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1 ic bacteria (e.g., Staphylococcus aureus and Pseudomonas aeruginosa).
2 positive Bacillus subtilis and Gram-negative Pseudomonas aeruginosa.
3 natural bispecific IgG1 candidate, targeting Pseudomonas aeruginosa.
4 ctamases, but is considered inactive against Pseudomonas aeruginosa.
5 duced and expressed in the outer membrane of Pseudomonas aeruginosa.
6 inhibits QS in opportunistic human pathogen Pseudomonas aeruginosa.
7 (ETA) is the most toxic virulence factor of Pseudomonas aeruginosa.
8 Paulo MBL (SPM-1) from beta-lactam-resistant Pseudomonas aeruginosa.
9 ic cells of the opportunistic human pathogen Pseudomonas aeruginosa.
10 ulence factors in the opportunistic pathogen Pseudomonas aeruginosa.
11 amma agonists attenuate biofilm formation by Pseudomonas aeruginosa.
12 ross some bacteria including Vibrio spp. and Pseudomonas aeruginosa.
13 omising therapeutic antibody targets against Pseudomonas aeruginosa.
14 ceftazidime-resistant Enterobacteriaceae or Pseudomonas aeruginosa.
15 jugation, and maintained in K pneumoniae and Pseudomonas aeruginosa.
16 MDR) gram-negative bacteria (GNB), including Pseudomonas aeruginosa.
17 fection assays with the pathogenic bacterium Pseudomonas aeruginosa.
18 nsors to trigger acute virulence programs in Pseudomonas aeruginosa.
19 olonisation by respiratory pathogens such as Pseudomonas aeruginosa.
20 ould be maintained when tested on pathogenic Pseudomonas aeruginosa.
21 ignature analysis for the bacterial pathogen Pseudomonas aeruginosa.
22 nescent strains of Staphylococcus aureus and Pseudomonas aeruginosa.
23 abolite by the opportunistic human pathogen, Pseudomonas aeruginosa.
24 e-associated pyochelin siderophore system in Pseudomonas aeruginosa.
25 m antibiotics in many Enterobacteriaceae and Pseudomonas aeruginosa.
26 e mass spectra of membrane grown biofilms of Pseudomonas aeruginosa.
27 le and cultures of Aspergillus fumigatus and Pseudomonas aeruginosa.
28 h a unique species-specific activity against Pseudomonas aeruginosa.
31 for the majority of Gram negative bacteria (Pseudomonas aeruginosa, 16-32 mug/mL, Klebsiella pneumon
32 broth microdilution (BMD) for 99 isolates of Pseudomonas aeruginosa, 26 Acinetobacter baumannii isola
33 (n=355) were Klebsiella pneumoniae (37%) and Pseudomonas aeruginosa (30%); 28% were ceftazidime-non-s
34 tive Staphylococcus aureus and gram-negative Pseudomonas aeruginosa (99.3 +/- 1.9% and 88.5 +/- 3.3%
36 at are notoriously problematic in hospitals: Pseudomonas aeruginosa, Acinetobacter baumannii, and Sta
37 lostridium difficile, Staphylococcus aureus, Pseudomonas aeruginosa, Acinetobacter baumannii, carbape
40 by the Gram-negative opportunistic pathogen, Pseudomonas aeruginosa Activation of phospholipase activ
42 healthy mouse corneas becomes vulnerable to Pseudomonas aeruginosa adhesion if it lacks the innate d
44 s this question in the pathogenic bacterium, Pseudomonas aeruginosa Although public goods producers w
45 gainst experimental endocarditis (EE) due to Pseudomonas aeruginosa, an archetype of difficult-to-tre
46 non-glucose-fermenting Gram-negative bacilli Pseudomonas aeruginosa and Acinetobacter baumannii are i
47 bial performance against two MDR isolates of Pseudomonas aeruginosa and Acinetobacter baumannii throu
48 ing Gram-negative bacilli (CPNFs), including Pseudomonas aeruginosa and Acinetobacter baumannii, is n
50 and model drug by LPS from F. tularensis vs Pseudomonas aeruginosa and by F. tularensis live bacteri
53 coccus pyogenes) and gram-negative bacteria (Pseudomonas aeruginosa and Escherichia coli) over time t
55 n of biofilms formed by the mucoid strain of Pseudomonas aeruginosa and investigated the commonality
56 is synthesized by the opportunistic pathogen Pseudomonas aeruginosa and is an important biofilm const
57 ed reservoirs, including electronic faucets (Pseudomonas aeruginosa and Legionella), decorative water
