ライフサイエンスコーパス: Pseudomonas

1 l details of the amyloid transporter FapF in Pseudomonas.

2 ced stomatal closing and more susceptible to Pseudomonas.

3 In conclusion, mucoid Pseudomonas adapted to the CF-lung remained able to inva

4 us aureus strain Y5 and ampicillin resistant Pseudomonas aerugenosa ATCC9027 strain, demonstrating po

5 Staphylococcus aureus (12.9%) and Pseudomonas aeruginosa (11.5%) were the most common path

6 (n=355) were Klebsiella pneumoniae (37%) and Pseudomonas aeruginosa (30%); 28% were ceftazidime-non-s

7 tive Staphylococcus aureus and gram-negative Pseudomonas aeruginosa (99.3 +/- 1.9% and 88.5 +/- 3.3%

8 for the detection of carbapenemase-producing Pseudomonas aeruginosa (CP-PA) and carbapenemase-produci

9 Infections by multidrug-resistant Pseudomonas aeruginosa (MDRPa) are an important cause of

10 Pseudomonas aeruginosa (PA) is a ubiquitous microbe.

11 he type I-F CRISPR adaptive immune system in Pseudomonas aeruginosa (PA14) consists of two CRISPR loc

12 y identified NB predicted to be specific for Pseudomonas aeruginosa (pyocin Sn) was produced and show

13 posed to include Acinetobacter baumannii and Pseudomonas aeruginosa (SNAP and POP studies).

14 The adaptive immune system in Pseudomonas aeruginosa (type I-F) relies on a 350 kDa CR

15 by the Gram-negative opportunistic pathogen, Pseudomonas aeruginosa Activation of phospholipase activ

16 Pseudomonas aeruginosa adaptation to survive in the host

17 healthy mouse corneas becomes vulnerable to Pseudomonas aeruginosa adhesion if it lacks the innate d

18 s this question in the pathogenic bacterium, Pseudomonas aeruginosa Although public goods producers w

19 bial performance against two MDR isolates of Pseudomonas aeruginosa and Acinetobacter baumannii throu

20 ing Gram-negative bacilli (CPNFs), including Pseudomonas aeruginosa and Acinetobacter baumannii, is n

21 erved against multidrug-resistant strains of Pseudomonas aeruginosa and Acinetobacter baumannii.

22 and model drug by LPS from F. tularensis vs Pseudomonas aeruginosa and by F. tularensis live bacteri

23 coccus pyogenes) and gram-negative bacteria (Pseudomonas aeruginosa and Escherichia coli) over time t

24 n of biofilms formed by the mucoid strain of Pseudomonas aeruginosa and investigated the commonality

25 is synthesized by the opportunistic pathogen Pseudomonas aeruginosa and is an important biofilm const

26 PPH assay and antibacterial activity against Pseudomonas aeruginosa and Staphylococcus aureus was ass

27 rial activity of raw rapeseed honeys against Pseudomonas aeruginosa and Staphylococcus aureus, with a

28 red by new data from 28 clinical isolates of Pseudomonas aeruginosa and strains evolved in laboratory

29 ough horizontal gene transfer by the species Pseudomonas aeruginosa and subsequently abundant P. aeru

30 obal sRNA interactions with their targets in Pseudomonas aeruginosa and verified the method with a kn

31 way infections by the opportunistic pathogen Pseudomonas aeruginosa are a major cause of mortality in

32 Staphylococci, Streptococcus pneumoniae and Pseudomonas aeruginosa are the leading isolates in ocula

33 Using Pseudomonas aeruginosa as a model system, the authors sh

34 between the two domains and is able to kill Pseudomonas aeruginosa at sub-micromolar concentrations.

35 the study was to assess the applicability of Pseudomonas aeruginosa ATCC9027 and its validated biolum

36 olymicrobial, with Staphylococcus aureus and Pseudomonas aeruginosa being the two most commonly isola

37 plant metabolites called flavonoids inhibit Pseudomonas aeruginosa biofilm formation by an unknown m

38 Pseudomonas aeruginosa biofilm infections are difficult

39 ial transcriptome map of the mature in vitro Pseudomonas aeruginosa biofilm model, revealing contempo

40 This paper studies the formation of a Pseudomonas aeruginosa biofilm on a Papyex graphite (PA)

41 We established a new approach to study Pseudomonas aeruginosa biofilm susceptibility on biotic

42 d healing, wild-type mice were infected with Pseudomonas aeruginosa biofilms and, akin to Nod2(-/-) m

43 on interact to shape competitive dynamics in Pseudomonas aeruginosa biofilms.

