ライフサイエンスコーパス: Q wave

1 /y (P<0.001) decline in those with any major Q wave.

2 negative predictive value compared with the Q wave.

3 is of myocardial infarction required new ECG Q waves.

4 tests, but the other developed anterolateral Q waves.

5 t ST-segment elevation as opposed to LBBB or Q waves.

6 ad transient ECG changes, and none developed Q-waves.

7 left bundle-branch block (4% versus 0%) and Q waves (5.3% versus 5.5%), serial cardiac indices, intr

8 Cirrhotics had a prolonged QTc interval, a Q wave, abnormal QRS axis deviation, ST segment depressi

9 persistent and transient ST segment and T or Q wave abnormalities discriminated those with from those

10 ST segment and T and Q wave abnormalities suggestive of myocardial ischemia o

11 ssociation class III angina in 36 (54%), non-Q wave acute myocardial infarction in 1, and acute pulmo

12 rtant variables were electrocardiogram (ECG) Q waves (adjusted chi-square=38.3, p <0.001) and periphe

13 .3%, 99.2%, and 70.8%, respectively, for the Q wave alone; 85.6%, 89%, and 92.7%, respectively, for t

14 e ability to detect a prior MI compared with Q waves alone by ECG.

15 rocardiograms (ExECGs); an uncomplicated non-Q wave AMI was diagnosed in two patients.

16 ears of age and those with Q wave versus non-Q wave AMI.

17 fects on LV remodeling in patients surviving Q-wave AMI.

18 e ratio in leads III to II, and ratio of the Q-wave amplitude in leads aVL to aVR, and a significantl

19 The Q-wave amplitude ratio in aVL/aVR was higher in the succ

20 91.4%, 89%, and 94.2%, respectively, for the Q wave and/or fQRS.

21 were classified by the presence of baseline Q waves and additionally into primary percutaneous coron

22 periprocedural infarctions (signified by new Q waves and CPK-MB >8xULN) are powerful determinants of

23 n did not modify the association of baseline Q waves and in-hospital outcomes (P interaction=0.918).

24 elevation declined as did the occurrence of Q waves and peak creatine kinase values.

25 ECG Q waves and/or evidence of PAD identified the most suita

26 arction was prospectively defined as: 1) new Q-wave and MB isoform of creatine kinase (CK-MB) elevati

27 rily to a lower incidence of peri-procedural Q-wave and non-Q-wave MI.

28 e, location, and transmural extent of healed Q-wave and non-Q-wave myocardial infarction can be accur

29 to predict death and the composite of death, Q-wave and non-Q-wave myocardial infarction, and emergen

30 or adverse events (defined as cardiac death, Q-wave and non-Q-wave myocardial infarction, or target v

31 nts with infarcts imaged at 3 months (13 non-Q-wave) and all of 19 imaged at 14 months (eight non-Q-w

32 adverse cardiac events (composite of death, Q-wave, and non-Q-wave myocardial infarction [MI], emerg

33 y (P<0.001) decline in those with subsequent Q-waves, and a 4.5%/y (P<0.001) decline in those with an

34 n occurred in the majority of patients after Q-wave anterior myocardial infarction.

35 Baseline Q waves, but not time to reperfusion, were associated wi

36 s (63) commented that "The Q-wave versus non-Q-wave categorization does not provide sufficient sensit

37 ients, with electrocardiographic evidence of Q waves corresponding to the CTO artery territory in onl

38 aracteristic of DMD patients, including deep Q-waves, diminished S:R ratios, polyphasic R-waves and f

39 negative deflections (eg, Q-wave EGMs), (3) Q-wave EGMs with superimposed RS deflections reflecting

40 large predominant negative deflections (eg, Q-wave EGMs), (3) Q-wave EGMs with superimposed RS defle

41 The influence of Q wave evolution on the use of invasive cardiac procedur

42 Although Q wave formation is rare, other ECG changes are common.

43 ely diagnose them; even the ECG criteria for Q wave formation signifying an important clinical event

44 ients in the successful group had an initial q wave in lead V1, as opposed to 6 (33%) in the unsucces

45 VR, a R/S ratio of > 2 in V1, and absence of q waves in lead V1 help identify appropriate candidates

46 They more often had new Q waves in two or more leads (51.9% vs. 31.5%, p < 0.04)

47 betes, previous acute myocardial infarction, Q wave infarction, and left-ventricular failure.