58 as Escherichia coli, Enterobacter aerogenes, Pseudomonas aeruginosa and Salmonella Typhimurium The ge
59 ocystitis; Coagulase negative Staphylococci, Pseudomonas aeruginosa and Staphylococcus aureus in kera
60 PPH assay and antibacterial activity against Pseudomonas aeruginosa and Staphylococcus aureus was ass
61 rial activity of raw rapeseed honeys against Pseudomonas aeruginosa and Staphylococcus aureus, with a
62 extran-coated nanoceria was examined against Pseudomonas aeruginosa and Staphylococcus epidermidis by
63 red by new data from 28 clinical isolates of Pseudomonas aeruginosa and strains evolved in laboratory
64 ough horizontal gene transfer by the species Pseudomonas aeruginosa and subsequently abundant P. aeru
65 opportunistic pathogens, namely the bacteria Pseudomonas aeruginosa and the fungus Aspergillus fumiga
66 obal sRNA interactions with their targets in Pseudomonas aeruginosa and verified the method with a kn
67 Escherichia coli, Staphylococcus aureus and Pseudomonas aeruginosa) and in vitro anti-proliferative
68 ith 294 isolates of Enterobacteriaceae spp., Pseudomonas aeruginosa, and Acinetobacter baumannii chos
69 (Staphylococcus aureus, Enterobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii).
71 apenem-resistant Acinetobacter baumannii and Pseudomonas aeruginosa, and carbapenem-resistant and thi
72 lococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebsiella pneumoniae, which
75 way infections by the opportunistic pathogen Pseudomonas aeruginosa are a major cause of mortality in
76 he fungus Candida albicans and the bacterium Pseudomonas aeruginosa are coisolated in the context of
77 Staphylococci, Streptococcus pneumoniae and Pseudomonas aeruginosa are the leading isolates in ocula
79 to Escherichia coli, Klebsiella species, or Pseudomonas aeruginosa at 130 VHA facilities from Januar
80 between the two domains and is able to kill Pseudomonas aeruginosa at sub-micromolar concentrations.
81 the study was to assess the applicability of Pseudomonas aeruginosa ATCC9027 and its validated biolum
82 olate bond in an engineered copper centre in Pseudomonas aeruginosa azurin by rational design of the
83 li, Salmonella enterica serovar Typhimurium, Pseudomonas aeruginosa, Bacillus subtilis, and Staphyloc
85 olymicrobial, with Staphylococcus aureus and Pseudomonas aeruginosa being the two most commonly isola
86 plant metabolites called flavonoids inhibit Pseudomonas aeruginosa biofilm formation by an unknown m
87 rophage migration inhibitory factor enhances Pseudomonas aeruginosa biofilm formation, potentially co
89 ion yellow 11), which targets amyloid in the Pseudomonas aeruginosa biofilm matrix through a diversit
90 ial transcriptome map of the mature in vitro Pseudomonas aeruginosa biofilm model, revealing contempo
93 d healing, wild-type mice were infected with Pseudomonas aeruginosa biofilms and, akin to Nod2(-/-) m
94 alize protein and metal distributions within Pseudomonas aeruginosa biofilms using imaging mass spect
97 ated the best antibacterial activity against Pseudomonas aeruginosa both in vitro and in vivo for tet
101 that enables cross-species interactions, as Pseudomonas aeruginosa cells also become attracted to th
102 ng approximately 90% of Escherichia coli and Pseudomonas aeruginosa cells within 90-120 and 5-30 min,
103 face glycosylation is modulated by IL-1R and Pseudomonas aeruginosa challenge but is insufficient for
104 vations link respiratory virus infection and Pseudomonas aeruginosa colonization in chronic lung dise
105 ophilia and mucin production associated with Pseudomonas aeruginosa colonization, which is associated
106 we report the crystal structure of LspA from Pseudomonas aeruginosa complexed with the antimicrobial
107 ng of a genetically detoxified exotoxin A of Pseudomonas aeruginosa covalently linked to Shigella fle