44 Moreover, Pseudomonas aeruginosa BioH is more highly expressed tha

45 ated the best antibacterial activity against Pseudomonas aeruginosa both in vitro and in vivo for tet

46 We describe for the first time how Pseudomonas aeruginosa can utilize human recombinant MIF

47 The N-acetylglucosaminidase NagZ of Pseudomonas aeruginosa catalyzes the first cytoplasmic s

48 that enables cross-species interactions, as Pseudomonas aeruginosa cells also become attracted to th

49 ng approximately 90% of Escherichia coli and Pseudomonas aeruginosa cells within 90-120 and 5-30 min,

50 face glycosylation is modulated by IL-1R and Pseudomonas aeruginosa challenge but is insufficient for

51 ne conductance regulator (CFTR) that reduces Pseudomonas aeruginosa culture positivity in CF patients

52 Ivacaftor produced rapid decreases in sputum Pseudomonas aeruginosa density that began within 48 hour

53 reted components by the pathogenic bacterium Pseudomonas aeruginosa during growth on a protein substr

54 The nefarious Gram-negative pathogen Pseudomonas aeruginosa encodes eleven LTs.

55 ysts of intracellular phenazine reduction in Pseudomonas aeruginosa Enzymatic assays in cell-free lys

56 Strains of the opportunistic pathogen Pseudomonas aeruginosa express one of five different typ

57 The opportunistic pathogen Pseudomonas aeruginosa forms antimicrobial resistant bio

58 ic coinfections of Staphylococcus aureus and Pseudomonas aeruginosa frequently fail to respond to ant

59 f Enterobacteriaceae, Acinetobacter spp, and Pseudomonas aeruginosa from 18 698 inpatients and 2923 h

60 ce variant of PTEN, were unable to eradicate Pseudomonas aeruginosa from the airways and could not ge

61 r interface and the active sites can abolish Pseudomonas aeruginosa growth in a defined medium with m

62 nization with other pathogens, in particular Pseudomonas aeruginosa Here, we demonstrate that CF mice

63 )F-fluoromaltotriose was also able to detect Pseudomonas aeruginosa in a clinically relevant mouse mo

64 apid, redox-active biomarker for identifying Pseudomonas aeruginosa in clinical infections.

65 red to those to the well-studied CF pathogen Pseudomonas aeruginosa In parallel, mice were also infec

66 Pseudomonas aeruginosa infection liberates transmissible

67 tactic gradients, and migrate in response to Pseudomonas aeruginosa infection of primary ALI barriers

68 leus, histamine blocker use, and respiratory Pseudomonas aeruginosa infection were associated with lo

69 the eye from pathogenic Candida albicans or Pseudomonas aeruginosa infection.

70 t poly(I:C), improved the host response to a Pseudomonas aeruginosa infection.

71 resistance during multidrug resistant (MDR)-Pseudomonas aeruginosa infections are limited.

72 Recurrent Pseudomonas aeruginosa infections coupled with robust, d

73 Pseudomonas aeruginosa is a Gram-negative bacterial path

74 Pseudomonas aeruginosa is a Gram-negative opportunistic

75 Pseudomonas aeruginosa is a Gram-negative, opportunistic

76 Pseudomonas aeruginosa is a leading cause of nosocomial

77 The opportunistic pathogen Pseudomonas aeruginosa is a major cause of sepsis in sev

78 Pseudomonas aeruginosa is a major cause of severe infect

79 Pseudomonas aeruginosa is a pathogenic gram-negative org

80 Pseudomonas aeruginosa is a significant contributor to r

81 Pseudomonas aeruginosa is a ubiquitous environmental org

82 Pseudomonas aeruginosa is among the leading causes of se

83 Pseudomonas aeruginosa is an important opportunistic hum

84 Pseudomonas aeruginosa is an opportunistic and frequentl

85 The ubiquitous bacterium Pseudomonas aeruginosa is an opportunistic pathogen that

86 nsporter FpvAI in the opportunistic pathogen Pseudomonas aeruginosa is hijacked to translocate the ba

87 resolution, we show that granule genesis in Pseudomonas aeruginosa is tightly organized under nitrog

88 ecretion system effector found in 90% of the Pseudomonas aeruginosa isolates.