48 -6.43), adjusted for age, sex, diabetes, and Q wave infarction.

49 t confidence interval, 1.37 to 2.26]) and of Q-wave infarction (2.08 [95 percent confidence interval,

50 y revascularization were at greater risk for Q-wave infarction and death than nonsmokers.

51 o reduced by 40 percent in patients with non-Q-wave infarction and those with chronic obstructive pul

52 The only subacute stent closure and non-Q-wave infarction occurred in a patient assigned to the

53 lso associated with a lower rate of nonfatal Q-wave infarction or cardiac arrest (RR, 0.67 [95% CI, 0

54 Models evaluating non-Q-wave infarction or requiring measurement of percent di

55 catheterization in the Veterans Affairs Non-Q-Wave Infarction Strategies in Hospital (VANQWISH) tria

56 The Veterans Affairs Non-Q-Wave Infarction Strategies In-Hospital (VANQWISH) stud

57 nvasive strategy in the Veterans Affairs Non-Q-Wave Infarction Strategies in-Hospital (VANQWISH) tria

58 The Veterans Affairs Non-Q-Wave Infarction Strategies In-Hospital (VANQWISH) tria

59 ls were for death, 0.85 and 7.9%; for death, Q-wave infarction, and bypass surgery, 0.77 and 13.2%; f

60 In patients with unstable angina and non-Q-wave infarction, angioscopic features of disruption, y

61 of diseased vessels, ejection fraction (EF), Q-wave infarction, in-hospital death, and initial therap

62 oad (for death, r = .37, P = .01; for death, Q-wave infarction, or bypass surgery, r = .58, P < .001)

63 of death and the composite outcome of death, Q-wave infarction, or emergency bypass surgery were deve

64 ents), within 72 hours of the onset of a non-Q-wave infarction.

65 atients with unstable angina and 10 with non-Q-wave infarction.

66 toward increased mortality occurred with non-Q wave infarctions and race other than Caucasian.

67 The prevalence of non-Q wave infarctions increased from 45% in 1994 to 63% in

68 Patients evolving non-Q wave infarctions were older and had increased comorbid

69 contributing to the higher prevalence of non-Q wave infarctions, shorter hospital stays and lower hos

70 (kappa 0.48), T inversion (kappa 0.52), and Q waves (kappa 0.44), good for bundle branch block (kapp

71 as a higher prevalence of periprocedural non-Q wave MI (28% vs. 16%, p = 0.009) in the multiple SVG g

72 vs. 0.06%, p = 0.009) and periprocedural non-Q wave MI (8.7% vs. 4.2%, p = 0.003) were more frequent

73 men and 976 men with unstable angina and non-Q wave MI at the time of enrollment were compared.

74 The proportion of unstable angina and non-Q wave MI for women was similar in the trial and Registr

75 mpared with men with unstable angina and non-Q wave MI have not been extensively studied.

76 s, bypass surgery in 13, arrhythmia in 3 and Q wave MI in 2.

77 2) The outcome with unstable angina and non-Q wave MI is related to severity of illness and not gend

78 rence in death (5.6% vs. 5.3%, p = 0.92) and Q wave MI rate (4.3% vs. 2.9%, p = 0.55) after the multi

79 roportionately more men in the trial had non-Q wave MI than men in the Registry.

80 evascularization for unstable angina and non-Q wave MI was similar for women and men.

81 able, the frequency of in-hospital death and Q wave MI was similar to that of a matched consecutive s

82 event-free survival (freedom from dealth or Q wave MI) and relief of angina; however, the need for r

83 At 1 year, the frequency of death, Q wave MI, CABG and severe angina at 1 year was similar

84 97.3%, with 2.7% major complications (death, Q wave MI, coronary artery bypass graft surgery [CABG]).

