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1 serotonin reuptake inhibitors (SSRIs; e.g., Prozac).
2 ansmission and is potentiated by fluoxetine (Prozac).
3 ppressed by therapeutic doses of fluoxetine (Prozac).
4 ive ingredient in the popular antidepressant Prozac.
5 effects of the serotonin reuptake inhibitor Prozac.
8 xetine, the active metabolite of fluoxetine (Prozac) and a state-dependent blocker of TREK channels.
10 rmaceuticals including Claritin, fluoxetine (Prozac), and [(18) F]DAA1106 were synthesized to show th
11 reuptake inhibitors, typified by fluoxetine (Prozac), and the more recently developed norepinephrine-
12 ine, citalopram, sertraline, and fluoxetine (Prozac), bound more avidly to SERT in the presence of Cl
13 ice with the antidepressant drug fluoxetine (Prozac) causes changes in C', C, and D site editing that
15 ral changes resulting from early fluoxetine (Prozac) exposure were different from those of ESC and, a
19 active ingredient of the antidepressant drug Prozac, inhibits reuptake of the neurotransmitter, serot
24 eiving chronic administration of fluoxetine (Prozac), others receiving a 5-HT(1A) receptor antagonist
25 tonin reuptake inhibitors (SSRI)-fluoxetine (Prozac), paroxetine (Paxil), and citalopram (Celexa)-sub
26 ive serotonin reuptake inhibitor fluoxetine (Prozac) potentiated the enhanced slowing response, and t
28 orters (SERTs) are targets for drugs such as Prozac that increase serotonin (5HT) levels by blocking
29 ve 5HT reuptake inhibitor (SSRI) fluoxetine (Prozac), the tricyclic antidepressant imipramine, and do
30 eta(1) receptor by the transporter inhibitor Prozac, the inhibition of the 5-hydroxytryptamine (5-HT)
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