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1 of SA, involved in response to both UV-C and Pseudomonas infection.
2 bute to the establishment and maintenance of Pseudomonas infection.
3 ion is required for the host defense against Pseudomonas infection.
4 l chemotherapeutic agents designed to combat Pseudomonas infection.
5 Weight loss was greatest 3 d after Pseudomonas infection.
6 tion designed to arrest tissue damage during Pseudomonas infection.
7 INA3 mRNA abundance decreases in response to Pseudomonas infection.
8 be partly responsible for the persistence of Pseudomonas infections.
9 e MMPs in regulating epithelial responses to Pseudomonas infection and show that a global genomics st
11 lymphocytic bronchiolitis, colonization with pseudomonas, infection, and BAL eosinophilia and neutrop
14 tauopathies such as Alzheimer disease, acute Pseudomonas infections cause a pathophysiological sequel
15 se-dependent respiratory burst stimulated by Pseudomonas infection contributes to host defense by mod
16 data on children participating in the Early Pseudomonas Infection Control trial who received standar
17 ly in macrophages also confers resistance to Pseudomonas infection, highlighting an important role fo
22 tration of long-lived cytotoxic agents after Pseudomonas infection may establish a molecular link to
25 ding that NAC1 mRNA abundance increases upon Pseudomonas infection, the SINA3 mRNA abundance decrease
28 severity of vaccine-specific Klebsiella plus Pseudomonas infections were not significantly different
29 e SlNAC1 gene is strongly upregulated during Pseudomonas infection, while repression of the NAC1 orth
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