85 uating patients with unstable angina and non-Q wave MI, little prospective information is available o

86 useful in risk stratifying patients with non-Q wave MI.

87 to the hospital with unstable angina or non-Q wave MI.

88 yzed 173 asymptomatic patients with previous Q-wave MI (>16 days) with echocardiographic quantitation

89 = 0.003), all MI (2.5% vs. 3.9%, p = 0.02), Q-wave MI (0.1% vs. 0.8%, p = 0.002), stent thrombosis (

90 ence in death (1.4% vs. 0.7%, p = 0.26), and Q-wave MI (1.2% vs. 0%, p = 0.02) was lower following mu

91 erence in death (2.2% versus 0.9%, P=.34) or Q-wave MI (1.4% versus 0.9%, P=.64) between the two grou

92 year mortality (2.5% vs. 3.5%, p = 0.49) or Q-wave MI (2.7% vs. 1.2%, p = 0.48), and the overall car

93 in 565 patients (54%) including death (9%), Q-wave MI (9%) and target vessel revascularization (36%)

94 Non-Q-wave MI (CK-MB > 3 times normal) occurred in 14% of pa

95 Non-Q-wave MI (CK-MB > or = 5 times normal) was more frequen

96 infarction [MI]) in patients who evolved non-Q-wave MI (NQMI) following thrombolytic therapy.

97 h a significant reduction in the risk of non-Q-wave MI (unadjusted odds ratio 0.18, 95% confidence in

98 e end point of death, postprocedural MI, non-Q-wave MI after PCI hospitalization, or urgent target-le

99 blation and stenting than PTCA, the rates of Q-wave MI and survival were device-independent.

100 alyzed 303 patients with previous (>16 days) Q-wave MI by ECG who underwent transthoracic echocardiog

101 it of normal (ULN) in 17.9% of patients, and Q-wave MI developed in 0.6%.

102 age of 69 years) with an initial recognized Q-wave MI from 1950 through 1989, we investigated time t

103 unclear if rates of long-term sequelae after Q-wave MI have improved.

104 cardial infarction [MI] through 30 days; non-Q-wave MI through 24 h; and ipsilateral stroke or neurol

105 The incidence of Q-wave MI was also higher with acolysis (5.4% versus 2.2

106 Q-wave MI was noted in 52% to 53%, and thrombolytic ther

107 8676 admissions with unstable angina or non-Q-wave MI were enumerated and, of these, 3318 patients w

108 butable mainly to a greater frequency of non-Q-wave MI with acolysis (19.6% versus 7.9%, P=0.03).

109 Non-Q-wave MI with CPK-MB >8x ULN was also a strong predicto

110 times normal), or type 3 (>8 times normal or Q-wave MI).

111 At six months, 6% of patients died, 1% had Q-wave MI, 17% had repeat TVR, and the overall rate of m

112 ristics, (2) a higher rate of procedural non-Q-wave MI, and (3) similar TLR and overall major cardiac

113 ts consisted of 3 deaths, 2 acute MIs, 1 non-Q-wave MI, and 3 cases of CHF.

114 ted electrocardiographic evidence of a prior Q-wave MI, but who lacked a history of this diagnosis.

115 The frequencies of in-hospital Q-wave MI, coronary artery bypass graft surgery and deat

116 y identified with coronary angiography after Q-wave MI, the culprit lesion after NQWMI has not been w

117 In-hospital composite cardiac events (death, Q-wave MI, urgent in-hospital revascularization) and 8 m

118 incidence of peri-procedural Q-wave and non-Q-wave MI.

119 reduced (nonspecified) composite of death or Q-wave MI.

120 There were 28 patients (16%) who had a non-Q-wave MI.

121 tine evaluation were defined as having a non-Q-wave MI.

122 included 246 patients with a first anterior Q-wave MI.

123 , and there is no specific ECG sign of a non-Q-wave MI.