89 robacteriaceae, Acinetobacter baumannii, and Pseudomonas aeruginosa isolates.

90 accounts for biofilm-mediated resistance to Pseudomonas aeruginosa killing.

91 Pseudomonas aeruginosa lacking the Pseudomonas prolyl-hy

92 ghput to sequence approximately 400 clinical Pseudomonas aeruginosa libraries and demonstrate excelle

93 A Pseudomonas aeruginosa mutant lacking all three known ir

94 nfections such as invasive aspergillosis and Pseudomonas aeruginosa occurred during hospitalization.

95 Growth of Pseudomonas aeruginosa on spermine requires a functional

96 fluorescent pseudomonads, such as pathogenic Pseudomonas aeruginosa or plant growth-promoting Pseudom

97 receptor, was induced by infection with live Pseudomonas aeruginosa or treatment of cells with its su

98 Here, we show that the bacterium Pseudomonas aeruginosa PA14 uses the cell-cell communica

99 In the pathogenic bacterium Pseudomonas aeruginosa PA14, antibiotics called phenazin

100 nd respiration in the opportunistic pathogen Pseudomonas aeruginosa PA14.

101 g protein (PumA) of the multi-drug resistant Pseudomonas aeruginosa PA7 strain.

102 The responses of Pseudomonas aeruginosa PAO1 and PA14 to a hexadecane-wat

103 The textile MFC used Pseudomonas aeruginosa PAO1 as a biocatalyst to generate

104 receptors and a single chemosensory pathway, Pseudomonas aeruginosa PAO1 has a much more complex chem

105 aves B-band LPS (O-specific antigen, OSA) of Pseudomonas aeruginosa PAO1.

106 Phis are hyperinflammatory and have impaired Pseudomonas aeruginosa phagocytosis, phenocopying CF MPh

107 The opportunistic pathogen Pseudomonas aeruginosa produces colorful redox-active me

108 The opportunistic pathogen Pseudomonas aeruginosa produces the cationic exopolysacc

109 This cascade consists of four Pseudomonas aeruginosa protein regulators (ExsADCE) that

110 e, the authors show that two DMAbs targeting Pseudomonas aeruginosa proteins confer protection agains

111 ontaining phosphodiesterase domains from the Pseudomonas aeruginosa proteins PA3825 (PA3825(EAL)) and

112 The outer membrane protein H (OprH) of Pseudomonas aeruginosa provides an increased stability t

113 Analysis of a Pseudomonas aeruginosa quiC1 gene knock-out demonstrates

114 co-infection of murine surgical wounds with Pseudomonas aeruginosa results in conversion of approxim

115 me-scale metabolic network reconstruction of Pseudomonas aeruginosa strain PA14 and an updated, expan

116 sted the individual oxidants on the virulent Pseudomonas aeruginosa strain PA14.

117 We demonstrate that the pathogenicity of Pseudomonas aeruginosa strains derived from acute clinic

118 ix mares were inoculated with lux-engineered Pseudomonas aeruginosa strains isolated from equine uter

119 ative organisms with higher activity towards Pseudomonas aeruginosa than the naturally-occurring AMP

120 In other genomes such as Pseudomonas aeruginosa the bioH gene is within a biotin

121 faucets were most frequently colonized, and Pseudomonas aeruginosa the predominant organism.

122 a chemotaxis-regulating methyltransferase in Pseudomonas aeruginosa This cocrystal structure, togethe

123 trate a 5-log increase in the sensitivity of Pseudomonas aeruginosa to ciprofloxacin.

124 s by RNA-Seq to characterize the response of Pseudomonas aeruginosa to external 0.5 mm CuSO4, a condi

125 Here, we experimentally evolved Pseudomonas aeruginosa to six 2-drug sequences.

126 tudying the sensitivities of a ohr mutant of Pseudomonas aeruginosa toward different hydroperoxides.

127 The Pseudomonas aeruginosa type III secretion system deliver

128 lus also are dispensable for activation of a Pseudomonas aeruginosa type VI secretion system (T6SS).

