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1 roteus-like (MR/P) fimbriae of uropathogenic Proteus mirabilis.
2 philus influenzae, Bacteroides fragilis, and Proteus mirabilis.
3 een of the isolates were E. coli and one was Proteus mirabilis.
4 ion, is a virulence factor for uropathogenic Proteus mirabilis.
5 acid decarboxylase that inhibits swarming in Proteus mirabilis.
6 d for swarming in the urinary tract pathogen Proteus mirabilis.
7 ity to a putrescine-deficient speA mutant of Proteus mirabilis.
8 es in Klebsiella spp., Escherichia coli, and Proteus mirabilis.
9  important virulence factor of uropathogenic Proteus mirabilis.
10  toxin to prevent urinary tract infection by Proteus mirabilis.
11 ifferentiation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by
12 (4.4%), Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and
13             Here we characterize PmDsbA from Proteus mirabilis, a bacterial pathogen increasingly ass
14                                              Proteus mirabilis, a cause of complicated urinary tract
15                                              Proteus mirabilis, a cause of complicated urinary tract
16  Recently, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated
17                                              Proteus mirabilis, a common cause of nosocomial and cath
18                                              Proteus mirabilis, a common cause of urinary tract infec
19                                              Proteus mirabilis, a gram-negative bacterium associated
20                                              Proteus mirabilis, a gram-negative bacterium, is a frequ
21                                              Proteus mirabilis, a Gram-negative bacterium, represents
22                                              Proteus mirabilis, a leading cause of catheter-associate
23                                              Proteus mirabilis alternates between motile and adherent
24                                              Proteus mirabilis, an etiologic agent of complicated uri
25 have shown previously using hyperflagellated Proteus mirabilis and a motile but non-swarming flgN tra
26 significant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia c
27  of Gram-negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates
28                       We focused on Lrp from Proteus mirabilis and E. coli, orthologs with 98% identi
29                          The closely related Proteus mirabilis and Enterobacterlaceae plasmid-encoded
30 of a urease-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was as
31  during UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition
32 operon as a major assimilatory checkpoint in Proteus mirabilis and other Gram-negative bacteria and e
33                                          The Proteus mirabilis and plasmid-encoded urease loci contai
34                  The urease-positive species Proteus mirabilis and Providencia stuartii are two of th
35 zation by urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occ
36 s II promoter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed det
37 cherichia coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by i
38 segregate from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcomp
39 ier studies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the
40 ct on the numbers of Salmonella typhimurium, Proteus mirabilis, and Escherichia coli internalized by
41 ization of wild-type Salmonella typhimurium, Proteus mirabilis, and Escherichia coli.
42 i, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).
43  as Klebsiella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhi
44 olates of Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associate
45 ted urinary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology i
46            Fimbriae of the human uropathogen Proteus mirabilis are the only characterized surface pro
47                                        Using Proteus mirabilis as a model, we investigate the role of
48 ol for application of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a se
49                                              Proteus mirabilis, associated with complicated urinary t
50                     One of the six predicted Proteus mirabilis autotransporters (ATs), ORF c2341, is
51 onstructed from the 50 %-identical TEM-1 and Proteus mirabilis beta-lactamases.
52  become encrusted and blocked by crystalline Proteus mirabilis biofilms.
53                                              Proteus mirabilis can rapidly utilize choline to enhance
54                                              Proteus mirabilis causes complicated urinary tract infec
55  fundamental behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to th
56 re also found in cell-free supernatants from Proteus mirabilis, Citrobacter freundii and Enterobacter
57           A total of 63 clinical isolates of Proteus mirabilis collected over a 19-month period were
58                                              Proteus mirabilis colonies exhibit striking geometric re
59                                              Proteus mirabilis commonly infects the complicated urina
60                                              Proteus mirabilis compromises the care of many patients
61 n CFT073, is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mira
62 ns), Listeria monocytogenes (three strains), Proteus mirabilis, Escherichia coli (three strains), and
63                                              Proteus mirabilis forms extensive crystalline biofilms o
64                                              Proteus mirabilis forms extensive crystalline biofilms o
65           In this study, we describe wosA, a Proteus mirabilis gene identified by its ability to incr
66                              In a search for Proteus mirabilis genes that were regulated by cell-to-c
67 rming motility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little und
68 -ray crystal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vit
69 se-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indicate that MrpB functions as the t
70 acteriaceae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that
71 , Seo et al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent i
72 hat provides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.
