamental behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to th

65 A total of 63 clinical isolates of Proteus mirabilis collected over a 19-month period were

66 Proteus mirabilis colonies exhibit striking geometric re

67 Proteus mirabilis commonly infects the complicated urina

68 Proteus mirabilis compromises the care of many patients

69 Proteus mirabilis forms extensive crystalline biofilms o

70 Proteus mirabilis forms extensive crystalline biofilms o

71 In this study, we describe wosA, a Proteus mirabilis gene identified by its ability to incr

72 In a search for Proteus mirabilis genes that were regulated by cell-to-c

73 rming motility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little und

74 -ray crystal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vit

75 , Seo et al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent i

76 hat provides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.

77 The enteric bacterium Proteus mirabilis is a common cause of complicated urina

78 Proteus mirabilis is a common cause of urinary tract inf

79 Proteus mirabilis is a common uropathogen in patients wi

80 Proteus mirabilis is a dimorphic motile bacterium well k

81 Proteus mirabilis is a dimorphic, motile bacterium often

82 The gram-negative enteric bacterium Proteus mirabilis is a frequent cause of urinary tract i

83 Proteus mirabilis is a Gram-negative bacterium that exis

84 Proteus mirabilis is a Gram-negative bacterium that unde

85 Proteus mirabilis is a model organism for urease-produci

86 Proteus mirabilis is a urinary tract pathogen and well k

87 Proteus mirabilis is a urinary tract pathogen that diffe

88 Proteus mirabilis is an opportunistic pathogen that is f

89 The enteric bacterium Proteus mirabilis is associated with a significant numbe

90 The bacterium Proteus mirabilis is capable of movement on solid surfac

91 Swarming by Proteus mirabilis is characterized by cycles of rapid an

92 rotease, ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with

93 loacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates)

94 Swarming colonies of independent Proteus mirabilis isolates recognize each other as forei

95 6 (3%) Klebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, w

96 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESB

97 gh level of similarity to sequences encoding Proteus mirabilis mannose-resistant fimbriae.

98 lular migration in a non-swarming but motile Proteus mirabilis mutant lacking the FIgN facilitator of

99 Molecular analyses have revealed that Proteus mirabilis possesses two genes, flaA and flaB, th

100 Uropathogenic Proteus mirabilis produces at least four types of fimbri

101 An isolate of Proteus mirabilis recovered from blood cultures of a dia

102 Escherichia coli, Klebsiella pneumoniae, and Proteus mirabilis remains unknown.

103 structures of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembran

104 y used to assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aerug

105 Proteus mirabilis swarming behavior is characterized by

106 Here, we identified a gene from Proteus mirabilis that encodes a 135-amino acid residue

107 MR/P fimbriae of uropathogenic Proteus mirabilis undergo invertible element-mediated ph

108 Proteus mirabilis urease catalyzes the hydrolysis of ure

109 Expression of Proteus mirabilis urease is governed by UreR, an AraC-li

110 We tested the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would

111 truncated form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and struc

112 Patients with Proteus mirabilis UTIs were more likely to have a foreig

113 higher than the averages were observed with Proteus mirabilis versus imipenem and with Klebsiella pn

114 to virulence in other pathogens, its role in Proteus mirabilis was investigated by constructing a str

115 A TnphoA mutant of Proteus mirabilis was isolated, which had lost the abili

116 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended

117 plementation studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) tr

118 Here we characterize PmDsbA from Proteus mirabilis, a bacterial pathogen increasingly ass

119 Proteus mirabilis, a cause of complicated urinary tract

120 Proteus mirabilis, a cause of complicated urinary tract

121 Recently, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated

122 Proteus mirabilis, a common cause of nosocomial and cath

123 Proteus mirabilis, a common cause of urinary tract infec

124 Proteus mirabilis, a gram-negative bacterium associated

125 Proteus mirabilis, a gram-negative bacterium, is a frequ

126 Proteus mirabilis, a Gram-negative bacterium, represents

127 Proteus mirabilis, a leading cause of catheter-associate

128 Proteus mirabilis, an etiologic agent of complicated uri

129 ier studies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the

130 ct on the numbers of Salmonella typhimurium, Proteus mirabilis, and Escherichia coli internalized by

131 ization of wild-type Salmonella typhimurium, Proteus mirabilis, and Escherichia coli.