129 and gene essentiality in the model organism Pseudomonas aeruginosa UCBPP-PA14.

130 activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infection, suggesti

131 ce in the opportunistic pathogenic bacterium Pseudomonas aeruginosa via an unknown pathway.

132 corneal epithelial barrier function against Pseudomonas aeruginosa was MyD88-dependent.

133 compared with that of its ortholog LecA from Pseudomonas aeruginosa We also investigated the utility

134 ococcus faecalis, Klebsiella pneumoniae, and Pseudomonas aeruginosa We therefore conclude that the un

135 Escherichia coli, Klebsiella pneumoniae, and Pseudomonas aeruginosa were the most common isolates.

136 patients infected with carbapenem-resistant Pseudomonas aeruginosa who were treated with ceftolozane

137 Escherichia coli, Staphylococcus aureus and Pseudomonas aeruginosa) and in vitro anti-proliferative

138 Staphylococcus aureus, Escherichia coli, and Pseudomonas aeruginosa) relative to controls.

139 ion by other uropathogens (Escherichia coli, Pseudomonas aeruginosa) was also explored, and thioridaz

140 old the minimal inhibitory concentration for Pseudomonas aeruginosa) were compared between the two gr

141 ic bacteria (e.g., Staphylococcus aureus and Pseudomonas aeruginosa).

142 for the majority of Gram negative bacteria (Pseudomonas aeruginosa, 16-32 mug/mL, Klebsiella pneumon

143 broth microdilution (BMD) for 99 isolates of Pseudomonas aeruginosa, 26 Acinetobacter baumannii isola

144 at are notoriously problematic in hospitals: Pseudomonas aeruginosa, Acinetobacter baumannii, and Sta

145 gainst experimental endocarditis (EE) due to Pseudomonas aeruginosa, an archetype of difficult-to-tre

146 ith 294 isolates of Enterobacteriaceae spp., Pseudomonas aeruginosa, and Acinetobacter baumannii chos

147 (Staphylococcus aureus, Enterobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii).

148 nslational modification in Escherichia coli, Pseudomonas aeruginosa, and Bacillus subtilis.

149 apenem-resistant Acinetobacter baumannii and Pseudomonas aeruginosa, and carbapenem-resistant and thi

150 lococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebsiella pneumoniae, which

151 ria-Staphylococcus aureus, Escherichia coli, Pseudomonas aeruginosa, and mycobacteria.

152 c bacterial strains: Legionella pneumophila, Pseudomonas aeruginosa, and Salmonella typhimurium.

153 For Pseudomonas aeruginosa, human monoclonal antibodies (mAb

154 ation with bacterial pathogens, particularly Pseudomonas aeruginosa, is the primary cause of morbidit

155 For Pseudomonas aeruginosa, it has long been known that intr

156 Here we defined PF orthologs in Pseudomonas aeruginosa, Moraxella catarrhalis, and Staph

157 arothermophilus) and gram-negative bacteria (Pseudomonas aeruginosa, Pseudomonas fluorescens, Salmone

158 hicillin-resistant Staphylococcus aureus and Pseudomonas aeruginosa, respectively, in catheter-associ

159 tunistic pathogens Aspergillus fumigatus and Pseudomonas aeruginosa, respectively.

160 ith Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphylococcus aureus (including

161 netobacter baumannii, Klebsiella pneumoniae, Pseudomonas aeruginosa, Staphylococcus aureus, and coagu

162 ebsiella pneumoniae, Legionella pneumophila, Pseudomonas aeruginosa, Stenotrophomonas maltophilia, Vi

163 ion of the peptide siderophore pyoverdine by Pseudomonas aeruginosa, under different nutrient-limitin

164 carbapenem-resistant Enterobacteriaceae and Pseudomonas aeruginosa, which are difficult to treat.

165 utrophil synthesis of LTB4 in the context of Pseudomonas aeruginosa-induced neutrophil transepithelia

166 Escherichia coli, Klebsiella pneumoniae, and Pseudomonas aeruginosa-reported here underscores the bro

167 e mass spectra of membrane grown biofilms of Pseudomonas aeruginosa.

168 le and cultures of Aspergillus fumigatus and Pseudomonas aeruginosa.

169 h a unique species-specific activity against Pseudomonas aeruginosa.