108 for the detection of carbapenemase-producing Pseudomonas aeruginosa (CP-PA) and carbapenemase-produci
109 ne conductance regulator (CFTR) that reduces Pseudomonas aeruginosa culture positivity in CF patients
111 Ivacaftor produced rapid decreases in sputum Pseudomonas aeruginosa density that began within 48 hour
112 actively synthesized proteins in nongrowing Pseudomonas aeruginosa, discovering a regulator whose in
113 reted components by the pathogenic bacterium Pseudomonas aeruginosa during growth on a protein substr
114 e rate by human neutrophil elastase (NE) and Pseudomonas aeruginosa elastase (PAE) by different mecha
116 ysts of intracellular phenazine reduction in Pseudomonas aeruginosa Enzymatic assays in cell-free lys
117 thal toxin, diphtheria toxin, cholera toxin, Pseudomonas aeruginosa exotoxin A, Botulinum neurotoxin,
119 large, non-Nocardia, or classically invasive Pseudomonas aeruginosa; for patients with low baseline v
121 ic coinfections of Staphylococcus aureus and Pseudomonas aeruginosa frequently fail to respond to ant
122 f Enterobacteriaceae, Acinetobacter spp, and Pseudomonas aeruginosa from 18 698 inpatients and 2923 h
123 ce variant of PTEN, were unable to eradicate Pseudomonas aeruginosa from the airways and could not ge
125 n B addition engendered Escherichia coli and Pseudomonas aeruginosa Gram-negative activity MIC's of 4
126 r interface and the active sites can abolish Pseudomonas aeruginosa growth in a defined medium with m
127 t of EGCG against the opportunistic pathogen Pseudomonas aeruginosa has been shown to involve disrupt
129 nization with other pathogens, in particular Pseudomonas aeruginosa Here, we demonstrate that CF mice
131 )F-fluoromaltotriose was also able to detect Pseudomonas aeruginosa in a clinically relevant mouse mo
133 de Staphylococci, Streptococcus pyogenes and Pseudomonas aeruginosa in blepharitis; Staphylococci, St
136 had lower sensitivities for the detection of Pseudomonas aeruginosa in comparison to the analogous 7-
137 red to those to the well-studied CF pathogen Pseudomonas aeruginosa In parallel, mice were also infec
140 utrophil synthesis of LTB4 in the context of Pseudomonas aeruginosa-induced neutrophil transepithelia
141 induced in mouse macrophages in response to Pseudomonas aeruginosa infection both in vivo and by iso
144 tactic gradients, and migrate in response to Pseudomonas aeruginosa infection of primary ALI barriers
145 leus, histamine blocker use, and respiratory Pseudomonas aeruginosa infection were associated with lo
146 p=0.037) when adjusted for sex, BMI, chronic Pseudomonas aeruginosa infection, FEV1/FVC (forced vital
152 rventions to treat multidrug-resistant (MDR) Pseudomonas aeruginosa infections are severely limited a
170 nsporter FpvAI in the opportunistic pathogen Pseudomonas aeruginosa is hijacked to translocate the ba
172 demonstrate that the Gram-negative pathogen Pseudomonas aeruginosa is susceptible to reactive oxygen
173 -GMP signaling in the opportunistic pathogen Pseudomonas aeruginosa is the transcription regulator Fl
174 resolution, we show that granule genesis in Pseudomonas aeruginosa is tightly organized under nitrog
175 ation with bacterial pathogens, particularly Pseudomonas aeruginosa, is the primary cause of morbidit
176 the time to detection (TTD) and growth of 2 Pseudomonas aeruginosa isolates in the presence of clini
182 tis; Staphylococci, Streptococus pneumoniae, Pseudomonas aeruginosa, Klebsiella pneumoniae and Escher
184 ghput to sequence approximately 400 clinical Pseudomonas aeruginosa libraries and demonstrate excelle
185 aling activity of Hfq from Escherichia coli, Pseudomonas aeruginosa, Listeria monocytogenes, Bacillus
187 built here an intact atomistic model of the Pseudomonas aeruginosa MexAB-OprM pump in a Gram-negativ
191 ) from E. coli B, E. coli 056, E. coli 0111, Pseudomonas aeruginosa NBRC 13743 and Hafnia alvei 1185.