124 The rate of the composite end point (death, Q-wave-MI and target lesion revascularization) at 1-year

125 h a lower incidence of procedure-related non-Q-wave MIs (duration of pretreatment <1 day, 29% had MI;

126 rdial infarction (MI); positive group, 4 non-Q-wave MIs and 12 myocardial revascularizations; nondiag

127 antly higher frequency of periprocedural non-Q-wave MIs, and 3) equivalent repeat revascularization r

128 ciated with a reduced risk of procedural non-Q-wave MIs.

129 farction (10.7% vs. 6.3%, p = 0.021) and non-Q wave myocardial infarction (9.6% vs. 4.9%, p = 0.006).

130 group: death (0.3% vs. 7.3%, p < 0.0001) and Q wave myocardial infarction (MI) (0.9% vs. 6.1%, p < 0.

131 of normal or with ECG changes diagnostic for Q wave myocardial infarction (MI) should be treated as p

132 (TIMI) IIIB trial of unstable angina and non-Q wave myocardial infarction (MI) were evaluated to dete

133 CG) in patients with unstable angina and non-Q wave myocardial infarction (MI).

134 Patients with unstable angina (UA) and non-Q wave myocardial infarction (NQMI) may sustain a small

135 d to compare outcomes of patients with a non-Q wave myocardial infarction (NQMI) who were randomized

136 lity in patients with unstable angina or non-Q wave myocardial infarction (NQMI).

137 lity in patients with unstable angina or non-Q wave myocardial infarction (NQMI).

138 pital and mid-term clinical outcomes (death, Q wave myocardial infarction [MI] and repeat revasculari

139 Directional atherectomy-related non-Q wave myocardial infarction appears to be platelet aggr

140 ere was no difference in cumulative death or Q wave myocardial infarction between the groups.

141 rocedure included death in 5.2% of patients, Q wave myocardial infarction in 1.3% and repeat bypass s

142 n 9.7% of patients, including death in 1.7%, Q wave myocardial infarction in 3.1% and emergency bypas

143 In-hospital complications included non-Q wave myocardial infarction in two patients and death a

144 In 597 patients with unstable angina or non-Q wave myocardial infarction participating in the Thromb

145 Non-Q wave myocardial infarction rates after PTCA were not a

146 IC trial confirmed the increased risk of non-Q wave myocardial infarction with directional atherectom

147 spital coronary artery bypass graft surgery, Q wave myocardial infarction) were chosen for analysis.

148 even additional patients (11%) developed non-Q wave myocardial infarction, and nine patients (9%) had

149 There were no deaths, no strokes, one Q wave myocardial infarction, and one sternal infection.

150 , including urgent bypass surgery, Q and non-Q wave myocardial infarction, dissection, acute occlusio

151 Patients with prior Q wave myocardial infarction, elevated troponin levels b

152 blockade on adverse outcomes, especially non-Q wave myocardial infarction, in patients undergoing dir

153 IMI-IIIB cohort) with unstable angina or non-Q wave myocardial infarction, who underwent predischarge

154 ents: one patient died, and two patients had Q wave myocardial infarction, with no emergency coronary

155 ency coronary artery bypass graft surgery or Q wave myocardial infarction.

156 ed acute ischemic complications, notably non-Q wave myocardial infarction.

157 Bolus and infusion of c7E3 reduced non-Q wave myocardial infarctions by 71% after atherectomy (

158 . 4.99%, p < 0.0001) and a trend toward more Q wave myocardial infarctions than after PTCA.

159 ter PTCA were not affected by c7E3, although Q wave myocardial infarctions were reduced from 2.6% to

160 In-hospital clinical complications including Q-wave myocardial infarction (2.9% versus 0.2%; P<0.001)

161 confidence interval, 1.9-3.3) and subsequent Q-wave myocardial infarction (hazard ratio, 2.7; 95% con

162 in 137 patients (13%) including death (8%), Q-wave myocardial infarction (MI) (2%) and coronary arte

163 The rate of periprocedural non-Q-wave myocardial infarction (MI) (creatine kinase-MB >

164 in-hospital outcome: 0.6% mortality rate, no Q-wave myocardial infarction (MI) and 0.6% rate of urgen

165 Short-term (<30 day) mortality after Q-wave myocardial infarction (MI) has declined over the

166 three groups included: negative group, 1 non-Q-wave myocardial infarction (MI); positive group, 4 non

167 g which patients with unstable angina or non-Q-wave myocardial infarction (NQMI) are likeliest to ben

168 We investigated whether patients with non-Q-wave myocardial infarction (NQMI) have more ischemic v

169 ity patterns in patients with an initial non-Q-wave myocardial infarction (NQWMI) as compared with th

170 with either unstable angina pectoris or non-Q-wave myocardial infarction (NQWMI) enrolled in TIMI II

171 nd characterize the culprit lesion after non-Q-wave myocardial infarction (NQWMI).