170 positive Bacillus subtilis and Gram-negative Pseudomonas aeruginosa.

171 natural bispecific IgG1 candidate, targeting Pseudomonas aeruginosa.

172 ctamases, but is considered inactive against Pseudomonas aeruginosa.

173 duced and expressed in the outer membrane of Pseudomonas aeruginosa.

174 inhibits QS in opportunistic human pathogen Pseudomonas aeruginosa.

175 ould be maintained when tested on pathogenic Pseudomonas aeruginosa.

176 ignature analysis for the bacterial pathogen Pseudomonas aeruginosa.

177 nescent strains of Staphylococcus aureus and Pseudomonas aeruginosa.

178 abolite by the opportunistic human pathogen, Pseudomonas aeruginosa.

179 e-associated pyochelin siderophore system in Pseudomonas aeruginosa.

180 m antibiotics in many Enterobacteriaceae and Pseudomonas aeruginosa.

181 large, non-Nocardia, or classically invasive Pseudomonas aeruginosa; for patients with low baseline v

182 ested that electrochemically active bacteria Pseudomonas and Acinetobacter transferred electrons extr

183 We found that Pseudomonas and Basidiomycota OTUs were associated with

184 mixture of 19 amino acids and glucose by two Pseudomonas and one Bacillus species isolated from groun

185 man hosts resist opportunistic infections by Pseudomonas and other pyochelin-expressing bacteria.

186 Several Pseudomonas and Xanthomonas species are plant pathogens

187 roinflammatory bacteria (eg, Staphylococcus, Pseudomonas, and Corynebacterium), anabolic remodeling w

188 us, Klebsiella, Ochrobactrum, Paenibacillus, Pseudomonas, and Ralstonia) confirmed that metal toleran

189 and unexpected microbial molecules including Pseudomonas-associated quinolones and rhamnolipids in fe

190 Pseudomonas cells arrive at a surface with low levels of

191 and cryo-electron tomography, we showed that Pseudomonas chlororaphis phage 201phi2-1 assembled a com

192 s Xanthomonas species, as well as HopQ1 from Pseudomonas, demonstrating widespread potential applicat

193 een shown to be resistant to Xanthomonas and Pseudomonas due to an immune response triggered by the b

194 vy chain-only antibodies (VHH) conjugated to Pseudomonas exotoxin A to deplete myeloid cells in vitro

195 Immunotoxins derived from Pseudomonas exotoxin are antibody-toxin fusion proteins

196 into the functional amyloid transporter from Pseudomonas, FapF.

197 bacteria Acinetobacter baylyi ADP1, not for Pseudomonas fluorescence, which exhibits limited chemota

198 putative periplasmic oxidoreductase PvdO of Pseudomonas fluorescens A506 is required for the final o

199 Probiotic baths of surface symbionts, Pseudomonas fluorescens and Flavobacterium johnsoniae we

200 Using Pseudomonas fluorescens as a model biofilm-forming bacte

201 Here, Pseudomonas fluorescens has been introduced to the syste

202 cillus subtilis, Lactobacillus rhamnosus and Pseudomonas fluorescens induces C. elegans stress resist

203 uminescence from the bioluminescent reporter Pseudomonas fluorescens M3A.

204 I, found in Pseudomonas stutzeri DSM4166 and Pseudomonas fluorescens SBW25, respectively.

205 domonas aeruginosa or plant growth-promoting Pseudomonas fluorescens The non-ribosomal peptide ferrib

206 carried out using 10(7) and 10(8) CFU mL(-1) Pseudomonas fluorescens to study the effects of the elec

207 opy, we investigated the interaction between Pseudomonas fluorescens, a biofilm-forming bacterium, an

208 m-negative bacteria (Pseudomonas aeruginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmone

209 d specifically the genera Pseudoalteromonas, Pseudomonas, Halomonas, and Cobetia.

210 mpositional shift, specifically in the genus Pseudomonas in Nod2(-/-) mice.

211 tration of long-lived cytotoxic agents after Pseudomonas infection may establish a molecular link to

212 We show that Pseudomonas invaded the host microbiota within three day

213 s (CF) patients, chronic airway infection by Pseudomonas leads to progressive lung destruction ultima

214 Here we report the crystal structure of a Pseudomonas malonate decarboxylase hetero-tetramer, as w

215 e authors present the crystal structure of a Pseudomonas MDC and give insights into its catalytic mec

216 ifically linked with infection attributed to Pseudomonas or Acinetobacter spp.