73                        The enteric bacterium Proteus mirabilis is a common cause of complicated urina
74                                              Proteus mirabilis is a common cause of urinary tract inf
75                                              Proteus mirabilis is a common uropathogen in patients wi
76                                              Proteus mirabilis is a dimorphic motile bacterium well k
77                                              Proteus mirabilis is a dimorphic, motile bacterium often
78          The gram-negative enteric bacterium Proteus mirabilis is a frequent cause of urinary tract i
79                                              Proteus mirabilis is a Gram-negative bacterium that exis
80                                              Proteus mirabilis is a Gram-negative bacterium that unde
81                                              Proteus mirabilis is a model organism for urease-produci
82                                              Proteus mirabilis is a urinary tract pathogen and well k
83                                              Proteus mirabilis is a urinary tract pathogen that diffe
84                                              Proteus mirabilis is an opportunistic pathogen that is f
85                        The enteric bacterium Proteus mirabilis is associated with a significant numbe
86                                The bacterium Proteus mirabilis is capable of movement on solid surfac
87                                  Swarming by Proteus mirabilis is characterized by cycles of rapid an
88 rotease, ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with
89 loacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates)
90             Swarming colonies of independent Proteus mirabilis isolates recognize each other as forei
91 6 (3%) Klebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, w
92 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESB
93 gh level of similarity to sequences encoding Proteus mirabilis mannose-resistant fimbriae.
94 lular migration in a non-swarming but motile Proteus mirabilis mutant lacking the FIgN facilitator of
95 ae (n = 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Steno
96        Molecular analyses have revealed that Proteus mirabilis possesses two genes, flaA and flaB, th
97                                Uropathogenic Proteus mirabilis produces at least four types of fimbri
98 kb PAI, designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella
99 scherichia coli 1021, Klebsiella pneumoniae, Proteus mirabilis, Providencia stuartii, and Pseudomonas
100 ibrary included probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcu
101 gram negative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pn
102                                An isolate of Proteus mirabilis recovered from blood cultures of a dia
103 Escherichia coli, Klebsiella pneumoniae, and Proteus mirabilis remains unknown.
104 structures of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembran
105  prevent colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia
106 y used to assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aerug
107  by Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subt
108                                              Proteus mirabilis swarming behavior is characterized by
109 een to identify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy su
110              Here, we identified a gene from Proteus mirabilis that encodes a 135-amino acid residue
111      In laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at a
112               MR/P fimbriae of uropathogenic Proteus mirabilis undergo invertible element-mediated ph
113                                              Proteus mirabilis urease catalyzes the hydrolysis of ure
114                                Expression of Proteus mirabilis urease is governed by UreR, an AraC-li
115   We tested the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would
116 truncated form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and struc
117                                Patients with Proteus mirabilis UTIs were more likely to have a foreig
118  higher than the averages were observed with Proteus mirabilis versus imipenem and with Klebsiella pn
119 e arfA hairpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella mult
120 to virulence in other pathogens, its role in Proteus mirabilis was investigated by constructing a str
121                           A TnphoA mutant of Proteus mirabilis was isolated, which had lost the abili
122 genesis of urinary tract infection caused by Proteus mirabilis, we constructed a nonmotile, nonswarmi
123 riminating strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between
124  tract pathogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable in
125 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended
126 plementation studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) tr

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