132 i, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

133 as Klebsiella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhi

134 olates of Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associate

135 Proteus mirabilis, associated with complicated urinary t

136 re also found in cell-free supernatants from Proteus mirabilis, Citrobacter freundii and Enterobacter

137 ns), Listeria monocytogenes (three strains), Proteus mirabilis, Escherichia coli (three strains), and

138 se-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indicate that MrpB functions as the t

139 acteriaceae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that

140 kb PAI, designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella

141 scherichia coli 1021, Klebsiella pneumoniae, Proteus mirabilis, Providencia stuartii, and Pseudomonas

142 ibrary included probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcu

143 gram negative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pn

144 prevent colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia

145 by Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subt

146 een to identify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy su

147 In laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at a

148 e arfA hairpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella mult

149 genesis of urinary tract infection caused by Proteus mirabilis, we constructed a nonmotile, nonswarmi

150 riminating strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between

151 tract pathogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable in

152 ity to a putrescine-deficient speA mutant of Proteus mirabilis.

153 es in Klebsiella spp., Escherichia coli, and Proteus mirabilis.

154 important virulence factor of uropathogenic Proteus mirabilis.

155 toxin to prevent urinary tract infection by Proteus mirabilis.

156 roteus-like (MR/P) fimbriae of uropathogenic Proteus mirabilis.

157 philus influenzae, Bacteroides fragilis, and Proteus mirabilis.

158 een of the isolates were E. coli and one was Proteus mirabilis.

159 ion, is a virulence factor for uropathogenic Proteus mirabilis.

160 acid decarboxylase that inhibits swarming in Proteus mirabilis.

161 d for swarming in the urinary tract pathogen Proteus mirabilis.

162 n CFT073, is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mira

163 l-Amino acid deaminase from Proteus myxofaciens (PmaLAAD) is a membrane flavoenzyme

164 membrane-bound LAADs mainly express in genus Proteus, Providencia and Morganella.

165 region of rtn is identical to the published Proteus sequence, with the exception of a single G inser

166 mophilus ducreyi is the newest member of the Proteus/Serratia family of pore-forming toxins.

167 nt aerobes were Escherichia coli (n = 8) and Proteus sp. (n = 7).

168 Most pathogenic Proteus species are primarily associated with urinary tr

169 cherichia coli, 21 Klebsiella species, and 6 Proteus species that were resistant to at least one ESC

170 toca, 16/17 with K. pneumoniae, and 0/1 with Proteus spp.

171 Escherichia coli, Klebsiella pneumoniae, and Proteus spp.

172 mograms and recovery of Escherichia coli and Proteus spp. from the livers of infected mice.

173 spp., 99.3%; Pseudomonas aeruginosa, 98.9%; Proteus spp., 100%; Acinetobacter spp., 98.4%; and Citro

174 e most frequently used codons in the AT-rich Proteus spp., AAA (lysine).

175 , Klebsiella oxytoca, Klebsiella pneumoniae, Proteus spp., Pseudomonas aeruginosa, and Serratia marce

176 In Proteus swarm cell extracts, all the FlhC was assembled

177 Each Proteus swarm colony terrace corresponds to one swarming

178 opsy samples obtained from patients with the Proteus syndrome and compared the resultant DNA sequence

179 Five had Proteus syndrome and one, a Proteus-like syndrome.

180 syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome and Proteus-like syndrome, collectively

181 r syndrome spectrum has broadened to include Proteus syndrome and Proteus-like syndromes.

182 issues and cell lines from patients with the Proteus syndrome harbored admixtures of mutant alleles t

183 clinical diagnosis of neurofibromatosis and Proteus syndrome has allowed advancements in the Elephan

184 Complications of Proteus syndrome include, among others, progressive skel

185 Proteus syndrome is a rare and sporadic disorder that ca

186 Proteus syndrome is an extremely rare disorder of mosaic

187 The Proteus syndrome is caused by a somatic activating mutat

188 The Proteus syndrome is characterized by the overgrowth of s

189 These findings extend the spectrum of Proteus syndrome pathological characteristics and sugges

190 Care of patients with Proteus syndrome presents significant challenges to both

191 Of 29 patients with the Proteus syndrome, 26 had a somatic activating mutation (

192 hs that covered his body: neurofibromatosis, Proteus syndrome, and a combination of childhood injury,

193 syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome, and Proteus-like syndrome.