172 in patients with unstable angina (UA) or non-Q-wave myocardial infarction (NQWMI).

173 c viable myocardium (IVM) than patients with Q-wave myocardial infarction (QMI).

174 QWMI) as compared with those with an initial Q-wave myocardial infarction (QWMI).

175 The in-hospital composite end point of death/Q-wave myocardial infarction (QWMI)/repeat revasculariza

176 (UFH) for management of unstable angina/non-Q-wave myocardial infarction (UA/NQMI).

177 One patient had a non-Q-wave myocardial infarction 24 h after the procedure.

178 The primary end point (all stroke, death, or Q-wave myocardial infarction [MI] through 30 days; non-Q

179 on rate (death, emergency bypass surgery, or Q-wave myocardial infarction [MI]) of 2.5%.

180 ital and long-term clinical outcomes (death, Q-wave myocardial infarction [MI], and repeat revascular

181 events (composite of death, Q-wave, and non-Q-wave myocardial infarction [MI], emergency bypass proc

182 iac event-free survival (freedom from death, Q-wave myocardial infarction and any coronary revascular

183 hospital major adverse cardiac event (death, Q-wave myocardial infarction and emergency coronary arte

184 s marred by a higher incidence of death, non-Q-wave myocardial infarction and major vascular and blee

185 nificant reduction in in-hospital mortality, Q-wave myocardial infarction and need for emergency bypa

186 An IPTE was strongly associated with Q-wave myocardial infarction and out-of-laboratory defin

187 -hospital and late clinical outcomes (death, Q-wave myocardial infarction and repeat revascularizatio

188 d transmural extent of healed Q-wave and non-Q-wave myocardial infarction can be accurately determine

189 Most patients with non-Q-wave myocardial infarction do not benefit from routine

190 trials of enoxaparin for unstable angina/non-Q-wave myocardial infarction have shown it to be superio

191 in 1 patient (1.4%), who died 2 weeks later; Q-wave myocardial infarction in 2 patients (2.8%); and n

192 ial infarction in 2 patients (2.8%); and non-Q-wave myocardial infarction in 8 patients (11.3%).

193 reduced the risk of death after spontaneous Q-wave myocardial infarction in BARI-eligible diabetic p

194 ents in patients with unstable angina or non-Q-wave myocardial infarction in the early phase.

195 reduced the risk of death after spontaneous Q-wave myocardial infarction in the patients with diabet

196 Non-Q-wave myocardial infarction is associated with higher i

197 Non-Q-wave myocardial infarction is usually managed accordin

198 s 100% with no in-hospital stent thrombosis, Q-wave myocardial infarction or death.

199 Q-wave myocardial infarction rate fell from 4.1% to 1.3%

200 ned 3171 patients with angina at rest or non-Q-wave myocardial infarction to receive either 1 mg of e

201 and in the absence of subsequent spontaneous Q-wave myocardial infarction was estimated with the use

202 001), and the five-year rates of spontaneous Q-wave myocardial infarction were 8 percent and 4 percen

203 h successful PCI and no emergency surgery or Q-wave myocardial infarction were followed for 38+/-25 m

204 Subjects with unstable angina or non-Q-wave myocardial infarction were randomized to atorvast

205 , 3,086 patients with unstable angina or non-Q-wave myocardial infarction were randomized within 4 da

206 ocardial infarction>>possibly related to non-Q-wave myocardial infarction>>no complication).

207 te coronary syndrome (unstable angina or non-Q-wave myocardial infarction).

