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1 eisseria meningitidis, Escherichia coli, and Pseudomonas aeruginosa infection).
2  (KO) mice with aerosolized LPS (100 mug) or Pseudomonas aeruginosa infection.
3 se is critical in the prognosis of pulmonary Pseudomonas aeruginosa infection.
4 eased in splenic natural killer cells during Pseudomonas aeruginosa infection.
5 f CF BAL fluid samples from patients without Pseudomonas aeruginosa infection.
6 ssociated with refractory CRS and persistent Pseudomonas aeruginosa infection.
7 ppaB signalling upon Gram-negative bacterium Pseudomonas aeruginosa infection.
8 cterial clearance and survival of mice after Pseudomonas aeruginosa infection.
9 's tautomerase activity in a murine model of Pseudomonas aeruginosa infection.
10  the eye from pathogenic Candida albicans or Pseudomonas aeruginosa infection.
11 ctors associated with both chronic and acute Pseudomonas aeruginosa infection.
12 t poly(I:C), improved the host response to a Pseudomonas aeruginosa infection.
13 xia succumbed to death during recovery after Pseudomonas aeruginosa infection.
14 rneal perforation in C57BL/6 (B6) mice after Pseudomonas aeruginosa infection.
15  interactions in response to acute pulmonary Pseudomonas aeruginosa infection.
16 tients with cystic fibrosis who have chronic Pseudomonas aeruginosa infection.
17 tibility of cystic fibrosis (CF) patients to Pseudomonas aeruginosa infection.
18 ramycin in patients with cystic fibrosis and Pseudomonas aeruginosa infection.
19 rse outcomes, particularly in the setting of Pseudomonas aeruginosa infection.
20 adult Swiss (HSD:ICR) mice is restored after Pseudomonas aeruginosa infection.
21 ronounced hypersusceptibility (80 to 90%) to Pseudomonas aeruginosa infection.
22 sponse of cystic fibrosis and sensitivity to Pseudomonas aeruginosa infections.
23 e challenged two strains of mice with lethal Pseudomonas aeruginosa infection 3 weeks after immunizat
24 ted (68 vs. 77%, p < 0.001), higher rates of Pseudomonas aeruginosa infection (71 vs. 65%, p < 0.01),
25               We investigated an outbreak of Pseudomonas aeruginosa infections after bronchoscopic pr
26 choice in the setting of multidrug-resistant Pseudomonas aeruginosa infections, although high doses o
27 fection among HIV-negative patients and with Pseudomonas aeruginosa infections among HIV-positive pat
28 nitial point of host-pathogen interaction in Pseudomonas aeruginosa infection, an important pathogen
29 e the in vivo functions of SPLUNC1 following Pseudomonas aeruginosa infection and to elucidate the un
30  of CF lung disease while adjusting for age, Pseudomonas aeruginosa infection, and cystic fibrosis tr
31 urine listeriosis model, the neutropenic rat Pseudomonas aeruginosa infection, and the mouse cecal li
32  resistance during multidrug resistant (MDR)-Pseudomonas aeruginosa infections are limited.
33 rventions to treat multidrug-resistant (MDR) Pseudomonas aeruginosa infections are severely limited a
34 esponse (within 24 hours) to an experimental Pseudomonas aeruginosa infection between vitamin A defic
35  induced in mouse macrophages in response to Pseudomonas aeruginosa infection both in vivo and by iso
36 r cystic fibrosis (CF) patients with chronic Pseudomonas aeruginosa infection, but there has not been
37                                              Pseudomonas aeruginosa infections can be virtually impos
38                                              Pseudomonas aeruginosa infection category in the year be
39                                    Recurrent Pseudomonas aeruginosa infections coupled with robust, d
40                 Furthermore, analysis of all Pseudomonas aeruginosa infections demonstrated no signif
41 y observed in clinical isolates from chronic Pseudomonas aeruginosa infections, enables bacteria to e
42 p=0.037) when adjusted for sex, BMI, chronic Pseudomonas aeruginosa infection, FEV1/FVC (forced vital
43                                              Pseudomonas aeruginosa infections generally persist desp
44                                              Pseudomonas aeruginosa infection has been demonstrated t
45                            Susceptibility to Pseudomonas aeruginosa infection has been linked to the
46 ns cells were induced into the cornea before Pseudomonas aeruginosa infection in BALB/c mice that are
47                           This suggests that Pseudomonas aeruginosa infection in CF reflects biofilm
48                      One of the hallmarks of Pseudomonas aeruginosa infection in cystic fibrosis (CF)
49 udy was to determine whether PLUNC can limit Pseudomonas aeruginosa infection in mice.
50 tor mutation to the onset and persistence of Pseudomonas aeruginosa infection in the airways, and her
51 w immunogenic antigens that diagnose initial Pseudomonas aeruginosa infections in patients with cysti
52 promoter exhibited an elevated resistance to Pseudomonas aeruginosa infection, indicating that these
53                             LPS treatment or Pseudomonas aeruginosa infection induces miR-301b expres
54                                              Pseudomonas aeruginosa infection is a clinically relevan
55  mucociliary clearance, and establishment of Pseudomonas aeruginosa infection is described.
56                             Although chronic Pseudomonas aeruginosa infection is the major cause of m
57  A novel immunochemical approach to diagnose Pseudomonas aeruginosa infections is reported, which is
58 ic endobronchiolitis compounded by recurring Pseudomonas aeruginosa infections is the major cause of
59                                              Pseudomonas aeruginosa infection liberates transmissible
60                                      Chronic Pseudomonas aeruginosa infection occurs in 75-90% of pat
61 cornea keratitis model was modified to study Pseudomonas aeruginosa infection of healing corneal epit
62 tactic gradients, and migrate in response to Pseudomonas aeruginosa infection of primary ALI barriers
63                         Here we investigated Pseudomonas aeruginosa infections of the cornea in Lum(-
64 was not altered by genotype, age, gender, or Pseudomonas aeruginosa infection, oral steroid treatment
65  the incidence or severity of Klebsiella and Pseudomonas aeruginosa infections, patients admitted to
66  Splenic natural killer cells activated upon Pseudomonas aeruginosa infection produced interferon-gam
67                      Prevention of pulmonary Pseudomonas aeruginosa infections represents a critical
68       With the rise of multidrug resistance, Pseudomonas aeruginosa infections require alternative th
69                                       During Pseudomonas aeruginosa infection, SCF (FBXL2) targets th
70 n cystic fibrosis (CF) patients with chronic Pseudomonas aeruginosa infection, the underlying mechani
71 nts who were ExBF >/=2 mo, and the number of Pseudomonas aeruginosa infections through the age of 2 y
72 leus, histamine blocker use, and respiratory Pseudomonas aeruginosa infection were associated with lo
73  bacterial outgrowth from cells subjected to Pseudomonas aeruginosa infection were used to determine
74 e synthesis and cell viability in a model of Pseudomonas aeruginosa infection where Ca(2+) concentrat

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