194 ent did not meet the diagnostic criteria for Proteus syndrome, he was found to have the c.49G>A, p.Gl

195 is the mildest molecularly confirmed case of Proteus syndrome, occurring in the absence of the charac

196 drome, Bannayan-Riley-Ruvalcaba syndrome and Proteus syndrome.

197 nt as a therapeutic option for patients with Proteus syndrome.

198 ling in cells and tissues from patients with Proteus syndrome.

199 onsible for the mosaic overgrowth condition, Proteus syndrome.

200 Merrick was in all likelihood suffering from Proteus syndrome.

201 DNA in 158 samples from 29 patients with the Proteus syndrome.

202 eria including Y. pestis, H. influenzae, and Proteus that cause plague, meningitis, and severe wound

203 sequence identities of 92% (Vibrio) and 98% (Proteus) to E. coli Lrp, including complete conservation

204 Proteus toxic agglutinin (Pta) represents a novel autotr

205 he HpmA hemolysin, a secreted cytotoxin, and proteus toxic agglutinin (Pta), a surface-associated cyt

206 the urinary tract, including a known toxin (Proteus toxic agglutinin) and the high pathogenicity isl

207 NA (eDNA) approach to detect the presence of Proteus using water samples collected from karst springs

208 udomonas aeruginosa, Enterococcus aerogenes, Proteus vulgaris and Enterobacter sakazakii) bacteria, w

209 The rtn gene, identified as coming from Proteus vulgaris ATCC 13315, is present in Escherichia c

210 Serratia marcescens, Erwinia carotovora, and Proteus vulgaris but not in several nonenteric bacteria.

211 We placed 43 isolates belonging to the Proteus vulgaris complex into proposed DNA groups (genom

212 of the tna operon of Escherichia coli and of Proteus vulgaris is induced by L-tryptophan.

213 species-specific class A beta-lactamase from Proteus vulgaris K1 was crystallized at pH 6.25 and its

214 oaceticus BD413, Vibrio cholerae El Tor, and Proteus vulgaris K80, were members of a previously descr

215 Proteus vulgaris L-amino acid deaminase (pvLAAD) belongs

216 ound in the O-polysaccharide of the LPS from Proteus vulgaris OX19 used in the Weil-Felix test, sugge

217 ith deletion constructs of the tna operon of Proteus vulgaris supported this interpretation.

218 e solved two x-ray crystal structures of the Proteus vulgaris tetrameric HigB-(HigA)2-HigB TA complex

219 ng of substrates and inhibitors to wild-type Proteus vulgaris tryptophan indole-lyase and to wild typ

220 Urinary tract infections caused by Proteus vulgaris typically form biofilms and are resista

221 Two commercial enzymes, chondroitinase ABC (Proteus vulgaris) and chondroitinase ACII (Arthrobacter

222 scherichia coli, Salmonella typhimurium, and Proteus vulgaris) We also isolated transposition events

223 Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

224 eas MICs for E. coli, Klebsiella pneumoniae, Proteus vulgaris, and Pseudomonas aeruginosa were > 100

225 of these enzymes, chondroitinase ABC I from Proteus vulgaris, has the broadest substrate specificity

226 of the PvuII plasmid pPvu1, originally from Proteus vulgaris, making this the first completely seque

227 erived from either Salmonella typhimurium or Proteus vulgaris, microorganisms that have diverged from

228 ression of the tryptophanase (tna) operon of Proteus vulgaris, short deletions were introduced in the

229 st Listeria monocytogenes, Escherichia coli, Proteus vulgaris, Staphylococcus aureus, and Candida alb

230 Serratia marcescens, Erwinia carotovora, and Proteus vulgaris, strongly suggesting that the physiolog

231 nzymes, cABCI and cABCII, were identified in Proteus vulgaris.

232 by expressing an L-amino acid deaminase from Proteus vulgaris.

233 nifestations, a reference to the ancient god Proteus, who could assume many forms and thus elude his

234 at the 5-HT3AB receptor (after the Greek god Proteus, who was able to change his shape and appearance