208 te coronary syndrome (unstable angina or non-Q-wave myocardial infarction).

209 ity and cardiac morbidity (cardiac arrest or Q-wave myocardial infarction).

210 rtality or cardiac events (cardiac arrest or Q-wave myocardial infarction).

211 nfarction and refractory ischaemia after non-Q-wave myocardial infarction, an acute coronary syndrome

212 ced major hemorrhage, 1575 (0.24%) developed Q-wave myocardial infarction, and 1321 (0.20%) suffered

213 h and the composite of death, Q-wave and non-Q-wave myocardial infarction, and emergency additional r

214 l success, death, any myocardial infarction, Q-wave myocardial infarction, and emergency coronary art

215 of 46 participants with RWMAs had documented Q-wave myocardial infarction, and three (7%) underwent c

216 ssified as Q-wave myocardial infarction, non-Q-wave myocardial infarction, and unstable angina, these

217 ter in-hospital combined major event (death, Q-wave myocardial infarction, emergent CABG; 10.8% versu

218 CE), defined as the occurrence of death, new Q-wave myocardial infarction, emergent coronary artery b

219 on of complications defined as either death, Q-wave myocardial infarction, emergent or urgent coronar

220 There was a high rate of non-Q-wave myocardial infarction, need for repeat revascular

221 Traditionally classified as Q-wave myocardial infarction, non-Q-wave myocardial infa

222 s no increase in major complications (death, Q-wave myocardial infarction, or emergent coronary arter

223 ploratory end point of death from any cause, Q-wave myocardial infarction, or ischemia-driven revascu

224 ts (defined as cardiac death, Q-wave and non-Q-wave myocardial infarction, or target vessel revascula

225 %; P=.23), and target-vessel failure (death, Q-wave myocardial infarction, or target-vessel revascula

226 erse cardiac events (the composite of death, Q-wave myocardial infarction, or target-vessel revascula

227 y end points were death from cardiac causes, Q-wave myocardial infarction, revascularization of the t

228 ients (4%) had the primary end point (death, Q-wave myocardial infarction, stroke, emergency coronary

229 One-year mortality, cerebrovascular events, Q-wave myocardial infarction, target vessel failure, and

230 onary intervention (PCI) (in-hospital death, Q-wave myocardial infarction, urgent or emergent coronar

231 kyphoplasty group had an intraoperative non-Q-wave myocardial infarction, which resolved and was att

232 ved in patients with unstable angina and non-Q-wave myocardial infarction, with initial LDL cholester

233 coronary angiography after uncomplicated non-Q-wave myocardial infarction.

234 d patients 24 to 72 hours after an acute non-Q-wave myocardial infarction.

235 patients with high-risk unstable angina/non-Q-wave myocardial infarction.

236 improve with declines in mortality rate and Q-wave myocardial infarction.

237 isease, left ventricular dysfunction, or non-Q-wave myocardial infarction.

238 vels in patients with unstable angina or non-Q-wave myocardial infarction.

239 gina and electrocardiographic changes or non-Q-wave myocardial infarction.

240 e no patients with in-hospital Q-wave or non-Q-wave myocardial infarction.

241 rial of patients with unstable angina or non-Q-wave myocardial infarction.

242 , both groups had similar rates of death and Q-wave myocardial infarction: 3.4% and 2.5% for the nega

243 with the incidence of procedure-related non-Q-wave myocardial infarctions (MIs).

244 Non-Q-wave myocardial infarctions occurred in 26% of patient

245 ) in-hospital deaths, 14 (12%) perioperative Q-wave myocardial infarctions, and 6 (5%) cerebrovascula

246 No deaths, strokes, Q-wave myocardial infarctions, or revascularization occu

247 undergone CABG but did not have spontaneous Q-wave myocardial infarctions, the corresponding relativ

248 Four patients died and four patients had non-Q-wave myocardial infarctions.

249 There were 2 operative deaths and 3 Q-wave myocardial infarctions.

250 6%), whereas 4 (2.4%) had periprocedural non-Q-wave myocardial infarctions.