192 nfections such as invasive aspergillosis and Pseudomonas aeruginosa occurred during hospitalization.
195 fluorescent pseudomonads, such as pathogenic Pseudomonas aeruginosa or plant growth-promoting Pseudom
196 receptor, was induced by infection with live Pseudomonas aeruginosa or treatment of cells with its su
197 ne reproductive pathogens (Escherichia coli, Pseudomonas aeruginosa, or Klebsiella pneumoniae) isolat
198 trains including Escherichia coli (E. coli), Pseudomonas aeruginosa (P. aeruginosa), Staphylococcus a
199 n-resistant Sthaphylococcus aureus, MRSA and Pseudomonas aeruginosa, P. aeruginosa) were analyzed in
202 ycobacterium marinum (Mm) (a model for Mtb), Pseudomonas aeruginosa (Pa), Legionella pneumophila (Lp)
207 e, we identified a tRNA methyltransferase in Pseudomonas aeruginosa PA14, trmJ, which confers resista
209 he type I-F CRISPR adaptive immune system in Pseudomonas aeruginosa (PA14) consists of two CRISPR loc
213 receptors and a single chemosensory pathway, Pseudomonas aeruginosa PAO1 has a much more complex chem
215 tailed bacterial viruses, or phages, such as Pseudomonas aeruginosa phage varphiKZ, have long genomes
216 Phis are hyperinflammatory and have impaired Pseudomonas aeruginosa phagocytosis, phenocopying CF MPh
218 mp MexGHI-OpmD in the opportunistic pathogen Pseudomonas aeruginosa Previous studies of P. aeruginosa
223 e, the authors show that two DMAbs targeting Pseudomonas aeruginosa proteins confer protection agains
224 ontaining phosphodiesterase domains from the Pseudomonas aeruginosa proteins PA3825 (PA3825(EAL)) and
226 arothermophilus) and gram-negative bacteria (Pseudomonas aeruginosa, Pseudomonas fluorescens, Salmone
227 al factors governing this critical period in Pseudomonas aeruginosa pulmonary pathogenesis when trans
228 y identified NB predicted to be specific for Pseudomonas aeruginosa (pyocin Sn) was produced and show
230 -heptyl-4-hydroxyquinoline-N-oxide (HQNO), a Pseudomonas aeruginosa quorum-sensing-regulated low-mole
232 Escherichia coli, Klebsiella pneumoniae, and Pseudomonas aeruginosa-reported here underscores the bro
233 hicillin-resistant Staphylococcus aureus and Pseudomonas aeruginosa, respectively, in catheter-associ
235 interactions with the respiratory pathogens Pseudomonas aeruginosa, respiratory syncytial virus and
236 co-infection of murine surgical wounds with Pseudomonas aeruginosa results in conversion of approxim
238 inding sites of the RNase E component of the Pseudomonas aeruginosa RNA degradosome, occluding them f
239 EF-Tu from the clinically relevant pathogen Pseudomonas aeruginosa shares over 84% sequence identity
243 ith Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphylococcus aureus (including
244 netobacter baumannii, Klebsiella pneumoniae, Pseudomonas aeruginosa, Staphylococcus aureus, and coagu
245 ebsiella pneumoniae, Legionella pneumophila, Pseudomonas aeruginosa, Stenotrophomonas maltophilia, Vi
246 me-scale metabolic network reconstruction of Pseudomonas aeruginosa strain PA14 and an updated, expan
249 We demonstrate that the pathogenicity of Pseudomonas aeruginosa strains derived from acute clinic
250 ix mares were inoculated with lux-engineered Pseudomonas aeruginosa strains isolated from equine uter
251 We used a collection of well characterized Pseudomonas aeruginosa strains, featuring distinct antib