217 aligenes (PpCutA) and a putative lipase from Pseudomonas pelagia (PpelaLip) were identified.

218 Pseudomonas phage LKA1 of the subfamily Autographivirina

219 nsferase-QueC homologs in Enterobacteria and Pseudomonas phage, and distant homologs in other phage a

220 Collective responses of Pseudomonas PMM/PGM to phosphosugar substrates and inhib

221 We investigated whether CF-adapted Pseudomonas populations invade the donor microbiota and

222 Pseudomonas aeruginosa lacking the Pseudomonas prolyl-hydroxylase domain-containing protein

223 antagonism by the plant commensal bacterium Pseudomonas protegens Consistent with the established ef

224 Here, we investigated the soil bacterium Pseudomonas protegens Pf-5, a strain remarkable for its

225 l metabolites produced by the soil bacterium Pseudomonas protegens.

226 sed on the in-silico search, a cutinase from Pseudomonas pseudoalcaligenes (PpCutA) and a putative li

227 In Pseudomonas putida biofilms, nutrient starvation trigger

228 . coli K-12) in mixtures with soil bacteria (Pseudomonas putida F1 and Bacillus subtilis 168).

229 Exposure of Pseudomonas putida F1 to 0.1, 1.0, and 5.0 g/L of nZVI c

230 en-gene operon that enables LA catabolism in Pseudomonas putida KT2440.

231 us irregularis and the rhizobacterial strain Pseudomonas putida KT2440.

232 ng LysR-type transcriptional regulators from Pseudomonas putida KT2440.

233 ing the only GGDEF/EAL response regulator in Pseudomonas putida, is transcriptionally regulated by Rp

234 The Pseudomonas quinolone signal (PQS) compound is a secrete

235 y directly interacting with the iron-binding Pseudomonas quinolone signal (PQS), a cell-cell signalli

236 rrF sRNAs also promote the production of the Pseudomonas quinolone signal (PQS), a quorum sensing mol

237 PqsR acts as the receptor for the Pseudomonas quinolone signal, and it controls the produc

238 e sequences matched 35% of the Illumina PMEZ Pseudomonas reads.

239 in several Gram-negative bacteria including Pseudomonas, Shewanella and Enterobacter.

240 nsposon mutagenesis sequencing (RB-TnSeq) in Pseudomonas simiae, a model root-colonizing bacterium, t

241 generated phnJL clones matched those of the Pseudomonas sp.

242 nt pathways of phenylalanine biosynthesis in Pseudomonas sp.

243 amine (BDMA) was identified in the genome of Pseudomonas sp. BIOMIG1, which is a bacterium present in

244 A reduction in phenylalanine deuteration in Pseudomonas sp. compared to that in E. coli is due to th

245 In 16S rRNA gene Illumina libraries, four Pseudomonas sp. operational taxonomic units surprisingly

246 oxic freshwater lake environments, and that Pseudomonas sp. populations are critical participants.

247 Escherichia coli used deuterated glucose and Pseudomonas sp. used deuterated naphthalene as sole carb

248 Typical wastewater microorganisms like Pseudomonas sp. were chosen for in-silico screening towa

249 Pseudomonas species and other aerobic bacteria have a bi

250 To confirm whether Pseudomonas species directly impair wound healing, wild-

251 ater deer harbored an increased abundance of Pseudomonas spp. and Acinetobacter spp., while milk from

252 Fluorescent Pseudomonas spp. are widely studied for their beneficial

253 To explore the genetic diversity of Pseudomonas spp. in tropical regions, we collected 76 is

254 onal genes also present in known fluorescent Pseudomonas spp. strains.

255 psychrotrophics, Brochothrix thermosphacta, Pseudomonas spp., and Enterobacteriaceae in AP meat comp

256 ltivation experiments revealed that in these Pseudomonas strains, cleavage of glycerolphosphorylcholi

257 Following LT, CF-adapted Pseudomonas strains, potentially originating from the si

258 aminoacid was carried out with a lipase from Pseudomonas stutzeri and a protease from Bacillus subtil

259 depolymerization of a native EPS produced by Pseudomonas stutzeri AS22.

