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1 QCA and IVUS studies were performed before and after int
2 QCA and optical coherence tomographic analyses were perf
3 QCA and QCU measurements of 1.5- to 2.5-mm residual lume
4 QCA can be used as an accurate method of poststent asses
5 QCA demonstrated a 15% increase in coronary artery diame
6 QCA measurements included minimal luminal diameter and d
7 QCA measurements included minimum lumen diameter (MLD) a
8 QCA systematic error (SE) varied from 0.01 for the Wikto
10 ation product, 3-quinolinecarboxylic acid (3-QCA), was identified by liquid chromatography high resol
11 oducts were formed via covalent binding of 3-QCA with DOM molecules of above-average O/C and H/C rati
14 y (tau = 15 s) reduced the gamma of 5-HT(3A)(QCA) receptors in inside-out patches, an effect reversed
15 exahydro-1H-cyclop enta[h]quinolin-3-one 3d (QCA-1093) as a novel nonsteroidal glucocorticoid recepto
18 (P < .001) with disagreement between CTA and QCA in multivariable analysis after controlling for sex,
20 stenosis between clinical interpretation and QCA was 8.2+/-8.4%, reflecting an average higher percent
21 here was a good correlation between MDCT and QCA percent stenosis (r = 0.75, p < 0.01, SEE = 15%).
25 entional quantitative coronary angiographic (QCA) and intravascular ultrasound (IVUS) predictors of r
26 lidity of quantitative coronary angiography (QCA) after stent placement has been questioned because t
28 performed quantitative coronary angiography (QCA) and intravascular ultrasound (IVUS) in 37 lesions w
29 Complete quantitative coronary angiography (QCA) and IVUS were obtained in 104 patients before and a
30 combined quantitative coronary angiography (QCA) and single-photon emission CT (SPECT) or QCA alone.
31 ent using quantitative coronary angiography (QCA) in 175 randomly selected patients undergoing electi
42 e >0.8 or quantitative coronary angiography [QCA] showing no percentage diameter stenosis >/=70% in 1
44 of the molecular quantum cellular automata (QCA) cell array by the electric field from the Fe(III)-R
46 ary diameter (2.69 +/- 0.33 mm) at baseline (QCA of three segments of LAD and three segments of left
48 he results demonstrated that type II binding QCA analogues were metabolically less stable (2- to 12-f
49 true" diameters of any stented lumen by both QCA and quantitative ultrasonic (QCU) measurement postst
53 e 16 (94%) transplant patients classified by QCA as having occlusive coronary artery disease and 29 o
54 were euthanized at day 28, and evaluation by QCA revealed a significant improvement in mean lumen los
56 nts were largest for intermediate lesions by QCA (50% to <70%), with variation existing across sites.
58 m lumen diameter (MLD) (2.26 +/- 0.82 mm) by QCA correlated less well with IVUS (2.8 +/- 0.82 mm, r =
59 gments with >20% coronary artery stenosis by QCA but also in 12 (15%) of 80 segments without angiogra
61 l lumen and arterial area); 2) follow-up DS (QCA lesion length); and 3) follow-up MLD (QCA lesion len
62 66 segments of the radial graft on the first QCA study was 0.170 mm (95% confidence interval [CI] 0.1
64 S (QCA lesion length); and 3) follow-up MLD (QCA lesion length and preinterventional MLD and DS and I
67 transformations, we synthesized a library of QCA compounds that could undergo N-dealkylation, O-dealk
68 Univariate and multivariate predictors of QCA restenosis (> or = 50% diameter stenosis at follow-u
76 lence of CAD was 39% (36 of 92) according to QCA and SPECT and 64% (59 of 92) according to QCA alone.
77 evaluate CTA diagnostic accuracy compared to QCA in patients according to calcium score and pre-test
79 476 Palmaz-Schatz stents for whom follow-up QCA data were available 5.5 +/- 4.8 months (mean +/- SD)
82 tive predictive values of MDCT compared with QCA for the detection of segments with significant (>50%
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