252 N prevented death due to pneumonia caused by Pseudomonas aeruginosa, Streptococcus pneumoniae, and As
253 to a burn infected with multidrug-resistant Pseudomonas aeruginosa substantially decreased bacterial
254 ative organisms with higher activity towards Pseudomonas aeruginosa than the naturally-occurring AMP
257 olution X-ray crystal structure of FliD from Pseudomonas aeruginosa, the first high-resolution struct
259 bound to the MLPs from Escherichia coli and Pseudomonas aeruginosa These new structures, along with
260 ubiquitous and opportunistic human pathogen Pseudomonas aeruginosa This bacterium is frequently adop
261 a chemotaxis-regulating methyltransferase in Pseudomonas aeruginosa This cocrystal structure, togethe
264 s by RNA-Seq to characterize the response of Pseudomonas aeruginosa to external 0.5 mm CuSO4, a condi
266 tudying the sensitivities of a ohr mutant of Pseudomonas aeruginosa toward different hydroperoxides.
267 ulence factors for many pathogens, including Pseudomonas aeruginosa Transcription of the major pilin
270 lus also are dispensable for activation of a Pseudomonas aeruginosa type VI secretion system (T6SS).
273 ion of the peptide siderophore pyoverdine by Pseudomonas aeruginosa, under different nutrient-limitin
274 activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infection, suggesti
276 la via trophic transfer from bacterial prey (Pseudomonas aeruginosa) versus direct uptake from growth
278 The discovery of therapies that modulate Pseudomonas aeruginosa virulence or that can eradicate c
279 augments host defense in sepsis and reduces Pseudomonas aeruginosa virulence through quorum sensing
282 human airway epithelial cells infected with Pseudomonas aeruginosa was applied to lung fibroblasts a
283 Aspergillus flavus and meropenem due to the Pseudomonas aeruginosa was initiated, the former necessi
287 ion by other uropathogens (Escherichia coli, Pseudomonas aeruginosa) was also explored, and thioridaz
288 compared with that of its ortholog LecA from Pseudomonas aeruginosa We also investigated the utility
289 ococcus faecalis, Klebsiella pneumoniae, and Pseudomonas aeruginosa We therefore conclude that the un
291 Staphylococcus aureus, Escherichia coli, and Pseudomonas aeruginosa were tested in triplicate using t
292 Escherichia coli, Klebsiella pneumoniae, and Pseudomonas aeruginosa were the most common isolates.
293 old the minimal inhibitory concentration for Pseudomonas aeruginosa) were compared between the two gr
294 me organisms, such as Klebsiella species and Pseudomonas aeruginosa, were lower in oncology than in n
295 carbapenem-resistant Enterobacteriaceae and Pseudomonas aeruginosa, which are difficult to treat.
296 vae are highly susceptible to infection with Pseudomonas aeruginosa, which can be almost fully rescue
297 patients infected with carbapenem-resistant Pseudomonas aeruginosa who were treated with ceftolozane
298 tified here melanogenic clinical isolates of Pseudomonas aeruginosa with large chromosomal deletions
299 erimentally evolved replicate populations of Pseudomonas aeruginosa with or without a community of th
300 ride, showed efficient biofilm inhibition of Pseudomonas aeruginosa without impairing its growth.
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