260 osphodiesterases, GlpQI and GlpQII, found in Pseudomonas stutzeri DSM4166 and Pseudomonas fluorescens

261 The ptxD gene, derived from Pseudomonas stutzeri WM88, that confers to cells the abi

262 esponse to the injection of avrRpm1-modified Pseudomonas syringae (P = 1.66e-08).

263 A previously unstudied Pseudomonas syringae (Psy) type III effector, HopBB1, in

264 per basal immunity to the bacterial pathogen Pseudomonas syringae Although SARD4 knockout plants show

265 mlo2 mlo6 mlo12 triple mutants, as shown for Pseudomonas syringae and Fusarium oxysporum.

266 cretion was enhanced in plants infected with Pseudomonas syringae and in response to treatment with s

267 rs during infection with the foliar pathogen Pseudomonas syringae and the vascular pathogen Ralstonia

268 Plant pathogenic bacteria, like Pseudomonas syringae and Xanthomonas campestris, use the

269 se (HR) typical of ETI is abolished when the Pseudomonas syringae effector AvrRpt2 is bacterially del

270 penetration, in this study we expressed the Pseudomonas syringae effector HopAI known to inactivate

271 JMJ27 is induced in response to virulent Pseudomonas syringae pathogens and is required for resis

272 rial causal agent of bleeding canker disease Pseudomonas syringae pv aesculi, and the bark-associated

273 secretion system-deficient bacterial strain Pseudomonas syringae pv tomato (Pst) DC3000 hrcC(-) and

274 avirulent strains of the bacterial pathogen Pseudomonas syringae pv tomato DC3000 results in a drast

275 nd resistance against the virulent bacterium Pseudomonas syringae pv tomato DC3000.

276 r, led to plants with enhanced resistance to Pseudomonas syringae pv.

277 Arabidopsis thaliana to the foliar pathogen Pseudomonas syringae pv.

278 ween Arabidopsis and its bacterial pathogen, Pseudomonas syringae pv.

279 The introduction of Pseudomonas syringae pv. actinidiae (Psa) severely damag

280 nst Xanthomonas citri subsp. citri (Xcc) and Pseudomonas syringae pv. phaseolicola (Psp) NPS3121.

281 The ethylene-forming enzyme (EFE) from Pseudomonas syringae pv. phaseolicola PK2 is a member of

282 creases the susceptibility of Arabidopsis to Pseudomonas syringae pv. tomato (Pst) DC3000 independent

283 ld-type plants, against avirulent strains of Pseudomonas syringae pv. tomato DC3000 (Pst) carrying Av

284 e resistance against the biotrophic bacteria Pseudomonas syringae pv. tomato DC3000 and for susceptib

285 tly increased upon infection with pathogenic Pseudomonas syringae pv. tomato DC3000 lacking hopQ1-1 [

286 merina BMM (PcBMM), but not to the bacterium Pseudomonas syringae pv. tomato DC3000 or to the oomycet

287 The agronomical relevant tomato-Pseudomonas syringae pv. tomato pathosystem is widely us

288 double mutant showed enhanced resistance to Pseudomonas syringae pv. tomato, which is consistent wit

289 that SA promotes the interaction between the Pseudomonas syringae type III effector AvrPtoB and NPR1.

290 natine (phytotoxin produced by the bacterium Pseudomonas syringae) or fusicoccin (a fungal toxin prod

291 ted in enhanced susceptibility to pathogenic Pseudomonas syringae, indicating functional redundancy i

292 radation, and susceptibility to the pathogen Pseudomonas syringae.

293 ontaining femtomolar INP concentrations from Pseudomonas syringae.

294 hallenged with the phytopathogenic bacterium Pseudomonas syringae.

295 ired for a complete defence response against Pseudomonas syringae.

296 1), as a factor enhancing resistance against Pseudomonas syringe pv tomato DC3000.

297 ct and quantify intracellular metabolites of Pseudomonas taiwanensis (P. taiwanensis) VLB120 to provi

298 Pseudomonas was replaced after 95 days by a microbiota d

299 nera (Vibrio, Flavobacterium, Tenacibaculum, Pseudomonas) with high louse burdens.

300 es an antimicrobial peptide-like response in Pseudomonas, with specific upregulation of membrane defe