1 nduction disorder, arrhythmias, or change in QRS axis.

2 n EP mechanism of the periodic transition in QRS axis.

3 RS morphology was accompanied by a change in QRS axis.

4      In 22 of 26 episodes, the transition in QRS axis coincided with the transient bifurcation of a p

5 a prolonged QTc interval, a Q wave, abnormal QRS axis deviation, ST segment depression and a patholog

6 he characteristic periodic transition of the QRS axis during TdP VT in the LQTS.

7 th inverted asymmetrical T-wave opposite the QRS axis in lead V5 or V6.

8 inverted asymmetrical T-wave opposite to the QRS axis in leads V5 and/or V6.

9 variate analysis demonstrated differences in QRS axis, limb (I, aVr), and precordial (V1, V2, V6) ECG

10 with a left bundle branch block and inferior QRS axis morphology.

11 atients with idiopathic VAs with an inferior QRS-axis morphology.

12 ve electrocardiographic traits (PR interval, QRS axis, QRS duration, and QTc interval) were evaluated

13                                  A change in QRS axis simultaneous with electrocardiographic waveform

14 ion, PR interval, QRS duration, QT interval, QRS axis, Sokolow-Lyon and Cornell voltages, and ST-segm

15  maps could be accurately constructed during QRS-axis transitions in surface ECGs.

16 st polymorphic VT, with an apparent shift in QRS axis, was due to a predominantly single localized ci