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1 roteus-like (MR/P) fimbriae of uropathogenic Proteus mirabilis.
2 philus influenzae, Bacteroides fragilis, and Proteus mirabilis.
3 een of the isolates were E. coli and one was Proteus mirabilis.
4 ion, is a virulence factor for uropathogenic Proteus mirabilis.
5 acid decarboxylase that inhibits swarming in Proteus mirabilis.
6 d for swarming in the urinary tract pathogen Proteus mirabilis.
7 ity to a putrescine-deficient speA mutant of Proteus mirabilis.
8 es in Klebsiella spp., Escherichia coli, and Proteus mirabilis.
9 important virulence factor of uropathogenic Proteus mirabilis.
10 toxin to prevent urinary tract infection by Proteus mirabilis.
11 ifferentiation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by
12 (4.4%), Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and
16 Recently, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated
25 have shown previously using hyperflagellated Proteus mirabilis and a motile but non-swarming flgN tra
26 significant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia c
27 of Gram-negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates
30 of a urease-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was as
31 during UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition
32 operon as a major assimilatory checkpoint in Proteus mirabilis and other Gram-negative bacteria and e
35 zation by urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occ
36 s II promoter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed det
37 cherichia coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by i
38 segregate from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcomp
39 ier studies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the
40 ct on the numbers of Salmonella typhimurium, Proteus mirabilis, and Escherichia coli internalized by
43 as Klebsiella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhi
44 olates of Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associate
45 ted urinary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology i
48 ol for application of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a se
55 fundamental behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to th
56 re also found in cell-free supernatants from Proteus mirabilis, Citrobacter freundii and Enterobacter
61 n CFT073, is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mira
62 ns), Listeria monocytogenes (three strains), Proteus mirabilis, Escherichia coli (three strains), and
67 rming motility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little und
68 -ray crystal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vit
69 se-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indicate that MrpB functions as the t
70 acteriaceae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that
71 , Seo et al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent i
72 hat provides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.
88 rotease, ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with
89 loacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates)
91 6 (3%) Klebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, w
92 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESB
94 lular migration in a non-swarming but motile Proteus mirabilis mutant lacking the FIgN facilitator of
95 ae (n = 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Steno
98 kb PAI, designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella
99 scherichia coli 1021, Klebsiella pneumoniae, Proteus mirabilis, Providencia stuartii, and Pseudomonas
100 ibrary included probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcu
101 gram negative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pn
104 structures of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembran
105 prevent colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia
106 y used to assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aerug
107 by Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subt
109 een to identify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy su
111 In laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at a
115 We tested the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would
116 truncated form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and struc
118 higher than the averages were observed with Proteus mirabilis versus imipenem and with Klebsiella pn
119 e arfA hairpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella mult
120 to virulence in other pathogens, its role in Proteus mirabilis was investigated by constructing a str
122 genesis of urinary tract infection caused by Proteus mirabilis, we constructed a nonmotile, nonswarmi
123 riminating strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between
124 tract pathogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable in
125 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended
126 plementation studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) tr
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