251 There were 3 postprocedural non-Q-wave myocardial infarctions.

252 Q waves on a 12-lead ECG are markers of a prior myocardi

253 al evidence of MI (clinical history of MI or Q waves on ECG).

254 sk of complications: ST-segment elevation or Q waves on the electrocardiogram thought to indicate acu

255 coronary artery bypass grafting, pathologic Q waves on the electrocardiogram, left bundle branch blo

256 on myocardial infarction patients, 36.9% had Q waves on their baseline ECG.

257 , oxygen saturation < or =94% (OR: 3.0), and Q-wave on the presenting electrocardiogram (OR: 2.8).

258 e studies have suggested that the absence of Q-waves on an electrocardiogram is due to incomplete occ

259 rdiogram, myocardial infarction confirmed by Q-waves on electrocardiogram or hospital records, angiog

260 anterior motion began mean 71 +/- 5 ms after Q-wave onset.

261 tion of serial evolution in ST segment, T or Q wave or left bundle branch block (LBBB) abnormalities

262 ecified as the development of new pathologic Q waves or creatine phosphokinase-MB isoenzyme elevation

263 ditional effect of evolving ST segment, T or Q waves or LBBB between serially obtained prehospital an

264 ical impulse, radiographic cardiomegaly, and q waves or left bundle branch block on an electrocardiog

265 Only 17 patients (2.0%) had new Q waves or left bundle-branch block after surgery; howev

266 V pathology included the following: syncope; Q waves or precordial QRS amplitudes <1.8 mV; 3 abnormal

267 (P<0.05) with ranked adverse outcome (death, Q-wave or creatine kinase >/= 3x normal myocardial infar

268 and there were no patients with in-hospital Q-wave or non-Q-wave myocardial infarction.

269 More men had ECG Q-wave progression, and more women had smaller ECG chang

270 Pathological early Q waves (QW) are associated with adverse outcomes in pat

271 8%, perioperative myocardial infarction (new Q wave) rate was 0.6%, and deep sternal wound infection

272 The presence of baseline Q waves, rather than time to treatment, was significantl

273 A Q-wave ratio of > 1.85 in aVL/aVR, a R/S ratio of > 2 in

274 of ischemia (ST elevation, T inversion, and Q waves, reader 1 kappa 0.40 to 0.69; reader 2 kappa 0.5

275 Neither the number of Q waves, residual ST-segment depression of >or=0.5 mm or

276 and all of 19 imaged at 14 months (eight non-Q-wave) showed hyperenhancement.

277 ECG changes were defined as Q waves, ST-segment depression, T-wave changes, ventricu

278 gment elevation of 6 mm or less, presence of Q waves, ST-segment elevation in only 2 leads, and alter

279 al emergency departments in Ontario, Canada, Q-waves, T-wave inversion, or ST-depression were present

280 st events occur without the development of a Q wave, the ECG will not definitively diagnose them; eve

281 Irrespective of the presence or absence of Q waves, the majority of patients with hyperenhancement

282 CK) MB fraction of at least 100 ng/mL or new Q waves through postoperative day 30.

283 , we evaluated the relationships of baseline Q waves, time from symptom onset, and reperfusion strate

284 The QRS interval, from the beginning of the Q wave to the end of the S wave on an electrocardiogram,

285 irst determining the mean time interval from Q-wave to SAM onset from multiple M-mode tracings.

286 eperfusion, and analyzed for the presence of Q waves using Selvester criteria.

287 versus > or = 70 years of age and those with Q wave versus non-Q wave AMI.

288 Moss (63) commented that "The Q-wave versus non-Q-wave categorization does not provide

289 n 191 (34.9%) patients, and an fQRS and/or a Q wave was present in 203 (42.3%) patients.

290 The Q wave was present in 71 (14.8%) patients, an fQRS was p

291 lysis, the periprocedural development of new Q waves was the most powerful independent determinant of

292 A prolonged QTc interval and Q wave were related to post-transplant cardiac events (p

293 Patients with Q waves were older (median age, 59 versus 57), were more

294 reater than 30 minutes or the development of Q-waves were identified and enumerated in 18 participati