ライフサイエンスコーパス: QRS complex

1 g responders (including patients with narrow QRS complexes).

2 n chronic systolic heart failure with a wide QRS complex.

3 oms, a reduced ejection fraction, and a wide QRS complex.

4 tients with a low ejection fraction and wide QRS complex.

5 the cardiac mortality compared with a narrow QRS complex.

6 ll-cause mortality rate compared with narrow QRS complex.

7 al) from each of nine unknown samples of the QRS complex.

8 ctor of incident CHD events in men with wide QRS complex.

9 ine, having either normal conduction or wide QRS complex.

10 ctuates cyclically back and forth across the QRS complex.

11 n a fairly constant position relative to the QRS complex.

12 n may depolarize tissue after the end of the QRS complex.

13 n negative deflection until greater than the QRS complex.

14 lectrocardiogram in the presence of a narrow QRS complex.

15 nt with (in the opposite direction from) the QRS complex.

16 d left ventricular (LV) function, and a wide QRS complex.

17 ation pattern was computed from the recorded QRS complexes.

18 Association class 3.4 +/- 0.5) and a widened QRS complex (184 +/- 31 ms) underwent robotic LV lead pl

19 Of the 10 impacts sustained during the QRS complex, 4 resulted in transient complete heart bloc

20 epressed ejection fraction (EF) and a narrow QRS complex, albeit in a small number of patients, and w

21 cles of mutant animals, including diminished QRS complex amplitude consistent with loss of electrical

22 n patients with heart failure (HF) with wide QRS complex and diminished left ventricular (LV) functio

23 were delivered at or before the onset of the QRS complex and never during the complex itself.

24 ature contractions with an elongation of the QRS complex and the hearts were more susceptible to isop

25 e., the spatial electrical angle between the QRS complex and the T-wave; p = 0.0005), wider QRS compl

26 nds (ms) after the end of the last conducted QRS complex and then scanned decrementally through that

27 G628S mice studied, primarily involving the QRS complex and, more rarely, T-wave morphology.

28 ndex, had elevated heart rate, had prolonged QRS complex, and had lower prevalence of history of prio

29 n a non-LBBB (left bundle branch block) wide QRS complex, and lower left ventricular ejection fractio

30 lethal cardiomyopathy associated with a wide QRS complex arrhythmia.

31 tion functional class I and II and with wide QRS complexes, carvedilol was associated with a 30% redu

32 ctionated late potentials (96+/-47 ms beyond QRS complex) clustering exclusively in the anterior aspe

33 lure, left ventricular dysfunction, and wide QRS complex compared with an ICD only.

34 The duration of the basal QRS complex does not reliably predict the clinical respo

35 llators (CRT-D) have a very wide (>/=180 ms) QRS complex duration (QRSD).

36 a premature ventricular depolarization or a QRS complex during ventricular tachycardia.

37 Fragmented QRS complexes (fQRSs), which include various RSR' patter

38 The sums of the amplitude of QRS complexes from the 12 ECG leads (Sigma QRS) were cor

39 The presence of notching on any QRS complex had 79% sensitivity and 65% specificity of (

40 he electrophysiological underpinnings of the QRS complex has become important not only to predict res

41 Some patients with narrow QRS complexes have echocardiographic evidence of left ve

42 he onset of or within the first 45 ms of the QRS complex in 16 animals.

43 ams revealed significant prolongation of the QRS complex in adult Cx43 -/+ mice (13.4+/-1.8 ms, n = 1

44 elatively fixed CI from the preceding normal QRS complex in most patients.

45 ventricular (LV) systolic function and broad QRS complex in the surface electrocardiogram (ECG).

46 However, low-voltage QRS complex is not a uniform finding with the infiltrati

47 sed by delayed ventricular contraction (wide QRS complex), is a common feature of cardiomyopathy and

48 The morphology of single paced QRS complexes may vary, depending on coupling interval,

49 In ICD patients with HF, a wide underlying QRS complex more than doubles the cardiac mortality comp

50 In the next 10 patients, the QRS complex morphology during ventricular overdrive paci

51 All patients with baseline normal QRS complexes (n=30) or left fascicular blocks (4 anteri

52 ct of ADS synchronized to normally conducted QRS complexes (NQRS) and to supraventricular complexes w

53 flected by the amplitude and duration of the QRS complex on the electrocardiogram (ECG).

54 The morphology of the QRS complexes on electrocardiogram is an excellent tool

55 t were significantly delayed with respect to QRS complex onset (3.7+/-0.7 ms in WT [n=6] and 6.5+/-0.

56 QRS complex onset to activation at the right ventricular

57 It is usually coincident with the QRS complex or appears on the ST segment or first half o

58 ed by difficulty in assessing: 1) changes in QRS complexes or P-waves that indicate fusion, and 2) th

59 ignificant with the duration of the filtered QRS complex (p < 0.001 for QRS duration, p < 0.01 for la

60 S complex and the T-wave; p = 0.0005), wider QRS complex (p = 0.004), longer QTrr (i.e., age- and gen

61 bserved an 80% reduction in amplitude of the QRS complex, profound systolic dysfunction, decreased co

62 is-unrelated bradycardia and PQ interval and QRS complex prolongation.

63 In individuals with wide QRS complex (QRSd > or =120 ms), similar analyses showed

64 on, salutary effects in patients with narrow QRS complexes remain to be demonstrated.

65 2 groups were detected for the PR interval, QRS complex, ST-segment duration, T-wave duration, QTc,

66 ncordant with (in the same direction as) the QRS complex; ST-segment depression of 1 mm or more in le

67 evidence of sinus bradycardia and fragmented QRS complex, supporting the critical role of Slc26a6 in

68 The ADFT(50) for QRS complex-synchronized shocks was 183+/-56 V, versus 1

69 The etiology of sustained monomorphic wide QRS complex tachycardia is often uncertain acutely.

70 apeutic agent for patients with regular wide QRS complex tachycardia.

71 gent for patients with undifferentiated wide QRS complex tachycardia.

72 ing in a type of VT with a relatively narrow QRS complex that mimics fascicular VT.

73 ntervals, PAWP was measured gated to the ECG QRS complex to calculate the QRS-gated DPD (diastolic pu

74 the electric delay from the beginning of the QRS complex to the local LV electrogram (QLV), was found

75 t of variation of the time interval from the QRS complex to the onset of expansion and to early diast

76 during normal sinus rhythm that reflects the QRS complex vector during prior periods of ventricular p

77 During pace mapping, the stimulus-QRS complex was longer at late potential sites, consiste

78 Low-voltage QRS complex was the sine qua non of infiltrative cardiom

79 Very late LAVA (>100 ms after QRS complex) were almost exclusively detected within the

80 chrony also occurs in patients with a narrow QRS complex, which suggests the potential usefulness of

81 ported were transient arrhythmia, reversible QRS-complex widening, transient hypotension and mild non

82 The isoelectric initial preexcited QRS complex with rSR' pattern in lead V1 of the surface

83 VCD did not differ from patients with narrow QRS complexes with regard to occurrence of tachycardias.